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The American Journal of Surgical Pathology, ISSN 0147-5185, 03/2017, Volume 41, Issue 3, pp. 326 - 333
Mismatch repair (MMR)-deficient endometrial carcinomas (ECs) bearing Lynch syndrome (LS)-associated germline mutations or sporadic MLH1 promoter... 
PD-L1 | mismatch repair | endometrial carcinoma | MLH1 hypermethylation | immunotherapy | Lynch syndrome | SURGERY | MICROSATELLITE INSTABILITY | INFILTRATING LYMPHOCYTES | IMMUNOSCORE | PATHOLOGY | LUNG-CANCER | PROGNOSTIC-FACTOR | IMMUNE-SYSTEM | MICROENVIRONMENT | COLORECTAL-CANCER | BLOCKADE | Immunohistochemistry | Neoplastic Syndromes, Hereditary - pathology | Prognosis | Follow-Up Studies | Endometrial Neoplasms - mortality | Colorectal Neoplasms - genetics | Colorectal Neoplasms, Hereditary Nonpolyposis - pathology | Humans | Middle Aged | Brain Neoplasms - pathology | Endometrial Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Neoplastic Syndromes, Hereditary - metabolism | Brain Neoplasms - metabolism | Neoplastic Syndromes, Hereditary - mortality | Colorectal Neoplasms, Hereditary Nonpolyposis - mortality | DNA Methylation | Endometrial Neoplasms - genetics | DNA Mismatch Repair | Biomarkers, Tumor - metabolism | Female | Neoplastic Syndromes, Hereditary - genetics | Brain Neoplasms - mortality | Colorectal Neoplasms - metabolism | Colorectal Neoplasms - mortality | Promoter Regions, Genetic | Colorectal Neoplasms, Hereditary Nonpolyposis - genetics | Brain Neoplasms - genetics | Colorectal Neoplasms, Hereditary Nonpolyposis - metabolism | B7-H1 Antigen - metabolism | MutL Protein Homolog 1 - genetics | Endometrial Neoplasms - pathology | Aged | Biomarkers, Tumor - genetics | Colorectal Neoplasms - pathology | Index Medicus
Journal Article
Best Practice & Research: Clinical Obstetrics & Gynaecology, ISSN 1521-6934, 2015, Volume 29, Issue 4, pp. 528 - 540
Journal Article
Journal Article
Anticancer Research, ISSN 0250-7005, 08/2009, Volume 29, Issue 8, pp. 2875 - 2884
Background: To identify immunosuppressive elements present in ovarian cancer associated ascites. Patients and Methods: Ascites and plasma were obtained from... 
Cell mediated immunity | Cytokines | Ascites | Chemokines | Ovarian cancer | DENDRITIC CELLS | PANCREATIC-CANCER | PERIPHERAL-BLOOD | chemokines | CYTOTOXIC T-LYMPHOCYTES | TUMOR-INFILTRATING LYMPHOCYTES | ascites | ADOPTIVE TRANSFER | ONCOLOGY | IN-VIVO | cytokines | cell mediated immunity | ANTIGEN-PRESENTING CELLS | PROGNOSTIC-SIGNIFICANCE | ENDOTHELIAL GROWTH-FACTOR | CD8-Positive T-Lymphocytes - pathology | Prognosis | Humans | Middle Aged | Cystadenocarcinoma, Serous - metabolism | Endometrial Neoplasms - metabolism | Ovarian Neoplasms - pathology | CD4-Positive T-Lymphocytes - pathology | Adenocarcinoma, Clear Cell - immunology | T-Lymphocytes, Regulatory - immunology | CD4-Positive T-Lymphocytes - immunology | Antigen-Presenting Cells - pathology | Endometrial Neoplasms - immunology | Flow Cytometry | Fallopian Tube Neoplasms - immunology | Aged, 80 and over | Ascites - immunology | CD8-Positive T-Lymphocytes - metabolism | Adult | Female | Ovarian Neoplasms - metabolism | Cystadenocarcinoma, Serous - immunology | Peritoneal Neoplasms - immunology | Peritoneal Neoplasms - pathology | Cystadenocarcinoma, Serous - secondary | Antigen-Presenting Cells - metabolism | Cytokines - metabolism | Adenocarcinoma, Clear Cell - metabolism | CD4-Positive T-Lymphocytes - metabolism | Fallopian Tube Neoplasms - pathology | Survival Rate | Chromium - metabolism | Antigen-Presenting Cells - immunology | Endometrial Neoplasms - secondary | Adenocarcinoma, Clear Cell - secondary | Ascites - metabolism | Peritoneal Neoplasms - metabolism | Aged | Fallopian Tube Neoplasms - metabolism | CD8-Positive T-Lymphocytes - immunology | Ascites - pathology | Ovarian Neoplasms - immunology | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 09/2017, Volume 23, Issue 9, pp. 1046 - 1054
It is generally assumed that recurrent mutations within a given cancer driver gene elicit similar drug responses. Cancer genome studies have identified... 
MEDICINE, RESEARCH & EXPERIMENTAL | BROMODOMAIN INHIBITORS | ENDOMETRIAL CARCINOMAS | UBIQUITIN LIGASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | TUMORS | CELL BIOLOGY | BREAST-CANCER | GENOMIC LANDSCAPE | PROSTATE-CANCER | RESISTANCE | MUTATIONS | Immunohistochemistry | Neoplasms, Cystic, Mucinous, and Serous - metabolism | Neoplasm Transplantation | Prostatic Neoplasms - metabolism | Immunoprecipitation | Apoptosis - drug effects | Heterocyclic Compounds, 3-Ring - pharmacology | Epigenesis, Genetic | Humans | Endometrial Neoplasms - metabolism | Drug Resistance, Neoplasm | Immunoblotting | Male | Molecular Targeted Therapy | Carcinoma, Endometrioid - metabolism | Neoplasms, Cystic, Mucinous, and Serous - genetics | Ubiquitination | Prostatic Neoplasms - genetics | Endometrial Neoplasms - genetics | Mass Spectrometry | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Chromatography, Liquid | Cullin Proteins - metabolism | Female | Nuclear Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | RNA-Binding Proteins - antagonists & inhibitors | Acetanilides - pharmacology | Carcinoma, Endometrioid - genetics | Carcinosarcoma - metabolism | Adenocarcinoma, Clear Cell - metabolism | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Reverse Transcriptase Polymerase Chain Reaction | Azepines - pharmacology | Transcription Factors - metabolism | Triazoles - pharmacology | Animals | Mice, Nude | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Carcinosarcoma - genetics | Cell Proliferation - drug effects | Mutation | Adenocarcinoma, Clear Cell - genetics | RNA-Binding Proteins - metabolism | Care and treatment | Gene mutations | Proteolysis | Physiological aspects | Genetic aspects | Research | Cancer | Ubiquitin | Biodegradation | Inhibitor drugs | Endometrial cancer | Genomes | Pharmacology | Substrates | Mutants | Degradation | Proteins | Sensitivity | Missense mutation | Inhibitors | Bet protein | Gene mapping | Prostate cancer | Prostate | Endometrium | Ubiquitin-protein ligase | Index Medicus
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2010, Volume 120, Issue 7, pp. 2319 - 2330
Journal Article
International Journal of Molecular Medicine, ISSN 1107-3756, 09/2017, Volume 40, Issue 3, pp. 587 - 606
Canonical WNT signaling through Frizzled and LRP5/6 receptors is transduced to the WNT/β-catenin and WNT/stabilization of proteins (STOP) signaling cascades to... 
regulatory T cells | FGF | angiogenesis | WNT5A | Notch | tumor microenvironment | cancer stem cells | Alzheimer's disease | myeloid-derived suppressor cells | epithelial-to-mesenchymal transition | Angiogenesis | Tumor microenvironment | Epithelial-to-mesenchymal transition | Myeloid-derived suppressor cells | Cancer stem cells | Regulatory T cells | MEDICINE, RESEARCH & EXPERIMENTAL | BONE-MINERAL DENSITY | NEURAL-TUBE DEFECTS | PLANAR CELL POLARITY | TCF SIGNALING PATHWAY | CANCER STEM-CELLS | DOMINANT ROBINOW SYNDROME | RECEPTOR TYROSINE KINASES | ACUTE LYMPHOBLASTIC-LEUKEMIA | FAMILIAL EXUDATIVE VITREORETINOPATHY | ENDOTHELIAL GROWTH-FACTOR | Prostatic Neoplasms - metabolism | Colorectal Neoplasms, Hereditary Nonpolyposis - genetics | Humans | Endometrial Neoplasms - metabolism | Male | Neoplasm Proteins - metabolism | Intellectual Disability - metabolism | Intellectual Disability - therapy | Prostatic Neoplasms - therapy | Colorectal Neoplasms, Hereditary Nonpolyposis - metabolism | Colorectal Neoplasms, Hereditary Nonpolyposis - therapy | Animals | Prostatic Neoplasms - genetics | Endometrial Neoplasms - genetics | Wnt Signaling Pathway - genetics | Endometrial Neoplasms - therapy | Female | Mutation | Neoplasm Proteins - genetics | Proteins | Tyrosine | Molecular genetics | Gene mutations | Colorectal cancer | Genetic aspects | Health aspects | Heparan sulfate | Phosphorylation | Pathogenesis | Genomics | Genes | Cardiovascular disease | Kinases | Defects | Cell adhesion & migration | Osteoporosis | Stem cells | Genetics | Ligands | Alzheimers disease
Journal Article