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1. Full Text Transforming Growth Factor-β Induced Warburg-Like Metabolic Reprogramming May Underpin the Development of Peritoneal Endometriosis
by Young, Vicky J and Brown, Jeremy K and Maybin, Jacqueline and Saunders, Philippa T. K and Duncan, W. Colin and Horne, Andrew W
The journal of clinical endocrinology and metabolism, ISSN 0021-972X, 09/2014, Volume 99, Issue 9, pp. 3450 - 3459
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | L-Lactate Dehydrogenase - metabolism | Epithelial Cells - metabolism | Endometriosis - genetics | Humans | Transforming Growth Factor beta1 - metabolism | Glucose Transporter Type 1 - metabolism | RNA, Messenger - metabolism | Glycolysis - genetics | Glycolysis - physiology | Peritoneum - pathology | Ascitic Fluid - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Isoenzymes - metabolism | Peritoneum - cytology | Female | Epithelial Cells - cytology | Protein-Serine-Threonine Kinases - metabolism | Endometrium - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Isoenzymes - genetics | Lactic Acid - metabolism | Oxidative Phosphorylation | Protein-Serine-Threonine Kinases - genetics | Endometriosis - pathology | L-Lactate Dehydrogenase - genetics | Glucose Transporter Type 1 - genetics | Endometriosis - metabolism | Endometrium - pathology | Peritoneum - metabolism | Cell Line, Transformed | Index Medicus | Abridged Index Medicus | Endocrine Research
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2. Full Text Role of nuclear progesterone receptor isoforms in uterine pathophysiology
by Patel, Bansari and Elguero, Sonia and Thakore, Suruchi and Dahoud, Wissam and Bedaiwy, Mohamed and Mesiano, Sam
Human reproduction update, ISSN 1355-4786, 2015, Volume 21, Issue 2, pp. 155 - 173
Uterine pathophysiology | Progesterone | Progesterone receptors | Life Sciences & Biomedicine | Obstetrics & Gynecology | Reproductive Biology | Science & Technology | Abortion, Habitual - pathology | Uterine Diseases - metabolism | Humans | Menstrual Cycle - metabolism | Uterus - metabolism | Endometrial Neoplasms - metabolism | Uterine Diseases - pathology | Uterine Cervical Neoplasms - pathology | Leiomyoma - metabolism | Abortion, Habitual - metabolism | Receptors, Progesterone - metabolism | Leiomyoma - pathology | Protein Isoforms - metabolism | Uterus - physiology | Uterine Cervical Neoplasms - metabolism | Female | Embryo Implantation - physiology | Signal Transduction | Endometriosis - pathology | Protein Isoforms - physiology | Receptors, Progesterone - physiology | Models, Biological | Endometriosis - metabolism | Progesterone - metabolism | Endometrial Neoplasms - pathology | Index Medicus | Reviews | uterine pathophysiology | progesterone | progesterone receptors
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3. Full Text Naringenin induces mitochondria-mediated apoptosis and endoplasmic reticulum stress by regulating MAPK and AKT signal transduction pathways in endometriosis cells
by Park, Sunwoo and Lim, Whasun and Bazer, Fuller W and Song, Gwonhwa
Molecular human reproduction, ISSN 1360-9947, 12/2017, Volume 23, Issue 12, pp. 842 - 854
Life Sciences & Biomedicine | Developmental Biology | Obstetrics & Gynecology | Reproductive Biology | Science & Technology | Transcription Factor CHOP - agonists | Transcription Factor CHOP - genetics | Endoribonucleases - genetics | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Stromal Cells - pathology | Humans | bcl-2-Associated X Protein - agonists | bcl-2 Homologous Antagonist-Killer Protein - genetics | Annexin A5 - genetics | Estrogen Antagonists - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Membrane Potential, Mitochondrial - drug effects | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Eukaryotic Initiation Factor-2 - agonists | Reactive Oxygen Species - agonists | Dose-Response Relationship, Drug | Heat-Shock Proteins - genetics | Stromal Cells - drug effects | Eukaryotic Initiation Factor-2 - metabolism | Female | Protein-Serine-Threonine Kinases - metabolism | bcl-2-Associated X Protein - genetics | Endometrium - metabolism | Cell Line | Endoplasmic Reticulum Stress - drug effects | Endoribonucleases - metabolism | Signal Transduction | Flavanones - pharmacology | Heat-Shock Proteins - metabolism | Stromal Cells - metabolism | Gene Expression Regulation | Protein-Serine-Threonine Kinases - genetics | bcl-2-Associated X Protein - metabolism | Endometriosis - pathology | Mitochondria - metabolism | Mitochondria - drug effects | Phosphatidylinositol 3-Kinases - genetics | Heat-Shock Proteins - agonists | Annexin A5 - metabolism | Eukaryotic Initiation Factor-2 - genetics | Endometriosis - metabolism | Mitogen-Activated Protein Kinases - genetics | Endometrium - pathology | Transcription Factor CHOP - metabolism | Mitogen-Activated Protein Kinases - metabolism | bcl-2 Homologous Antagonist-Killer Protein - agonists | Index Medicus
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4. Full Text Unus pro omnibus, omnes pro uno: A novel, evidence-based, unifying theory for the pathogenesis of endometriosis
by Laganà, Antonio Simone and Vitale, Salvatore Giovanni and Salmeri, Francesca Maria and Triolo, Onofrio and Ban Frangež, Helena and Vrtačnik-Bokal, Eda and Stojanovska, Lily and Apostolopoulos, Vasso and Granese, Roberta and Sofo, Vincenza
Medical hypotheses, ISSN 0306-9877, 2017, Volume 103, pp. 10 - 20
Internal Medicine | Endometriosis | Embryology | Proteins homeodomain | Wingless type proteins | Embryonic stem cells | Body patterning | Endometriosis - genetics | Homeodomain Proteins - metabolism | Humans | Uterus - metabolism | Stem Cells - cytology | Stem Cells - metabolism | Wnt Proteins - metabolism | Peritoneum - pathology | Immune System | Female | beta Catenin - chemistry | Cell Differentiation | Endometriosis - physiopathology | Endometrium - metabolism | Models, Theoretical | Menstruation | Cytokines - metabolism | Signal Transduction | Inflammation | Cell Adhesion Molecules - metabolism | Organogenesis | Animals | Endometrium - physiopathology | Mesoderm - metabolism | Mice | Matrix Metalloproteinases - metabolism | Bone Marrow Cells - metabolism | Developmental biology | Stem cells | Proteins | Physiological aspects | Index Medicus
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5. Full Text IL-27 triggers IL-10 production in Th17 cells via a c-Maf/ROR gamma t/Blimp-1 signal to promote the progression of endometriosis
by Chang, Kai-Kai and Liu, Li-Bing and Jin, Li-Ping and Zhang, Bing and Mei, Jie and Li, Hui and Wei, Chun-Yan and Zhou, Wen-Jie and Zhu, Xiao-Yong and Shao, Jun and Li, Da-Jin and Li, Ming-Qing
Cell death & disease, ISSN 2041-4889, 03/2017, Volume 8, Issue 3, pp. e2666 - e2666
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Stromal Cells - pathology | Humans | CD4-Positive T-Lymphocytes - pathology | Interleukins - metabolism | Th17 Cells - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Up-Regulation - physiology | Interleukin-10 - metabolism | Adult | Female | Cell Differentiation - physiology | Interleukin-6 - metabolism | Repressor Proteins - metabolism | Endometrium - metabolism | Macrophages - pathology | Stromal Cells - metabolism | CD4-Positive T-Lymphocytes - metabolism | Endometriosis - pathology | Disease Progression | Macrophages - metabolism | Endometriosis - metabolism | Proto-Oncogene Proteins c-maf - metabolism | Endometrium - pathology | Transforming Growth Factor beta - metabolism | Positive Regulatory Domain I-Binding Factor 1 | Index Medicus | Original
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6. Full Text Endometrial and peritoneal expression of aromatase, cytokines, and adhesion factors in women with endometriosis
by Kyama, Cleophas M., M.Sc and Overbergh, Lutgart, Ph.D and Mihalyi, Attila, Ph.D and Meuleman, Christel, M.D and Mwenda, Jason M., Ph.D and Mathieu, Chantal, M.D. Ph.D and D'Hooghe, Thomas M., M.D. Ph.D
Fertility and sterility, ISSN 0015-0282, 2008, Volume 89, Issue 2, pp. 301 - 310
Internal Medicine | Obstetrics and Gynecology | endometriosis | peritoneum | cytokines | mRNA | Endometrium | Gynecology. Andrology. Obstetrics | Biological and medical sciences | Medical sciences | Non tumoral diseases | Female genital diseases | Receptors, CCR2 - genetics | Uterine Diseases - metabolism | Cell Adhesion Molecules - genetics | Endometriosis - genetics | Vimentin - metabolism | Humans | Aromatase - metabolism | Interleukin-1beta - genetics | RNA, Messenger - metabolism | Aromatase - genetics | Interleukin-1beta - metabolism | Vimentin - genetics | Luteal Phase - metabolism | Adult | Female | Menstruation - genetics | Vascular Cell Adhesion Molecule-1 - genetics | Chemokine CCL5 - metabolism | Cytokines - genetics | Endometrium - metabolism | Cytokines - metabolism | Luteal Phase - genetics | Cell Adhesion Molecules - metabolism | Menstruation - metabolism | Receptors, CCR2 - metabolism | Chemokine CCL5 - genetics | Endometriosis - metabolism | Uterine Diseases - genetics | Vascular Cell Adhesion Molecule-1 - metabolism | Peritoneum - metabolism | Women | Endometriosis | Cytokines | RNA | Index Medicus
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