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Circulation, ISSN 0009-7322, 02/2015, Volume 131, Issue 6, pp. 550 - 559
BACKGROUND—Characterization of myocardial structural changes in heart failure with preserved ejection fraction (HFpEF) has been hindered by the limited... 
heart failure, diastolic | endothelium | pathology | hypertrophy | fibrosis | autopsy | microvessels | PROGNOSTIC IMPACT | CARDIAC & CARDIOVASCULAR SYSTEMS | IDIOPATHIC DILATED CARDIOMYOPATHY | CARDIAC STRUCTURE | RESTING TENSION | LEFT-VENTRICLE | ACTIVE RELAXATION | DIASTOLIC DYSFUNCTION | FLOW RESERVE | HYPERTROPHIC CARDIOMYOPATHY | PERIPHERAL VASCULAR DISEASE | ARTERY-DISEASE | Immunohistochemistry | Heart - anatomy & histology | Humans | Heart Failure - physiopathology | Microvessels - pathology | Cardiomegaly - epidemiology | Cardiomegaly - pathology | Male | Reference Values | Cause of Death | Autopsy | Coronary Artery Disease - pathology | Aged, 80 and over | Electrocardiography | Female | Endomyocardial Fibrosis - pathology | Heart Failure - diagnosis | Heart Failure - epidemiology | Coronary Vessels - pathology | Echocardiography | Comorbidity | Endomyocardial Fibrosis - epidemiology | Organ Size | Myocardium - pathology | Heart Failure - pathology | Stroke Volume | Myocytes, Cardiac - pathology | Algorithms | Diabetes Mellitus - epidemiology | Aged | Coronary Artery Disease - epidemiology | Endomyocardial Fibrosis - physiopathology | Postmortem Changes | Heart failure | Analysis | Research | Cardiac output | Risk factors | Endothelium | Hypertrophy | Index Medicus | Abridged Index Medicus | coronary microvessel | diastolic heart failure
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2007, Volume 13, Issue 8, pp. 952 - 961
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2008, Volume 105, Issue 35, pp. 13027 - 13032
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 09/2017, Volume 491, Issue 2, pp. 329 - 336
Hyperglycemia plays a crucial role in the pathogenesis of diabetic complications; however, the mechanisms underlying diabetic cardiac fibrosis remain unclear.... 
MicroRNA | EndMT | Notch2 | Fibrosis | High glucose | TARGET | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | MICRORNAS | DIABETIC CARDIOMYOPATHY | BIOPHYSICS | ALAGILLE-SYNDROME | EXPRESSION | Dependovirus - genetics | Cadherins - metabolism | Genetic Vectors - administration & dosage | Humans | Aortic Valve - drug effects | Diabetes Mellitus, Experimental - genetics | Male | MicroRNAs - metabolism | Diabetes Mellitus, Experimental - chemically induced | Cadherins - genetics | Endomyocardial Fibrosis - prevention & control | Dependovirus - metabolism | Genetic Vectors - chemistry | Signal Transduction | Endothelial Cells - metabolism | Genetic Vectors - metabolism | Glucose - pharmacology | Heart Ventricles - metabolism | MicroRNAs - genetics | Calcium-Binding Proteins - genetics | Platelet Endothelial Cell Adhesion Molecule-1 - genetics | Streptozocin | Vimentin - metabolism | Actins - metabolism | Receptor, Notch2 - genetics | Actins - genetics | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Vimentin - genetics | Epithelial-Mesenchymal Transition | Diabetic Cardiomyopathies - prevention & control | Endomyocardial Fibrosis - genetics | Endomyocardial Fibrosis - chemically induced | Endomyocardial Fibrosis - pathology | Diabetes Mellitus, Experimental - metabolism | Heart Ventricles - pathology | Calcium-Binding Proteins - metabolism | Cell Line | Mice, Inbred C57BL | Gene Expression Regulation | Receptor, Notch2 - metabolism | Aortic Valve - cytology | Aortic Valve - metabolism | Fibronectins - metabolism | Diabetic Cardiomyopathies - genetics | Animals | Endothelial Cells - cytology | Diabetes Mellitus, Experimental - pathology | Diabetic Cardiomyopathies - pathology | Fibronectins - genetics | Diabetic Cardiomyopathies - chemically induced | Endothelial Cells - drug effects | Fibronectins | Hyperglycemia | Stem cells | Glucose | Muscle proteins | Dextrose | Endothelium | Index Medicus
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2013, Volume 65, pp. 108 - 119
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 9, pp. e74535 - e74535
Senescence is a recognized mechanism of cardiovascular diseases; however, its contribution to myocardial fibrosis and rupture after infarction and the... 
APOPTOSIS | PREMATURE SENESCENCE | MULTIDISCIPLINARY SCIENCES | VASCULAR CELL SENESCENCE | PHENOTYPE | TUMOR-SUPPRESSOR | MICE | MODEL | INFLAMMATORY RESPONSE | P53 | DELETION | Myocardial Infarction - genetics | RNA, Small Interfering - genetics | Adenoviridae | Endomyocardial Fibrosis - etiology | Tumor Suppressor Protein p53 - antagonists & inhibitors | Male | Tumor Suppressor Protein p53 - genetics | Myocardial Infarction - pathology | Myocardium - metabolism | Endomyocardial Fibrosis - genetics | Collagen - genetics | Endomyocardial Fibrosis - pathology | Disease Models, Animal | Fibroblasts - metabolism | Cellular Senescence - genetics | Signal Transduction | Cells, Cultured | Gene Expression Regulation | Myocardium - pathology | Myocardial Infarction - metabolism | Fibroblasts - pathology | Tumor Suppressor Protein p53 - deficiency | Mice, Knockout | Collagen - metabolism | Myocardial Infarction - complications | Animals | Endomyocardial Fibrosis - metabolism | Mice | Cell Hypoxia - genetics | Genetic Vectors | Cell Movement | RNA, Small Interfering - metabolism | Heart | Collagen | Adenoviruses | Fibrosis | Tumor proteins | Health aspects | Heart attack | Myocardial infarction | Regulators | Senescence | Immunoglobulins | Matrix metalloproteinases | Heart attacks | p53 Protein | Rupture | siRNA | Macrophages | Genotype & phenotype | Hospitals | Education | Fibroblasts | Hypoxia | Infiltration | Infarction | Diabetes | Laboratory animals | Cardiovascular diseases | Heart diseases | Index Medicus
Journal Article
Blood, ISSN 0006-4971, 07/2017, Volume 130, Issue 2, pp. 205 - 213
Journal Article