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Nucleic acids research, ISSN 1362-4962, 2017, Volume 45, Issue 17, pp. 9901 - 9916
Journal Article
Nature (London), ISSN 1476-4687, 2015, Volume 526, Issue 7571, pp. 136 - 139
Journal Article
Archives of pathology & laboratory medicine (1976), ISSN 0003-9985, 2014, Volume 138, Issue 8, pp. 1015 - 1026
Journal Article
Cell (Cambridge), ISSN 0092-8674, 2016, Volume 167, Issue 1, pp. 233 - 247.e17
Journal Article
Cell reports (Cambridge), ISSN 2211-1247, 2015, Volume 13, Issue 1, pp. 70 - 79
The human bacterial pathogen Helicobacter pylori exhibits genotoxic properties that promote gastric carcinogenesis. H. pylori introduces DNA double strand... 
HOST-CELLS | DAMAGE RESPONSE | MUTAGENESIS | ACTIVATION | HELICOBACTER-PYLORI | CELL BIOLOGY | Chromatin - metabolism | Interleukin-8 - genetics | RNA, Small Interfering - genetics | Endonucleases - genetics | Epithelial Cells - metabolism | Inhibitor of Apoptosis Proteins - genetics | Humans | Transcriptional Activation | Helicobacter pylori - genetics | Helicobacter pylori - pathogenicity | Type IV Secretion Systems - genetics | NF-kappa B - metabolism | Endonucleases - metabolism | I-kappa B Proteins - metabolism | DNA Breaks, Double-Stranded | I-kappa B Proteins - genetics | Transcription Factor RelA - genetics | DNA-Binding Proteins - metabolism | DNA End-Joining Repair | Inhibitor of Apoptosis Proteins - metabolism | Interleukin-8 - metabolism | Nuclear Proteins - genetics | Chromatin - chemistry | Type IV Secretion Systems - metabolism | NF-KappaB Inhibitor alpha | Signal Transduction | Cell Survival | Helicobacter pylori - growth & development | Epithelial Cells - pathology | Nuclear Proteins - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Host-Pathogen Interactions | Integrin beta1 - metabolism | Transcription Factors - metabolism | NF-kappa B - genetics | Transcription Factor RelA - metabolism | Epithelial Cells - microbiology | Cell Line, Tumor | Integrin beta1 - genetics | RNA, Small Interfering - metabolism
Journal Article
Aging cell, ISSN 1474-9718, 2015, Volume 14, Issue 4, pp. 644 - 658
Summary The healthspan of mice is enhanced by killing senescent cells using a transgenic suicide gene. Achieving the same using small molecules would have a... 
dasatinib | plasminogen‐activated inhibitor | dependence receptors | quercetin | ephrins | PI3K delta | p21 | Dasatinib | Dependence receptors | Ephrins | Plasminogen-activated inhibitor | Quercetin | P21 | ENDOTHELIAL DYSFUNCTION | PLASMINOGEN-ACTIVATOR INHIBITOR-1 | EXPRESSION PROFILES | plasminogen-activated inhibitor | CELLULAR SENESCENCE | CANCER-THERAPY | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | LUNG-CANCER | SET ENRICHMENT ANALYSIS | GENE-EXPRESSION | IONIZING-RADIATION | TUMOR-GROWTH | Carotid Arteries - drug effects | Endonucleases - genetics | Plasminogen Activator Inhibitor 2 - genetics | Transcriptome | Gene Expression Profiling | Adipocytes - drug effects | Aging - genetics | Osteoporosis - metabolism | Ephrins - metabolism | Plasminogen Activator Inhibitor 2 - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Osteoporosis - genetics | Intervertebral Disc - pathology | Fibroblasts - metabolism | Endothelial Cells - metabolism | Intervertebral Disc - chemistry | Fibroblasts - pathology | Mesenchymal Stem Cells - pathology | Mice, Knockout | Dasatinib - pharmacology | Osteoporosis - pathology | Ephrins - genetics | Fibroblasts - drug effects | Mice | Endothelial Cells - pathology | bcl-X Protein - metabolism | Aging - metabolism | Aging - drug effects | bcl-X Protein - genetics | Cellular Senescence - drug effects | Phosphatidylinositol 3-Kinases - metabolism | Endonucleases - metabolism | DNA-Binding Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Mesenchymal Stem Cells - drug effects | Mesenchymal Stem Cells - metabolism | Heart - physiopathology | Cellular Senescence - genetics | Osteoporosis - prevention & control | DNA-Binding Proteins - genetics | Adipocytes - pathology | Aging - pathology | Phosphatidylinositol 3-Kinases - genetics | Animals | Quercetin - pharmacology | Adipocytes - metabolism | Heart - drug effects | Carotid Arteries - pathology | Intervertebral Disc - drug effects | Drug Combinations | Endothelial Cells - drug effects | Original
Journal Article
Nature (London), ISSN 1476-4687, 2018, Volume 554, Issue 7690, pp. 112 - 117
Many craniofacial disorders are caused by heterozygous mutations in general regulators of housekeeping cellular functions such as transcription or ribosome... 
PATHOGENESIS | CELLS | PROTEIN | ENDONUCLEASE | RNA HELICASE DDX21 | DNA | MULTIDISCIPLINARY SCIENCES | PREVENTION | TREACHER-COLLINS-SYNDROME | NEURAL CREST | P53 | Xenopus | Chromatin - metabolism | Cell Nucleolus - pathology | Embryonic Stem Cells - cytology | Mandibulofacial Dysostosis - embryology | Humans | Protein Transport - drug effects | DNA-Directed RNA Polymerases - deficiency | RNA, Ribosomal - genetics | RNA Polymerase I - antagonists & inhibitors | Mandibulofacial Dysostosis - genetics | Cell Nucleus - metabolism | Skull - pathology | Nuclear Proteins - deficiency | Cell Nucleolus - drug effects | Stress, Physiological - drug effects | Neural Crest - enzymology | Benzothiazoles - pharmacology | Phenotype | Zebrafish Proteins - deficiency | Ribosomes - genetics | Mice | DNA Damage | HeLa Cells | Embryonic Stem Cells - metabolism | RNA Helicases - metabolism | Mandibulofacial Dysostosis - pathology | Neural Crest - pathology | Ribosomal Proteins - biosynthesis | Ribosomes - metabolism | DNA, Ribosomal - metabolism | Phosphoproteins - metabolism | Zebrafish - embryology | Cell Nucleus - pathology | Cell Nucleolus - metabolism | DEAD-box RNA Helicases - deficiency | DEAD-box RNA Helicases - metabolism | Nuclear Proteins - genetics | DNA, Ribosomal - genetics | Cell Nucleolus - genetics | Ribosomal Proteins - genetics | Tumor Suppressor Protein p53 - metabolism | RNA, Ribosomal - metabolism | Nuclear Proteins - metabolism | Phosphoproteins - genetics | Organ Specificity | DEAD-box RNA Helicases - genetics | Animals | Phosphoproteins - deficiency | RNA, Ribosomal - biosynthesis | Cell Nucleus - drug effects | Naphthyridines - pharmacology | Apoptosis | Nucleolus organizer region | Genetic aspects | Gene mutations | Observations | Neural crest | Craniofacial dysostosis | Chromatin | Transcription | Genomics | p53 Protein | Genes | DNA damage | Ribosomal DNA | Disorders | Homeostasis | Biosynthesis | Genomes | DNA-directed RNA polymerase | Proteins | DNA helicase | Nucleoli | Embryogenesis | Cell activation | RNA processing | Chemical synthesis | Deoxyribonucleic acid--DNA | Anemia | rRNA | RNA polymerase | Ribonucleic acid--RNA | Embryonic growth stage | DNA biosynthesis | Craniofacial growth | Stem cells | Mutation | Developmental disabilities | Tumors | RNA helicase
Journal Article
Journal Article