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International Journal of Developmental Neuroscience, ISSN 0736-5748, 02/2016, Volume 48, pp. 58 - 70
The endoplasmic reticulum (ER) is tasked, among many other functions, with preventing excitotoxicity from killing neurons following neonatal hypoxia-ischemia... 
Cytoplasmic shedding | Neonatal hypoxia-ischemia | Cell death | Macrozeiosis | Programmed necrosis | Endoplasmic reticulum stress | Unfolded protein response | BRAIN-INJURY | INDUCED APOPTOSIS | NEURODEGENERATION | DEVELOPMENTAL BIOLOGY | NEUROSCIENCES | CELL-DEATH | ENCEPHALOPATHY | BIOMARKERS | MESSENGER-RNA | MITOCHONDRIAL DYSFUNCTION | RAT BRAIN | Neurons - pathology | Calpain - metabolism | eIF-2 Kinase - metabolism | Humans | Male | Hypoxia-Ischemia, Brain - pathology | Mitochondria - ultrastructure | Endoplasmic Reticulum - ultrastructure | Endoplasmic Reticulum - pathology | Caspases - metabolism | Neurons - ultrastructure | Neuroprotective Agents - pharmacology | Time Factors | Brain - ultrastructure | Indoles - pharmacology | Microfilament Proteins - metabolism | Gene Expression Regulation, Developmental - physiology | eIF-2 Kinase - genetics | Animals, Newborn | Imidazoles - pharmacology | Mitochondria - pathology | Hypoxia-Ischemia, Brain - complications | Animals | Carrier Proteins - metabolism | Necrosis - etiology | Brain - pathology | Mice | Apoptosis - physiology | Endoplasmic Reticulum Stress - physiology | Infants (Newborn) | Mitochondrial DNA | Ischemia | Neurons | Necrosis | Index Medicus | Programmed Necrosis | Neonatal Hypoxia-Ischemia | Endoplasmic Reticulum Stress | Cell Death | Unfolded Protein Response | Cytoplasmic Shedding
Journal Article
Nature, ISSN 0028-0836, 05/2011, Volume 473, Issue 7348, pp. 528 - 531
Journal Article
Journal Article
Journal Article
PLoS Medicine, ISSN 1549-1277, 03/2008, Volume 5, Issue 3, pp. 0440 - 0460
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2015, Volume 35, Issue 1, pp. 60 - 70
OBJECTIVE—S-Adenosylhomocysteine (SAH) is a better predictor of cardiovascular disease than homocysteine is, and it has been implicated in mediating the... 
S-adenosylhomocysteine | endoplasmic reticulum stress | epigenenomics | Atherosclerosis | atherosclerosis | HOMOCYSTEINE | DNA HYPOMETHYLATION | CORONARY-ANGIOGRAPHY | CYSTATHIONINE BETA-SYNTHASE | XENOPUS-LAEVIS | APOE-DEFICIENT MICE | GENE-EXPRESSION | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | VASCULAR ENDOTHELIAL-CELLS | HEMATOLOGY | Up-Regulation | Adenosylhomocysteinase - antagonists & inhibitors | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Epigenesis, Genetic | Aortic Diseases - blood | Endoplasmic Reticulum - metabolism | Male | Endoplasmic Reticulum Stress - genetics | Endoplasmic Reticulum - pathology | Heat-Shock Proteins - genetics | Atherosclerosis - enzymology | RNA Interference | Time Factors | Adenosylhomocysteinase - genetics | Aortic Diseases - enzymology | CCAAT-Enhancer-Binding Proteins - genetics | Binding Sites | Aortic Diseases - pathology | Disease Models, Animal | CCAAT-Enhancer-Binding Proteins - metabolism | Cell Line | Promoter Regions, Genetic | Atherosclerosis - pathology | S-Adenosylhomocysteine - blood | Heat-Shock Proteins - metabolism | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Adenosylhomocysteinase - metabolism | Adenosine - pharmacology | Plaque, Atherosclerotic | Mice, Knockout | Atherosclerosis - blood | Gene Expression Regulation, Enzymologic | Aortic Diseases - genetics | Animals | Histone-Lysine N-Methyltransferase - metabolism | Adenosine - analogs & derivatives | Apolipoproteins E - genetics | Histones - metabolism | Methylation | RNA, Small Interfering - administration & dosage | Index Medicus
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 10/2008, Volume 178, Issue 8, pp. 838 - 846
Rationale The molecular pathomechanisms underlying idiopathic pulmonary fibrosis (IPF) are elusive, but chronic epithelial injury has recently been suggested... 
C/EBP-homologous protein | X-box binding protein-1 | Type II alveolar epithelial cell | Interstitial lung disease | Lung fibrosis | LUNG | OXIDATIVE STRESS | ACTIVATION | INTERSTITIAL PNEUMONIA | lung fibrosis | CELL-DEATH | PATHOGENESIS | UNFOLDED PROTEIN RESPONSE | interstitial lung disease | RESPIRATORY SYSTEM | CHOP | MYOFIBROBLASTS | MUTATIONS | type II alveolar epithelial cell | CRITICAL CARE MEDICINE | Immunohistochemistry | Humans | Middle Aged | Oxidative Stress - physiology | Endoplasmic Reticulum - metabolism | Male | Pulmonary Fibrosis - genetics | Pulmonary Disease, Chronic Obstructive - pathology | Activating Transcription Factor 6 - genetics | X-Box Binding Protein 1 | RNA - genetics | Activating Transcription Factor 6 - biosynthesis | Endoplasmic Reticulum - pathology | Pulmonary Alveoli - metabolism | Female | Pulmonary Fibrosis - metabolism | Pulmonary Disease, Chronic Obstructive - metabolism | Pulmonary Disease, Chronic Obstructive - genetics | In Situ Nick-End Labeling | Severity of Illness Index | Gene Expression | Pulmonary Alveoli - pathology | Activating Transcription Factor 4 - genetics | Transcription Factors - biosynthesis | Pulmonary Fibrosis - pathology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Regulatory Factor X Transcription Factors | Blotting, Western | Activating Transcription Factor 4 - biosynthesis | Apoptosis - physiology | DNA-Binding Proteins - biosynthesis | Index Medicus | Abridged Index Medicus | EBP-homologous protein | E. Interstitial Lung Disease
Journal Article