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1. Full Text Increased Alzheimer's disease–like pathology in the APP/ PS1ΔE9 mouse model lacking Nrf2 through modulation of autophagy
by Joshi, Gururaj and Gan, Kok Ann and Johnson, Delinda A and Johnson, Jeffrey A
Neurobiology of aging, ISSN 0197-4580, 2015, Volume 36, Issue 2, pp. 664 - 679
Neurology | Internal Medicine | p62/SQSTM | mTOR pathway | Amyloid β peptide | Autophagy | Amyloid precursor protein (APP) | MTOR pathway | P62/SQSTM | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Geriatrics & Gerontology | Science & Technology | NF-E2-Related Factor 2 - physiology | Alzheimer Disease - therapy | Mice, Transgenic | Molecular Targeted Therapy | Endosomes - metabolism | Alzheimer Disease - pathology | Animals | Lysosomes - metabolism | Gene Deletion | Amyloid beta-Peptides - metabolism | Amyloid beta-Protein Precursor - metabolism | NF-E2-Related Factor 2 - genetics | Autophagy - genetics | Alzheimer Disease - genetics | Disease Models, Animal | Index Medicus
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2. Full Text Intraneuronal β-amyloid accumulation and synapse pathology in Alzheimer's disease
by Gouras, Gunnar K and Tampellini, Davide and Takahashi, Reisuke H and Capetillo-Zarate, Estibaliz
Acta neuropathologica, ISSN 0001-6322, 05/2010, Volume 119, Issue 5, pp. 523 - 541
Tau | Synapse | Amyloid | Head injury | Endosome | Dementia pugilistica | Pathology | Neurosciences | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Brain - metabolism | Neurons - pathology | Synapses - metabolism | Humans | Alzheimer Disease - metabolism | Amyloid beta-Peptides - metabolism | Brain - pathology | tau Proteins - metabolism | Neurons - metabolism | Synapses - pathology | Alzheimer Disease - pathology | Index Medicus | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Neurovetenskaper
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3. Full Text Perforin and granzymes: Function, dysfunction and human pathology
by Voskoboinik, Ilia and Whisstock, James C and Trapani, Joseph A
Nature reviews. Immunology, ISSN 1474-1733, 06/2015, Volume 15, Issue 6, pp. 388 - 400
Life Sciences & Biomedicine | Immunology | Science & Technology | T-Lymphocytes, Cytotoxic - immunology | Cell Communication - immunology | Endosomes - immunology | Perforin - genetics | Immune System Diseases - genetics | Infection - immunology | Cytosol - immunology | Humans | Perforin - chemistry | Immune System Diseases - immunology | Inflammation - immunology | Exocytosis - physiology | Polymorphism, Genetic | Granzymes - chemistry | Secretory Vesicles - immunology | Animals | Apoptosis - immunology | Neoplasms - genetics | Neoplasms - immunology | Killer Cells, Natural - immunology | Granzymes - immunology | Perforin - immunology | Cellular proteins | Chronic diseases | Protein biosynthesis | Development and progression | Genetic aspects | Properties | Health aspects | Index Medicus
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4. Full Text Intraneuronal aggregation of the β-CTF fragment of APP (C99) induces Aβ-independent lysosomal-autophagic pathology
by Lauritzen, Inger and Pardossi-Piquard, Raphaëlle and Bourgeois, Alexandre and Pagnotta, Sophie and Biferi, Maria-Grazia and Barkats, Martine and Lacor, Pascale and Klein, William and Bauer, Charlotte and Checler, Frederic
Acta neuropathologica, ISSN 0001-6322, 8/2016, Volume 132, Issue 2, pp. 257 - 276
Aggregation | Pathology | Neurosciences | C99 | Medicine & Public Health | Triple-transgenic mouse | Lysosomes | Alzheimer | Autophagy | γ-Secretase inhibition | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neurons - pathology | Mice, Inbred C57BL | Mice, Transgenic | Autophagy - physiology | Endosomes - metabolism | Alzheimer Disease - pathology | Brain - metabolism | Amyloid Precursor Protein Secretases - metabolism | Animals | Lysosomes - metabolism | Alzheimer Disease - metabolism | Amyloid beta-Peptides - metabolism | Amyloid beta-Protein Precursor - metabolism | Brain - pathology | Neurons - metabolism | Disease Models, Animal | Index Medicus | Life Sciences | Original Paper
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5. Full Text Down Syndrome Fibroblast Model of Alzheimer-Related Endosome Pathology: Accelerated Endocytosis Promotes Late Endocytic Defects
by Cataldo, Anne M and Mathews, Paul M and Boiteau, Anne Boyer and Hassinger, Linda C and Peterhoff, Corrinne M and Jiang, Ying and Mullaney, Kerry and Neve, Rachael L and Gruenberg, Jean and Nixon, Ralph A
The American journal of pathology, ISSN 0002-9440, 08/2008, Volume 173, Issue 2, pp. 370 - 384
Life Sciences & Biomedicine | Pathology | Science & Technology | Biological and medical sciences | Investigative techniques, diagnostic techniques (general aspects) | Medical sciences | Pathology. Cytology. Biochemistry. Spectrometry. Miscellaneous investigative techniques | Chromosome aberrations | Medical genetics | Hydrolases - metabolism | rab GTP-Binding Proteins - metabolism | Down Syndrome - pathology | Lysophospholipids - metabolism | Humans | Middle Aged | Cells, Cultured | Endosomes - pathology | Fibroblasts - pathology | Monoglycerides - metabolism | rab5 GTP-Binding Proteins - metabolism | Alzheimer Disease - pathology | Endocytosis - physiology | Biological Transport, Active | Aged, 80 and over | Aged | Index Medicus | Abridged Index Medicus | Regular
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6. Full Text Niemann–Pick type C disease: cellular pathology and pharmacotherapy
by Wheeler, Simon and Sillence, Dan J
Journal of neurochemistry, ISSN 0022-3042, 06/2020, Volume 153, Issue 6, pp. 674 - 692
cholesterol | endocytosis | Niemann‐Pick | GBA2 | lysosome | glucosylceramide | Biochemistry & Molecular Biology | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Spleen | Signs and symptoms | Lysosomes | Sphingolipids | Motor task performance | Organelles | Autophagy | Cholesterol | Proteins | Pathology | Malfunctions | Endocytosis | Mitochondria | Pathways | Cell death | Npc1 protein | Metabolic pathways | Mutation | Phagocytosis | Endosomes | Index Medicus
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