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1. Full Text Oncometabolite 2-Hydroxyglutarate Is a Competitive Inhibitor of α-Ketoglutarate-Dependent Dioxygenases
by Xu, Yan-hui and Xu, Wei and Yang, Chen and Yang, Ying and Yang, Hui and Liu, Li-xia and Liu, Ying and Liu, Jing and Wang, Bin and Wang, Ping and Wang, Pu and Kim, Se-Hee and Ito, Shinsuke and Xiao, Meng-Tao and Jiang, Wen-qing and Zhang, Yi and Zhang, Jin-ye and Frye, Stephen and Lei, Qun-ying and Guan, Kun-Liang and Zhao, Shi-min and Xiong, Yue
Cancer Cell, ISSN 1535-6108, 2011, Volume 19, Issue 1, pp. 17 - 30
and mutations occur frequently in gliomas and acute myeloid leukemia, leading to simultaneous loss and gain of activities in the production of α-ketoglutarate... 
BREAST-CANCER | TRANSCRIPTIONAL ACTIVITY | IDH2 MUTATIONS | ONCOLOGY | PROLYL HYDROXYLATION | INTEGRATED GENOMIC ANALYSIS | 2-OXOGLUTARATE OXYGENASES | ACUTE MYELOID-LEUKEMIA | HIF-ALPHA | HISTONE DEMETHYLATION | FAMILY | CELL BIOLOGY | Dioxygenases - metabolism | Histone Demethylases - antagonists & inhibitors | Gene Expression - genetics | Caenorhabditis elegans Proteins - chemistry | Humans | Ketoglutaric Acids - chemistry | Glioma - genetics | F-Box Proteins | Oxidoreductases, N-Demethylating - antagonists & inhibitors | Ketoglutaric Acids - pharmacology | Cytosine - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Glutarates - chemistry | Oxidoreductases, N-Demethylating - metabolism | DNA-Binding Proteins - antagonists & inhibitors | Glioma - enzymology | Endostatins - metabolism | Models, Molecular | Isocitrate Dehydrogenase - genetics | Histone Demethylases - metabolism | Dioxygenases - antagonists & inhibitors | Amino Acid Substitution - physiology | Procollagen-Proline Dioxygenase - genetics | Cell Line, Tumor | Isocitrate Dehydrogenase - metabolism | Glutarates - pharmacology | Histones - metabolism | Jumonji Domain-Containing Histone Demethylases - metabolism | Caenorhabditis elegans - enzymology | Cytosine - analogs & derivatives | Gene Expression - drug effects | Caenorhabditis elegans Proteins - metabolism | Isocitrate Dehydrogenase - antagonists & inhibitors | Glioma - metabolism | Procollagen-Proline Dioxygenase - metabolism | DNA-Binding Proteins - metabolism | Mixed Function Oxygenases | Biocatalysis - drug effects | Jumonji Domain-Containing Histone Demethylases - antagonists & inhibitors | Jumonji Domain-Containing Histone Demethylases - chemistry | Procollagen-Proline Dioxygenase - antagonists & inhibitors | Ketoglutaric Acids - metabolism | Oxalates - pharmacology | Binding, Competitive | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Catalytic Domain | Proto-Oncogene Proteins - genetics | Hypoxia-Inducible Factor-Proline Dioxygenases | DNA-Binding Proteins - genetics | Homeodomain Proteins - genetics | Animals | 5-Methylcytosine - metabolism | Caenorhabditis elegans Proteins - antagonists & inhibitors | Glutarates - metabolism
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2. Full Text Corneal angiogenic privilege: Angiogenic and antiangiogenic factors in corneal avascularity, vasculogenesis, and wound healing (An American Ophthalmological Society Thesis)
by Azar, Dimitri T
Transactions of the American Ophthalmological Society, ISSN 0065-9533, 2006, Volume 104, pp. 264 - 302
To determine the molecular basis of corneal avascularity during wound healing and determine the role of angiogenic and antiangiogenic factors in corneal... 
Microtubule-Associated Proteins - metabolism | Nerve Growth Factors - metabolism | Vascular Endothelial Growth Factor A - metabolism | Neoplasm Proteins - metabolism | Cornea - blood supply | Angiogenesis Inducing Agents - metabolism | Fibroblast Growth Factor 2 - metabolism | Angiostatins - metabolism | Cornea - pathology | Fluorescent Antibody Technique, Indirect | Disease Models, Animal | Serpins - metabolism | Endostatins - metabolism | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | Matrix Metalloproteinase 14 - metabolism | Blotting, Western | Cornea - metabolism | Microscopy, Confocal | Angiogenesis Inhibitors - metabolism | Animals | Corneal Neovascularization - metabolism | Eye Proteins - metabolism | Mice | Wound Healing - physiology | Angiogenic Proteins - metabolism | Corneal Neovascularization - pathology | 2006 Theses
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3. Matricryptins derived from collagens and proteoglycans
by Ricard-Blum, Sylvie and Ballut, Lionel
Frontiers in Bioscience, ISSN 1093-9946, 01/2011, Volume 16, Issue 2, pp. 674 - 697
Controlled proteolysis of extracellular matrix components releases bioactive fragments or unmasks cryptic sites that play key roles in controlling various... 
Matricryptic sites | Extracellular matrix | Matricryptins | Proteoglycans | Review | Collagens | BIOCHEMISTRY & MOLECULAR BIOLOGY | IV COLLAGEN | BASEMENT-MEMBRANE COLLAGEN | HUMAN POLYMORPHONUCLEAR NEUTROPHILS | CELL BIOLOGY | BREAST-CANCER CELLS | TISSUE-INJURY RESPONSES | RECOMBINANT HUMAN ENDOSTATIN | SUPPRESS TUMOR-GROWTH | EXTRACELLULAR-MATRIX | FOCAL ADHESION KINASE | ANGIOGENESIS INHIBITOR ENDOSTATIN | Microtubule-Associated Proteins - metabolism | Angiogenesis Inhibitors - physiology | Humans | Extracellular Matrix - metabolism | Neoplasm Proteins - metabolism | Collagen Type II - metabolism | Antineoplastic Agents - pharmacology | Collagen Type XVIII - metabolism | Calcium-Binding Proteins - metabolism | Collagen Type IV - metabolism | Peptide Fragments - metabolism | Collagen Type I - metabolism | Glycosaminoglycans - metabolism | Proteoglycans - metabolism | Receptors, Cell Surface - metabolism | Peptide Fragments - biosynthesis | Collagen - metabolism | Animals | Heparan Sulfate Proteoglycans - metabolism | Hyaluronic Acid - metabolism | Peptide Fragments - therapeutic use | Wound Healing - physiology | Matrix Metalloproteinases - metabolism | Extracellular Matrix - pathology | Neovascularization, Physiologic
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4. Full Text IL-32 promotes angiogenesis
by Nold-Petry, C.A and Rudloff, I and Baumer, Y and Ruvo, M and Marasco, D and Botti, P and Farkas, L and Cho, S.X and Zepp, J.A and Azam, T and Dinkel, H and Palmer, B.E and Boisvert, W.A and Cool, C.D and Taraseviciene-Stewart, L and Heinhuis, B and Joosten, L.A and Dinarello, C.A and Voelkel, N.F and Nold, M.F
Journal of Immunology, ISSN 0022-1767, 2014, Volume 192, Issue 2, pp. 589 - 602
IL-32 is a multifaceted cytokine with a role in infections, autoimmune diseases, and cancer, and it exerts diverse functions, including aggravation of... 
PROINFLAMMATORY CYTOKINE | ACTIVATION | PULMONARY-HYPERTENSION | INFLAMMATION | ENDOTHELIAL-CELLS | PLEXIFORM LESIONS | GROWTH | FOCAL ADHESION KINASE | IMMUNOLOGY | NF-KAPPA-B | INTERLEUKIN-32 EXPRESSION | Human Umbilical Vein Endothelial Cells | Humans | Transforming Growth Factor beta1 - metabolism | Activins - metabolism | Vascular Endothelial Growth Factor A - metabolism | Glioblastoma - embryology | Interferon-gamma - metabolism | Integrin alphaVbeta3 - metabolism | Interleukins - metabolism | Neovascularization, Pathologic - pathology | Matrix Metalloproteinase 9 - metabolism | Receptors, Vascular Endothelial Growth Factor - metabolism | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Interleukin-8 - metabolism | Nitrogen Oxides - metabolism | Endostatins - metabolism | Cells, Cultured | Familial Primary Pulmonary Hypertension | Hypertension, Pulmonary - metabolism | Animals | Glioblastoma - pathology | Mice | Neovascularization, Pathologic - metabolism | Apoptosis - physiology | Hypertension, Pulmonary - pathology
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5. Full Text Cathepsin S Controls Angiogenesis and Tumor Growth via Matrix-derived Angiogenic Factors
by Bing Wang and Jiusong Sun and Shiro Kitamoto and Min Yang and Anders Grubb and Harold A. Chapman and Raghu Kalluri and Guo-Ping Shi and Division of Clinical Chemistry and Pharmacology and Avdelningen för klinisk kemi och farmakologi and Lund University and Lunds universitet
Journal of Biological Chemistry, ISSN 0021-9258, 03/2006, Volume 281, Issue 9, pp. 6020 - 6029
The cysteine protease cathepsin S is highly expressed in malignant tissues. By using a mouse model of multistage murine pancreatic islet cell carcinogenesis in... 
CANCER-CELLS | ENDOTHELIAL-CELL PROLIFERATION | FACTOR-BETA | COLLAGEN | LAMININ-5 GAMMA-2 CHAIN | INTEGRIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | CYSTATIN-C | GENETIC-HETEROGENEITY | PROTEOLYSIS | CYSTEINE PROTEASES | Neoplasms - metabolism | Autoantigens - metabolism | Cell Proliferation | Pancreatic Neoplasms - metabolism | Humans | Neovascularization, Pathologic | Extracellular Matrix - metabolism | Recombinant Fusion Proteins - metabolism | Extracellular Matrix - chemistry | Angiogenesis Inducing Agents - metabolism | Cystatin C | Collagen Type IV - metabolism | Peptide Fragments - metabolism | Endostatins - metabolism | Mice, Inbred C57BL | Pancreatic Neoplasms - pathology | Mice, Transgenic | Protease Inhibitors - metabolism | Survival Rate | Cystatins - metabolism | Cell Adhesion Molecules - metabolism | Mice, Knockout | Angiogenesis Inhibitors - metabolism | Animals | Cathepsins - genetics | Cystatins - genetics | Cathepsins - metabolism | Recombinant Fusion Proteins - genetics | Mice | Neoplasms - pathology | Medicinal Chemistry | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Farmakologi och toxikologi | Pharmacology and Toxicology | Läkemedelskemi
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6. Full Text Distinct platelet packaging, release, and surface expression of proangiogenic and antiangiogenic factors on different platelet stimuli
by Chatterjee, Madhumita and Huang, Zhangsen and Zhang, Wei and Jiang, Lei and Hultenby, Kjell and Zhu, Linjing and Hu, Hu and Nilsson, Gunnar P and Li, Nailin
Blood, ISSN 0006-4971, 04/2011, Volume 117, Issue 14, pp. 3907 - 3911
The present study characterized platelet secretion and surface expression of proangiogenic stromal cell-derived factor-1 alpha (SDF-1 alpha) and vascular... 
PROGENITOR CELLS | ADHESION | ATHEROGENESIS | ACTIVATION | ANGIOGENESIS | VIVO | INFLAMMATION | FACTOR-I | HEMATOLOGY | GRANULES | ENDOSTATIN | Membrane Glycoproteins - metabolism | Endostatins - metabolism | Humans | Middle Aged | Receptor, PAR-1 - agonists | Platelet Activation - physiology | Male | Vascular Endothelial Growth Factor A - metabolism | Blood Platelets - secretion | Angiogenesis Inhibitors - metabolism | Carrier Proteins - metabolism | Chemokine CXCL12 - metabolism | Peptides - metabolism | Blood Platelets - metabolism | Angiogenesis Inducing Agents - metabolism | Platelet Membrane Glycoproteins - metabolism | Fluorescent Antibody Technique | Adult | Female | Platelet Activation - drug effects | Blood Platelets - drug effects | In Vitro Techniques | Oligopeptides - pharmacology
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7. Full Text Angiogenesis in chronic liver disease and its complications
by Coulon, Stephanie and Heindryckx, Femke and Geerts, Anja and Van Steenkiste, Christophe and Colle, Isabelle and Van Vlierberghe, Hans
Liver International, ISSN 1478-3223, 02/2011, Volume 31, Issue 2, pp. 146 - 162
Nowadays, liver cancer, cirrhosis and other liver‐related diseases are the fifth most common cause of mortality in the UK. Furthermore, chronic liver diseases... 
chronic liver diseases | non‐alcoholic steatohepatitis | portal hypertension | angiogenesis | hepatocellular carcinoma | Angiogenesis | Chronic liver diseases | Hepatocellular carcinoma | Non-alcoholic steatohepatitis | Portal hypertension | ADVANCED HEPATOCELLULAR-CARCINOMA | HYPOXIA-INDUCIBLE FACTOR | PORTAL HYPERTENSIVE-RATS | ENDOTHELIAL-GROWTH-FACTOR | NONALCOHOLIC STEATOHEPATITIS | FACTOR RECEPTOR | PHASE-II TRIAL | HEPATIC STELLATE CELLS | TYROSINE KINASE INHIBITOR | HYPERDYNAMIC SPLANCHNIC CIRCULATION | GASTROENTEROLOGY & HEPATOLOGY | non-alcoholic steatohepatitis | Microvessels - physiopathology | Hypertension, Portal - physiopathology | Cadherins - metabolism | Hypertension, Portal - complications | Humans | Fatty Liver - complications | Liver Neoplasms - complications | Vascular Endothelial Growth Factor A - metabolism | Hypoxia-Inducible Factor 1 - metabolism | Integrins - metabolism | Neovascularization, Pathologic - etiology | Fibroblast Growth Factors - metabolism | Carcinoma, Hepatocellular - drug therapy | Receptors, Vascular Endothelial Growth Factor - metabolism | Carcinoma, Hepatocellular - complications | Liver Neoplasms - physiopathology | Non-alcoholic Fatty Liver Disease | Angiogenesis Inhibitors - therapeutic use | Neovascularization, Pathologic - physiopathology | Angiostatins - metabolism | Angiopoietins - metabolism | Carcinoma, Hepatocellular - physiopathology | Endostatins - metabolism | Liver Cirrhosis - complications | Fatty Liver - physiopathology | Liver Neoplasms - drug therapy | Disease Progression | Platelet-Derived Growth Factor - metabolism | Models, Biological | Signal Transduction - physiology | Liver Cirrhosis - physiopathology | Chronic Disease | Thrombospondins - metabolism | Hypertension | Complications and side effects | Liver cancer | Development and progression | Mortality
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8. Full Text Increased endogenous H2S generation by CBS, CSE, and 3MST gene therapy improves ex vivo renovascular relaxation in hyperhomocysteinemia
by Utpal Sen and Pushpakumar B. Sathnur and Sourav Kundu and Srikanth Givvimani and Denise M. Coley and Paras K. Mishra and Natia Qipshidze and Neetu Tyagi and Naira Metreveli and Suresh C. Tyagi
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 07/2012, Volume 303, Issue 1, pp. 41 - 51
Hydrogen sulfide (H2S) has recently been identified as a regulator of various physiological events, including vasodilation, angiogenesis, antiapoptotic, and... 
Hydrogen sulfide | Cystathionine γ-lyase | Cystathionine β-synthase | 3-mercaptopyruvate sulfurtransferase | Mitophagy | homocysteine | cystathionine gamma-lyase | OXIDATIVE STRESS | PHYSIOLOGY | ENDOTHELIAL DYSFUNCTION | DIABETIC-NEPHROPATHY | ENDOPLASMIC-RETICULUM STRESS | SMOOTH-MUSCLE-CELL | HYDROGEN-SULFIDE | CELL BIOLOGY | MATRIX METALLOPROTEINASES | mitophagy | hydrogen sulfide | EXTRACELLULAR-MATRIX | cystathionine beta-synthase | TIMP-1 EXPRESSION | Hydrogen Sulfide - metabolism | Genetic Therapy | Vascular Endothelial Growth Factor A - biosynthesis | Reactive Oxygen Species - metabolism | Hyperhomocysteinemia - therapy | Hyperhomocysteinemia - metabolism | Cystathionine gamma-Lyase - metabolism | Homocysteine - metabolism | Hypertension, Renovascular - genetics | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Forkhead Transcription Factors - metabolism | Renal Artery - metabolism | Cystathionine gamma-Lyase - genetics | Cystathionine beta-Synthase - genetics | Proto-Oncogene Proteins c-akt - metabolism | Organ Culture Techniques | Endostatins - biosynthesis | Mice, Inbred C57BL | Cells, Cultured | Sulfurtransferases - genetics | Hypertension, Renovascular - therapy | Cystathionine beta-Synthase - metabolism | Matrix Metalloproteinase 13 - metabolism | Animals | Hyperhomocysteinemia - genetics | Mice | Forkhead Box Protein O3 | Platelet Endothelial Cell Adhesion Molecule-1 - biosynthesis | Vascular System Injuries | Sulfurtransferases - metabolism | cystathionine γ-lyase | cystathionine β-synthase
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9. Full Text Generation and degradation of human endostatin proteins by various proteinases
by Ferreras, Mercedes and Felbor, Ute and Lenhard, Thomas and Olsen, Bjorn R and Delaissé, Jean-Marie
FEBS Letters, ISSN 0014-5793, 2000, Volume 486, Issue 3, pp. 247 - 251
The angiogenesis inhibitor endostatin is a fragment of the NC1 domain of collagen XVIII. The generation of endostatin has been investigated only in murine... 
Angiogenesis | Collagen XVIII | NC1 | Proteinase | Endostatin | hNC1, human NC1 | hES, human endostatin | MMP, matrix metalloproteinase | mNC1, murine NC1 | DTT, dithiothreitol | angiogenesis | BIOCHEMISTRY & MOLECULAR BIOLOGY | XVIII COLLAGEN | CELL BIOLOGY | ALPHA-1(XVIII) | CHAIN | MATRIX METALLOPROTEINASES | BIOPHYSICS | proteinase | endostatin | NCl | CELL-MIGRATION | ANGIOSTATIN | collagen XVIII | ANGIOGENESIS INHIBITOR ENDOSTATIN | TUMOR-GROWTH | EXPRESSION | CARCINOMA | Humans | Collagen - chemistry | Sequence Analysis, Protein | Cysteine Endopeptidases - metabolism | Protein Structure, Tertiary | Recombinant Proteins - metabolism | Cell Line | Endopeptidases - metabolism | Cathepsin L | Peptide Fragments - metabolism | Cathepsin K | Collagen Type XVIII | Electrophoresis, Polyacrylamide Gel | Recombinant Proteins - chemistry | Cathepsin B - metabolism | Cathepsin D - metabolism | Peptide Fragments - analysis | Collagen - metabolism | Peptide Fragments - chemistry | Angiogenesis Inhibitors - metabolism | Cathepsins - metabolism | Aspartic Acid Endopeptidases - metabolism | Endostatins | Serine Endopeptidases - metabolism | Matrix Metalloproteinases - metabolism
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10. Full Text Tumstatin, an Endothelial Cell-Specific Inhibitor of Protein Synthesis
by Yohei Maeshima and Akulapalli Sudhakar and Julie C. Lively and Kohjiro Ueki and Surender Kharbanda and C. Ronald Kahn and Nahum Sonenberg and Richard O. Hynes and Raghu Kalluri
Science, ISSN 0036-8075, 1/2002, Volume 295, Issue 5552, pp. 140 - 143
Tumstatin is a 28-kilodalton fragment of type IV collagen that displays both anti-angiogenic and proapoptotic activity. Here we show that tumstatin functions... 
Phosphorylation | Messenger RNA | Protein synthesis | Long term potentiation | Reports | Angiogenesis inhibitors | Endothelial cells | Medical schools | Integrins | Apoptosis | Tumors | RAPAMYCIN | TARGET | ANGIOGENESIS | CAP-DEPENDENT INITIATION | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | KINASE | MICE | DISTINCT ANTITUMOR PROPERTIES | TRANSLATION | ENDOSTATIN | Endothelium, Vascular - cytology | Protein Kinases - metabolism | Autoantigens - metabolism | Protein-Tyrosine Kinases - metabolism | Humans | Protein-Serine-Threonine Kinases | Molecular Sequence Data | Endothelium, Vascular - drug effects | Peptide Fragments - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Phosphoproteins - metabolism | RNA Caps - metabolism | RNA, Messenger - metabolism | Eukaryotic Initiation Factor-4E | Focal Adhesion Kinase 1 | Collagen Type IV - chemistry | Collagen Type IV - pharmacology | Cattle | Protein Synthesis Inhibitors - pharmacology | Receptors, Vitronectin - metabolism | Tumor Cells, Cultured | Autoantigens - pharmacology | Collagen Type IV - metabolism | Proto-Oncogene Proteins - metabolism | Amino Acid Sequence | Signal Transduction | RNA, Messenger - genetics | Cells, Cultured | Focal Adhesion Protein-Tyrosine Kinases | Autoantigens - chemistry | Enzyme Activation - drug effects | Peptide Initiation Factors - metabolism | Proto-Oncogene Proteins c-akt | Adaptor Proteins, Signal Transducing | Protein Kinase Inhibitors | Animals | Carrier Proteins - metabolism | Endothelium, Vascular - metabolism | Protein Biosynthesis - drug effects | Mice | TOR Serine-Threonine Kinases | Proteins | Synthesis | Organic compounds | Cellular biology | Cells
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11. Full Text Angiogenic Properties of Human Dental Pulp Stem Cells
by Bronckaers, Annelies and Hilkens, Petra and Fanton, Yanick and Struys, Tom and Gervois, Pascal and Politis, Constantinus and Martens, Wendy and Lambrichts, Ivo
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e71104
Angiogenesis, the formation of capillaries from pre-existing blood vessels, is a key process in tissue engineering. If blood supply cannot be established... 
FIBROBLAST GROWTH FACTOR-2 | ACTIVATION | REGENERATION | MULTIDISCIPLINARY SCIENCES | OSTEOBLASTS | PHENOTYPE | PROLIFERATION | MECHANISMS | DIFFERENTIATION | STROMAL CELLS | EXPRESSION | Immunohistochemistry | Neovascularization, Physiologic - drug effects | Nitriles - pharmacology | Humans | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Culture Media, Conditioned - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor A - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Stem Cells - metabolism | Vascular Endothelial Growth Factor A - genetics | Young Adult | Plasminogen Activator Inhibitor 1 - metabolism | Angiogenesis Inducing Agents - metabolism | Chorioallantoic Membrane - drug effects | Chemokine CCL2 - metabolism | Angiogenesis Inducing Agents - pharmacology | Plasminogen Activator Inhibitor 1 - genetics | Chromones - pharmacology | Chorioallantoic Membrane - blood supply | Cell Line | Butadienes - pharmacology | Endostatins - metabolism | Cells, Cultured | Dental Pulp - cytology | Morpholines - pharmacology | Chemokine CCL2 - genetics | Reverse Transcriptase Polymerase Chain Reaction | Chick Embryo | Endostatins - genetics | Cell Movement - drug effects | Neovascularization, Physiologic - physiology | Animals | Endothelial Cells - cytology | Adolescent | Culture Media, Conditioned - metabolism | Endothelial Cells - drug effects | Viral antibodies | Tissue engineering | Analysis | Stem cells | Antibodies | Neovascularization | Vascular endothelial growth factor | Endothelium | Cell proliferation | Biomedical research | Heart attacks | Transplants & implants | Endostatin | AKT protein | mRNA | Kinases | Blood | Angiogenesis | Lysates | Engineering | Ischemia | Cryopreservation | Rodents | Bone marrow | Conditioning | Capillaries | Stroke | Nutrient transport | Polyamide-imides | Teeth | Wounds | Extracellular signal-regulated kinase | Blood vessels | Endothelial cells | 1-Phosphatidylinositol 3-kinase | Inhibitors | Chorioallantoic membrane | Plasminogen activator inhibitors | Spinal cord injuries | Microvasculature | Cell migration | Monocyte chemoattractant protein 1 | Dental pulp | Pulp
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12. Full Text Expression of angiogenesis-related factors in human corneas after cultivated oral mucosal epithelial transplantation
by Chen, Hung-Chi Jesse and Yeh, Lung-Kun and Tsai, Yueh-Ju and Lai, Chyong-Huey and Chen, Chi-Chun and Lai, Jui-Yang and Sun, Chi-Chin and Chang, Grace and Hwang, Tsann-Long and Chen, Jan-Kan and Ma, David Hui-Kang
Investigative Ophthalmology and Visual Science, ISSN 0146-0404, 08/2012, Volume 53, Issue 9, pp. 5615 - 5623
PURPOSE. We analyzed the expression of angiogenesis-related factors in corneal tissues that had undergone previously autologous cultivated oral mucosal... 
SHEETS IN-VITRO | FACTOR VEGF | STEM-CELL DEFICIENCY | MATRIX METALLOPROTEINASES | SURFACE RECONSTRUCTION | IMMUNOHISTOCHEMICAL LOCALIZATION | OPHTHALMOLOGY | HUMAN AMNIOTIC MEMBRANE | NEOVASCULARIZATION | ENDOTHELIAL GROWTH-FACTOR | CULTURED CORNEAL | Cornea - surgery | Postoperative Complications - etiology | Burns, Chemical - metabolism | Humans | Middle Aged | Postoperative Complications - metabolism | Male | Mouth Mucosa - transplantation | Transplantation, Autologous | Nerve Growth Factors - metabolism | Vascular Endothelial Growth Factor A - metabolism | Eye Burns - pathology | Cornea - blood supply | Interleukin 1 Receptor Antagonist Protein - metabolism | Fibroblast Growth Factor 2 - metabolism | Adult | Retrospective Studies | Corneal Neovascularization - etiology | Postoperative Complications - pathology | Serpins - metabolism | Endostatins - metabolism | Tissue Culture Techniques | Eye Burns - surgery | Treatment Outcome | Burns, Chemical - surgery | Epithelium - transplantation | Cornea - metabolism | Corneal Neovascularization - metabolism | Eye Proteins - metabolism | Adolescent | Burns, Chemical - pathology | Eye Burns - metabolism | Signal Transduction - physiology | Corneal Neovascularization - pathology | Clinical Trials, Phase I as Topic
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13. Full Text Mitochondrial division/mitophagy inhibitor (MDIVI) ameliorates pressure overload induced heart failure
by Givvimani, Srikanth and Munjal, Charu and Tyagi, Neetu and Sen, Utpal and Metreveli, Naira and Tyagi, Suresh C
PLoS ONE, ISSN 1932-6203, 03/2012, Volume 7, Issue 3, p. e32388
Background: We have previously reported the role of anti-angiogenic factors in inducing the transition from compensatory cardiac hypertrophy to heart failure... 
APOPTOSIS | OXIDATIVE STRESS | ISCHEMIC CARDIOMYOPATHY | ANGIOGENESIS | MATRIX-METALLOPROTEINASE | MULTIDISCIPLINARY SCIENCES | CARDIOVASCULAR-DISEASE | DEATH | AUTOPHAGY | FISSION | MAMMALIAN-CELLS | Immunohistochemistry | Mitochondria, Heart - metabolism | Gene Expression - drug effects | Apoptosis - drug effects | Vascular Endothelial Growth Factor A - metabolism | Mitochondria, Heart - drug effects | Vascular Endothelial Growth Factor A - genetics | Electron Transport Complex IV - metabolism | Heart Failure - prevention & control | Matrix Metalloproteinase 9 - metabolism | Matrix Metalloproteinase 9 - genetics | Myocardium - metabolism | Tissue Inhibitor of Metalloproteinase-3 - metabolism | Angios