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Cancer Cell, ISSN 1535-6108, 2011, Volume 19, Issue 1, pp. 17 - 30
and mutations occur frequently in gliomas and acute myeloid leukemia, leading to simultaneous loss and gain of activities in the production of α-ketoglutarate... 
BREAST-CANCER | TRANSCRIPTIONAL ACTIVITY | IDH2 MUTATIONS | ONCOLOGY | PROLYL HYDROXYLATION | INTEGRATED GENOMIC ANALYSIS | 2-OXOGLUTARATE OXYGENASES | ACUTE MYELOID-LEUKEMIA | HIF-ALPHA | HISTONE DEMETHYLATION | FAMILY | CELL BIOLOGY | Dioxygenases - metabolism | Histone Demethylases - antagonists & inhibitors | Gene Expression - genetics | Caenorhabditis elegans Proteins - chemistry | Humans | Ketoglutaric Acids - chemistry | Glioma - genetics | F-Box Proteins | Oxidoreductases, N-Demethylating - antagonists & inhibitors | Ketoglutaric Acids - pharmacology | Cytosine - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Glutarates - chemistry | Oxidoreductases, N-Demethylating - metabolism | DNA-Binding Proteins - antagonists & inhibitors | Glioma - enzymology | Endostatins - metabolism | Models, Molecular | Isocitrate Dehydrogenase - genetics | Histone Demethylases - metabolism | Dioxygenases - antagonists & inhibitors | Amino Acid Substitution - physiology | Procollagen-Proline Dioxygenase - genetics | Cell Line, Tumor | Isocitrate Dehydrogenase - metabolism | Glutarates - pharmacology | Histones - metabolism | Jumonji Domain-Containing Histone Demethylases - metabolism | Caenorhabditis elegans - enzymology | Cytosine - analogs & derivatives | Gene Expression - drug effects | Caenorhabditis elegans Proteins - metabolism | Isocitrate Dehydrogenase - antagonists & inhibitors | Glioma - metabolism | Procollagen-Proline Dioxygenase - metabolism | DNA-Binding Proteins - metabolism | Mixed Function Oxygenases | Biocatalysis - drug effects | Jumonji Domain-Containing Histone Demethylases - antagonists & inhibitors | Jumonji Domain-Containing Histone Demethylases - chemistry | Procollagen-Proline Dioxygenase - antagonists & inhibitors | Ketoglutaric Acids - metabolism | Oxalates - pharmacology | Binding, Competitive | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Catalytic Domain | Proto-Oncogene Proteins - genetics | Hypoxia-Inducible Factor-Proline Dioxygenases | DNA-Binding Proteins - genetics | Homeodomain Proteins - genetics | Animals | 5-Methylcytosine - metabolism | Caenorhabditis elegans Proteins - antagonists & inhibitors | Glutarates - metabolism
Journal Article
Frontiers in Bioscience, ISSN 1093-9946, 01/2011, Volume 16, Issue 2, pp. 674 - 697
Journal Article
Journal of Immunology, ISSN 0022-1767, 2014, Volume 192, Issue 2, pp. 589 - 602
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 03/2006, Volume 281, Issue 9, pp. 6020 - 6029
Journal Article
Liver International, ISSN 1478-3223, 02/2011, Volume 31, Issue 2, pp. 146 - 162
Nowadays, liver cancer, cirrhosis and other liver‐related diseases are the fifth most common cause of mortality in the UK. Furthermore, chronic liver diseases... 
chronic liver diseases | non‐alcoholic steatohepatitis | portal hypertension | angiogenesis | hepatocellular carcinoma | Angiogenesis | Chronic liver diseases | Hepatocellular carcinoma | Non-alcoholic steatohepatitis | Portal hypertension | ADVANCED HEPATOCELLULAR-CARCINOMA | HYPOXIA-INDUCIBLE FACTOR | PORTAL HYPERTENSIVE-RATS | ENDOTHELIAL-GROWTH-FACTOR | NONALCOHOLIC STEATOHEPATITIS | FACTOR RECEPTOR | PHASE-II TRIAL | HEPATIC STELLATE CELLS | TYROSINE KINASE INHIBITOR | HYPERDYNAMIC SPLANCHNIC CIRCULATION | GASTROENTEROLOGY & HEPATOLOGY | non-alcoholic steatohepatitis | Microvessels - physiopathology | Hypertension, Portal - physiopathology | Cadherins - metabolism | Hypertension, Portal - complications | Humans | Fatty Liver - complications | Liver Neoplasms - complications | Vascular Endothelial Growth Factor A - metabolism | Hypoxia-Inducible Factor 1 - metabolism | Integrins - metabolism | Neovascularization, Pathologic - etiology | Fibroblast Growth Factors - metabolism | Carcinoma, Hepatocellular - drug therapy | Receptors, Vascular Endothelial Growth Factor - metabolism | Carcinoma, Hepatocellular - complications | Liver Neoplasms - physiopathology | Non-alcoholic Fatty Liver Disease | Angiogenesis Inhibitors - therapeutic use | Neovascularization, Pathologic - physiopathology | Angiostatins - metabolism | Angiopoietins - metabolism | Carcinoma, Hepatocellular - physiopathology | Endostatins - metabolism | Liver Cirrhosis - complications | Fatty Liver - physiopathology | Liver Neoplasms - drug therapy | Disease Progression | Platelet-Derived Growth Factor - metabolism | Models, Biological | Signal Transduction - physiology | Liver Cirrhosis - physiopathology | Chronic Disease | Thrombospondins - metabolism | Hypertension | Complications and side effects | Liver cancer | Development and progression | Mortality
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 07/2012, Volume 303, Issue 1, pp. 41 - 51
Hydrogen sulfide (H2S) has recently been identified as a regulator of various physiological events, including vasodilation, angiogenesis, antiapoptotic, and... 
Hydrogen sulfide | Cystathionine γ-lyase | Cystathionine β-synthase | 3-mercaptopyruvate sulfurtransferase | Mitophagy | homocysteine | cystathionine gamma-lyase | OXIDATIVE STRESS | PHYSIOLOGY | ENDOTHELIAL DYSFUNCTION | DIABETIC-NEPHROPATHY | ENDOPLASMIC-RETICULUM STRESS | SMOOTH-MUSCLE-CELL | HYDROGEN-SULFIDE | CELL BIOLOGY | MATRIX METALLOPROTEINASES | mitophagy | hydrogen sulfide | EXTRACELLULAR-MATRIX | cystathionine beta-synthase | TIMP-1 EXPRESSION | Hydrogen Sulfide - metabolism | Genetic Therapy | Vascular Endothelial Growth Factor A - biosynthesis | Reactive Oxygen Species - metabolism | Hyperhomocysteinemia - therapy | Hyperhomocysteinemia - metabolism | Cystathionine gamma-Lyase - metabolism | Homocysteine - metabolism | Hypertension, Renovascular - genetics | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Forkhead Transcription Factors - metabolism | Renal Artery - metabolism | Cystathionine gamma-Lyase - genetics | Cystathionine beta-Synthase - genetics | Proto-Oncogene Proteins c-akt - metabolism | Organ Culture Techniques | Endostatins - biosynthesis | Mice, Inbred C57BL | Cells, Cultured | Sulfurtransferases - genetics | Hypertension, Renovascular - therapy | Cystathionine beta-Synthase - metabolism | Matrix Metalloproteinase 13 - metabolism | Animals | Hyperhomocysteinemia - genetics | Mice | Forkhead Box Protein O3 | Platelet Endothelial Cell Adhesion Molecule-1 - biosynthesis | Vascular System Injuries | Sulfurtransferases - metabolism | cystathionine γ-lyase | cystathionine β-synthase
Journal Article
FEBS Letters, ISSN 0014-5793, 2000, Volume 486, Issue 3, pp. 247 - 251
Journal Article
Science, ISSN 0036-8075, 1/2002, Volume 295, Issue 5552, pp. 140 - 143
Tumstatin is a 28-kilodalton fragment of type IV collagen that displays both anti-angiogenic and proapoptotic activity. Here we show that tumstatin functions... 
Phosphorylation | Messenger RNA | Protein synthesis | Long term potentiation | Reports | Angiogenesis inhibitors | Endothelial cells | Medical schools | Integrins | Apoptosis | Tumors | RAPAMYCIN | TARGET | ANGIOGENESIS | CAP-DEPENDENT INITIATION | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | KINASE | MICE | DISTINCT ANTITUMOR PROPERTIES | TRANSLATION | ENDOSTATIN | Endothelium, Vascular - cytology | Protein Kinases - metabolism | Autoantigens - metabolism | Protein-Tyrosine Kinases - metabolism | Humans | Protein-Serine-Threonine Kinases | Molecular Sequence Data | Endothelium, Vascular - drug effects | Peptide Fragments - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Phosphoproteins - metabolism | RNA Caps - metabolism | RNA, Messenger - metabolism | Eukaryotic Initiation Factor-4E | Focal Adhesion Kinase 1 | Collagen Type IV - chemistry | Collagen Type IV - pharmacology | Cattle | Protein Synthesis Inhibitors - pharmacology | Receptors, Vitronectin - metabolism | Tumor Cells, Cultured | Autoantigens - pharmacology | Collagen Type IV - metabolism | Proto-Oncogene Proteins - metabolism | Amino Acid Sequence | Signal Transduction | RNA, Messenger - genetics | Cells, Cultured | Focal Adhesion Protein-Tyrosine Kinases | Autoantigens - chemistry | Enzyme Activation - drug effects | Peptide Initiation Factors - metabolism | Proto-Oncogene Proteins c-akt | Adaptor Proteins, Signal Transducing | Protein Kinase Inhibitors | Animals | Carrier Proteins - metabolism | Endothelium, Vascular - metabolism | Protein Biosynthesis - drug effects | Mice | TOR Serine-Threonine Kinases | Proteins | Synthesis | Organic compounds | Cellular biology | Cells
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e71104
Angiogenesis, the formation of capillaries from pre-existing blood vessels, is a key process in tissue engineering. If blood supply cannot be established... 
FIBROBLAST GROWTH FACTOR-2 | ACTIVATION | REGENERATION | MULTIDISCIPLINARY SCIENCES | OSTEOBLASTS | PHENOTYPE | PROLIFERATION | MECHANISMS | DIFFERENTIATION | STROMAL CELLS | EXPRESSION | Immunohistochemistry | Neovascularization, Physiologic - drug effects | Nitriles - pharmacology | Humans | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Culture Media, Conditioned - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor A - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Stem Cells - metabolism | Vascular Endothelial Growth Factor A - genetics | Young Adult | Plasminogen Activator Inhibitor 1 - metabolism | Angiogenesis Inducing Agents - metabolism | Chorioallantoic Membrane - drug effects | Chemokine CCL2 - metabolism | Angiogenesis Inducing Agents - pharmacology | Plasminogen Activator Inhibitor 1 - genetics | Chromones - pharmacology | Chorioallantoic Membrane - blood supply | Cell Line | Butadienes - pharmacology | Endostatins - metabolism | Cells, Cultured | Dental Pulp - cytology | Morpholines - pharmacology | Chemokine CCL2 - genetics | Reverse Transcriptase Polymerase Chain Reaction | Chick Embryo | Endostatins - genetics | Cell Movement - drug effects | Neovascularization, Physiologic - physiology | Animals | Endothelial Cells - cytology | Adolescent | Culture Media, Conditioned - metabolism | Endothelial Cells - drug effects | Viral antibodies | Tissue engineering | Analysis | Stem cells | Antibodies | Neovascularization | Vascular endothelial growth factor | Endothelium | Cell proliferation | Biomedical research | Heart attacks | Transplants & implants | Endostatin | AKT protein | mRNA | Kinases | Blood | Angiogenesis | Lysates | Engineering | Ischemia | Cryopreservation | Rodents | Bone marrow | Conditioning | Capillaries | Stroke | Nutrient transport | Polyamide-imides | Teeth | Wounds | Extracellular signal-regulated kinase | Blood vessels | Endothelial cells | 1-Phosphatidylinositol 3-kinase | Inhibitors | Chorioallantoic membrane | Plasminogen activator inhibitors | Spinal cord injuries | Microvasculature | Cell migration | Monocyte chemoattractant protein 1 | Dental pulp | Pulp
Journal Article
Investigative Ophthalmology and Visual Science, ISSN 0146-0404, 08/2012, Volume 53, Issue 9, pp. 5615 - 5623
Journal Article