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Nature Cell Biology, ISSN 1465-7392, 10/2011, Volume 13, Issue 10, pp. 1202 - 1213
Angiogenesis, the growth of new blood vessels, involves specification of endothelial cells to tip cells and stalk cells, which is controlled by Notch... 
LYMPHATIC ENDOTHELIUM | KINASE-ACTIVITY | VASCULAR NETWORK FORMATION | ENDOTHELIAL-GROWTH-FACTOR | FACTOR-C | MOUSE EMBRYOS | EXTRACELLULAR-MATRIX | MONOCLONAL-ANTIBODY | TUMOR ANGIOGENESIS | FACTOR RECEPTOR-3 | CELL BIOLOGY | Neovascularization, Physiologic - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Receptors, Notch - metabolism | Humans | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Receptors, Notch - antagonists & inhibitors | Transfection | RNA Interference | Time Factors | Forkhead Transcription Factors - metabolism | Vascular Endothelial Growth Factor D - genetics | Phosphatidylinositol 3-Kinase - metabolism | Vascular Endothelial Growth Factor Receptor-3 - deficiency | Transduction, Genetic | Retinal Vessels - metabolism | Endothelial Cells - metabolism | Rhombencephalon - blood supply | Gene Expression Regulation | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Forkhead Transcription Factors - genetics | Vascular Endothelial Growth Factor C - genetics | Mice, Knockout | Antibodies - pharmacology | Macrophages - metabolism | Vascular Endothelial Growth Factor D - metabolism | Animals | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Vascular Endothelial Growth Factor C - metabolism | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Vascular Endothelial Growth Factor B - genetics | Mice | Neovascularization, Pathologic - metabolism | Protein Kinase Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-3 - genetics | Endothelial Cells - drug effects | Physiological aspects | Blood vessels | Cellular signal transduction | Growth factor receptors | Research | Vascular endothelial growth factor | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2010, Volume 107, Issue 6, pp. 2425 - 2430
Vascular endothelial growth factors (VEGFs) regulate blood and lymph vessel formation through activation of three receptor tyrosine kinases, VEGFR-1, -2, and... 
Proteins | Angiogenesis | Receptors | Vascular endothelial growth factors | Endothelial growth factors | Ligands | Dimers | Chimeras | Monomers | Crystal structure | Vascular endothelial growth factor receptor-2 | Lymphangiogenesis | Vascular endothelial growth factor C | ACTIVATION | COMPLEX | FACTOR-C | angiogenesis | CRYSTAL-STRUCTURE | MULTIDISCIPLINARY SCIENCES | ORF-VIRUS | TYROSINE KINASES | vascular endothelial growth factor receptor-2 | VASCULAR-PERMEABILITY | FAMILY-MEMBER | lymphangiogenesis | KINASE DOMAIN RECEPTOR | vascular endothelial growth factor C | Humans | Crystallography, X-Ray | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Recombinant Fusion Proteins - metabolism | Spodoptera | Vascular Endothelial Growth Factor Receptor-2 - genetics | Vascular Endothelial Growth Factor A - chemistry | Vascular Endothelial Growth Factor C - chemistry | Vascular Endothelial Growth Factor Receptor-1 - genetics | Protein Structure, Tertiary | Cell Line | Cell Survival | Models, Molecular | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Binding Sites - genetics | Recombinant Fusion Proteins - chemistry | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Vascular Endothelial Growth Factor Receptor-1 - chemistry | Vascular Endothelial Growth Factor C - genetics | Vascular Endothelial Growth Factor Receptor-3 - chemistry | Vascular Endothelial Growth Factor Receptor-2 - chemistry | Animals | Hydrogen Bonding | Vascular Endothelial Growth Factor C - metabolism | Protein Binding | Recombinant Fusion Proteins - genetics | Protein Conformation | Kinetics | Mutation | Vascular Endothelial Growth Factor Receptor-3 - genetics | Development and progression | Research | Growth factor receptors | Neovascularization | Properties | Vascular endothelial growth factor | Protein binding | Lymphangioma | Biological Sciences
Journal Article
Angiogenesis, ISSN 0969-6970, 6/2012, Volume 15, Issue 2, pp. 171 - 185
Pharmacological inhibition of VEGF-A has proven to be effective in inhibiting angiogenesis and vascular leak associated with cancers and various eye diseases.... 
Biomedicine | Age-related macular degeneration | Cancer Research | Oncology | Affinity | Ophthalmology | VEGF receptor | Cardiology | Aflibercept | Placental growth factor | Biomedicine general | Cell Biology | ANGIOGENESIS | EFFICACY | MACULAR DEGENERATION | PHASE-II | ANTIBODY | FACTOR THERAPY | EYE | IN-VITRO | PEGAPTANIB | RETINAL DISEASES | PERIPHERAL VASCULAR DISEASE | Angiogenesis Inhibitors - immunology | Receptors, Vascular Endothelial Growth Factor | Vascular Endothelial Growth Factor B - antagonists & inhibitors | Recombinant Fusion Proteins - pharmacology | Endothelial Cells - metabolism | Humans | Angiogenesis Inhibitors - pharmacology | Vascular Endothelial Growth Factor B - immunology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Vascular Endothelial Growth Factor A - immunology | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor Receptor-2 - immunology | Enzyme Activation - drug effects | Bevacizumab | Endothelial Cells - immunology | Vascular Endothelial Growth Factor B - metabolism | Antibodies, Monoclonal, Humanized - pharmacology | HEK293 Cells | Ranibizumab | Vascular Endothelial Growth Factor Receptor-1 - immunology | Antibodies, Monoclonal, Humanized - immunology | Original Paper
Journal Article
Circulation Research, ISSN 0009-7330, 04/2017, Volume 120, Issue 9, pp. 1414 - 1425
RATIONALE:Vascular endothelial growth factor (VEGF) is the main driver of angiogenesis and vascular permeability via VEGF receptor 2 (VEGFR2), whereas... 
Blood vessels | vascular leakage | VE-Cadherin | vascular biology | VEGF receptor regulation | REGRESSION | LYMPHATIC ENDOTHELIUM | CARDIAC & CARDIOVASCULAR SYSTEMS | RECEPTOR VEGFR-3 | ANGIOGENESIS | ENDOTHELIAL-GROWTH-FACTOR | FACTOR-C | MOUSE EMBRYOS | blood vessels | IN-VIVO | PERIPHERAL VASCULAR DISEASE | ADHERENS JUNCTIONS | HEMATOLOGY | Cadherins - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Neovascularization, Pathologic | Capillary Permeability - drug effects | Male | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Carcinoma, Lewis Lung - blood supply | Adherens Junctions - metabolism | Antigens, CD - metabolism | Vascular Endothelial Growth Factor Receptor-2 - genetics | Transfection | Female | Vascular Endothelial Growth Factor A - pharmacology | Tight Junctions - metabolism | Vascular Endothelial Growth Factor Receptor-3 - deficiency | Retinal Vessels - metabolism | Endothelial Cells - metabolism | Mice, Inbred C57BL | Vascular Endothelial Growth Factor Receptor-2 - deficiency | Cells, Cultured | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Genotype | Retinal Vessels - drug effects | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Carcinoma, Lewis Lung - metabolism | Mice, Knockout | Phenotype | Animals | Signal Transduction - drug effects | Lung - blood supply | Neovascularization, Physiologic | Vascular Endothelial Growth Factor Receptor-3 - genetics | Endothelial Cells - drug effects | Leakage | Phosphorylation | Kidneys | Cell junctions | Permeability | Gene deletion | Cadherin | Endothelial cells | Vascular endothelial growth factor receptors | Gene silencing | Angiogenesis | Clonal deletion | Rodents | Fibrinogen | Localization | Vascular endothelial growth factor | Tumors
Journal Article
Nature, ISSN 0028-0836, 04/2012, Volume 484, Issue 7392, pp. 110 - 114
Developing tissues and growing tumours produce vascular endothelial growth factors (VEGFs), leading to the activation of the corresponding receptors in... 
TIP CELL-FORMATION | DLL4 | PATHWAYS | ACTIVATION | BEVACIZUMAB | MULTIDISCIPLINARY SCIENCES | GROWTH | MOUSE | LIGAND | INHIBIT | D114 | Human Umbilical Vein Endothelial Cells | Neovascularization, Physiologic - drug effects | Up-Regulation | Vascular Endothelial Growth Factor Receptor-3 - biosynthesis | Receptors, Notch - metabolism | Humans | Male | Intracellular Signaling Peptides and Proteins - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor Receptor-2 - genetics | Intracellular Signaling Peptides and Proteins - deficiency | Membrane Proteins - deficiency | Receptors, Notch - antagonists & inhibitors | HEK293 Cells | Female | Transcription, Genetic | Membrane Proteins - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Endothelial Cells - metabolism | Membrane Proteins - genetics | Vascular Endothelial Growth Factor Receptor-2 - deficiency | Cells, Cultured | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Neovascularization, Physiologic - physiology | Animals | Signal Transduction - drug effects | Endothelial Cells - cytology | Models, Biological | Mice | Protein Kinase Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-3 - genetics | Endothelial Cells - drug effects | Cellular control mechanisms | Physiological aspects | Growth factor receptors | Neovascularization | Research | Vascular endothelial growth factor | Endothelium | Angiogenesis | Ligands | Kinases | Rodents
Journal Article
Annals of the New York Academy of Sciences, ISSN 0077-8923, 05/2008, Volume 1131, Issue 1, pp. 225 - 234
The metastatic spread of tumor cells is the most lethal aspect of cancer and often occurs via the lymphatic vasculature. Both experimental tumor models and... 
lymphangiogenesis | lymphatic vessel | cancer | angiogenesis | lymph node | Angiogenesis | Lymph node | Lymphangiogenesis | Lymphatic vessel | Cancer | ENDOTHELIAL-GROWTH-FACTOR | FACTOR-C EXPRESSION | MULTIDISCIPLINARY SCIENCES | VEGF-C | MELANOMA METASTASIS | CELL LUNG-CANCER | BREAST-CANCER | NODE METASTASIS | RECEPTOR TYROSINE KINASES | PROMOTES TUMOR-METASTASIS | IN-VIVO | Lymph Nodes - pathology | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - pathology | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor Receptor-2 - immunology | Vascular Endothelial Growth Factor A - genetics | Neovascularization, Pathologic - pathology | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Lymph Nodes - blood supply | Lymphatic Metastasis - pathology | Neovascularization, Pathologic - prevention & control | Vascular Endothelial Growth Factor D - genetics | Signal Transduction | Lymphangiogenesis - physiology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Vascular Endothelial Growth Factor C - genetics | Vascular Endothelial Growth Factor D - metabolism | Animals | Lymphatic Metastasis - prevention & control | Models, Biological | Vascular Endothelial Growth Factor C - metabolism | Lymphatic Vessels - pathology | Vascular Endothelial Growth Factor Receptor-3 - genetics | Endothelial growth factors | Metastasis
Journal Article
1999, Current topics in microbiology and immunology, ISBN 3540647317, Volume 237, 189
Book
Journal of Biological Chemistry, ISSN 0021-9258, 2017, Volume 292, Issue 31, pp. 12787 - 12800
Stress-induced p38 mitogen-activated protein kinase (MAPK) activity is implicated in pathological remodeling in the heart. For example, constitutive p38 MAPK... 
Endothelium, Vascular - cytology | Myocardial Ischemia - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Myocytes, Cardiac - secretion | Male | Sp1 Transcription Factor - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Vascular Endothelial Growth Factor A - secretion | Vascular Endothelial Growth Factor Receptor-2 - genetics | Cell Hypoxia | Sp1 Transcription Factor - antagonists & inhibitors | Mitogen-Activated Protein Kinase 14 - metabolism | RNA Interference | Vascular Endothelial Growth Factor A - agonists | Human Umbilical Vein Endothelial Cells - cytology | Vascular Endothelial Growth Factor Receptor-2 - agonists | Mitogen-Activated Protein Kinase 14 - chemistry | Animals, Newborn | Recombinant Proteins - metabolism | Myocytes, Cardiac - cytology | Cells, Cultured | Gene Expression Regulation | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Mice, Transgenic | Mitogen-Activated Protein Kinase 14 - genetics | Rats, Sprague-Dawley | Mice, Knockout | Myocardial Ischemia - pathology | Myocardial Revascularization | Myocytes, Cardiac - pathology | Animals | Sp1 Transcription Factor - genetics | Endothelium, Vascular - metabolism | Myocytes, Cardiac - metabolism | Endothelium, Vascular - pathology | Enzyme Activation | Sus scrofa | Crosses, Genetic | cross-talk | angiogenesis | cardiomyocyte | vascular endothelial growth factor (VEGF) | cardiac hypertrophy | p38 MAPK | Cell Biology
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 5178 - 16
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2015, Volume 290, Issue 26, pp. 16451 - 16462
Background: Vascular endothelial growth factor (VEGF) requires heparin-like molecules for full activity. Results: VEGF, VEGF receptor-2, and neuropilin-1... 
ISOFORMS | TYROSINE KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | heparin-binding protein | neuropilin-1 | vascular endothelial growth factor (VEGF) | EXTRACELLULAR DOMAIN | surface plasmon resonance (SPR) | VEGF receptor | PATHOLOGICAL ANGIOGENESIS | TUMOR ANGIOGENESIS | MICE LACKING |