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2006, ISBN 9780470012949, xvii, 350 p., [8] p. of plates
Book
Cell stem cell, ISSN 1934-5909, 2015, Volume 16, Issue 1, pp. 51 - 66
Mesenchymal stem cells (MSCs) reside in the perivascular niche of many organs, including kidney, lung, liver, and heart, although their roles in these tissues are poorly understood... 
REGULATOR | ORIGIN | SONIC HEDGEHOG | PATHWAY | BONE-MARROW NICHE | MYOFIBROBLASTS | NG2 PROTEOGLYCAN | EXPRESSION | MESENCHYMAL STEM-CELLS | GROWTH FACTOR-AA | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Organ Specificity - drug effects | Diphtheria Toxin - pharmacology | Neovascularization, Physiologic - drug effects | Pericytes - drug effects | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Fibrosis - metabolism | Cell Lineage - drug effects | Myofibroblasts - metabolism | Mesenchymal Stromal Cells - cytology | Mesenchymal Stromal Cells - ultrastructure | Kruppel-Like Transcription Factors - metabolism | Pericytes - pathology | Bone Marrow Cells - drug effects | Colony-Forming Units Assay | Aorta - physiopathology | Homeostasis - drug effects | Heart Ventricles - pathology | Receptor, Platelet-Derived Growth Factor beta - metabolism | Mesenchymal Stromal Cells - drug effects | Endothelial Cells - metabolism | Aorta - drug effects | Pericytes - metabolism | Cells, Cultured | Proteoglycans - metabolism | Antigens - metabolism | Aorta - pathology | Myofibroblasts - cytology | Animals | Heart Ventricles - physiopathology | Cell Differentiation - drug effects | Endothelial Cells - cytology | Blood Vessels - drug effects | Mice | Stem Cell Niche - drug effects | Zinc Finger Protein GLI1 | Fibrosis - pathology | Bone Marrow Cells - metabolism | Endothelial Cells - drug effects | Heart Ventricles - drug effects
Journal Article
Bioimpacts, ISSN 2228-5660, 12/2018, Volume 8, Issue Suppl 1, pp. S1 - S129
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2015, Volume 112, Issue 40, pp. 12516 - 12521
Human pluripotent stem cell-based in vitro models that reflect human physiology have the potential to reduce the number of drug failures in clinical trials and offer a cost-effective approach... 
Organoid | Toxicology | Differentiation | Tissue engineering | Machine learning | toxicology | HUMAN NEOCORTEX | tissue engineering | DEVELOPMENTAL NEUROTOXICITY | differentiation | HUMAN BRAIN | MULTIDISCIPLINARY SCIENCES | CLASSIFICATION | FATTY-ACIDS | machine learning | CANCER | organoid | IN-VITRO | HUMAN CEREBRAL-CORTEX | MICROGLIA | GENE-EXPRESSION | Embryonic Stem Cells - metabolism | Microglia - metabolism | Embryonic Stem Cells - cytology | Humans | Brain - growth & development | Support Vector Machine | Neural Stem Cells - cytology | Xenobiotics - pharmacology | Brain - metabolism | Neurogenesis - genetics | Mesenchymal Stromal Cells - cytology | Xenobiotics - classification | Gene Expression Regulation, Developmental | Cell Differentiation | Neurogenesis - drug effects | Culture Media, Serum-Free - pharmacology | Gene Ontology | Polyethylene Glycols - pharmacology | Microglia - cytology | Mesenchymal Stromal Cells - drug effects | Brain - cytology | Pluripotent Stem Cells - cytology | Tissue Engineering - methods | Microglia - drug effects | Endothelial Cells - metabolism | Cells, Cultured | Neural Stem Cells - drug effects | Mesenchymal Stromal Cells - metabolism | Cell Communication - genetics | Macrophages - cytology | Pluripotent Stem Cells - metabolism | Macrophages - metabolism | Embryonic Stem Cells - drug effects | Endothelial Cells - cytology | Models, Biological | Pluripotent Stem Cells - drug effects | Cell Communication - drug effects | Macrophages - drug effects | Hydrogels - pharmacology | Neural Stem Cells - metabolism | Endothelial Cells - drug effects | Biological Sciences
Journal Article
Cancer Chemotherapy and Pharmacology, ISSN 0344-5704, 8/2011, Volume 68, Issue 2, pp. 445 - 455
The natural flavonoid fisetin was recently identified as a lead compound that stabilizes endothelial cell microtubules... 
Fisetin | Lewis lung carcinoma | Angiogenesis | Antitumour activity | Cyclophosphamide | Medicine & Public Health | Cancer Research | Oncology | Cytotoxicity | EA·hy 926 endothelial cells | Pharmacology/Toxicology | Flavonoid | EA•hy 926 endothelial cells | APOPTOSIS | ANTIINFLAMMATORY ACTIVITY | CELL-CYCLE ARREST | PROLIFERATION | FLUOROURACIL | CANCER | IN-VITRO | ONCOLOGY | ENDOTHELIAL-CELLS | PHARMACOLOGY & PHARMACY | EA.hy 926 endothelial cells | INHIBITORS | NIH 3T3 Cells | Cyclophosphamide - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - administration & dosage | Tubulin Modulators - pharmacology | Humans | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Antineoplastic Agents, Alkylating - pharmacology | Flavonoids - adverse effects | Antineoplastic Agents, Alkylating - administration & dosage | Cyclophosphamide - adverse effects | Cyclophosphamide - therapeutic use | Flavonoids - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Angiogenesis Inhibitors - administration & dosage | Antineoplastic Agents, Phytogenic - administration & dosage | Angiogenesis Inhibitors - therapeutic use | Flavonoids - administration & dosage | Tubulin Modulators - administration & dosage | Female | Flavonoids - pharmacology | Antineoplastic Agents, Phytogenic - therapeutic use | Angiogenesis Inhibitors - adverse effects | Antineoplastic Agents, Phytogenic - adverse effects | Cell Line | Cell Survival - drug effects | Tubulin Modulators - adverse effects | Mice, Inbred C57BL | Angiogenesis Inhibitors - pharmacology | Tubulin Modulators - therapeutic use | Carcinoma, Lewis Lung - drug therapy | Antineoplastic Agents, Alkylating - therapeutic use | Cell Movement - drug effects | Animals | Tumor Burden - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Endothelial Cells - cytology | Neovascularization, Pathologic - drug therapy | Cyclophosphamide - pharmacology | Carcinoma, Lewis Lung - pathology | Cell Proliferation - drug effects | Mice | Antineoplastic Agents, Alkylating - adverse effects | Antineoplastic Agents, Phytogenic - pharmacology | Cell Cycle - drug effects | Endothelial Cells - drug effects | Antimitotic agents | Flavonoids | Flavones | Lung cancer | Bioflavonoids | Accountants | Drug therapy, Combination | Universities and colleges | Antineoplastic agents | Endothelium | Tumors | Index Medicus | cytology | Antineoplastic Agents, Phytogenic | pathology | Cell Proliferation | Endothelial Cells | Tubulin Modulators | Neovascularization, Pathologic | fisetin | administration & dosage | pharmacology | flavonoid | Carcinoma, Lewis Lung | Antineoplastic Agents, Alkylating | cytotoxicity | drug therapy | Cell Survival | angiogenesis | Antineoplastic Combined Chemotherapy Protocols | drug effects | Tumor Burden | Angiogenesis Inhibitors | EA.hy 926 | Cell Cycle | antitumour activity | adverse effects | therapeutic use | Cell Movement
Journal Article
Cell stem cell, ISSN 1934-5909, 2017, Volume 20, Issue 4, pp. 490 - 504.e5
.... Here, we used comparison of induced pluripotent stem cell-derived endothelial cells (iPSC-ECs) from three families with unaffected mutation carriers... 
cell survival | bone morphogenetic protein receptor 2 | unaffected mutation carrier | cell adhesion | penetrance | cell signaling | transcriptomic analysis | pulmonary arterial hypertension | endothelial dysfunction | induced pluripotent stem cell-derived endothelial cell | MIGRATION | PATHOGENESIS | ANGIOGENESIS | GENE | INTEGRIN | ARTERIAL-HYPERTENSION | MOUSE | RECEPTOR | DYSFUNCTION | EXPRESSION | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Neovascularization, Physiologic - drug effects | Humans | Hypertension, Pulmonary - prevention & control | Base Sequence | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Induced Pluripotent Stem Cells - cytology | Induced Pluripotent Stem Cells - metabolism | Cell Survival - drug effects | Bone Morphogenetic Protein Receptors, Type II - genetics | Induced Pluripotent Stem Cells - drug effects | Endothelial Cells - metabolism | Neovascularization, Physiologic - genetics | Hypertension, Pulmonary - genetics | Cell Adhesion - drug effects | Mutation - genetics | Bone Morphogenetic Protein 4 - pharmacology | Gene Editing | Cell Shape - drug effects | Gene Expression Regulation - drug effects | Cell Movement - drug effects | Sequence Analysis, RNA | Signal Transduction - drug effects | Endothelial Cells - cytology | Heterozygote | Hypertension, Pulmonary - pathology | Smad Proteins - metabolism | Endothelial Cells - drug effects | Pulmonary arterial hypertension | induced pluripotent stem cell derived endothelial cell
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2015, Volume 112, Issue 50, pp. 15408 - 15413
Journal Article
PLoS ONE, ISSN 1932-6203, 2009, Volume 4, Issue 12, p. e8443
.... Moreover, to fully understand the beneficial effects of stem cell therapy, investigators must be able to track the functional biology and physiology of transplanted cells in living subjects over time. Methodology... 
PROGENITOR CELLS | SURVIVAL | MORPHOGENESIS | MULTIDISCIPLINARY SCIENCES | CORD BLOOD | ISCHEMIA | EXTRACELLULAR-MATRIX | PERIPHERAL-BLOOD | CARDIOMYOCYTES | NEOVASCULARIZATION | TRANSPLANTATION | Embryo, Mammalian - drug effects | Embryonic Stem Cells - metabolism | Neovascularization, Physiologic - drug effects | Transcription, Genetic - drug effects | Embryonic Stem Cells - cytology | Oligonucleotide Array Sequence Analysis | Recovery of Function - drug effects | Humans | Endothelial Cells - transplantation | Myocardial Contraction - drug effects | Gene Expression Profiling | Tissue Survival - drug effects | Embryo, Mammalian - metabolism | Stem Cell Transplantation | Cell Differentiation - genetics | Myocardial Infarction - therapy | Myocardial Infarction - pathology | Polymerase Chain Reaction | Female | Myocardial Infarction - physiopathology | Collagen - pharmacology | Cell Line | Cell Survival - drug effects | Reproducibility of Results | Endothelial Cells - metabolism | Embryo, Mammalian - blood supply | Ventricular Function, Left - drug effects | Mice, SCID | Gene Expression Regulation - drug effects | Animals | Embryonic Stem Cells - drug effects | Cell Differentiation - drug effects | Endothelial Cells - cytology | Mice | Endothelial Cells - drug effects | Stem cells | Genetic aspects | Transplantation | Genetic transcription | Embryonic stem cells | Heart attack | Endothelium | Myocardial infarction | Heart | Cell culture | Therapy | Transcription | Embryo cells | Stem cell transplantation | Cardiovascular disease | Genomes | Angiogenesis | Allografts | Ischemia | Cell fate | Fibroblasts | Bone marrow | Extracellular matrix | Cardiology | Heart diseases | Cytokines | Developmental biology | Blood vessels | Cardiomyocytes | Embryo fibroblasts | Embryos | Coronary artery disease | Endothelial cells | Medicine | DNA microarrays | Collagen | Infarction | In vivo methods and tests | Umbilical cord | Differentiation | Umbilical vein | Apoptosis
Journal Article
Acta Biomaterialia, ISSN 1742-7061, 04/2012, Volume 8, Issue 4, pp. 1440 - 1449
.... However, few in vitro studies have been performed to identify the effects of environmental elasticity on the differentiation of MSC into vascular cell types... 
Mesenchymal stem cell | Elasticity | Vascular differentiation | Nanofiber | 3-D matrix | MATERIALS SCIENCE, BIOMATERIALS | STIFFNESS | ENGINEERING, BIOMEDICAL | EXTRACELLULAR-MATRIX | SCAFFOLD | Cross-Linking Reagents - pharmacology | Up-Regulation - radiation effects | Tensile Strength - drug effects | Elastic Modulus - drug effects | Polyethylene Glycols - chemistry | Methacrylates - chemistry | Spectroscopy, Fourier Transform Infrared | Tissue Scaffolds - chemistry | Polymerization - drug effects | Mesenchymal Stromal Cells - cytology | Ultraviolet Rays | Time Factors | Polymerase Chain Reaction | Compressive Strength - drug effects | Compressive Strength - radiation effects | Hydrogel, Polyethylene Glycol Dimethacrylate - pharmacology | Porosity - drug effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Biomarkers - metabolism | Cell Differentiation - radiation effects | Nanofibers - chemistry | Mesenchymal Stromal Cells - drug effects | Nanofibers - ultrastructure | Polymerization - radiation effects | Endothelial Cells - metabolism | Mesenchymal Stromal Cells - metabolism | Rats | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Elasticity - drug effects | Endothelial Cells - radiation effects | Materials Testing | Elasticity - radiation effects | Muscle, Smooth, Vascular - cytology | Tensile Strength - radiation effects | Porosity - radiation effects | Elastic Modulus - radiation effects | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Animals | Cell Differentiation - drug effects | Endothelial Cells - cytology | Gene Expression Regulation - radiation effects | Endothelial Cells - drug effects | Actin | Polyethylene glycol | Stem cells | Smooth muscle | Muscle proteins | Polyols | Nanotechnology | Endothelium | mesenchymal stem cell | vascular differentiation | 3D matrix | nanofiber
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 4, p. e35685
We have previously shown that mesenchymal stem cells (MSC) improve function upon integration in ischemic myocardium... 
MIGRATION | SURVIVAL | BAD | PHOSPHORYLATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | H9C2 CELLS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INHIBIT APOPTOSIS | CHEMOKINE RECEPTORS | STROMAL CELLS | Neovascularization, Physiologic - drug effects | Gene Expression - drug effects | Apoptosis - drug effects | Caspase 3 - metabolism | Culture Media, Conditioned - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Proto-Oncogene Proteins c-akt - genetics | Mesenchymal Stromal Cells - cytology | Caspase 3 - genetics | Proto-Oncogene Proteins c-akt - metabolism | Cell Survival - drug effects | Endothelial Cells - metabolism | Cytokines - secretion | Mesenchymal Stromal Cells - metabolism | Chemotaxis - drug effects | Mesenchymal Stromal Cells - secretion | Phosphatidylinositol 3-Kinases - genetics | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Endothelial Cells - cytology | Dogs | Mice | Primary Cell Culture | Cytokines - biosynthesis | Cytokines - pharmacology | Endothelial Cells - drug effects | Cytokines | Comparative analysis | Vascular endothelial growth factor | Stem cells | Apoptosis | Heart | Phosphorylation | Leukocyte migration | Mesenchyme | Intracellular signalling | AKT protein | Kinases | Macrophages | Caspase-3 | Proteins | Angiogenesis | Signal transduction | Mitochondria | Cell growth | Ischemia | Rodents | Cell cycle | Tumor necrosis factor-TNF | Conditioning | Localization | Cardiology | Growth factors | Extracellular signal-regulated kinase | Caspase | Cardiomyocytes | Inflammation | Ribonucleic acid--RNA | Chemotaxis | Endothelial cells | 1-Phosphatidylinositol 3-kinase | Polymerase chain reaction | Inhibitors | γ-Interferon | Myocardium | Hypoxia | Interferon | Laboratory animals | Cell migration | Monocyte chemoattractant protein 1 | RNA | Ribonucleic acid
Journal Article
Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 11/2007, Volume 34, Issue 11, pp. 1109 - 1115
SUMMARY • It has been well established that oestrogens can increase the number of endothelial progenitor cells (EPC... 
coronary artery disease | vasculogenesis | resveratrol | endothelial progenitor cells | Vasculogenesis | Coronary artery disease | Endothelial progenitor cells | Resveratrol | MORTALITY | REENDOTHELIALIZATION | PHYSIOLOGY | BONE-MARROW | TRANSPLANTATION | STATIN THERAPY | INCREASE | PHARMACOLOGY & PHARMACY | RED WINE | EXPRESSION | CONSUMPTION | PROGRESSION | Cell Survival - drug effects | Neovascularization, Physiologic - drug effects | Endothelial Cells - metabolism | Humans | Cells, Cultured | Vascular Endothelial Growth Factor A - metabolism | G1 Phase - drug effects | Stilbenes - pharmacology | Cell Adhesion - drug effects | Stem Cells - metabolism | Dose-Response Relationship, Drug | Cell Movement - drug effects | Time Factors | Cell Differentiation - drug effects | Stem Cells - drug effects | Resting Phase, Cell Cycle - drug effects | Cell Proliferation - drug effects | S Phase - drug effects | Phytoestrogens - pharmacology | Endothelial Cells - drug effects | Cell Survival, drug effects | G1 Phase, drug effects | Vascular Endothelial Growth Factor A, metabolism | Cell Proliferation, drug effects | Cell Adhesion, drug effects | Stem Cells, metabolism | Endothelial Cells, metabolism | Phytoestrogens, pharmacology | S Phase, drug effects | Endothelial Cells, drug effects | Cell Movement, drug effects | Cell Differentiation, drug effects | Neovascularization, Physiologic, drug effects | G0 Phase, drug effects | Stilbenes, pharmacology | Stem Cells, drug effects | Endothelial growth factors | Analysis
Journal Article
Nature cell biology, ISSN 1476-4679, 2015, Volume 17, Issue 8, pp. 994 - 1003
The use of human pluripotent stem cells for in vitro disease modelling and clinical applications requires protocols that convert these cells into relevant adult cell types... 
DIRECTED DIFFERENTIATION | IN-VIVO | MOUSE | DEFINITIVE ENDODERM | GROWTH-FACTOR | HUMAN BLASTOCYSTS | STROMAL CELLS | WNT | CULTURE | LINES | CELL BIOLOGY | Coculture Techniques | Humans | Endothelial Cells - transplantation | Glycogen Synthase Kinase 3 beta | Cell Lineage - drug effects | Dose-Response Relationship, Drug | Human Umbilical Vein Endothelial Cells - physiology | Transfection | Time Factors | Gene Expression Regulation, Developmental | Transcription, Genetic | Myocytes, Smooth Muscle - drug effects | Proto-Oncogene Proteins c-sis - pharmacology | Myocytes, Smooth Muscle - transplantation | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Muscle, Smooth, Vascular - physiology | Muscle, Smooth, Vascular - drug effects | Biomarkers - metabolism | Cell Line | Induced Pluripotent Stem Cells - enzymology | Induced Pluripotent Stem Cells - drug effects | Induced Pluripotent Stem Cells - physiology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Myocytes, Smooth Muscle - enzymology | Induced Pluripotent Stem Cells - transplantation | Myocytes, Smooth Muscle - physiology | Gene Expression Profiling - methods | Muscle, Smooth, Vascular - transplantation | Glycogen Synthase Kinase 3 - metabolism | Mice, SCID | Muscle, Smooth, Vascular - cytology | Metabolomics - methods | Bone Morphogenetic Protein 4 - pharmacology | Phenotype | Animals | Wnt Signaling Pathway - drug effects | Cell Differentiation - drug effects | Mice, Inbred NOD | Protein Kinase Inhibitors - pharmacology | Endothelial Cells - enzymology | Muscle, Smooth, Vascular - enzymology | Neovascularization, Physiologic | Endothelial Cells - drug effects | Usage | Cell research | Growth | Stem cells | Muscle cells | Research | Cell differentiation | Endothelium
Journal Article