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Nature cell biology, ISSN 1476-4679, 07/2015, Volume 17, Issue 8, pp. 994 - 1003
The use of human pluripotent stem cells for in vitro disease modelling and clinical applications requires protocols that convert these cells into relevant adult cell types... 
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Coculture Techniques | Humans | Endothelial Cells - transplantation | Glycogen Synthase Kinase 3 beta | Cell Lineage - drug effects | Dose-Response Relationship, Drug | Human Umbilical Vein Endothelial Cells - physiology | Transfection | Time Factors | Gene Expression Regulation, Developmental | Transcription, Genetic | Myocytes, Smooth Muscle - drug effects | Proto-Oncogene Proteins c-sis - pharmacology | Myocytes, Smooth Muscle - transplantation | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Muscle, Smooth, Vascular - physiology | Muscle, Smooth, Vascular - drug effects | Biomarkers - metabolism | Cell Line | Induced Pluripotent Stem Cells - enzymology | Induced Pluripotent Stem Cells - drug effects | Induced Pluripotent Stem Cells - physiology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Myocytes, Smooth Muscle - enzymology | Induced Pluripotent Stem Cells - transplantation | Myocytes, Smooth Muscle - physiology | Gene Expression Profiling - methods | Muscle, Smooth, Vascular - transplantation | Glycogen Synthase Kinase 3 - metabolism | Mice, SCID | Muscle, Smooth, Vascular - cytology | Metabolomics - methods | Bone Morphogenetic Protein 4 - pharmacology | Phenotype | Animals | Wnt Signaling Pathway - drug effects | Cell Differentiation - drug effects | Mice, Inbred NOD | Protein Kinase Inhibitors - pharmacology | Endothelial Cells - enzymology | Muscle, Smooth, Vascular - enzymology | Neovascularization, Physiologic | Endothelial Cells - drug effects | Usage | Cell research | Growth | Stem cells | Muscle cells | Research | Cell differentiation | Endothelium | Index Medicus
Journal Article
Journal of the American Heart Association, ISSN 2047-9980, 02/2016, Volume 5, Issue 2, p. n/a
.... Methods and Results We explored the impact of TMAO on endothelial and smooth muscle cell function... 
atherosclerosis | cardiovascular disease | inflammation | endothelial cell | nuclear factor‐κB signaling | vascular smooth muscle cell | leukocyte adhesion | trimethylamine N‐oxide | Endothelial cell | Trimethylamine N-oxide | Nuclear factor-κB signaling | Atherosclerosis | Cardiovascular disease | Leukocyte adhesion | Vascular smooth muscle cell | Inflammation | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Atherosclerosis - genetics | Coculture Techniques | Humans | Myocytes, Smooth Muscle - pathology | Aortitis - enzymology | NF-kappa B - metabolism | Aortitis - chemically induced | Leukocytes - enzymology | Atherosclerosis - enzymology | Receptors, LDL - deficiency | Female | Aortitis - pathology | Myocytes, Smooth Muscle - drug effects | Aorta - enzymology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Receptors, LDL - genetics | Atherosclerosis - pathology | Genetic Predisposition to Disease | Myocytes, Smooth Muscle - enzymology | Aorta - drug effects | Atherosclerosis - chemically induced | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Aortitis - genetics | Cell Adhesion - drug effects | Mice, Knockout | Aorta - pathology | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Signal Transduction - drug effects | Choline | Leukocytes - drug effects | Enzyme Activation | Methylamines - toxicity | Endothelial Cells - pathology | Endothelial Cells - enzymology | Muscle, Smooth, Vascular - enzymology | Endothelial Cells - drug effects | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
Journal Article
Journal Article
Clinical and experimental pharmacology & physiology, ISSN 0305-1870, 10/2008, Volume 35, Issue 10, pp. 1156 - 1163
SUMMARY 1 Fabry disease results from an X‐linked mutation in the lysosomal α‐galactosidase A (Gla) gene. Defective Gla results in multi‐organ accumulation of... 
Fabry disease | endothelium | globotriaosylceramide | α‐galactosidase A | Globotriaosylceramide | α-galactosidase A | Endothelium | Pharmacology & Pharmacy | Physiology | Life Sciences & Biomedicine | Science & Technology | alpha-Galactosidase - genetics | Vascular Diseases - pathology | Blood Pressure - genetics | Fabry Disease - genetics | Male | Fabry Disease - enzymology | alpha-Galactosidase - physiology | Endothelium, Vascular - enzymology | Fabry Disease - pathology | Cell Membrane - pathology | Blood Pressure - physiology | Cell Membrane - metabolism | Aorta, Thoracic - pathology | Disease Models, Animal | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Vascular Diseases - genetics | Vascular Diseases - enzymology | Mice, Knockout | Aorta, Thoracic - enzymology | Cell Membrane - enzymology | Animals | Endothelial Cells - cytology | Endothelium, Vascular - metabolism | Glycosphingolipids - metabolism | Endothelium, Vascular - pathology | Mice | Endothelial Cells - pathology | In Vitro Techniques | Endothelial Cells - enzymology | Vascular Diseases, pathology | Aorta, Thoracic, pathology | Endothelial Cells, cytology | Endothelium, Vascular, metabolism | Cell Membrane, enzymology | Aorta, Thoracic, metabolism | Endothelial Cells, pathology | alpha-Galactosidase, genetics | Glycosphingolipids, metabolism | Endothelial Cells, enzymology | Endothelium, Vascular, enzymology | Endothelium, Vascular, pathology | Cell Membrane, pathology | Blood Pressure, physiology | Cell Membrane, metabolism | Vascular Diseases, enzymology | Fabry Disease, pathology | Fabry Disease, enzymology | Aorta, Thoracic, enzymology | Blood Pressure, genetics | alpha-Galactosidase, physiology | Vascular Diseases, genetics | Fabry Disease, genetics | Index Medicus
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 03/2005, Volume 111, Issue 9, pp. 1114 - 1120
Background - The function of bone marrow - derived endothelial progenitor cells (EPCs) in repair of ischemic tissue been the subject of intense scrutiny... 
Myocardial infarction | Nitric oxide synthase | Ischemia | Blood cells | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Phosphorylation | Capillaries - pathology | Bone Marrow Cells - enzymology | Nitric Oxide Synthase - physiology | Male | Stem Cells - cytology | Nitric Oxide Synthase - genetics | Nitric Oxide Synthase Type II | Cytokines - physiology | Cell Hypoxia | Myocardial Reperfusion Injury - pathology | Blood Cells - cytology | Stem Cells - enzymology | Myocardial Infarction - pathology | Bone Marrow Transplantation | Myocardial Ischemia - physiopathology | Coronary Circulation | Bone Marrow Cells - cytology | Mice, Inbred C57BL | Nitric Oxide Synthase Type III | Myocardium - pathology | Nitric Oxide - physiology | Radiation Chimera | Mice, Inbred Strains | Cells, Cultured - enzymology | Mice, Knockout | Blood Cells - enzymology | Myocardium - enzymology | Animals | Nitric Oxide Synthase - deficiency | Endothelial Cells - cytology | Ischemic Preconditioning, Myocardial | Mice | Protein Processing, Post-Translational | Endothelial Cells - enzymology | Vascular Endothelial Growth Factor A - physiology | Myocardial Reperfusion Injury - prevention & control | Neovascularization, Physiologic | Cell Movement | Index Medicus | Abridged Index Medicus
Journal Article
Molecular cell, ISSN 1097-2765, 11/2011, Volume 44, Issue 4, pp. 545 - 558
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2010, Volume 176, Issue 3, pp. 1494 - 1504
Journal Article
Circulation research, ISSN 1524-4571, 08/2014, Volume 115, Issue 4, pp. 450 - 459
...). The rapid increase in arginase activity in human aortic endothelial cells exposed to oxidized low-density lipoprotein (OxLDL... 
atherosclerosis | nitric oxide synthase type III | oxidized low density lipoprotein | mitochondrial processing peptidase | mitochondria | arginase | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Metalloendopeptidases - genetics | Mitochondria - enzymology | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Arginase - metabolism | Molecular Sequence Data | Male | Metalloendopeptidases - metabolism | rho-Associated Kinases - antagonists & inhibitors | Cytosol - enzymology | Atherosclerosis - enzymology | Transfection | RNA Interference | Time Factors | rho-Associated Kinases - metabolism | Aortic Diseases - prevention & control | Aortic Diseases - enzymology | Lipoproteins, LDL - metabolism | Aorta - physiopathology | Aorta - enzymology | Aortic Diseases - pathology | Disease Models, Animal | Metalloendopeptidases - antagonists & inhibitors | Amino Acid Sequence | Atherosclerosis - pathology | Scavenger Receptors, Class E - genetics | Signal Transduction | Atherosclerosis - physiopathology | Aorta - drug effects | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Arginase - genetics | Mitochondria - drug effects | Mice, Knockout | Protein Transport | Aorta - pathology | Aortic Diseases - genetics | Aortic Diseases - physiopathology | Animals | Apolipoproteins E - genetics | Protein Kinase Inhibitors - pharmacology | Enzyme Activation | Scavenger Receptors, Class E - deficiency | Endothelial Cells - enzymology | Atherosclerosis - prevention & control | Endothelial Cells - drug effects | Index Medicus
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 11/2012, Volume 287, Issue 47, pp. 39753 - 39765
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 06/2014, Volume 63, Issue 24, pp. 2734 - 2741
Journal Article