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The Journal of experimental medicine, ISSN 1540-9538, 2014, Volume 211, Issue 6, pp. 1153 - 1166
Journal Article
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2015, Volume 112, Issue 50, pp. 15408 - 15413
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2012, Volume 287, Issue 33, pp. 28017 - 28026
.... A reduction of IFN-gamma-producing CD4(+) T cells is observed in DMF-treated patients and may contribute to its clinical efficacy... 
TRANSCRIPTIONAL ACTIVATION | MULTIPLE-SCLEROSIS | PHOSPHORYLATION | PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOTHELIAL-CELLS | GENE-EXPRESSION | INDUCTION | T-BET | P65 | INNATE IMMUNITY | Dendritic Cells - immunology | Th1 Cells - immunology | MAP Kinase Signaling System - immunology | Th1 Cells - metabolism | Ribosomal Protein S6 Kinases, 90-kDa - metabolism | Th17 Cells - metabolism | Transcription Factor RelA - immunology | Th17 Cells - cytology | Phosphorylation - immunology | B7-1 Antigen - biosynthesis | Phosphorylation - drug effects | B7-2 Antigen - immunology | Dimethyl Fumarate | Immunosuppressive Agents - pharmacology | Interleukin-6 - metabolism | Dendritic Cells - metabolism | Mitogen-Activated Protein Kinase 3 - immunology | B7-1 Antigen - immunology | Cells, Cultured | Histocompatibility Antigens Class II - biosynthesis | Interleukin-12 - metabolism | Ribosomal Protein S6 Kinases, 90-kDa - immunology | Cell Differentiation - immunology | Animals | B7-2 Antigen - biosynthesis | Histocompatibility Antigens Class II - immunology | MAP Kinase Signaling System - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Transcription Factor RelA - metabolism | Cell Differentiation - drug effects | Fumarates - pharmacology | Interleukin-12 - immunology | Interleukin-6 - immunology | Th17 Cells - immunology | Dendritic Cells - cytology | Mice | Th1 Cells - cytology | Multiple Sclerosis | MAP Kinases (MAPKs) | Immunology | Dendritic Cells | Antigen Presentation | NF-Kappa B (NF-KB) | Cytokine
Journal Article
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2010, Volume 207, Issue 4, pp. 681 - 688
Peripheral immune tolerance is generally thought to result from cross-presentation of tissue-derived proteins by quiescent tissue-resident dendritic cells to self-reactive T cells that have escaped... 
PROMISCUOUS GENE-EXPRESSION | DELETIONAL TOLERANCE | MEDICINE, RESEARCH & EXPERIMENTAL | DENDRITIC CELLS | THYMIC EPITHELIAL-CELLS | IMMUNOTHERAPY | IN-VIVO | DIFFERENTIATION | IMMUNOLOGY | CD8(+) T-CELLS | SELF-TOLERANCE | CROSS-PRESENTATION | Autoantigens - metabolism | Cell Proliferation | Membrane Glycoproteins - metabolism | Gene Expression - genetics | Autoantigens - genetics | Monophenol Monooxygenase - metabolism | Histocompatibility Antigens Class I - metabolism | Lymphocyte Activation - immunology | Immune Tolerance - immunology | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Gene Expression - immunology | Receptors, Antigen, T-Cell - immunology | Monophenol Monooxygenase - genetics | Epitopes, T-Lymphocyte - immunology | Neoplasm Proteins - genetics | Antigens, Neoplasm - genetics | Endothelial Cells - metabolism | Stromal Cells - metabolism | Histocompatibility Antigens Class I - immunology | Glutamate Decarboxylase - genetics | Immunophenotyping | Mice, Transgenic | Stromal Cells - immunology | Histocompatibility Antigens Class I - genetics | Lymph Nodes - immunology | Transcription Factors - genetics | Antigen Presentation - immunology | Lymph Nodes - cytology | Membrane Glycoproteins - genetics | Mice, Knockout | Endothelial Cells - immunology | Animals | Monophenol Monooxygenase - immunology | T-Lymphocytes - cytology | Endothelial Cells - cytology | Autoantigens - immunology | MART-1 Antigen | T-Lymphocytes - immunology | Mice | Mice, Inbred BALB C | Receptors, Antigen, T-Cell - genetics | Epitopes, T-Lymphocyte - metabolism | Stromal Cells - cytology | Brief Definitive Report
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 11, p. e15531
Background: There is consensus that experimental autoimmune encephalomyelitis (EAE) can be mediated by myelin specific T cells of Th1 as well as of Th17... 
MYELIN OLIGODENDROCYTE GLYCOPROTEIN | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | CENTRAL-NERVOUS-SYSTEM | MICE | CYTOKINE | VASCULAR ENDOTHELIAL-CELLS | INDUCTION | EXPRESSION | T-CELLS | CUTTING EDGE | Cell Proliferation | Spleen - immunology | Encephalomyelitis, Autoimmune, Experimental - metabolism | Coculture Techniques | Interleukin-17 - immunology | Encephalomyelitis, Autoimmune, Experimental - immunology | Th17 Cells - transplantation | Adoptive Transfer | Interferon-gamma - metabolism | Th1 Cells - immunology | Th1 Cells - transplantation | Brain - metabolism | Th1 Cells - metabolism | Astrocytes - immunology | Th17 Cells - metabolism | Encephalomyelitis, Autoimmune, Experimental - genetics | Spleen - pathology | Mice, Inbred C57BL | Cells, Cultured | Myelin-Oligodendrocyte Glycoprotein | Mice, Transgenic | Mice, Knockout | Cell Differentiation - immunology | Interleukin-17 - metabolism | Myelin-Associated Glycoprotein - immunology | Animals | Spleen - metabolism | Interferon-gamma - immunology | Brain - pathology | Myelin Proteins | Th17 Cells - immunology | Mice | Cytotoxicity, Immunologic - immunology | Brain - immunology | Astrocytes - metabolism | Autoimmunity | Encephalomyelitis | Cytokines | Genetic engineering | B cells | T cells | Myelin proteins | Biotechnology | Neurosciences | Multiple sclerosis | Oligodendrocyte-myelin glycoprotein | Pathogenesis | Neurobiology | Helper cells | Central nervous system | Cytotoxicity | Lymphocytes T | Biology | Adoptive transfer | T-cell receptor | Lymphocytes | Rodents | Fibroblasts | Lesions | Localization | Antigens | Myelin | Astrocytes | Neutrophils | Transgenic mice | Glycoproteins | Gene expression | Experimental allergic encephalomyelitis | Interleukin 17 | CD4 antigen | Studies | RAG2 protein | Lymphopenia | Biopsy | Production methods | γ-Interferon | Cell lines | Interferon
Journal Article
Immunology, ISSN 1365-2567, 2010, Volume 129, Issue 3, pp. 363 - 374
...‐parenchymal liver cells (NPC) as a major component of the innate and adaptive immune system of the liver... 
sinusoidal endothelial cells | Toll‐like receptor | innate immunity | Kupffer cells | Innate immunity | Sinusoidal endothelial cells | Toll-like receptor | TOLERANCE | TRAUMA-HEMORRHAGE | IMMUNOLOGY | REPLICATION | HEPATITIS-C VIRUS | SINUSOIDAL ENDOTHELIAL-CELLS | LIPOPOLYSACCHARIDE | MICE | UP-REGULATION | T-CELLS | Tumor Necrosis Factor-alpha - metabolism | Interferon-alpha - pharmacology | Interferon-beta - immunology | Gene Expression - genetics | Dendritic Cells - immunology | Culture Media, Conditioned - pharmacology | Antigens, CD - metabolism | CD4-Positive T-Lymphocytes - immunology | Histocompatibility Antigens Class I - metabolism | Liver - immunology | Lymphocyte Activation - immunology | Gene Expression - immunology | Antibodies - immunology | Toll-Like Receptor 4 - agonists | Dendritic Cells - drug effects | Histocompatibility Antigens Class II - metabolism | Kupffer Cells - metabolism | Toll-Like Receptor 3 - genetics | Toll-Like Receptors - metabolism | Interleukin-6 - metabolism | Dendritic Cells - metabolism | Interferon-alpha - metabolism | Virus Replication - drug effects | Poly I-C - pharmacology | Endothelial Cells - metabolism | Mice, Inbred C57BL | Toll-Like Receptor 4 - genetics | Virus Replication - immunology | Toll-Like Receptor 3 - agonists | Encephalomyocarditis virus - immunology | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Toll-Like Receptors - agonists | Interferon-alpha - immunology | Kupffer Cells - drug effects | Antibodies - pharmacology | Endothelial Cells - immunology | Immunity, Innate - immunology | Animals | Interferon-beta - metabolism | Interferon-beta - pharmacology | Toll-Like Receptors - genetics | T-Lymphocytes - immunology | Liver - cytology | Mice | CD8-Positive T-Lymphocytes - immunology | Endothelial Cells - drug effects | Kupffer Cells - immunology | Medical colleges | Immune response | Dendritic cells | Interferon beta | Cross infection | Nosocomial infections | Biological response modifiers | T cells | Endothelium | Antigens | Ligands | T cell receptors | Immune system | Original
Journal Article