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Journal of Molecular Biology, ISSN 0022-2836, 2002, Volume 316, Issue 3, pp. 769 - 787
Vascular endothelial growth factor (VEGF) is a potent endothelial cell-specific mediator of angiogenesis and vasculogenesis. VEGF is involved pathologically in... 
protein structure | NMR | angiogenesis | vascular endothelial growth factor | peptide antagonist | Angiogenesis | Peptide antagonist | Protein structure | Vascular endothelial growth factor | MOLECULAR-DYNAMICS | TRIPLE-RESONANCE EXPERIMENTS | CRYSTAL-STRUCTURE | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROTEIN-STRUCTURE DETERMINATION | RECEPTOR-BINDING DOMAIN | BIOACTIVE CONFORMATION | ANGSTROM RESOLUTION | DE-NOVO DESIGN | IMPROVED SENSITIVITY | INTERPROTON DISTANCE RESTRAINTS | Disulfides - metabolism | Receptors, Vascular Endothelial Growth Factor | Vascular Endothelial Growth Factor A | Endothelial Growth Factors - metabolism | Vascular Endothelial Growth Factors | Humans | Molecular Sequence Data | Receptors, Growth Factor - antagonists & inhibitors | Crystallography, X-Ray | Endothelial Growth Factors - chemistry | Proto-Oncogene Proteins - chemistry | Vascular Endothelial Growth Factor Receptor-1 | Lymphokines - genetics | Peptides - chemical synthesis | Peptides - metabolism | Alanine - genetics | Solutions | Lymphokines - antagonists & inhibitors | Protein Structure, Quaternary | Lymphokines - metabolism | Nuclear Magnetic Resonance, Biomolecular | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Binding Sites | Dimerization | Protein Structure, Tertiary | Proto-Oncogene Proteins - metabolism | Amino Acid Sequence | Proto-Oncogene Proteins - antagonists & inhibitors | Bacteriophages - chemistry | Peptides - chemistry | Protein Structure, Secondary | Models, Molecular | Alanine - metabolism | Mutation - genetics | Receptor Protein-Tyrosine Kinases - metabolism | Receptors, Growth Factor - chemistry | Peptides - pharmacology | Endothelial Growth Factors - antagonists & inhibitors | Lymphokines - chemistry | Endothelial Growth Factors - genetics | Hydrophobic and Hydrophilic Interactions | Receptor Protein-Tyrosine Kinases - chemistry | Receptors, Growth Factor - metabolism
Journal Article
Nature Reviews Cancer, ISSN 1474-175X, 10/2004, Volume 4, Issue 10, pp. 806 - 813
Many solid tumours show an increased interstitial fluid pressure (IFP), which forms a barrier to transcapillary transport. This barrier is an obstacle in... 
RADIATION RESPONSE | DRUG PENETRATION | SOLID TUMORS | ONCOLOGY | COLLAGEN GEL CONTRACTION | PERICYTE RECRUITMENT | HUMAN OSTEOSARCOMA XENOGRAFTS | BLOOD-VESSELS | EXPERIMENTAL CARCINOMA | ENDOTHELIAL GROWTH-FACTOR | BASEMENT-MEMBRANE | Extracellular Fluid - physiology | Humans | Alprostadil - pharmacology | Neoplasms - blood supply | Bradykinin - antagonists & inhibitors | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Hydrostatic Pressure | Neoplasms - drug therapy | Hyaluronoglucosaminidase - pharmacology | Animals | Biological Transport | Signal Transduction - drug effects | Transforming Growth Factor beta - antagonists & inhibitors | Antineoplastic Agents - pharmacokinetics | Mice | Neoplasms - physiopathology | Niacinamide - pharmacology | Platelet-Derived Growth Factor - antagonists & inhibitors | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Care and treatment | Research | Health aspects | Methods | Cancer | Alprostadil/pharmacology | Niacinamide/pharmacology | Bradykinin/antagonists & inhibitors | Signal Transduction/drug effects | Hyaluronoglucosaminidase/pharmacology | Neoplasms/blood supply/drug therapy/physiopathology | Tumor Necrosis Factor-alpha/antagonists & inhibitors | Platelet-Derived Growth Factor/antagonists & inhibitors | Antineoplastic Agents/pharmacokinetics | Extracellular Fluid/physiology | Transforming Growth Factor beta/antagonists & inhibitors | Research Support; Non-U.S. Gov't | Vascular Endothelial Growth Factor A/antagonists & inhibitors
Journal Article
Retina, ISSN 0275-004X, 04/2011, Volume 31, Issue 4, pp. 662 - 668
Purpose: To assess the rate of infectious endophthalmitis and to describe the clinical and microbiological features of eyes that develop clinically suspected... 
VERTEPORFIN | INJECTION | SAFETY | bevacizumab | OPHTHALMOLOGY | endophthalmitis | intravitreal injection | anti-vascular endothelial growth factor | pegaptanib | age-related macular degeneration | ranibizumab
Journal Article
Annals of Oncology, ISSN 0923-7534, 03/2014, Volume 25, Issue 3, pp. 552 - 563
Journal Article
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 02/2014, Volume 348, Issue 2, pp. 236 - 245
Rheumatoid arthritis (RA) is a chronic autoimmune disease with high morbidity and mortality. Within the inflammatory milieu, resident fibroblast-like... 
OVEREXPRESSION | CIGARETTE-SMOKING | MMP EXPRESSION | VEGF | DIOXIN RECEPTOR | PHARMACOLOGY & PHARMACY | LIGAND | METALLOPROTEINASES | INHIBITORY FACTOR | T-CELLS | 2,3,7,8-TETRACHLORODIBENZO-P-DIOXIN | Receptors, Aryl Hydrocarbon - antagonists & inhibitors | Humans | Synovial Membrane - pathology | Vascular Endothelial Growth Factor A - metabolism | Epiregulin | Interleukin-1beta - genetics | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Promoter Regions, Genetic - drug effects | Vascular Endothelial Growth Factor A - genetics | Vascular Endothelial Growth Factor A - secretion | Arthritis, Rheumatoid - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Arthritis, Rheumatoid - drug therapy | Interleukin-1beta - metabolism | Gelatinases - metabolism | Interleukin-1beta - antagonists & inhibitors | Glycoproteins - genetics | Basic Helix-Loop-Helix Transcription Factors - genetics | Gelatinases - antagonists & inhibitors | Receptors, Aryl Hydrocarbon - genetics | Recombinant Proteins - chemistry | Cell Movement - drug effects | Fibroblast Growth Factor 2 - antagonists & inhibitors | Fibroblast Growth Factor 2 - genetics | Epidermal Growth Factor - secretion | Glycoproteins - metabolism | Synovial Membrane - immunology | Synovial Membrane - drug effects | Amphiregulin | Molecular Targeted Therapy | Intercellular Signaling Peptides and Proteins - metabolism | EGF Family of Proteins | Fibroblast Growth Factor 2 - metabolism | Receptors, Aryl Hydrocarbon - metabolism | Indoles - pharmacology | Antirheumatic Agents - pharmacology | Recombinant Proteins - metabolism | Cytokines - metabolism | Epidermal Growth Factor - genetics | Purines - pharmacology | Cells, Cultured | Gene Silencing | Glycoproteins - antagonists & inhibitors | Intercellular Signaling Peptides and Proteins - genetics | Epidermal Growth Factor - metabolism | Basic Helix-Loop-Helix Transcription Factors - antagonists & inhibitors | Down-Regulation - drug effects | Arthritis, Rheumatoid - pathology | Response Elements - drug effects | Synovial Membrane - secretion | Gelatinases - genetics | Epidermal Growth Factor - antagonists & inhibitors | Cell Proliferation - drug effects | Cytokines - antagonists & inhibitors | Arthritis, Rheumatoid - immunology
Journal Article
Angiogenesis, ISSN 0969-6970, 10/2013, Volume 16, Issue 4, pp. 903 - 917
Hypoxia-inducible factors (HIFs) and STAT-3 play essential roles in angiogenesis. HIF-1α and STAT-3 are clients of the heat shock protein 90 (HSP90). We... 
HSP90 | Angiogenesis | Biomedicine | Cancer Research | Oncology | HIF-1α | STAT-3 | Ophthalmology | Cardiology | Biomedicine general | Cell Biology | CELLS | HIF-1 alpha | HEAT-SHOCK-PROTEIN-90 | HYPOXIA | HIF-1 | THERAPY | O-2 | PERIPHERAL VASCULAR DISEASE | TUMOR-GROWTH | EXPRESSION | Adenocarcinoma - pathology | Humans | Vesicular Transport Proteins - biosynthesis | Antineoplastic Agents - therapeutic use | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | Gene Knockdown Techniques | Ribonuclease, Pancreatic - biosynthesis | RNA, Messenger - biosynthesis | Vesicular Transport Proteins - antagonists & inhibitors | Laminin | Platelet-Derived Growth Factor - biosynthesis | Rectal Neoplasms - genetics | STAT3 Transcription Factor - physiology | Rectal Neoplasms - pathology | Ribonuclease, Pancreatic - antagonists & inhibitors | Rectal Neoplasms - drug therapy | STAT3 Transcription Factor - genetics | Transforming Growth Factor beta1 - biosynthesis | Triazoles - therapeutic use | Proteoglycans | Specific Pathogen-Free Organisms | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Down-Regulation | Ribonuclease, Pancreatic - genetics | Angiogenesis Inhibitors - pharmacology | Adenocarcinoma - drug therapy | Chick Embryo | Adenocarcinoma - blood supply | Fibroblast Growth Factor 2 - antagonists & inhibitors | Fibroblast Growth Factor 2 - genetics | HSP90 Heat-Shock Proteins - antagonists & inhibitors | Mice, Nude | Cell Line, Tumor | Human Umbilical Vein Endothelial Cells | Vascular Endothelial Growth Factor A - biosynthesis | Transforming Growth Factor beta1 - antagonists & inhibitors | HSP90 Heat-Shock Proteins - physiology | Gene Expression Regulation, Neoplastic | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Platelet-Derived Growth Factor - genetics | HEK293 Cells | Angiogenesis Inhibitors - therapeutic use | Female | Antineoplastic Agents - pharmacology | Hypoxia-Inducible Factor 1, alpha Subunit - physiology | Chorioallantoic Membrane - blood supply | Colorectal Neoplasms - blood supply | Vesicular Transport Proteins - genetics | Fibroblast Growth Factor 2 - biosynthesis | Transforming Growth Factor beta1 - genetics | Triazoles - pharmacology | Xenograft Model Antitumor Assays | Animals | Collagen | Colorectal Neoplasms - pathology | STAT3 Transcription Factor - antagonists & inhibitors | Platelet-Derived Growth Factor - antagonists & inhibitors | Drug Combinations
Journal Article
Journal of Medicinal Chemistry, ISSN 0022-2623, 05/2018, Volume 61, Issue 9, pp. 4135 - 4154
Journal Article
Journal of Biochemistry, ISSN 0021-924X, 01/2013, Volume 153, Issue 1, pp. 13 - 19
Vascular endothelial growth factors (VEGFs) belong to the platelet-derived growth factor supergene family, and they play central roles in the regulation of... 
VEGFR | hypoxia | preeclampsia | VEGF | tumor angiogenesis | SUPPRESSES TUMOR-GROWTH | CELLS | TYROSINE KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | BONE-MARROW | VEGF RECEPTOR-1 | BEVACIZUMAB PLUS IRINOTECAN | NUTRIENT STARVATION | DNA-SYNTHESIS | FLT-1 | IN-VIVO | Neoplasms - metabolism | Vascular Endothelial Growth Factor Receptor-1 - antagonists & inhibitors | Humans | Male | Neoplasm Proteins - antagonists & inhibitors | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Molecular Targeted Therapy | Neoplasm Proteins - metabolism | Vascular Endothelial Growth Factor A - chemistry | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Angiogenesis Inhibitors - therapeutic use | Female | Pre-Eclampsia - metabolism | Lymphangiogenesis | Angiogenesis Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Neoplasm Proteins - chemistry | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Vascular Endothelial Growth Factor Receptor-1 - chemistry | Neoplasms - drug therapy | Pregnancy | Vascular Endothelial Growth Factor Receptor-2 - chemistry | Animals | Pre-Eclampsia - drug therapy | Signal Transduction - drug effects | Neovascularization, Pathologic - drug therapy | Neovascularization, Pathologic - metabolism | Neovascularization, Physiologic | JB Reviews
Journal Article
Nature Cell Biology, ISSN 1465-7392, 10/2011, Volume 13, Issue 10, pp. 1202 - 1213
Angiogenesis, the growth of new blood vessels, involves specification of endothelial cells to tip cells and stalk cells, which is controlled by Notch... 
LYMPHATIC ENDOTHELIUM | KINASE-ACTIVITY | VASCULAR NETWORK FORMATION | ENDOTHELIAL-GROWTH-FACTOR | FACTOR-C | MOUSE EMBRYOS | EXTRACELLULAR-MATRIX | MONOCLONAL-ANTIBODY | TUMOR ANGIOGENESIS | FACTOR RECEPTOR-3 | CELL BIOLOGY | Neovascularization, Physiologic - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Receptors, Notch - metabolism | Humans | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Receptors, Notch - antagonists & inhibitors | Transfection | RNA Interference | Time Factors | Forkhead Transcription Factors - metabolism | Vascular Endothelial Growth Factor D - genetics | Phosphatidylinositol 3-Kinase - metabolism | Vascular Endothelial Growth Factor Receptor-3 - deficiency | Transduction, Genetic | Retinal Vessels - metabolism | Endothelial Cells - metabolism | Rhombencephalon - blood supply | Gene Expression Regulation | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Forkhead Transcription Factors - genetics | Vascular Endothelial Growth Factor C - genetics | Mice, Knockout | Antibodies - pharmacology | Macrophages - metabolism | Vascular Endothelial Growth Factor D - metabolism | Animals | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Vascular Endothelial Growth Factor C - metabolism | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Vascular Endothelial Growth Factor B - genetics | Mice | Neovascularization, Pathologic - metabolism | Protein Kinase Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-3 - genetics | Endothelial Cells - drug effects | Physiological aspects | Blood vessels | Cellular signal transduction | Growth factor receptors | Research | Vascular endothelial growth factor | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
Pancreas, ISSN 0885-3177, 04/2013, Volume 42, Issue 3, pp. 411 - 421
Objectives: Pancreatic endocrine tumors (PETs) share numerous features with gastrointestinal neuroendocrine (carcinoid) tumors. Targets of novel therapeutic... 
Pancreatic endocrine tumors | Molecular analysis | pancreatic endocrine tumors | GROWTH-FACTOR-RECEPTOR | GASTROENTEROPANCREATIC NEUROENDOCRINE TUMORS | KRAS MUTATION | IN-SITU-HYBRIDIZATION | GEFITINIB THERAPY | METASTATIC COLORECTAL-CANCER | CELL LUNG-CANCER | BREAST-CANCER | GASTROENTEROLOGY & HEPATOLOGY | molecular analysis | GENE COPY NUMBER | CLINICAL-RESPONSE | Immunohistochemistry | Receptor, Epidermal Growth Factor - genetics | Receptor, IGF Type 1 - metabolism | Proto-Oncogene Proteins p21(ras) | Pyrazoles - therapeutic use | Receptor, IGF Type 1 - antagonists & inhibitors | Pancreatic Neoplasms - metabolism | Receptor, ErbB-2 - genetics | Humans | Carcinoid Tumor - metabolism | Biomarkers, Tumor - metabolism | Receptors, Somatostatin - antagonists & inhibitors | Receptor, ErbB-2 - antagonists & inhibitors | Receptors, Somatostatin - metabolism | Imidazoles - therapeutic use | Protein-Serine-Threonine Kinases - metabolism | Cell Survival - drug effects | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Carcinoid Tumor - drug therapy | ras Proteins - antagonists & inhibitors | In Situ Hybridization, Fluorescence | Carcinoid Tumor - genetics | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Drug Therapy - methods | HSP90 Heat-Shock Proteins - antagonists & inhibitors | Cell Line, Tumor | HSP90 Heat-Shock Proteins - metabolism | Biomarkers, Tumor - genetics | Mutation | Benzoquinones - therapeutic use | Indazoles - therapeutic use | Vascular Endothelial Growth Factor Receptor-1 - antagonists & inhibitors | ras Proteins - genetics | Triazines - therapeutic use | Receptor, ErbB-2 - metabolism | ras Proteins - metabolism | Proto-Oncogene Proteins c-kit - metabolism | Proto-Oncogene Proteins c-kit - antagonists & inhibitors | Pancreatic Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - metabolism | Receptor, Platelet-Derived Growth Factor alpha - antagonists & inhibitors | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Proto-Oncogene Proteins c-kit - genetics | Lactams, Macrocyclic - therapeutic use | Biomarkers, Tumor - antagonists & inhibitors | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Pancreatic Neoplasms - genetics | Proto-Oncogene Proteins - genetics | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Receptors, Transforming Growth Factor beta - metabolism | Protein Kinase Inhibitors - therapeutic use | Receptor, Platelet-Derived Growth Factor alpha - genetics | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Index Medicus
Journal Article
Cancer Research, ISSN 0008-5472, 06/2008, Volume 68, Issue 12, pp. 4774 - 4782
Inhibition of tumor angiogenesis through blockade of the vascular endothelial growth factor (VEGF) signaling pathway is a novel treatment modality in oncology.... 
CELLS | POTENT | ONCOLOGY | PATHWAY | CRYSTAL-STRUCTURE | VEGF | TYROSINE KINASES | TUMOR ANGIOGENESIS | CANCER | PERICYTES | ENDOTHELIAL GROWTH-FACTOR | Endothelium, Vascular - cytology | Pericytes - drug effects | Apoptosis - drug effects | Humans | Pericytes - cytology | Rats, Inbred F344 | Indoles - chemical synthesis | Endothelium, Vascular - drug effects | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor Receptor-2 - immunology | Enzyme Inhibitors - chemical synthesis | Neoplasms, Experimental - pathology | Tissue Distribution | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Enzyme Inhibitors - pharmacokinetics | Female | Molecular Structure | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Receptors, Platelet-Derived Growth Factor - antagonists & inhibitors | Signal Transduction | Pericytes - metabolism | Cells, Cultured | Receptors, Fibroblast Growth Factor - antagonists & inhibitors | Rats | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Enzyme Inhibitors - therapeutic use | Xenograft Model Antitumor Assays | Animals | Endothelium, Vascular - metabolism | Mice, Nude | Blood Coagulation Factors - antagonists & inhibitors | Receptors, Fibroblast Growth Factor - metabolism | Indoles - pharmacokinetics | Indoles - therapeutic use | Cell Proliferation - drug effects | Mice | Neoplasms, Experimental - metabolism | Neoplasms, Experimental - drug therapy
Journal Article
Journal Article