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1. Full Text Andrographolide inhibits hypoxia‐induced hypoxia‐inducible factor 1α and endothelin 1 expression through the heme oxygenase 1/CO/cGMP/MKP‐5 pathways in EA.hy926 cells
by Lin, Hung‐Chih and Su, Shih‐Li and Lin, Wan‐Chun and Lin, Ai‐Hsuan and Yang, Ya‐Chen and Lii, Chong‐Kuei and Chen, Haw‐Wen
Environmental Toxicology, ISSN 1520-4081, 03/2018, Volume 33, Issue 3, pp. 269 - 279
Andrographolide is a potent anti‐inflammatory agent found in Andrographis paniculata . Endothelin 1 (ET‐1) is an endothelium‐derived vasoconstrictor with... 
endothelin 1 | mitogen‐activated protein kinase phosphatase 5 | hypoxia‐inducible factor 1α | heme oxygenase 1 | andrographolide | hypoxia-inducible factor 1α | mitogen-activated protein kinase phosphatase 5 | ACTIVATED PROTEIN-KINASE | MAP KINASE PHOSPHATASE-5 | ATHEROSCLEROSIS | WATER RESOURCES | MECHANISMS | INDUCTION | ENVIRONMENTAL SCIENCES | INFLAMMATION | MONOXIDE | DISEASE | GENE-EXPRESSION | TOXICOLOGY | hypoxia-inducible factor 1 alpha | COBALT | Index Medicus
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2. Full Text Endothelin‐1 increases CHSY‐1 expression in aortic endothelial cells via transactivation of transforming growth factor β type I receptor induced by type B receptor endothelin‐1
by Seif, Faezeh and Little, Peter J and Niayesh‐Mehr, Reyhaneh and Zamanpour, Masoumeh and Babaahmadi‐Rezaei, Hossein
Journal of Pharmacy and Pharmacology, ISSN 0022-3573, 06/2019, Volume 71, Issue 6, pp. 988 - 995
Objectives TGF-beta through hyperelongation of glycosaminoglycan (GAG) chains leads to binding of low-density lipoproteins to the proteoglycans. The vasoactive... 
atherosclerosis | Smad2C | TGF | receptor | endothelin‐1 | transactivation | ETB receptor | TGF-beta | ELONGATION | PROTEIN-COUPLED RECEPTORS | ETA-RECEPTOR | PHARMACOLOGY & PHARMACY | endothelin-1 | EGF RECEPTOR | Phosphorylation | Matrix metalloproteinases | Glycosaminoglycans | Endothelin | Transforming growth factor | Cytochalasin D | Matrix metalloproteinase | Kinases | Western blotting | Proteins | Time dependence | Signal transduction | Actin | Vasoactive agents | Smad2 protein | Atherosclerosis | Aorta | Metalloproteinase | Inhibition | Growth factors | Proteoglycans | Polymerization | Endothelial cells | Rho-associated kinase | Intermediates | Signaling | Lipoproteins | Inhibitors | Arteriosclerosis | Cytoskeleton | Index Medicus
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3. Full Text Neural peptidase endothelin-converting enzyme 1 regulates endothelin 1-induced pruritus
by Kido-Nakahara, Makiko and Buddenkotte, Jörg and Kempkes, Cordula and Ikoma, Akihiko and Cevikbas, Ferda and Akiyama, Tasuku and Nunes, Frank and Seeliger, Stephan and Hasdemir, Burcu and Mess, Christian and Buhl, Timo and Sulk, Mathias and Müller, Frank-Ulrich and Metze, Dieter and Bunnett, Nigel W and Bhargava, Aditi and Carstens, Earl and Furue, Masutaka and Steinhoff, Martin
Journal of Clinical Investigation, ISSN 0021-9738, 06/2014, Volume 124, Issue 6, pp. 2683 - 2695
In humans, pruritus (itch) is a common but poorly understood symptom in numerous skin and systemic diseases. Endothelin 1 (ET-1) evokes histamine-independent... 
Metalloendopeptidases - genetics | Up-Regulation | Skin - metabolism | Humans | Male | Metalloendopeptidases - metabolism | Pruritus - genetics | Aspartic Acid Endopeptidases - genetics | Endothelin-Converting Enzymes | MAP Kinase Signaling System | Adult | Female | Pruritus - etiology | Skin - pathology | Metalloendopeptidases - antagonists & inhibitors | Signal Transduction | Aspartic Acid Endopeptidases - antagonists & inhibitors | Endothelin-1 - genetics | Endothelin-1 - metabolism | Mice, Inbred C57BL | Endothelin-1 - administration & dosage | Mice, Knockout | Pruritus - metabolism | Animals | Aspartic Acid Endopeptidases - metabolism | Skin - innervation | Receptor, Endothelin A - metabolism | Mice | Ganglia, Spinal - metabolism | Physiological aspects | Cellular signal transduction | Pruritus | Proteases | Neurons | Health aspects | Signal transduction | Enzymes | Pain | Nervous system | Neuropeptides | Software | Recycling | Kinases | Laboratory animals | Autoimmune diseases | Age | Index Medicus | Abridged Index Medicus
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4. Full Text Ambrisentan for the treatment of pulmonary arterial hypertension: Results of the ambrisentan in pulmonary arterial hypertension, randomized, double-Blind, placebo-controlled, multicenter, efficacy (ARIES) study 1 and 2
by Galiè, Nazzareno and Olschewski, Horst and Oudiz, Ronald J and Torres, Fernando and Frost, Adaani and Ghofrani, Hossein A and Badesch, David B and McGoon, Michael D and McLaughlin, Vallerie V and Roecker, Ellen B and Gerber, Michael J and Dufton, Christopher and Wiens, Brian L and Rubin, Lewis J and Ambrisentan Pulm Arterial Hyperten and Ambrisentan in Pulmonary Arterial Hypertension, Randomized, Double-Blind, Placebo-Controlled, Multicenter, Efficacy Studies (ARIES) Group
Circulation, ISSN 0009-7322, 06/2008, Volume 117, Issue 23, pp. 3010 - 3019
Background - Ambrisentan is a propanoic acid-based, A-selective endothelin receptor antagonist for the once-daily treatment of pulmonary arterial hypertension.... 
Drugs | Hypertension | Receptors | Endothelin | Pulmonary | Ambrisentan | INHALED ILOPROST | CONTROLLED-TRIAL | CARDIAC & CARDIOVASCULAR SYSTEMS | BRAIN NATRIURETIC PEPTIDE | receptors | drugs | ambrisentan | pulmonary | CONTINUOUS INTRAVENOUS EPOPROSTENOL | endothelin | THERAPY | 6-MINUTE WALK | PROSTACYCLIN ANALOG | DISEASE | PERIPHERAL VASCULAR DISEASE | hypertension | BOSENTAN | Double-Blind Method | Follow-Up Studies | Administration, Oral | Humans | Middle Aged | Phenylpropionates - administration & dosage | Kaplan-Meier Estimate | Hypertension, Pulmonary - physiopathology | Male | Treatment Outcome | Activities of Daily Living | Dyspnea | Exercise | Pyridazines - administration & dosage | Placebos | Quality of Life | Female | Phenylpropionates - adverse effects | Aged | Hypertension, Pulmonary - drug therapy | Endothelin Receptor Antagonists | Natriuretic Peptide, Brain - blood | Pyridazines - adverse effects | Care and treatment | Dosage and administration | Research | Pulmonary hypertension | Index Medicus | Abridged Index Medicus
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5. Full Text Andrographolide inhibits hypoxia‐induced HIF‐1α‐driven endothelin 1 secretion by activating Nrf2/HO‐1 and promoting the expression of prolyl hydroxylases 2/3 in human endothelial cells
by Lin, Hung‐Chih and Su, Shih‐Li and Lu, Chia‐Yang and Lin, Ai‐Hsuan and Lin, Wan‐Chun and Liu, Chin‐San and Yang, Ya‐Chen and Wang, Hsiu‐Miao and Lii, Chong‐Kuei and Chen, Haw‐Wen
Environmental Toxicology, ISSN 1520-4081, 03/2017, Volume 32, Issue 3, pp. 918 - 930
Andrographolide, the main bioactive component of the medicinal plant Andrographis paniculata , has been shown to possess potent anti‐inflammatory activity.... 
heme oxygenase 1 (HO‐1) | prolyl hydroxylases (PHDs) | Nrf2 | endothelin 1 (ET‐1) | hypoxia‐inducible factor 1α (HIF‐1α) | andrographolide | hypoxia-inducible factor 1α (HIF-1α) | endothelin 1 (ET-1) | heme oxygenase 1 (HO-1) | Heme Oxygenase-1 - metabolism | NF-E2-Related Factor 2 - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Prolyl Hydroxylases - metabolism | Humans | RNA, Messenger - metabolism | Cell Hypoxia | Heme Oxygenase-1 - genetics | RNA Interference | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Prolyl Hydroxylases - genetics | NF-E2-Related Factor 2 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Diterpenes - pharmacology | Cell Line | Cell Survival - drug effects | Hydroxylation | Endothelial Cells - metabolism | Endothelin-1 - genetics | Endothelin-1 - metabolism | Heme Oxygenase-1 - antagonists & inhibitors | Cobalt - toxicity | Signal Transduction - drug effects | Endothelial Cells - cytology | NF-E2-Related Factor 2 - metabolism | RNA, Small Interfering - metabolism | Index Medicus
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6. Full Text Immediate and Catastrophic Antibody‐Mediated Rejection in a Lung Transplant Recipient With Anti–Angiotensin II Receptor Type 1 and Anti–Endothelin‐1 Receptor Type A Antibodies
by Cozzi, E and Calabrese, F and Schiavon, M and Feltracco, P and Seveso, M and Carollo, C and Loy, M and Cardillo, M and Rea, F
American Journal of Transplantation, ISSN 1600-6135, 02/2017, Volume 17, Issue 2, pp. 557 - 564
Preexisting donor‐specific anti‐ HLA antibodies ( DSAs ) have been associated with reduced survival of lung allografts. However, antibodies with specificities... 
rejection: antibody‐mediated | clinical research/practice | lung transplantation/pulmonology | alloantibody | ABMR | histocompatibility | immunobiology | rejection: antibody-mediated (ABMR) | HEART | AUTOANTIBODIES | SURGERY | A RECEPTOR | MANAGEMENT | INTERNATIONAL-SOCIETY | POSTTRANSPLANT | NON-HLA-ANTIBODIES | POLYMORPHISM | DYSFUNCTION | ALLOGRAFTS | TRANSPLANTATION | Prognosis | Humans | HLA Antigens - immunology | Graft Rejection - etiology | Graft Survival | Male | Receptor, Endothelin A - immunology | Lung Transplantation - adverse effects | Transplant Recipients | Cystic Fibrosis - surgery | Isoantibodies - immunology | Adult | Receptor, Angiotensin, Type 1 - immunology | Tissue Donors | Postoperative Complications | Viral antibodies | Transplantation of organs, tissues, etc | Antibodies | Cystic fibrosis | Endothelin | Angiotensin | Transplants & implants | Immunoglobulins
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7. Full Text β-Arrestin 1 is required for endothelin-1-induced NF-κB activation in ovarian cancer cells
by Cianfrocca, Roberta and Tocci, Piera and Semprucci, Elisa and Spinella, Francesca and Di Castro, Valeriana and Bagnato, Anna and Rosanò, Laura
Life Sciences, ISSN 0024-3205, 11/2014, Volume 118, Issue 2, pp. 179 - 184
In epithelial ovarian cancer (EOC), activation of endothelin-1 (ET-1)/endothelin A receptor (ET R) signalling is linked to many tumor promoting effects, such... 
NF-κB | β-Arrestin-1 | Endothelin-1 | Ovarian cancer | PATHWAYS | MEDICINE, RESEARCH & EXPERIMENTAL | LOCALIZATION | PHOSPHORYLATION | beta-Arrestin-1 | NUCLEAR EXPORT SIGNAL | RECEPTOR | IDENTIFICATION | G-PROTEIN | IN-VIVO | PHARMACOLOGY & PHARMACY | NF-kappa beta | TRANSCRIPTIONAL RESPONSE | Neoplasms, Glandular and Epithelial - pathology | Endothelin-1 - metabolism | Humans | Gene Expression Regulation, Neoplastic | Ovarian Neoplasms - pathology | Neoplasms, Glandular and Epithelial - metabolism | NF-kappa B - metabolism | Receptor, Endothelin A - genetics | Arrestins - metabolism | Ovarian Neoplasms - genetics | Neoplasms, Glandular and Epithelial - genetics | Receptor, Endothelin B - genetics | Models, Biological | beta-Arrestin 1 | beta-Arrestins | Carcinoma, Ovarian Epithelial | Cell Line, Tumor | Female | Receptor, Endothelin A - metabolism | Ovarian Neoplasms - metabolism | Receptor, Endothelin B - metabolism | Genes, Reporter | RNA, Small Interfering - metabolism | Index Medicus
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8. Full Text Endothelin-1 induces hypoxia inducible factor 1α expression in pulmonary artery smooth muscle cells
by Li, Manxiang and Li, Zongfang and Liu, Yuan and Liu, Yun and Jin, Faguang and Sun, Xiuzhen and Fang, Ping and Shi, Hongyang and Jiang, Xingtang
FEBS Letters, ISSN 0014-5793, 11/2012, Volume 586, Issue 21, pp. 3888 - 3893
► Endothelin-1 posttranscriptionally increases HIF1α expression by suppressing proteasome-dependent HIF1α degradation. ► Endothelin-1 stimulates calcineurin... 
RACK1 | HIF1α | Calcineurin | Endothelin-1 | Proteasome | HSP90 | ACTIVATION | MANAGEMENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | INTRACELLULAR CA2 | INTERMITTENT HYPOXIA | INHIBITION | BIOPHYSICS | HIF1 alpha | DEGRADATION | HIF-1-ALPHA | HYPERTENSION | Phosphorylation | Receptors, Endothelin - genetics | Cyclosporine - pharmacology | GTP-Binding Proteins - genetics | Protease Inhibitors - pharmacology | Pulmonary Artery - metabolism | Calcineurin - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Receptors for Activated C Kinase | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Endothelin-1 - genetics | Endothelin-1 - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Gene Silencing | Receptors, Endothelin - metabolism | Rats | Signal Transduction - genetics | Pulmonary Artery - drug effects | Rats, Sprague-Dawley | Pulmonary Artery - cytology | Gene Expression Regulation - drug effects | Leupeptins - pharmacology | Animals | Signal Transduction - drug effects | Protein Biosynthesis - drug effects | Primary Cell Culture | Proteasome Endopeptidase Complex - metabolism | Calcineurin - metabolism | GTP-Binding Proteins - metabolism | Index Medicus
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9. Full Text Andrographolide inhibits hypoxia-induced hypoxia-inducible factor 1[alpha] and endothelin 1 expression through the heme oxygenase 1/CO/cGMP/MKP-5 pathways in EA.hy926 cells
by Lin, Hung-Chih and Su, Shih-Li and Lin, Wan-Chun and Lin, Ai-Hsuan and Yang, Ya-Chen and Lii, Chong-Kuei and Chen, Haw-Wen
Environmental Toxicology, ISSN 1520-4081, 03/2018, Volume 33, Issue 3, p. 269
Andrographolide is a potent anti-inflammatory agent found in Andrographis paniculata. Endothelin 1 (ET-1) is an endothelium-derived vasoconstrictor with... 
Endothelin | Phosphatases | Mitogens | Protein kinases | Heme | Endothelium | Damping | Hypoxia-inducible factor 1 | Activation | Kinases | Guanylate cyclase | Phosphatase | Epithelia | MAP kinase phosphatase | Proteins | Hypoxia-inducible factor 1a | Specificity | Transfection | Pathways | Cyclic GMP | Inhibition | Pretreatment | Hypoxia-inducible factors | Tyrosine | Threonine | Secretion | MAP kinase | Inflammation | siRNA | Endothelin 1 | Cells | Vanadate | Oxygenase | Inhibitors | Protein kinase | Hypoxia | Protein-tyrosine-phosphatase
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10. Full Text Low Blood Pressure in Endothelial Cell–Specific Endothelin 1 Knockout Mice
by Kisanuki, Yaz Y and Emoto, Noriaki and Ohuchi, Takashi and Widyantoro, Bambang and Yagi, Keiko and Nakayama, Kazuhiko and Kedzierski, Rafal M and Hammer, Robert E and Yanagisawa, Hiromi and Williams, S. Clay and Richardson, James A and Suzuki, Takashi and Yanagisawa, Masashi
Hypertension, ISSN 0194-911X, 07/2010, Volume 56, Issue 1, pp. 121 - 128
Endothelin (ET) 1 is a potent vasoconstrictor peptide produced by vascular endothelial cells and implicated in various pathophysiologic states involving... 
Hypertension | Blood pressure | Cre/loxP | Gene knockout | Endothelium | SYSTEM | endothelium | blood pressure | DUCT-SPECIFIC KNOCKOUT | SODIUM RETENTION | RATS | cre/loxP | ANGIOTENSIN-II | gene knockout | ETB | ETA | EXPERIMENTAL-HYPERTENSION | B RECEPTORS | IN-VIVO | DEFICIENT | PERIPHERAL VASCULAR DISEASE | hypertension | EXPRESSION | Hypotension - pathology | Immunohistochemistry | Receptor, Endothelin B - biosynthesis | RNA, Messenger - genetics | Endothelium, Vascular - physiopathology | Gene Expression Regulation | Proteoglycans - biosynthesis | Receptor, Endothelin A - genetics | Reverse Transcriptase Polymerase Chain Reaction | Immunoenzyme Techniques | Mice, Knockout | Receptor, Endothelin A - biosynthesis | Receptor, Endothelin B - genetics | Animals | Hypotension - metabolism | Hypotension - physiopathology | Endothelium, Vascular - metabolism | Alleles | Endothelium, Vascular - pathology | Blood Pressure - physiology | Mice | Proteoglycans - genetics | Disease Models, Animal | Index Medicus
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11. Full Text Systematic Evaluation of Endothelin 1 Measurement Relative to Traditional and Modern Biomarkers for Clinical Assessment and Prognosis in Patients With Chronic Systolic Heart Failure Serial Measurement and Multimarker Testing
by Gaggin, HK and Truong, QA and Gandhi, PU and Motiwala, SR and Belcher, AM and Weiner, RB and Baggish, AL and Januzzi, JL
AMERICAN JOURNAL OF CLINICAL PATHOLOGY, ISSN 0002-9173, 05/2017, Volume 147, Issue 5, pp. 461 - 472
Objectives: To define the role of single or serial measurement of endothelin 1 (ET-1) for prognostication beyond traditional and modern markers of risk in... 
Heart failure | Prognosis | Endothelin | MANAGEMENT | ENDOGENOUS ENDOTHELIN | ANGIOTENSIN-II | PATHOLOGY | NATRIURETIC PEPTIDE | Multimarker panel | HYPERTROPHY | THERAPY PROTECT | TASK-FORCE | Systolic | DYSFUNCTION | RECEPTORS | Monitoring | Immunoassay | Echocardiography | Humans | Middle Aged | Proportional Hazards Models | Male | Biomarkers - blood | Endothelin-1 - blood | Peptide Fragments - blood | Female | Aged | Heart Failure, Systolic - blood | Chronic Disease | Natriuretic Peptide, Brain - blood | Angiotensin-converting enzyme inhibitors | Brain natriuretic peptide | Endothelin 1 | Reclassification | Risk factors | Troponin | Troponin I | Peptidyl-dipeptidase A | Calcium-binding protein | Angiotensin | Biomarkers | Ventricle | Health risk assessment | Heart diseases | Index Medicus | Abridged Index Medicus
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12. Full Text EP02.01: Autoantibodies against the Endothelin‐1 receptor type A and the Angiotensin II receptor type 1 in patients with pregnancy induced hypertension and pre‐eclampsia
by Tørring, N and Larsen, S and Wallukat, G and Schimke, I and Sandager, A and Uldbjerg, N
Ultrasound in Obstetrics & Gynecology, ISSN 0960-7692, 10/2018, Volume 52, Issue S1, pp. 196 - 196
Hypertension | Autoimmunity | Care and treatment | Endothelin | Autoantibodies | Pregnant women | Angiotensin | Pregnancy | Eclampsia | Angiotensin II
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13. Full Text An arterial-specific enhancer of the human endothelin converting enzyme 1 (ECE1) gene is synergistically activated by Sox17, FoxC2, and Etv2
by Robinson, Ashley S and Materna, Stefan C and Barnes, Ralston M and De Val, Sarah and Xu, Shan-Mei and Black, Brian L
Developmental Biology, ISSN 0012-1606, 11/2014, Volume 395, Issue 2, pp. 379 - 389
Endothelin-converting enzyme-1 (Ece-1), a crucial component of the Endothelin signaling pathway, is required for embryonic development and is an important... 
FoxC2 | Ece-1 | Embryo | Endothelin | Mouse | Sox17 | Etv2 | Vasculogenesis | Vein | Artery | VASCULAR DEVELOPMENT | DEVELOPMENT IN-VIVO | DEVELOPMENTAL BIOLOGY | HAEMOGENIC ENDOTHELIUM | CELL DEVELOPMENT | FORKHEAD TRANSCRIPTION FACTORS | HEMATOPOIETIC DEVELOPMENT | ENDOTHELIN-CONVERTING ENZYME-1 | MESSENGER-RNA | SIGNALING PATHWAY | EXPRESSION | Metalloendopeptidases - genetics | Humans | Metalloendopeptidases - metabolism | DNA Primers - genetics | Aspartic Acid Endopeptidases - genetics | SOXF Transcription Factors - metabolism | Endothelin-Converting Enzymes | Endocardium - metabolism | Forkhead Transcription Factors - metabolism | Cloning, Molecular | Electrophoretic Mobility Shift Assay | Indoles | Endothelium, Vascular - embryology | Galactosides | Endocardium - embryology | Mice, Transgenic | Transcription Factors - metabolism | Animals | Mutagenesis | Endothelium, Vascular - metabolism | Aspartic Acid Endopeptidases - metabolism | Fluorescent Antibody Technique | SOX Transcription Factors - metabolism | Enhancer Elements, Genetic - genetics | Mice | Enzymes | Embryonic development | Genes | Genetic research | Genetic engineering | Cell differentiation | Index Medicus | mouse | vasculogenesis | vein | embryo | artery
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14. Full Text Involvement of Endothelin 1 in Remote Preconditioning-Induced Cardioprotection through connexin 43 and Akt/GSK-3β Signaling Pathway
by Zhang, Min and Gu, Wei Wei and Hong, Xing yu
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 10941 - 12
The present study was aimed to explore the role of endothelins in remote preconditioning (RP)induced myocardial protection in ischemia-reperfusion (IR) injury.... 
GLYCOGEN-SYNTHASE KINASE-3-BETA | ISCHEMIA/REPERFUSION INJURY | GAP-JUNCTIONS | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | MYOCARDIAL-ISCHEMIA | NITRIC-OXIDE | ETA-RECEPTOR | REPERFUSION | EXPRESSION | PROTECTS | Endothelin | Contractility | Endothelin ETB receptors | Caspase | AKT protein | Endothelin 1 | Connexin 43 | Caspase-3 | Muscle contraction | Troponin T | Heart rate | Signal transduction | Troponin | Reperfusion | Ischemia | Triphenyl tetrazolium chloride | Calcium-binding protein | Rodents | Blood pressure | Ventricle | Apoptosis
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