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1. Full Text Andrographolide inhibits hypoxia‐induced hypoxia‐inducible factor 1α and endothelin 1 expression through the heme oxygenase 1/CO/cGMP/MKP‐5 pathways in EA.hy926 cells
by Lin, Hung‐Chih and Su, Shih‐Li and Lin, Wan‐Chun and Lin, Ai‐Hsuan and Yang, Ya‐Chen and Lii, Chong‐Kuei and Chen, Haw‐Wen
Environmental toxicology, ISSN 1520-4081, 03/2018, Volume 33, Issue 3, pp. 269 - 279
endothelin 1 | mitogen‐activated protein kinase phosphatase 5 | hypoxia‐inducible factor 1α | heme oxygenase 1 | andrographolide | hypoxia-inducible factor 1α | mitogen-activated protein kinase phosphatase 5 | Physical Sciences | Toxicology | Life Sciences & Biomedicine | Water Resources | Environmental Sciences & Ecology | Science & Technology | Environmental Sciences | Diterpenes - pharmacology | Cell Line | Cyclic GMP - pharmacology | Heme Oxygenase-1 - metabolism | Anti-Inflammatory Agents - pharmacology | Dual-Specificity Phosphatases - metabolism | Endothelin-1 - metabolism | Humans | Thionucleotides - pharmacology | Carbon Monoxide - metabolism | Cell Hypoxia | Cyclic GMP - analogs & derivatives | Cyclic GMP - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Mitogen-Activated Protein Kinase Phosphatases - metabolism | Index Medicus
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2. Full Text Neural peptidase endothelin-converting enzyme 1 regulates endothelin 1-induced pruritus
by Kido-Nakahara, Makiko and Buddenkotte, Jörg and Kempkes, Cordula and Ikoma, Akihiko and Cevikbas, Ferda and Akiyama, Tasuku and Nunes, Frank and Seeliger, Stephan and Hasdemir, Burcu and Mess, Christian and Buhl, Timo and Sulk, Mathias and Müller, Frank-Ulrich and Metze, Dieter and Bunnett, Nigel W and Bhargava, Aditi and Carstens, Earl and Furue, Masutaka and Steinhoff, Martin
The Journal of clinical investigation, ISSN 0021-9738, 06/2014, Volume 124, Issue 6, pp. 2683 - 2695
Metalloendopeptidases - genetics | Up-Regulation | Skin - metabolism | Humans | Male | Metalloendopeptidases - metabolism | Pruritus - genetics | Aspartic Acid Endopeptidases - genetics | Endothelin-Converting Enzymes | MAP Kinase Signaling System | Adult | Female | Pruritus - etiology | Skin - pathology | Metalloendopeptidases - antagonists & inhibitors | Signal Transduction | Aspartic Acid Endopeptidases - antagonists & inhibitors | Endothelin-1 - genetics | Endothelin-1 - metabolism | Mice, Inbred C57BL | Endothelin-1 - administration & dosage | Mice, Knockout | Pruritus - metabolism | Animals | Aspartic Acid Endopeptidases - metabolism | Skin - innervation | Receptor, Endothelin A - metabolism | Mice | Ganglia, Spinal - metabolism | Physiological aspects | Cellular signal transduction | Pruritus | Proteases | Neurons | Health aspects | Signal transduction | Enzymes | Pain | Nervous system | Neuropeptides | Software | Recycling | Kinases | Laboratory animals | Autoimmune diseases | Age | Index Medicus | Abridged Index Medicus
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3. Full Text Andrographolide inhibits hypoxia‐induced HIF‐1α‐driven endothelin 1 secretion by activating Nrf2/HO‐1 and promoting the expression of prolyl hydroxylases 2/3 in human endothelial cells
by Lin, Hung‐Chih and Su, Shih‐Li and Lu, Chia‐Yang and Lin, Ai‐Hsuan and Lin, Wan‐Chun and Liu, Chin‐San and Yang, Ya‐Chen and Wang, Hsiu‐Miao and Lii, Chong‐Kuei and Chen, Haw‐Wen
Environmental toxicology, ISSN 1520-4081, 03/2017, Volume 32, Issue 3, pp. 918 - 930
heme oxygenase 1 (HO‐1) | prolyl hydroxylases (PHDs) | Nrf2 | endothelin 1 (ET‐1) | hypoxia‐inducible factor 1α (HIF‐1α) | andrographolide | hypoxia-inducible factor 1α (HIF-1α) | endothelin 1 (ET-1) | heme oxygenase 1 (HO-1) | Heme Oxygenase-1 - metabolism | NF-E2-Related Factor 2 - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Prolyl Hydroxylases - metabolism | Humans | RNA, Messenger - metabolism | Cell Hypoxia | Heme Oxygenase-1 - genetics | RNA Interference | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Prolyl Hydroxylases - genetics | NF-E2-Related Factor 2 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Diterpenes - pharmacology | Cell Line | Cell Survival - drug effects | Hydroxylation | Endothelial Cells - metabolism | Endothelin-1 - genetics | Endothelin-1 - metabolism | Heme Oxygenase-1 - antagonists & inhibitors | Cobalt - toxicity | Signal Transduction - drug effects | Endothelial Cells - cytology | NF-E2-Related Factor 2 - metabolism | RNA, Small Interfering - metabolism | Index Medicus
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4. Full Text Endothelin 1 in cancer: Biological implications and therapeutic opportunities
by Rosanò, Laura and Spinella, Francesca and Bagnato, Anna
Nature reviews. Cancer, ISSN 1474-175X, 09/2013, Volume 13, Issue 9, pp. 637 - 651
Life Sciences & Biomedicine | Oncology | Science & Technology | Endothelin-1 - antagonists & inhibitors | Neoplasms - metabolism | Autocrine Communication | Cell Proliferation | Paracrine Communication | Signal Transduction | Cell Survival | Endothelin-1 - metabolism | Humans | Receptors, Endothelin - metabolism | Tumor Microenvironment | Neoplasms - drug therapy | Neovascularization, Pathologic - metabolism | Lymphangiogenesis | Endothelin | Genetic aspects | Cellular signal transduction | Research | Health aspects | Risk factors | Cancer | Index Medicus
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5. Full Text Immediate and Catastrophic Antibody‐Mediated Rejection in a Lung Transplant Recipient With Anti–Angiotensin II Receptor Type 1 and Anti–Endothelin‐1 Receptor Type A Antibodies
by Cozzi, E and Calabrese, F and Schiavon, M and Feltracco, P and Seveso, M and Carollo, C and Loy, M and Cardillo, M and Rea, F
American journal of transplantation, ISSN 1600-6135, 02/2017, Volume 17, Issue 2, pp. 557 - 564
alloantibody | histocompatibility | immunobiology | rejection: antibody‐mediated (ABMR) | clinical research/practice | lung transplantation/pulmonology | rejection: antibody-mediated (ABMR) | Transplantation | Life Sciences & Biomedicine | Surgery | Science & Technology | Prognosis | Humans | HLA Antigens - immunology | Graft Rejection - etiology | Graft Survival | Male | Receptor, Endothelin A - immunology | Lung Transplantation - adverse effects | Transplant Recipients | Cystic Fibrosis - surgery | Isoantibodies - immunology | Adult | Receptor, Angiotensin, Type 1 - immunology | Tissue Donors | Postoperative Complications | Viral antibodies | Transplantation of organs, tissues, etc | Antibodies | Cystic fibrosis | Endothelin | Angiotensin | Transplants & implants | Immunoglobulins
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6. Full Text β-Arrestin 1 is required for endothelin-1-induced NF-κB activation in ovarian cancer cells
by Cianfrocca, Roberta and Tocci, Piera and Semprucci, Elisa and Spinella, Francesca and Di Castro, Valeriana and Bagnato, Anna and Rosanò, Laura
Life sciences (1973), ISSN 0024-3205, 11/2014, Volume 118, Issue 2, pp. 179 - 184
NF-κB | β-Arrestin-1 | Endothelin-1 | Ovarian cancer | Pharmacology & Pharmacy | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Neoplasms, Glandular and Epithelial - pathology | Endothelin-1 - metabolism | Humans | Gene Expression Regulation, Neoplastic | Ovarian Neoplasms - pathology | Neoplasms, Glandular and Epithelial - metabolism | NF-kappa B - metabolism | Receptor, Endothelin A - genetics | Arrestins - metabolism | Ovarian Neoplasms - genetics | Neoplasms, Glandular and Epithelial - genetics | Receptor, Endothelin B - genetics | Models, Biological | beta-Arrestin 1 | beta-Arrestins | Carcinoma, Ovarian Epithelial | Cell Line, Tumor | Female | Receptor, Endothelin A - metabolism | Ovarian Neoplasms - metabolism | Receptor, Endothelin B - metabolism | Genes, Reporter | RNA, Small Interfering - metabolism | Index Medicus
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7. Full Text Endothelin-1 induces hypoxia inducible factor 1α expression in pulmonary artery smooth muscle cells
by Li, Manxiang and Liu, Yuan and Jin, Faguang and Sun, Xiuzhen and Li, Zongfang and Liu, Yun and Fang, Ping and Shi, Hongyang and Jiang, Xingtang
FEBS letters, ISSN 0014-5793, 11/2012, Volume 586, Issue 21, pp. 3888 - 3893
RACK1 | HIF1α | Calcineurin | Endothelin-1 | Proteasome | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Phosphorylation | Receptors, Endothelin - genetics | Cyclosporine - pharmacology | GTP-Binding Proteins - genetics | Protease Inhibitors - pharmacology | Pulmonary Artery - metabolism | Calcineurin - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Receptors for Activated C Kinase | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Endothelin-1 - genetics | Endothelin-1 - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Gene Silencing | Receptors, Endothelin - metabolism | Rats | Signal Transduction - genetics | Pulmonary Artery - drug effects | Rats, Sprague-Dawley | Pulmonary Artery - cytology | Gene Expression Regulation - drug effects | Leupeptins - pharmacology | Animals | Signal Transduction - drug effects | Protein Biosynthesis - drug effects | Primary Cell Culture | Proteasome Endopeptidase Complex - metabolism | Calcineurin - metabolism | GTP-Binding Proteins - metabolism | Index Medicus
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8. Full Text Image mean square displacement to study the lateral mobility of Angiotensin II type 1 and Endothelin 1 type A receptors on living cells
by Planes, Nadir and Vanderheyden, Patrick P.M.L and Gratton, Enrico and Caballero‐George, Catherina
Microscopy research and technique, ISSN 1059-910X, 04/2020, Volume 83, Issue 4, pp. 381 - 392
total internal reflection fluorescence microscopy | Angiotensin II type 1 receptor | image mean square displacement | Endothelin 1 type A receptor | G‐protein‐coupled receptor | diffusion coefficient | Life Sciences & Biomedicine | Microscopy | Technology | Anatomy & Morphology | Biology | Life Sciences & Biomedicine - Other Topics | Science & Technology | Binding | Membranes | Endothelin | Molecular interactions | Fluorescence | Mobility | Reflection | Endothelin 1 | Diffusivity | Proteins | Receptors | Membrane fluidity | Angiotensin AT1 receptors | Cells (biology) | Information processing | Ligands | Qualitative analysis | Receptor mechanisms | Diffusion | Coordination compounds | Angiotensin II | Fluorescence microscopy | Index Medicus
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