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HUMAN MOLECULAR GENETICS, ISSN 0964-6906, 07/2016, Volume 25, Issue 13, pp. 2862 - 2872
Journal Article
Journal Article
Brain, ISSN 0006-8950, 2012, Volume 135, Issue 6, pp. 1964 - 1980
Ambivalent effects of interleukin-6 on the pathogenesis of ischaemic stroke have been reported. However, to date, the long-term actions of interleukin-6 after... 
cerebral ischaemia | inflammation | regeneration | angiogenesis | interleukin-6 | FOCAL CEREBRAL-ISCHEMIA | NEUROSCIENCES | CLINICAL NEUROLOGY | ENDOTHELIAL PROGENITOR CELLS | IN-VITRO | BRAIN ISCHEMIA | NITRIC-OXIDE | GENE-EXPRESSION | PHYSICAL-ACTIVITY | RAT-BRAIN | GROWTH-FACTOR | INFLAMMATORY CYTOKINES | Angiogenic Proteins - genetics | Infarction, Middle Cerebral Artery - physiopathology | Oligonucleotide Array Sequence Analysis | Cytokine Receptor gp130 - genetics | Embryo, Mammalian | Gene Expression Profiling | Green Fluorescent Proteins - genetics | Receptor, trkB - genetics | Bone Marrow Transplantation - methods | Infarction, Middle Cerebral Artery - complications | Perfusion Imaging | Cytokines - genetics | Interleukin-6 - metabolism | STAT3 Transcription Factor - genetics | Disease Models, Animal | Cytokine Receptor gp130 - metabolism | Interleukin-6 - genetics | Endothelin-1 - genetics | Hypoxia - complications | Signal Transduction - genetics | Analysis of Variance | Brain - pathology | Mice | Neovascularization, Pathologic - metabolism | Endothelial Cells - pathology | Angiogenic Proteins - metabolism | Gait Disorders, Neurologic - etiology | Mice, Knockout - genetics | Cerebral Cortex - cytology | Neovascularization, Pathologic - etiology | Tetrazolium Salts | Thiazoles | Microfilament Proteins - metabolism | Neurons - drug effects | STAT3 Transcription Factor - metabolism | Calcium-Binding Proteins - metabolism | Green Fluorescent Proteins - metabolism | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Endothelin-1 - metabolism | Gene Expression Regulation - genetics | Cells, Cultured | Neuroglia - physiology | Infarction, Middle Cerebral Artery - pathology | Glucose - deficiency | Rotarod Performance Test | Animals | Infarction, Middle Cerebral Artery - surgery | Signal Transduction - physiology | Receptor, trkB - metabolism | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e85318
Hepatocarcinogenesis commonly involves the gradual progression from hepatitis to fibrosis and cirrhosis, and ultimately to hepatocellular carcinoma (HCC).... 
TRANSGENIC ZEBRAFISH | IN-VITRO | B-VIRUS X | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | LIVER-SPECIFIC EXPRESSION | GROWTH-FACTOR | HUMAN HEPATOCELLULAR-CARCINOMA | HEPATITIS-B | MYC EXPRESSION | Endothelin-1 - antagonists & inhibitors | Cell Proliferation | Fatty Liver - pathology | Humans | Fatty Liver - complications | MicroRNAs - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - genetics | Liver Neoplasms - etiology | Carcinoma, Hepatocellular - genetics | HEK293 Cells | Liver Cirrhosis - metabolism | Liver Neoplasms - pathology | Carcinoma, Hepatocellular - etiology | Proto-Oncogene Proteins c-akt - metabolism | Liver Cirrhosis - genetics | Fatty Liver - genetics | Gene Expression | Fatty Liver - metabolism | Liver Neoplasms - genetics | Unfolded Protein Response - genetics | Endothelin-1 - genetics | Endothelin-1 - metabolism | Liver Cirrhosis - complications | Zebrafish Proteins - metabolism | Zebrafish Proteins - antagonists & inhibitors | Zebrafish | Phosphatidylinositol 3-Kinases - genetics | Animals | Cell Cycle | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Liver Cirrhosis - pathology | MicroRNAs - genetics | Protein Kinase Inhibitors - pharmacology | Zebrafish Proteins - genetics | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Carcinoma, Hepatocellular - metabolism | Cell Movement | Genetically modified animals | Animal genetic engineering | Endothelin | T cells | Glycogen | Liver cirrhosis | Cell proliferation | Cell culture | Liver | Genes | Xenotransplantation | Hyperplasia | Hepatocellular carcinoma | AKT protein | Transgenic fish | Hepatitis | Liver cancer | Cell growth | Protein folding | Rodents | Xenografts | Cell cycle | Endothelin 1 | Gene expression | Ribonucleic acid--RNA | Bile duct | 1-Phosphatidylinositol 3-kinase | Steatosis | Cirrhosis | Inhibitors | Hepatocytes | Fibrosis | Cell migration | RNA | Ribonucleic acid
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, p. e0166433
Background Alternatively activated (M2) macrophages are phenotypically characterized by the expression of specific markers, mainly macrophage scavenger... 
EPITHELIAL-MESENCHYMAL TRANSITION | CANCER-CELLS | FIBROSIS | MULTIDISCIPLINARY SCIENCES | DISEASE | TISSUE-REPAIR | TUMOR-ASSOCIATED MACROPHAGES | SKIN | SYSTEMIC-SCLEROSIS | POLARIZATION | PLASTICITY | Antigens, CD - immunology | Tetradecanoylphorbol Acetate - pharmacology | Monocytes - cytology | Endothelin-1 - pharmacology | Humans | Lectins, C-Type - immunology | Matrix Metalloproteinase 9 - immunology | Monocytes - immunology | Receptor, Endothelin A - immunology | Lectins, C-Type - genetics | Antigens, CD - genetics | Transforming Growth Factor beta1 - immunology | Interleukin-4 - pharmacology | Chemokine CCL22 - immunology | Matrix Metalloproteinase 9 - genetics | Scavenger Receptors, Class A - genetics | Scavenger Receptors, Class A - immunology | Antigens, Differentiation, Myelomonocytic - immunology | Macrophages - immunology | Cell Line | Gene Expression Regulation | Transforming Growth Factor beta1 - genetics | Macrophages - cytology | Receptor, Endothelin A - genetics | Sulfonamides - pharmacology | Receptors, Cell Surface - immunology | Antigens, Differentiation, Myelomonocytic - genetics | Monocytes - drug effects | Chemokine CCL22 - genetics | Endothelin Receptor Antagonists - pharmacology | Phenotype | Mannose-Binding Lectins - genetics | Mannose-Binding Lectins - immunology | Cell Differentiation - drug effects | Interleukin-10 - genetics | Macrophages - drug effects | Primary Cell Culture | Macrophage Activation - drug effects | Interleukin-10 - immunology | Receptors, Cell Surface - genetics | Type 2 diabetes | Endothelin | Systemic scleroderma | Interleukins | Genes | Scleroderma (Disease) | Genetic aspects | Macrophages | Transforming growth factors | Gene expression | Sugars | Monosaccharides | Laboratories | Immunocytochemistry | Pathogenesis | Interleukin | Mannose | Cancer therapies | Western blotting | Genotype & phenotype | Receptors | Interleukin 4 | Peripheral blood | Aging | Scavenger receptors | Metalloproteinase | Diabetes mellitus (non-insulin dependent) | Growth factors | Transforming growth factor-b1 | Cytokines | Internal medicine | Diabetes mellitus | Markers | Rheumatology | 12-O-Tetradecanoylphorbol-13-acetate | Cultures | Endothelin 1 | Gelatinase B | Polymerase chain reaction | Medicine | Studies | CD163 antigen | Monocytes | Systemic sclerosis | Interleukin 10 | Fibrosis | Acetic acid
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, p. e0166984
Birth defects are among the leading causes of infant mortality and contribute substantially to illness and long-term disability. Defects in Bone Morphogenetic... 
BRANCHIAL ARCH | DLX GENES | HEAD SKELETON | ALAGILLE-SYNDROME | DOWNS-SYNDROME | MULTIDISCIPLINARY SCIENCES | AURICULOCONDYLAR SYNDROME | LEFT-RIGHT ASYMMETRY | REPEAT PROTEIN | HUMAN JAGGED1 | NEURAL CREST CELLS | Jagged-1 Protein - metabolism | Zebrafish - embryology | Intercellular Signaling Peptides and Proteins - metabolism | Somites - embryology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Nuclear Proteins - biosynthesis | Smad5 Protein - metabolism | Smad1 Protein - genetics | Smad5 Protein - genetics | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Gene Expression Regulation, Developmental - physiology | Zebrafish Proteins - biosynthesis | Basic Helix-Loop-Helix Transcription Factors - genetics | Endothelin-1 - genetics | Endothelin-1 - metabolism | Zebrafish Proteins - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Repressor Proteins - genetics | Jagged-1 Protein - genetics | Zebrafish - genetics | Body Patterning - physiology | Animals | Transforming Growth Factor beta - genetics | Facial Bones - enzymology | Smad1 Protein - metabolism | Signal Transduction - physiology | Zebrafish Proteins - genetics | Transforming Growth Factor beta - metabolism | Bone morphogenetic proteins | Genetic aspects | Research | Craniofacial abnormalities | Neural crest | Pattern formation | Jaw | Transcription factors | Endothelin | Arches | Congenital defects | Alagille syndrome | Genomics | Transforming growth factor-b | Genomes | Somites | Kinases | Defects | Proteins | Cartilage | Signal transduction | Pathways | Growth factors | Territory | Patterning | Mandible | Interference | Craniofacial syndromes | Zebrafish | Infant mortality | Birth defects | Endothelin 1 | Gene expression | Embryos | Mutants | Cleft lip/palate | Craniofacial growth | Signaling | Scaffolding | In vivo methods and tests | Notch protein
Journal Article
European Journal of Oral Sciences, ISSN 0909-8836, 06/2010, Volume 118, Issue 3, pp. 213 - 220
Jagomägi T, Nikopensius T, Krjutškov K, Tammekivi V, Viltrop T, Saag M, Metspalu A. MTHFR and MSX1 contribute to the risk of nonsyndromic cleft lip/palate. Eur... 
Arrayed Primer Extension method (APEX) | nonsyndromic cleft lip with or without cleft palate | single nucleotide polymorphism (SNP) | case–control association study | genotyping | Case-control association study | Genotyping | Nonsyndromic cleft lip with or without cleft palate | Single nucleotide polymorphism (SNP) | case-control association study | CHILEAN POPULATION | ORAL CLEFTS | CANDIDATE GENES | LOCI | PALATE | LIP | LINKAGE DISEQUILIBRIUM | POLYMORPHISMS | DENTISTRY, ORAL SURGERY & MEDICINE | IRF6 | ASSOCIATION | Cell Adhesion Molecules - genetics | Gene Frequency - genetics | Humans | Cleft Palate - genetics | Immunoglobulins - genetics | Case-Control Studies | Guanine | Nectins | Haplotypes - genetics | Methylenetetrahydrofolate Reductase (NADPH2) - genetics | MSX1 Transcription Factor - genetics | Cleft Palate - etiology | Jagged-2 Protein | Cytosine | Endothelin-1 - genetics | Membrane Proteins - genetics | Risk Factors | Intercellular Signaling Peptides and Proteins - genetics | Thymine | Adenine | Interferon Regulatory Factors - genetics | Proto-Oncogene Proteins - genetics | Chromosome Mapping | Epistasis, Genetic - genetics | Transcription Factors - genetics | Cleft Lip - genetics | Polymorphism, Single Nucleotide - genetics | Cleft Lip - etiology | Estonia | Anopheles | Cleft lip | Universities and colleges
Journal Article
American Journal of Obstetrics and Gynecology, ISSN 0002-9378, 2010, Volume 203, Issue 4, pp. 361.e1 - 361.e30
Objective We sought to determine whether maternal/fetal single-nucleotide polymorphisms (SNPs) in candidate genes are associated with preterm prelabor rupture... 
Obstetrics and Gynecology | DNA variants | haplotype | high dimensional biology | prematurity | preterm prelabor rupture of membranes | chorioamnionitis | parturition | genetic association study | genotype | genomics | matrix metalloproteinase | single-nucleotide polymorphism | extracellular matrix | INTERLEUKIN-1 RECEPTOR ANTAGONIST | FACTOR-ALPHA GENE | THAN-G POLYMORPHISM | MIDTRIMESTER AMNIOTIC-FLUID | OBSTETRICS & GYNECOLOGY | FALSE DISCOVERY RATE | TUMOR-NECROSIS-FACTOR | PREMATURE-RUPTURE | SINGLE NUCLEOTIDE POLYMORPHISM | HUMAN UTERINE CERVIX | EHLERS-DANLOS-SYNDROME | Haplotypes | Humans | Receptors, Corticotropin-Releasing Hormone - genetics | Male | Autoantigens - genetics | Mothers | Receptors, Prostaglandin E - genetics | Case-Control Studies | Tissue Inhibitor of Metalloproteinase-2 - genetics | alpha-Defensins - genetics | Fetal Membranes, Premature Rupture - genetics | Adult | Female | Fetus | Collagen - genetics | Infant, Newborn | Procollagen | Genetic Association Studies | Endothelin-1 - genetics | Gene Frequency | Collagen Type I | Genotype | Sequence Analysis, DNA | Pregnancy | Chorioamnionitis - pathology | Protein Isoforms | Collagen Type IV - genetics | Models, Genetic | Polymorphism, Single Nucleotide | Receptors, Prostaglandin E, EP1 Subtype | Genetic research | Physiological aspects | Genomics | DNA | Public health | Chorioamnionitis | MMP | SNP | pPROM
Journal Article
Nature Communications, ISSN 2041-1723, 04/2015, Volume 6, Issue 1, p. 6853
Journal Article