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1. Full Text A multi-ethnic genome-wide association study identifies novel loci for non-syndromic cleft lip with or without cleft palate on 2p24.2, 17q23 and 19q13
by Leslie, EJ and Carlson, JC and Shaffer, JR and Feingold, E and Wehby, G and Laurie, CA and Jain, D and Laurie, CC and Doheny, KF and McHenry, T and Resick, J and Sanchez, C and Jacobs, J and Emanuele, B and Vieira, AR and Neiswanger, K and Lidral, AC and Valencia-Ramirez, LC and Lopez-Palacio, AM and Valencia, DR and Arcos-Burgos, M and Czeizel, AE and Field, LL and Padilla, CD and Maria, E and Cutiongco-de la Paz, C and Deleyiannis, F and Christensen, K and Munger, RG and Lie, RT and Wilcox, A and Romitti, PA and Castilla, EE and Mereb, JC and Poletta, FA and Orioli, IM and Carvalho, FM and Hecht, JT and Blanton, SH and Buxo, CJ and Butali, A and Mossey, PA and Adeyemo, WL and James, O and Braimah, RO and Aregbesola, BS and Eshete, MA and Abate, F and Koruyucu, M and Seymen, F and Ma, L and de Salamanca, JE and Weinberg, SM and Moreno, L and Murray, JC and Marazita, ML
HUMAN MOLECULAR GENETICS, ISSN 0964-6906, 07/2016, Volume 25, Issue 13, pp. 2862 - 2872
Orofacial clefts (OFCs), which include non-syndromic cleft lip with or without cleft palate (CL/P), are among the most common birth defects in humans,... 
POPULATION | GENE | VARIANTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | CAUSE VAN | RISK | SUSCEPTIBILITY LOCUS | MUTATIONS | ENDOTHELIN-1 | EXPRESSION | CANCER | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Humans | Risk Factors | Asian Continental Ancestry Group - genetics | Male | Cleft Palate - genetics | Genetic Loci | Chromosomes, Human, Pair 17 - genetics | Chromosomes, Human, Pair 19 - genetics | Chromosomes, Human, Pair 2 - genetics | Cleft Lip - genetics | Ethnic Groups | Polymorphism, Single Nucleotide - genetics | Female | African Continental Ancestry Group - genetics | Association Studies
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2. Full Text Endothelin promotes colorectal tumorigenesis by activating YAP/TAZ
by Wang, Zhen and Liu, Peng and Zhou, Xin and Wang, Tianxiang and Feng, Xu and Sun, Yi-Ping and Xiong, Yue and Yuan, Hai-Xin and Guan, Kun-Liang
Cancer Research, ISSN 0008-5472, 05/2017, Volume 77, Issue 9, pp. 2413 - 2423
Endothelin receptor A (ETAR) promotes tumorigenesis by stimulating cell proliferation, migration, and survival. However, the mechanism of ETAR in promoting... 
BREAST-CANCER | CONTROLLED-TRIAL | TISSUE GROWTH-FACTOR | RECEPTOR ANTAGONISTS | PULMONARY-HYPERTENSION | ONCOLOGY | ORGAN SIZE CONTROL | HIPPO PATHWAY | FACTOR EXPRESSION | RESISTANT PROSTATE-CANCER | ZIBOTENTAN ZD4054 | Cell Proliferation - genetics | Colonic Neoplasms - genetics | Endothelin-1 - genetics | rho GTP-Binding Proteins - genetics | HCT116 Cells | Humans | Carcinogenesis - genetics | Endothelin-1 - administration & dosage | Gene Expression Regulation, Neoplastic | GTP-Binding Protein alpha Subunits - genetics | Phosphoproteins - genetics | Receptor, Endothelin A - genetics | Cell Movement - genetics | Colonic Neoplasms - pathology | Adaptor Proteins, Signal Transducing - genetics | Intracellular Signaling Peptides and Proteins - genetics | Cell proliferation | Endothelin | Cell survival | Activation | Endothelin 1 | Dephosphorylation | Yes-associated protein | Signal transduction | Signaling | Cell growth | Colon cancer | Pathways | Transcription activation | Tumor suppressor genes | Tumorigenesis | Colon | Cell migration | Cancer | Guanosinetriphosphatase
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3. Full Text Essential role of interleukin-6 in post-stroke angiogenesis
by Gertz, Karen and Kronenberg, Golo and Kälin, Roland E and Baldinger, Tina and Werner, Christian and Balkaya, Mustafa and Eom, Gina D and Hellmann-Regen, Julian and Kröber, Jan and Miller, Kelly R and Lindauer, Ute and Laufs, Ulrich and Dirnagl, Ulrich and Heppner, Frank L and Endres, Matthias
Brain, ISSN 0006-8950, 2012, Volume 135, Issue 6, pp. 1964 - 1980
Ambivalent effects of interleukin-6 on the pathogenesis of ischaemic stroke have been reported. However, to date, the long-term actions of interleukin-6 after... 
cerebral ischaemia | inflammation | regeneration | angiogenesis | interleukin-6 | FOCAL CEREBRAL-ISCHEMIA | NEUROSCIENCES | CLINICAL NEUROLOGY | ENDOTHELIAL PROGENITOR CELLS | IN-VITRO | BRAIN ISCHEMIA | NITRIC-OXIDE | GENE-EXPRESSION | PHYSICAL-ACTIVITY | RAT-BRAIN | GROWTH-FACTOR | INFLAMMATORY CYTOKINES | Angiogenic Proteins - genetics | Infarction, Middle Cerebral Artery - physiopathology | Oligonucleotide Array Sequence Analysis | Cytokine Receptor gp130 - genetics | Embryo, Mammalian | Gene Expression Profiling | Green Fluorescent Proteins - genetics | Receptor, trkB - genetics | Bone Marrow Transplantation - methods | Infarction, Middle Cerebral Artery - complications | Perfusion Imaging | Cytokines - genetics | Interleukin-6 - metabolism | STAT3 Transcription Factor - genetics | Disease Models, Animal | Cytokine Receptor gp130 - metabolism | Interleukin-6 - genetics | Endothelin-1 - genetics | Hypoxia - complications | Signal Transduction - genetics | Analysis of Variance | Brain - pathology | Mice | Neovascularization, Pathologic - metabolism | Endothelial Cells - pathology | Angiogenic Proteins - metabolism | Gait Disorders, Neurologic - etiology | Mice, Knockout - genetics | Cerebral Cortex - cytology | Neovascularization, Pathologic - etiology | Tetrazolium Salts | Thiazoles | Microfilament Proteins - metabolism | Neurons - drug effects | STAT3 Transcription Factor - metabolism | Calcium-Binding Proteins - metabolism | Green Fluorescent Proteins - metabolism | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Endothelin-1 - metabolism | Gene Expression Regulation - genetics | Cells, Cultured | Neuroglia - physiology | Infarction, Middle Cerebral Artery - pathology | Glucose - deficiency | Rotarod Performance Test | Animals | Infarction, Middle Cerebral Artery - surgery | Signal Transduction - physiology | Receptor, trkB - metabolism | Original
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4. Full Text Overexpression of endothelin 1 triggers hepatocarcinogenesis in zebrafish and promotes cell proliferation and migration through the AKT pathway
by Lu, Jeng-Wei and Liao, Chung-Yi and Yang, Wan-Yu and Lin, Yueh-Min and Jin, Shiow-Lian Catherine and Wang, Horng-Dar and Yuh, Chiou-Hwa
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e85318
Hepatocarcinogenesis commonly involves the gradual progression from hepatitis to fibrosis and cirrhosis, and ultimately to hepatocellular carcinoma (HCC).... 
TRANSGENIC ZEBRAFISH | IN-VITRO | B-VIRUS X | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | LIVER-SPECIFIC EXPRESSION | GROWTH-FACTOR | HUMAN HEPATOCELLULAR-CARCINOMA | HEPATITIS-B | MYC EXPRESSION | Endothelin-1 - antagonists & inhibitors | Cell Proliferation | Fatty Liver - pathology | Humans | Fatty Liver - complications | MicroRNAs - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - genetics | Liver Neoplasms - etiology | Carcinoma, Hepatocellular - genetics | HEK293 Cells | Liver Cirrhosis - metabolism | Liver Neoplasms - pathology | Carcinoma, Hepatocellular - etiology | Proto-Oncogene Proteins c-akt - metabolism | Liver Cirrhosis - genetics | Fatty Liver - genetics | Gene Expression | Fatty Liver - metabolism | Liver Neoplasms - genetics | Unfolded Protein Response - genetics | Endothelin-1 - genetics | Endothelin-1 - metabolism | Liver Cirrhosis - complications | Zebrafish Proteins - metabolism | Zebrafish Proteins - antagonists & inhibitors | Zebrafish | Phosphatidylinositol 3-Kinases - genetics | Animals | Cell Cycle | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Liver Cirrhosis - pathology | MicroRNAs - genetics | Protein Kinase Inhibitors - pharmacology | Zebrafish Proteins - genetics | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Carcinoma, Hepatocellular - metabolism | Cell Movement | Genetically modified animals | Animal genetic engineering | Endothelin | T cells | Glycogen | Liver cirrhosis | Cell proliferation | Cell culture | Liver | Genes | Xenotransplantation | Hyperplasia | Hepatocellular carcinoma | AKT protein | Transgenic fish | Hepatitis | Liver cancer | Cell growth | Protein folding | Rodents | Xenografts | Cell cycle | Endothelin 1 | Gene expression | Ribonucleic acid--RNA | Bile duct | 1-Phosphatidylinositol 3-kinase | Steatosis | Cirrhosis | Inhibitors | Hepatocytes | Fibrosis | Cell migration | RNA | Ribonucleic acid
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5. Full Text Prioritizing individual genetic variants after kernel machine testing using variable selection
by He, Qianchuan and Cai, Tianxi and Liu, Yang and Zhao, Ni and Harmon, Quaker E and Almli, Lynn M and Binder, Elisabeth B and Engel, Stephanie M and Ressler, Kerry J and Conneely, Karen N and Lin, Xihong and Wu, Michael C
Genetic Epidemiology, ISSN 0741-0395, 12/2016, Volume 40, Issue 8, pp. 722 - 731
ABSTRACT Kernel machine learning methods, such as the SNP‐set kernel association test (SKAT), have been widely used to test associations between traits and... 
KNIFE | genetic association studies | variable selection | kernel machine methods | set‐based | set-based | REGRESSION | RARE | CANCER | GENETICS & HEREDITY | MATHEMATICAL & COMPUTATIONAL BIOLOGY | OUTCOMES | REGULARIZATION | STRESS | EXPRESSION | GENOME-WIDE ASSOCIATION | Data Interpretation, Statistical | Phenotype | Wounds and Injuries - genetics | Genetic Association Studies | Methylenetetrahydrofolate Reductase (NADPH2) - genetics | Endothelin-1 - genetics | Genetic Markers - genetics | Humans | Polymorphism, Single Nucleotide - genetics | Models, Genetic | Birth Weight - genetics | Selection, Genetic - genetics | Genetic research | Single nucleotide polymorphisms | Analysis | Machine learning | Genotype & phenotype | Polymorphism | Genetic association studies | Kernel machine methods | Set-based | Variable selection
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6. Full Text Alternatively activated (M2) macrophage phenotype is inducible by endothelin-1 in cultured human macrophages
by Soldano, Stefano and Pizzorni, Carmen and Paolino, Sabrina and Trombetta, Amelia Chiara and Montagna, Paola and Brizzolara, Renata and Ruaro, Barbara and Sulli, Alberto and Cutolo, Maurizio
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, p. e0166433
Background Alternatively activated (M2) macrophages are phenotypically characterized by the expression of specific markers, mainly macrophage scavenger... 
EPITHELIAL-MESENCHYMAL TRANSITION | CANCER-CELLS | FIBROSIS | MULTIDISCIPLINARY SCIENCES | DISEASE | TISSUE-REPAIR | TUMOR-ASSOCIATED MACROPHAGES | SKIN | SYSTEMIC-SCLEROSIS | POLARIZATION | PLASTICITY | Antigens, CD - immunology | Tetradecanoylphorbol Acetate - pharmacology | Monocytes - cytology | Endothelin-1 - pharmacology | Humans | Lectins, C-Type - immunology | Matrix Metalloproteinase 9 - immunology | Monocytes - immunology | Receptor, Endothelin A - immunology | Lectins, C-Type - genetics | Antigens, CD - genetics | Transforming Growth Factor beta1 - immunology | Interleukin-4 - pharmacology | Chemokine CCL22 - immunology | Matrix Metalloproteinase 9 - genetics | Scavenger Receptors, Class A - genetics | Scavenger Receptors, Class A - immunology | Antigens, Differentiation, Myelomonocytic - immunology | Macrophages - immunology | Cell Line | Gene Expression Regulation | Transforming Growth Factor beta1 - genetics | Macrophages - cytology | Receptor, Endothelin A - genetics | Sulfonamides - pharmacology | Receptors, Cell Surface - immunology | Antigens, Differentiation, Myelomonocytic - genetics | Monocytes - drug effects | Chemokine CCL22 - genetics | Endothelin Receptor Antagonists - pharmacology | Phenotype | Mannose-Binding Lectins - genetics | Mannose-Binding Lectins - immunology | Cell Differentiation - drug effects | Interleukin-10 - genetics | Macrophages - drug effects | Primary Cell Culture | Macrophage Activation - drug effects | Interleukin-10 - immunology | Receptors, Cell Surface - genetics | Type 2 diabetes | Endothelin | Systemic scleroderma | Interleukins | Genes | Scleroderma (Disease) | Genetic aspects | Macrophages | Transforming growth factors | Gene expression | Sugars | Monosaccharides | Laboratories | Immunocytochemistry | Pathogenesis | Interleukin | Mannose | Cancer therapies | Western blotting | Genotype & phenotype | Receptors | Interleukin 4 | Peripheral blood | Aging | Scavenger receptors | Metalloproteinase | Diabetes mellitus (non-insulin dependent) | Growth factors | Transforming growth factor-b1 | Cytokines | Internal medicine | Diabetes mellitus | Markers | Rheumatology | 12-O-Tetradecanoylphorbol-13-acetate | Cultures | Endothelin 1 | Gelatinase B | Polymerase chain reaction | Medicine | Studies | CD163 antigen | Monocytes | Systemic sclerosis | Interleukin 10 | Fibrosis | Acetic acid
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7. Full Text Wdr68 mediates dorsal and ventral patterning events for craniofacial development
by Alvarado, Estibaliz and Yousefelahiyeh, Mina and Alvarado, Greg and Shang, Robin and Whitman, Taryn and Martinez, Andrew and Yu, Yang and Pham, Annie and Bhandari, Anish and Wang, Bingyan and Nissen, Robert M
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, p. e0166984
Birth defects are among the leading causes of infant mortality and contribute substantially to illness and long-term disability. Defects in Bone Morphogenetic... 
BRANCHIAL ARCH | DLX GENES | HEAD SKELETON | ALAGILLE-SYNDROME | DOWNS-SYNDROME | MULTIDISCIPLINARY SCIENCES | AURICULOCONDYLAR SYNDROME | LEFT-RIGHT ASYMMETRY | REPEAT PROTEIN | HUMAN JAGGED1 | NEURAL CREST CELLS | Jagged-1 Protein - metabolism | Zebrafish - embryology | Intercellular Signaling Peptides and Proteins - metabolism | Somites - embryology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Nuclear Proteins - biosynthesis | Smad5 Protein - metabolism | Smad1 Protein - genetics | Smad5 Protein - genetics | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Gene Expression Regulation, Developmental - physiology | Zebrafish Proteins - biosynthesis | Basic Helix-Loop-Helix Transcription Factors - genetics | Endothelin-1 - genetics | Endothelin-1 - metabolism | Zebrafish Proteins - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Repressor Proteins - genetics | Jagged-1 Protein - genetics | Zebrafish - genetics | Body Patterning - physiology | Animals | Transforming Growth Factor beta - genetics | Facial Bones - enzymology | Smad1 Protein - metabolism | Signal Transduction - physiology | Zebrafish Proteins - genetics | Transforming Growth Factor beta - metabolism | Bone morphogenetic proteins | Genetic aspects | Research | Craniofacial abnormalities | Neural crest | Pattern formation | Jaw | Transcription factors | Endothelin | Arches | Congenital defects | Alagille syndrome | Genomics | Transforming growth factor-b | Genomes | Somites | Kinases | Defects | Proteins | Cartilage | Signal transduction | Pathways | Growth factors | Territory | Patterning | Mandible | Interference | Craniofacial syndromes | Zebrafish | Infant mortality | Birth defects | Endothelin 1 | Gene expression | Embryos | Mutants | Cleft lip/palate | Craniofacial growth | Signaling | Scaffolding | In vivo methods and tests | Notch protein
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8. Full Text MTHFR and MSX1 contribute to the risk of nonsyndromic cleft lip/palate
by Jagomägi, Triin and Nikopensius, Tiit and Krjutškov, Kaarel and Tammekivi, Veronika and Viltrop, Triin and Saag, Mare and Metspalu, Andres
European Journal of Oral Sciences, ISSN 0909-8836, 06/2010, Volume 118, Issue 3, pp. 213 - 220
Jagomägi T, Nikopensius T, Krjutškov K, Tammekivi V, Viltrop T, Saag M, Metspalu A. MTHFR and MSX1 contribute to the risk of nonsyndromic cleft lip/palate. Eur... 
Arrayed Primer Extension method (APEX) | nonsyndromic cleft lip with or without cleft palate | single nucleotide polymorphism (SNP) | case–control association study | genotyping | Case-control association study | Genotyping | Nonsyndromic cleft lip with or without cleft palate | Single nucleotide polymorphism (SNP) | case-control association study | CHILEAN POPULATION | ORAL CLEFTS | CANDIDATE GENES | LOCI | PALATE | LIP | LINKAGE DISEQUILIBRIUM | POLYMORPHISMS | DENTISTRY, ORAL SURGERY & MEDICINE | IRF6 | ASSOCIATION | Cell Adhesion Molecules - genetics | Gene Frequency - genetics | Humans | Cleft Palate - genetics | Immunoglobulins - genetics | Case-Control Studies | Guanine | Nectins | Haplotypes - genetics | Methylenetetrahydrofolate Reductase (NADPH2) - genetics | MSX1 Transcription Factor - genetics | Cleft Palate - etiology | Jagged-2 Protein | Cytosine | Endothelin-1 - genetics | Membrane Proteins - genetics | Risk Factors | Intercellular Signaling Peptides and Proteins - genetics | Thymine | Adenine | Interferon Regulatory Factors - genetics | Proto-Oncogene Proteins - genetics | Chromosome Mapping | Epistasis, Genetic - genetics | Transcription Factors - genetics | Cleft Lip - genetics | Polymorphism, Single Nucleotide - genetics | Cleft Lip - etiology | Estonia | Anopheles | Cleft lip | Universities and colleges
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9. Full Text A genetic association study of maternal and fetal candidate genes that predispose to preterm prelabor rupture of membranes (PROM)
by Romero, Roberto, MD and Friel, Lara A., MD, PhD and Velez Edwards, Digna R., PhD and Kusanovic, Juan Pedro, MD and Hassan, Sonia S., MD and Mazaki-Tovi, Shali, MD and Vaisbuch, Edi, MD and Kim, Chong Jai, MD and Erez, Offer, MD and Chaiworapongsa, Tinnakorn, MD and Pearce, Brad D., PhD and Bartlett, Jacquelaine, MS and Salisbury, Benjamin A., PhD and Anant, Madan Kumar, PhD and Vovis, Gerald F., PhD and Lee, Min Seob, PhD and Gomez, Ricardo, MD and Behnke, Ernesto, MD and Oyarzun, Enrique, MD and Tromp, Gerard, PhD and Williams, Scott M., PhD and Menon, Ramkumar, PhD
American Journal of Obstetrics and Gynecology, ISSN 0002-9378, 2010, Volume 203, Issue 4, pp. 361.e1 - 361.e30
Objective We sought to determine whether maternal/fetal single-nucleotide polymorphisms (SNPs) in candidate genes are associated with preterm prelabor rupture... 
Obstetrics and Gynecology | DNA variants | haplotype | high dimensional biology | prematurity | preterm prelabor rupture of membranes | chorioamnionitis | parturition | genetic association study | genotype | genomics | matrix metalloproteinase | single-nucleotide polymorphism | extracellular matrix | INTERLEUKIN-1 RECEPTOR ANTAGONIST | FACTOR-ALPHA GENE | THAN-G POLYMORPHISM | MIDTRIMESTER AMNIOTIC-FLUID | OBSTETRICS & GYNECOLOGY | FALSE DISCOVERY RATE | TUMOR-NECROSIS-FACTOR | PREMATURE-RUPTURE | SINGLE NUCLEOTIDE POLYMORPHISM | HUMAN UTERINE CERVIX | EHLERS-DANLOS-SYNDROME | Haplotypes | Humans | Receptors, Corticotropin-Releasing Hormone - genetics | Male | Autoantigens - genetics | Mothers | Receptors, Prostaglandin E - genetics | Case-Control Studies | Tissue Inhibitor of Metalloproteinase-2 - genetics | alpha-Defensins - genetics | Fetal Membranes, Premature Rupture - genetics | Adult | Female | Fetus | Collagen - genetics | Infant, Newborn | Procollagen | Genetic Association Studies | Endothelin-1 - genetics | Gene Frequency | Collagen Type I | Genotype | Sequence Analysis, DNA | Pregnancy | Chorioamnionitis - pathology | Protein Isoforms | Collagen Type IV - genetics | Models, Genetic | Polymorphism, Single Nucleotide | Receptors, Prostaglandin E, EP1 Subtype | Genetic research | Physiological aspects | Genomics | DNA | Public health | Chorioamnionitis | MMP | SNP | pPROM
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10. Full Text Developmental genetic bases behind the independent origin of the tympanic membrane in mammals and diapsids
by Kitazawa, Taro and Takechi, Masaki and Hirasawa, Tatsuya and Adachi, Noritaka and Narboux-Nême, Nicolas and Kume, Hideaki and Maeda, Kazuhiro and Hirai, Tamami and Miyagawa-Tomita, Sachiko and Kurihara, Yukiko and Hitomi, Jiro and Levi, Giovanni and Kuratani, Shigeru and Kurihara, Hiroki
Nature Communications, ISSN 2041-1723, 04/2015, Volume 6, Issue 1, p. 6853
The amniote middle ear is a classical example of the evolutionary novelty. Although paleontological evidence supports the view that mammals and diapsids... 
INACTIVATION | JAW | A RECEPTOR | MULTIDISCIPLINARY SCIENCES | TETRAPOD EAR | DEFICIENT | MICE | ENDOTHELIN-1 | MIDDLE-EAR EVOLUTION | EXPRESSION | MOLECULAR-MECHANISMS | Paired Box Transcription Factors - genetics | Ambystoma mexicanum - genetics | Embryo, Mammalian | Homeodomain Proteins - metabolism | Lizards - genetics | Molecular Sequence Data | Goosecoid Protein - genetics | Mice - embryology | Goosecoid Protein - metabolism | Mandible - embryology | Sharks - embryology | Gene Expression Regulation, Developmental | Base Sequence | Tympanic Membrane - embryology | Sharks - genetics | Mice - genetics | Endothelin-1 - genetics | Endothelin-1 - metabolism | Maxilla - embryology | Transcription Factors - genetics | Receptor, Endothelin A - genetics | Ambystoma mexicanum - embryology | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Animals | Lizards - embryology | Embryo, Nonmammalian | Receptor, Endothelin A - metabolism | Paired Box Transcription Factors - metabolism | Evolution, Molecular | Life Sciences | Morphogenesis | Neurons and Cognition | Neurobiology | Development Biology | Sexual reproduction | Reproductive Biology | Embryology and Organogenesis
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