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Journal Article
Experimental Eye Research, ISSN 0014-4835, 11/2018, Volume 176, pp. 1 - 9
Hypertension is an independent risk factor for diabetic retinopathy, yet anti-hypertensive medications such as blockade of angiotensin II do not completely... 
Hypertension | Diabetic retinopathy | Endothelin | VEGF | Diabetes | Vascular leakage | Retinal pigment epithelium | CELLS | ACTIVATION | ANGIOGENESIS | PREVALENCE | MODEL | DISEASE | OPHTHALMOLOGY | GROWTH-FACTOR | EXPRESSION | PROGRESSION | RETINAL-PIGMENT EPITHELIUM | Retinal Pigment Epithelium - metabolism | Retina - metabolism | Intravitreal Injections | Streptozocin | Rats, Inbred WKY | Ocular Hypertension - metabolism | Blood-Retinal Barrier - drug effects | Oligopeptides - therapeutic use | Ocular Hypertension - prevention & control | Endothelin A Receptor Antagonists - metabolism | Diabetes Mellitus, Experimental - prevention & control | Endothelin-2 - genetics | Blood Pressure - drug effects | Endothelin-2 - metabolism | Real-Time Polymerase Chain Reaction | Endothelin A Receptor Antagonists - therapeutic use | Fluorescent Antibody Technique, Indirect | Rats, Inbred SHR | Endothelin B Receptor Antagonists - therapeutic use | Choroid - metabolism | Enzyme-Linked Immunosorbent Assay | Endothelin-1 - genetics | Endothelin-1 - metabolism | Rats | Endothelin B Receptor Antagonists - metabolism | Antihypertensive Agents - therapeutic use | Diabetic Retinopathy - metabolism | Diabetic Retinopathy - prevention & control | Animals | Piperidines - therapeutic use | Peptides, Cyclic - therapeutic use | Endothelial growth factors | RNA | Angiotensin | Endothelium
Journal Article
Reproduction, Fertility and Development, ISSN 1031-3613, 2016, Volume 28, Issue 6, pp. 673 - 681
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/2005, Volume 25, Issue 18, pp. 4540 - 4549
Regardless of proximal cause, photoreceptor injury or disease almost invariably leads to the activation of Muller cells, the principal glial cells in the... 
Retinal detachment | Endothelin | Photoreceptor | Retinal degeneration | Light damage | Microarray | Retinal disease | CELLS | retinal degeneration | retinal disease | MULLER | photoreceptor | RAT RETINA | SPINAL-CORD | ASTROCYTES | NEUROSCIENCES | light damage | DEGENERATION | endothelin | microarray | B RECEPTORS | GENE-EXPRESSION | retinal detachment | UP-REGULATION | Retinal Diseases - genetics | Receptors, Endothelin - genetics | Genomics | Nerve Tissue Proteins - deficiency | Molecular Sequence Data | Peptides, Cyclic - pharmacology | Retinal Diseases - metabolism | Dihydroorotase - deficiency | Endothelins - metabolism | Glial Fibrillary Acidic Protein - metabolism | Aspartate Carbamoyltransferase - deficiency | Time Factors | Retinal Diseases - etiology | Endothelin-2 - genetics | Protein Binding - drug effects | Endothelin-2 - metabolism | In Situ Hybridization - methods | Endothelin Receptor Antagonists | Disease Models, Animal | Peripherins | Blotting, Western - methods | Gene Expression Regulation - genetics | Oligonucleotide Array Sequence Analysis - methods | Fluorescent Antibody Technique - methods | Light - adverse effects | Mice, Inbred C57BL | Receptors, Endothelin - metabolism | Carbamoyl-Phosphate Synthase (Glutamine-Hydrolyzing) - deficiency | Gene Expression Regulation - physiology | Signal Transduction - genetics | Mice, Knockout | Immunohistochemistry - methods | Ceruloplasmin - genetics | Animals | Annexin A2 - genetics | Intermediate Filament Proteins - deficiency | Photoreceptor Cells - physiopathology | Ceruloplasmin - metabolism | Neuroglia - metabolism | Signal Transduction - physiology | Blotting, Northern - methods | Mice | Annexin A2 - metabolism | Membrane Glycoproteins - deficiency | Protein Binding - physiology | Neurobiology of Disease
Journal Article
Journal Article
Journal of Physiology and Biochemistry, ISSN 1138-7548, 12/2016, Volume 72, Issue 4, pp. 763 - 779
Decline in oxygen availability experienced under hypobaric hypoxia (HH) mediates imbalance in lung fluid clearance and is a causative agent of acute lung... 
Biomedicine, general | Animal Physiology | Biomedicine | Human Physiology | Nanocurcumin | Vasoconstriction | Hypoxia | Lung injury | CLEARANCE | APOPTOSIS | PHYSIOLOGY | CURCUMIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | PEROXIDATION | PATHOGENESIS | OVEREXPRESSION | HYPERTROPHY | ENDOTHELIN-1 | ALTITUDE PULMONARY-EDEMA | HYPERTENSION | Hypoxia - drug therapy | Sodium-Potassium-Exchanging ATPase - genetics | Endothelin-3 - genetics | Acute Lung Injury - genetics | Male | Proto-Oncogene Proteins c-akt - genetics | Protective Agents - pharmacology | Endothelin-2 - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Oxidation-Reduction - drug effects | Acute Lung Injury - drug therapy | Endothelin-2 - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Nanostructures - therapeutic use | Disease Models, Animal | Endothelin-3 - metabolism | Biomarkers - metabolism | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Endothelin-1 - genetics | Endothelin-1 - metabolism | Curcumin - pharmacology | Rats | Hypoxia - complications | Receptor, Endothelin A - genetics | Acute Lung Injury - pathology | Rats, Sprague-Dawley | Gene Expression Regulation - drug effects | Sodium-Potassium-Exchanging ATPase - metabolism | Hypoxia - genetics | Receptor, Endothelin B - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Hypoxia - pathology | Nanostructures - chemistry | Receptor, Endothelin A - metabolism | Receptor, Endothelin B - metabolism | Acute Lung Injury - etiology | Mitogen-Activated Protein Kinase 1 - metabolism | Acute respiratory distress syndrome | Respiratory agents | Adenosine triphosphatase | Histochemistry
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 12/2014, Volume 171, Issue 24, pp. 5555 - 5572
Journal Article
Journal Article
The Journal of Physiology, ISSN 0022-3751, 10/2011, Volume 589, Issue 19, pp. 4671 - 4679
Journal Article
Journal Article
Reproduction, Fertility and Development, ISSN 1031-3613, 08/2008, Volume 20, Issue 7, pp. 750 - 759
Endothelins (ETs) have been shown to have mitogenic effects on prostate cells in vitro . To study a relationship between ETs and prostate growth, we... 
CELLS | LOCALIZATION | REGIONAL DIFFERENCES | URINARY-BLADDER | SUBTYPES | DEVELOPMENTAL BIOLOGY | CANCER | FUNCTIONAL-PROPERTIES | DENSITY | REPRODUCTIVE BIOLOGY | ZOOLOGY | DEVELOPMENTAL-CHANGES | PROGRESSION | Receptors, Endothelin - genetics | Humans | Rats, Inbred F344 | Prostate - growth & development | Male | DNA Primers - genetics | Endothelins - metabolism | RNA, Messenger - metabolism | Prostate - metabolism | Base Sequence | Endothelin-2 - genetics | Endothelin-2 - metabolism | Gene Expression | Prostate - anatomy & histology | Signal Transduction | Endothelin-1 - genetics | Endothelin-1 - metabolism | RNA, Messenger - genetics | Receptors, Endothelin - metabolism | Organ Size | Rats | Receptor, Endothelin A - genetics | Endothelins - genetics | Receptor, Endothelin B - genetics | Animals | Receptor, Endothelin A - metabolism | Receptor, Endothelin B - metabolism | Receptor, Endothelin A, genetics | Prostate, growth and development | Prostate, anatomy and histology | Endothelin-1, genetics | Endothelins, genetics | RNA, Messenger, genetics | Endothelins, metabolism | RNA, Messenger, metabolism | Prostate, metabolism | Receptor, Endothelin A, metabolism | DNA Primers, genetics | Receptor, Endothelin B, genetics | Receptors, Endothelin, metabolism | Receptor, Endothelin B, metabolism | Endothelin-2, metabolism | Endothelin-2, genetics | Receptors, Endothelin, genetics | Endothelin-1, metabolism
Journal Article