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Nature medicine, ISSN 1546-170X, 06/2015, Volume 21, Issue 7, pp. 760 - 768
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 01/2018, Volume 314, Issue 1, pp. H52 - H64
Hyperglycemia-induced production of endothelin (ET)-1 is a hallmark of endothelial dysfunction in diabetes. Although the detrimental vascular effects of... 
Endothelial dysfunction | Hyperglycemia | Endothelin-1 | TRAF3 interacting protein 2 | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Inflammation - chemically induced | Coculture Techniques | Humans | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Male | Monocytes - metabolism | Inflammation - metabolism | I-kappa B Kinase - metabolism | Inflammation Mediators - metabolism | Female | Glucose - toxicity | Diabetes Mellitus, Experimental - metabolism | Diabetic Angiopathies - genetics | Tumor Necrosis Factor Receptor-Associated Peptides and Proteins - genetics | Diabetic Angiopathies - chemically induced | Diabetic Angiopathies - metabolism | Cytokines - metabolism | Endothelial Cells - metabolism | Endothelin-1 - genetics | Endothelin-1 - metabolism | Mitogen-Activated Protein Kinase 8 - metabolism | Cells, Cultured | Diabetes Mellitus, Type 1 - genetics | Cell Adhesion | Cell Adhesion Molecules - metabolism | Endothelin-1 - toxicity | Tumor Necrosis Factor Receptor-Associated Peptides and Proteins - metabolism | Animals | Signal Transduction - drug effects | Adaptor Proteins, Signal Transducing - genetics | Inflammation - genetics | Mice, Inbred NOD | Adaptor Proteins, Signal Transducing - metabolism | Endothelial Cells - drug effects | Reactive oxygen species | Vascular cell adhesion molecule 1 | Transcription | Activation | Glucose | NAD(P)H oxidase | Proteins | Aorta | Inhibition | Chemical synthesis | Intercellular adhesion molecule 1 | Complications | Adapters | Diabetes mellitus | JNK protein | Pharmacology | Inflammation | Endothelin 1 | Tumor necrosis factor-α | IL-1β | Adhesion | Endothelial cells | Endothelium | E-selectin | Molecular modelling | Monocyte chemoattractant protein | Diabetes | Monocyte chemoattractant protein 1 | Index Medicus | hyperglycemia | endothelin-1 | endothelial dysfunction
Journal Article
Journal Article
Journal of vascular research, ISSN 1018-1172, 10/2009, Volume 46, Issue 6, pp. 561 - 571
Background: Alterations in pulmonary blood flow are often associated with the initiation and progression of pulmonary vascular disease. However, the cellular... 
Research Paper | Vascular remodeling | Shear stress | Endothelin | Vascular tone | Nitric oxide | Pulmonary hypertension | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Endothelial Growth Factors - metabolism | Phosphorylation | Thromboxane B2 - metabolism | Cell Proliferation | Cadherins - metabolism | Muscle, Smooth, Vascular - metabolism | Pulsatile Flow | Coculture Techniques | Actins - metabolism | Myocytes, Smooth Muscle - pathology | Hypertension, Pulmonary - physiopathology | Stress, Mechanical | Vascular Endothelial Growth Factor A - metabolism | Myosin Heavy Chains - metabolism | Antigens, CD - metabolism | Pulmonary Artery - metabolism | Cattle | Vasodilator Agents - metabolism | Nitric Oxide Synthase Type III - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Myocytes, Smooth Muscle - metabolism | Animals, Newborn | Endothelial Cells - metabolism | Endothelin-1 - metabolism | Cells, Cultured | Hypertension, Pulmonary - metabolism | Prostaglandins F - metabolism | Mechanotransduction, Cellular | Muscle, Smooth, Vascular - pathology | Animals | Vasoconstrictor Agents - metabolism | Endothelial Cells - pathology | Nitric Oxide - metabolism | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Index Medicus
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 1522-1539, 12/2010, Volume 299, Issue 6, pp. H1959 - H1967
We showed previously that vascular smooth muscle cells (VMSC) from spontaneously hypertensive rats (SHR) exhibit increased proliferation. The present study was... 
Vascular smooth muscle cell proliferation | Oxidative stress | Spontaneously hypertensive rat | Extracellular signal-regulated kinase 1/2 | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Free Radical Scavengers - pharmacology | Phosphorylation | Muscle, Smooth, Vascular - metabolism | Oxidative Stress | src-Family Kinases | Protein-Tyrosine Kinases - metabolism | Hyperplasia | Myocytes, Smooth Muscle - pathology | NADPH Oxidases - metabolism | Rats, Inbred WKY | Angiotensin II Type 1 Receptor Blockers - pharmacology | Receptor, Epidermal Growth Factor - metabolism | Endothelin A Receptor Antagonists | Superoxides - metabolism | Receptor, Angiotensin, Type 1 - drug effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Rats, Inbred SHR | Angiotensin II - metabolism | Signal Transduction | Endothelin-1 - metabolism | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Epidermal Growth Factor - metabolism | Endothelin B Receptor Antagonists | Hypertension - pathology | Hypertension - metabolism | Muscle, Smooth, Vascular - pathology | Animals | Receptor, Angiotensin, Type 1 - metabolism | Epidermal Growth Factor - antagonists & inhibitors | Receptor, Endothelin A - metabolism | Cell Proliferation - drug effects | Receptor, Endothelin B - metabolism | Mitogen-Activated Protein Kinases - metabolism | Protein-Tyrosine Kinases - antagonists & inhibitors | Hypertension | Cell proliferation | Peptides | Physiological aspects | Research | Vascular smooth muscle | Health aspects | Studies | Rodents | Insulin-like growth factors | Comparative analysis | Kinases | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, pp. e72354 - e72354
Objective: Mitochondrial transcription factor A (TFAM) is normally bound to and remains associated with mitochondrial DNA (mtDNA) when released from damaged... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | HMGB2 Protein - metabolism | Dendritic Cells - immunology | Humans | Male | NF-kappa B - metabolism | Phosphatidylinositol 3-Kinases - metabolism | DNA-Binding Proteins - metabolism | Mitochondrial Proteins - metabolism | HMGB1 Protein - metabolism | Dendritic Cells - metabolism | Cell Line | DNA, Mitochondrial - metabolism | Signal Transduction | Spleen - cytology | DNA, Mitochondrial - immunology | Transcription Factors - metabolism | Animals | Spleen - metabolism | Models, Biological | CpG Islands | Protein Binding | Mice | Toll-Like Receptor 9 - metabolism | Mitogen-Activated Protein Kinases - metabolism | Cell culture | Veterinary colleges | Transcription factors | Endothelin | Acidification | Critical care | Mitochondrial DNA | Cell interactions | Kinases | Sulfates | Veterinary medicine | Proteins | Signal transduction | Receptors | Illnesses | Ischemia | Splenocytes | Toll-like receptors | Pretreatment | Deoxyribonucleic acid--DNA | Enzyme-linked immunosorbent assay | CpG islands | Enzymes | NF-κB protein | FLT3L protein | Immune response | Cytokines | Dendritic cells | Multiple organ dysfunction syndrome | Inflammation | Hazards | Tumor necrosis factor-α | Trauma | TLR9 protein | 1-Phosphatidylinositol 3-kinase | Signaling | Sleep | Converting | Equivalence | In vivo methods and tests | Heparin | Laboratory animals | Recognition | Endosomes | Index Medicus | Deoxyribonucleic acid | DNA | Sulfate
Journal Article
Journal Article
Journal Article