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PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e87936
The growing incidence of chronic kidney disease remains a global health problem. Obesity is a major risk factor for type-2 diabetes and renal impairment.... 
CHRONIC HEART-FAILURE | TISSUE INFLAMMATION | ATRIAL-NATRIURETIC-PEPTIDE | WAIST CIRCUMFERENCE | GLUCOSE-METABOLISM | MULTIDISCIPLINARY SCIENCES | IN-VIVO | ENHANCES INSULIN SENSITIVITY | FACTOR-KAPPA-B | CHRONIC KIDNEY-DISEASE | HYPERTENSIVE-RATS | Tumor Necrosis Factor-alpha - metabolism | Atrial Natriuretic Factor - metabolism | Endothelin-1 - metabolism | Diabetic Nephropathies - metabolism | Rats | Male | Dinoprost - metabolism | Interleukins - metabolism | Hemin - metabolism | Rats, Zucker | Inflammation - metabolism | Kidney - metabolism | Macrophages - metabolism | Animals | Diabetic Nephropathies - physiopathology | Chemokine CCL3 - metabolism | Adiposity - physiology | Dinoprost - analogs & derivatives | Blood Glucose - metabolism | Heme Oxygenase (Decyclizing) - metabolism | Extracellular Matrix Proteins - metabolism | Kidney - physiopathology | Inflammation - physiopathology | Type 2 diabetes | Adipose tissues | Fibronectins | Obesity | Endothelin | Cytokines | Diabetic nephropathies | Macrophages | Risk factors | Glucose metabolism | Chronic kidney failure | Collagen | Heme | Natriuretic peptides | Adipose tissue | Syngeneic grafts | Selectivity | Adipocytes | Fibronectin | Proteins | Genotype & phenotype | Cyclic GMP | Fibroblasts | Physiology | Carbon monoxide | Public health | Pulmonary arteries | Endothelin 1 | Tumor necrosis factor-α | Metabolism | Gene expression | Studies | Oxygenase | Hemin | Nephropathy | Fibrosis | Interleukin 10 | Infiltration | Renal function | Paracrine signalling | Adiponectin | Interleukin 6 | Rodents | Diaphragm (anatomy) | Extracellular matrix | Tumor necrosis factor-TNF | Lesions | Inducers | Creatinine | Hypertension | Excretion | Kidneys | Diabetes mellitus | Markers | Health risks | Inflammation | Morbidity | Scaffolding | Insulin resistance | Diaphragm | Diabetes | Proteinuria
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e87548
Background: Inhibition of the transporter-mediated hepatobiliary elimination of bile salts is a putative mechanism for liver toxicity observed with some... 
BILE-ACID TRANSPORT | IN-VITRO | RAT HEPATOCYTES | PULMONARY ARTERIAL-HYPERTENSION | SALT EXPORT PUMP | BILIARY-EXCRETION | MULTIDISCIPLINARY SCIENCES | HEPATIC-UPTAKE | KNOCKOUT MICE | FAMILIAL INTRAHEPATIC CHOLESTASIS | P-GLYCOPROTEIN | ATP Binding Cassette Transporter, Sub-Family G, Member 2 | Humans | Middle Aged | ATP-Binding Cassette, Sub-Family B, Member 1 - metabolism | Bile - metabolism | Male | Hepatocytes - metabolism | Pyridazines - pharmacology | Neoplasm Proteins - metabolism | Organic Anion Transporters - metabolism | Liver - drug effects | Organic Anion Transporters, Sodium-Independent - metabolism | ATP-Binding Cassette Transporters - metabolism | Adult | Female | Membrane Transport Proteins - metabolism | Endothelin Receptor Antagonists | Hepatocytes - drug effects | Solute Carrier Organic Anion Transporter Family Member 1B3 | Taurocholic Acid - pharmacology | Liver - metabolism | Receptors, Endothelin - metabolism | Thiophenes - pharmacology | Bile Acids and Salts - metabolism | Pyrimidines - pharmacology | Sulfonamides - pharmacology | Symporters - metabolism | Isoxazoles - pharmacology | Phenylpropionates - pharmacology | Bile - drug effects | Solute Carrier Organic Anion Transporter Family Member 1b1 | Multidrug Resistance-Associated Proteins - metabolism | Organic Anion Transporters, Sodium-Dependent - metabolism | Drug resistance in microorganisms | Endothelin | Bile acids | Research | Analysis | Salts | Transplants & implants | Toxicity | Pathogenesis | Liver | Gallbladder diseases | Accumulation | Bioaccumulation | Rodents | Protein transport | P-Glycoprotein | Efflux | Polypeptides | Mortality | Multidrug resistance | Glycoprotein | Substrate inhibition | Breast cancer | Substrates | Bile salts | Organic anion transporting polypeptide | Molecular modelling | Acids | Sodium | Hepatocytes | Transporter | Cancer | Bile
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2015, Volume 21, Issue 7, pp. 760 - 768
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 01/2012, Volume 46, Issue 1, pp. 14 - 22
Endothelin-1 is a potent vasoactive peptide that occurs in chronically high levels in humans with pulmonary hypertension and in animal models of the disease.... 
Pulmonary artery smooth muscle | Inflammation | Endothelin | Hyaluronic acid | Unfolded protein response | ER STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | HYALURONAN SYNTHESIS | RATS | ATHEROSCLEROSIS | ENDOPLASMIC-RETICULUM STRESS | PROINFLAMMATORY CYTOKINES | HYPOXIA | hyaluronic acid | CELL BIOLOGY | unfolded protein response | endothelin | inflammation | RESPIRATORY SYSTEM | pulmonary artery smooth muscle | DISEASE | HYPERTENSION | PROGRESSION | Phosphorylation - physiology | Arteritis - pathology | Muscle, Smooth, Vascular - metabolism | Myocytes, Smooth Muscle - pathology | Arteritis - metabolism | Monocytes - metabolism | DNA-Binding Proteins - metabolism | Phosphorylation - genetics | Pulmonary Artery - metabolism | Endothelin A Receptor Antagonists | Myocytes, Smooth Muscle - metabolism | Cytokines - metabolism | Unfolded Protein Response - genetics | Endothelin-1 - genetics | Endothelin-1 - metabolism | Rats | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - metabolism | Signal Transduction - genetics | Regulatory Factor X Transcription Factors | Transcription Factors - metabolism | Activating Transcription Factor 6 - metabolism | Muscle, Smooth, Vascular - pathology | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Hyaluronic Acid - metabolism | Receptor, Endothelin A - metabolism | Signal Transduction - physiology | Receptor, Endothelin B - metabolism | Unfolded Protein Response - physiology | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/2004, Volume 24, Issue 21, pp. 5016 - 5021
The regenerative capacity of the CNS is extremely limited. The reason for this is unclear, but glial cell involvement has been suspected, and oligodendrocytes... 
Vimentin | Regeneration | Endothelin B receptor | Astrocytes | Intermediate filaments | Hippocampus | GFAP | endothelin B receptor | hippocampus | regeneration | INJURY | vimentin | NEUROSCIENCES | NEURITE OUTGROWTH | ELECTRON-MICROSCOPIC ANALYSIS | ADULT-RAT | DISEASE | REACTIVE ASTROCYTES | astrocytes | DENTATE GYRUS | intermediate filaments | INTERMEDIATE-FILAMENTS | TRANSGENIC MICE | DEVELOP | Up-Regulation | Hypertrophy - prevention & control | Vimentin - metabolism | Mice, Inbred C57BL | Nerve Regeneration - physiology | Cells, Cultured | Cytoplasm - ultrastructure | Brain Injuries - physiopathology | Glial Fibrillary Acidic Protein - metabolism | Hypertrophy - pathology | Hypertrophy - metabolism | Astrocytes - ultrastructure | Hippocampus - metabolism | Animals | Hippocampus - ultrastructure | Mice | Receptor, Endothelin B - metabolism | Brain Injuries - pathology | Glutamate-Ammonia Ligase - metabolism | Entorhinal Cortex - injuries | Astrocytes - metabolism | Plasticity | Development | Repair | Neurosciences | Nerve Regeneration/physiology | Astrocytes/metabolism/ultrastructure | Endothelin B/metabolism | Cells | Inbred C57BL | Cytoplasm/ultrastructure | Glial Fibrillary Acidic Protein/metabolism | Brain Injuries/pathology/physiopathology | Receptor | Cultured | Hypertrophy/metabolism/pathology/prevention & control | Entorhinal Cortex/injuries | Glutamate-Ammonia Ligase/metabolism | Hippocampus/metabolism/ultrastructure | Vimentin/metabolism | Neurovetenskaper
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2016, Volume 291, Issue 9, pp. 4323 - 4333
Invadosomes are actin-rich membrane protrusions that degrade the extracellular matrix to drive tumor cell invasion. Key players in invadosome formation are... 
Receptors, Lysophosphatidic Acid - metabolism | Lysophospholipids - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | Extracellular Matrix - metabolism | Melanoma - enzymology | Podosomes - enzymology | Neoplasm Proteins - antagonists & inhibitors | Receptors, Lysophosphatidic Acid - agonists | Receptors, Lysophosphatidic Acid - genetics | cdc42 GTP-Binding Protein - metabolism | rhoA GTP-Binding Protein - metabolism | rhoA GTP-Binding Protein - genetics | Endothelins - metabolism | Neoplasm Proteins - metabolism | Time-Lapse Imaging | Podosomes - metabolism | RNA Interference | cdc42 GTP-Binding Protein - antagonists & inhibitors | Fluorescence Resonance Energy Transfer | Receptors, Lysophosphatidic Acid - antagonists & inhibitors | Neoplasm Proteins - genetics | Biomarkers - metabolism | Melanoma - metabolism | Recombinant Proteins - metabolism | rac1 GTP-Binding Protein - agonists | rhoA GTP-Binding Protein - antagonists & inhibitors | Recombinant Proteins - genetics | Melanoma - pathology | cdc42 GTP-Binding Protein - agonists | Hydrolysis | Podosomes - pathology | Microscopy, Confocal | Neoplasm Proteins - agonists | cdc42 GTP-Binding Protein - genetics | Receptors, G-Protein-Coupled - antagonists & inhibitors | rac1 GTP-Binding Protein - antagonists & inhibitors | Cell Line, Tumor | Luminescent Proteins - genetics | Receptors, G-Protein-Coupled - genetics | Extracellular Matrix - pathology | Microscopy, Fluorescence | rac1 GTP-Binding Protein - metabolism | Luminescent Proteins - metabolism | rac1 GTP-Binding Protein - genetics | CDC42 | invadopodia | biosensor | fluorescence resonance energy transfer (FRET) | calcium intracellular release | G protein-coupled receptor (GPCR) | Rho (Rho GTPase) | phosphatidylinositide 3-kinase (PI 3-kinase) | imaging | Rac (Rac GTPase) | Cell Biology
Journal Article
Canadian Journal of Physiology and Pharmacology, ISSN 0008-4212, 06/2010, Volume 88, Issue 6, pp. 682 - 691
To evaluate potential mechanisms of clinical hepatotoxicity, 4 endothelin receptor antagonists (ERAs) were examined for substrate activity and inhibition of... 
Hepatic transport | BSEP | Endothelin | Transporter substrate | Pulmonary artery hypertension | Endothelin receptor antagonists | OATP | PHARMACOKINETIC INTERACTION | PHYSIOLOGY | pulmonary artery hypertension | RAT HEPATOCYTES | PULMONARY ARTERIAL-HYPERTENSION | SALT EXPORT PUMP | transporter substrate | HEPATIC-UPTAKE | endothelin receptor antagonists | CLINICAL-PRACTICE GUIDELINES | endothelin | CYCLOSPORINE-A | THERAPY | BILIARY-EXCRETION | KETOCONAZOLE | PHARMACOLOGY & PHARMACY | hepatic transport | Antihypertensive Agents - pharmacology | Estradiol - analogs & derivatives | Taurocholic Acid - metabolism | Humans | Middle Aged | ATP-Binding Cassette, Sub-Family B, Member 1 - metabolism | Male | Thiophenes - metabolism | Antihypertensive Agents - metabolism | Hepatocytes - metabolism | Pyridazines - pharmacology | ATP-Binding Cassette, Sub-Family B, Member 1 - antagonists & inhibitors | Organic Anion Transporters - metabolism | Pyrimidines - metabolism | Young Adult | Phenylpropionates - metabolism | Symporters - antagonists & inhibitors | ATP-Binding Cassette Transporters - metabolism | Adult | Female | Pyridazines - metabolism | Biological Transport - drug effects | Cell Culture Techniques | Endothelin Receptor Antagonists | Hepatocytes - drug effects | Estradiol - metabolism | Organic Anion Transporters - antagonists & inhibitors | Isoxazoles - metabolism | Organic Anion Transporters, Sodium-Dependent - antagonists & inhibitors | Cells, Cultured | Thiophenes - pharmacology | Multidrug Resistance-Associated Proteins - antagonists & inhibitors | Pyrimidines - pharmacology | Sulfonamides - pharmacology | Symporters - metabolism | Isoxazoles - pharmacology | Phenylpropionates - pharmacology | ATP Binding Cassette Subfamily B Member 11 | Enkephalin, D-Penicillamine (2,5)- - metabolism | Adolescent | Sulfonamides - metabolism | Aged | Multidrug Resistance-Associated Proteins - metabolism | Organic Anion Transporters, Sodium-Dependent - metabolism | ATP-Binding Cassette Transporters - antagonists & inhibitors | Cell receptors | Genetic aspects | Research | Diagnosis | Properties | Health aspects | Liver cells | Pulmonary hypertension
Journal Article
Oncogene, ISSN 0950-9232, 06/2016, Volume 35, Issue 26, pp. 3432 - 3442
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 02/2015, Volume 172, Issue 4, pp. 1005 - 1019
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 12/2010, Volume 299, Issue 6, pp. 1959 - 1967
We showed previously that vascular smooth muscle cells (VMSC) from spontaneously hypertensive rats (SHR) exhibit increased proliferation. The present study was... 
Vascular smooth muscle cell proliferation | Oxidative stress | Spontaneously hypertensive rat | Extracellular signal-regulated kinase 1/2 | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | PROTEIN | SPONTANEOUSLY HYPERTENSIVE-RATS | KINASE | spontaneously hypertensive rat | TYPE-1 RECEPTOR | vascular smooth muscle cell proliferation | ENDOTHELIN MESSENGER-RNA | ENHANCED EXPRESSION | PERIPHERAL VASCULAR DISEASE | ANGIOTENSIN-II RECEPTORS | extracellular signal-regulated kinase 1/2 | ADENYLATE-CYCLASE | oxidative stress | Free Radical Scavengers - pharmacology | Phosphorylation | Muscle, Smooth, Vascular - metabolism | Oxidative Stress | src-Family Kinases | Protein-Tyrosine Kinases - metabolism | Hyperplasia | Myocytes, Smooth Muscle - pathology | NADPH Oxidases - metabolism | Rats, Inbred WKY | Angiotensin II Type 1 Receptor Blockers - pharmacology | Receptor, Epidermal Growth Factor - metabolism | Endothelin A Receptor Antagonists | Superoxides - metabolism | Receptor, Angiotensin, Type 1 - drug effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Rats, Inbred SHR | Angiotensin II - metabolism | Signal Transduction | Endothelin-1 - metabolism | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Epidermal Growth Factor - metabolism | Endothelin B Receptor Antagonists | Hypertension - pathology | Hypertension - metabolism | Muscle, Smooth, Vascular - pathology | Animals | Receptor, Angiotensin, Type 1 - metabolism | Epidermal Growth Factor - antagonists & inhibitors | Receptor, Endothelin A - metabolism | Cell Proliferation - drug effects | Receptor, Endothelin B - metabolism | Mitogen-Activated Protein Kinases - metabolism | Protein-Tyrosine Kinases - antagonists & inhibitors | Hypertension | Cell proliferation | Peptides | Physiological aspects | Research | Vascular smooth muscle | Health aspects
Journal Article
Experimental Neurology, ISSN 0014-4886, 2005, Volume 195, Issue 2, pp. 293 - 304
Bone morphogenetic proteins (BMPs) are a large class of secreted factors, which serve as modulators of development in multiple organ systems, including the... 
Noggin | Spinal cord | Endothelin | OPC | BMP | Injury | Neural stem cell | Transplantation | Differentiation | neural stem cell | differentiation | noggin | BMP SIGNALING INHIBITION | BONE MORPHOGENETIC PROTEINS | NEUROSCIENCES | endothelin | CONTUSION INJURIES | ADULT-RAT | STRUCTURAL BASIS | transplantation | GROWTH | GENE-EXPRESSION | spinal cord | injury | CYSTINE KNOT PROTEIN | LINEAGE | Nestin | O Antigens - metabolism | Embryo, Mammalian | Microtubule-Associated Proteins - metabolism | Vimentin - metabolism | Rats, Inbred F344 | Phosphopyruvate Hydratase - metabolism | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Stem Cells - metabolism | Bone Morphogenetic Proteins - metabolism | Transfection - methods | Time Factors | Reverse Transcriptase Polymerase Chain Reaction - methods | Female | Membrane Proteins - metabolism | Neurons - metabolism | Bone Morphogenetic Proteins - genetics | Cell Differentiation - physiology | Green Fluorescent Proteins - metabolism | Spinal Cord Injuries - metabolism | Bone Morphogenetic Protein 2 | Ectodysplasins | Cells, Cultured | Spinal Cord Injuries - surgery | Gene Expression Regulation - physiology | Proteoglycans - metabolism | Rats | Antigens - metabolism | Oligopeptides - metabolism | Immunohistochemistry - methods | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Electrophoretic Mobility Shift Assay - methods | Stem Cell Transplantation - methods | Animals | Carrier Proteins - metabolism | Cell Count - methods | Transforming Growth Factor beta - genetics | Neuroglia - metabolism | Blotting, Northern - methods | Intermediate Filament Proteins - metabolism | Transforming Growth Factor beta - metabolism | Neurons | Stem cells | Bone morphogenetic proteins | Spinal cord injuries | Universities and colleges
Journal Article
Journal of Vascular Research, ISSN 1018-1172, 10/2009, Volume 46, Issue 6, pp. 561 - 571
Background: Alterations in pulmonary blood flow are often associated with the initiation and progression of pulmonary vascular disease. However, the cellular... 
Research Paper | Vascular remodeling | Shear stress | Endothelin | Vascular tone | Nitric oxide | Pulmonary hypertension | CELLS | PHYSIOLOGY | FLUID SHEAR-STRESS | NITRIC-OXIDE SYNTHASE | MYOGENIC RESPONSE | MECHANISMS | BLOOD-FLOW | VASCULAR ENDOTHELIUM | HEMODYNAMICS | PERIPHERAL VASCULAR DISEASE | HYPERTENSION | EXPRESSION | Endothelial Growth Factors - metabolism | Phosphorylation | Thromboxane B2 - metabolism | Cell Proliferation | Cadherins - metabolism | Muscle, Smooth, Vascular - metabolism | Pulsatile Flow | Coculture Techniques | Actins - metabolism | Myocytes, Smooth Muscle - pathology | Hypertension, Pulmonary - physiopathology | Stress, Mechanical | Vascular Endothelial Growth Factor A - metabolism | Myosin Heavy Chains - metabolism | Antigens, CD - metabolism | Pulmonary Artery - metabolism | Cattle | Vasodilator Agents - metabolism | Nitric Oxide Synthase Type III - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Myocytes, Smooth Muscle - metabolism | Animals, Newborn | Endothelial Cells - metabolism | Endothelin-1 - metabolism | Cells, Cultured | Hypertension, Pulmonary - metabolism | Prostaglandins F - metabolism | Mechanotransduction, Cellular | Muscle, Smooth, Vascular - pathology | Animals | Vasoconstrictor Agents - metabolism | Endothelial Cells - pathology | Nitric Oxide - metabolism | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology
Journal Article