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Acta Neuropathologica, ISSN 0001-6322, 9/2011, Volume 122, Issue 3, pp. 293 - 311
Cerebrovascular lesions related to congophilic amyloid angiopathy (CAA) often accompany deposition of β-amyloid (Aβ) in Alzheimer’s disease (AD), leading to... 
Pathology | Neurosciences | Congophilic amyloid angiopathy | Medicine & Public Health | Alzheimer’s disease | Astrocytes | Cerebral glucose metabolism | Alzheimer's disease | MICROVASCULAR PATHOLOGY | ALZHEIMERS-DISEASE | NEUROVASCULAR MECHANISMS | GLUCOSE-UTILIZATION | PATHOLOGY | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | CLINICAL NEUROLOGY | MOUSE MODEL | ENDOTHELIAL-CELLS | A-BETA | SMOOTH-MUSCLE-CELLS | RAT-BRAIN | Microdialysis - methods | Humans | Astrocytes - pathology | Dystroglycans - metabolism | Glucose Transporter Type 1 - metabolism | Muscle, Smooth - ultrastructure | Glial Fibrillary Acidic Protein - metabolism | Microscopy, Electron, Scanning - methods | Amyloid beta-Peptides - genetics | Amyloid beta-Peptides - metabolism | Cell Culture Techniques | Disease Models, Animal | Endothelium - pathology | Mice, Transgenic | Cerebral Arteries - ultrastructure | Basement Membrane - metabolism | Disease Progression | Symporters - metabolism | Astrocytes - ultrastructure | Blood-Brain Barrier - pathology | Cerebral Arteries - metabolism | Brain - pathology | Glucose - metabolism | Plaque, Amyloid - metabolism | Mice | Astrocytes - metabolism | Blood-Brain Barrier - physiopathology | Basement Membrane - pathology | Cerebral Amyloid Angiopathy - complications | Monocarboxylic Acid Transporters - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Hemorrhage - etiology | Amyloid beta-Protein Precursor - metabolism | Lactase - metabolism | Cerebral Amyloid Angiopathy - genetics | Astrocytes - drug effects | Plaque, Amyloid - pathology | Gene Expression Regulation - genetics | Hemorrhage - metabolism | Muscle, Smooth - metabolism | Cerebrovascular Disorders - etiology | Cerebral Amyloid Angiopathy - pathology | Animals | Endothelium - metabolism | Glucose Transporter Type 1 - genetics | Symporters - genetics | Cerebral Arteries - pathology | Laminin - metabolism | Monocarboxylic Acid Transporters - genetics | Cerebrovascular Disorders - pathology | Hemorrhage - pathology | Muscle, Smooth - pathology | Index Medicus | Glucose transporter | Leakage | Brain | Neurodegenerative diseases | Cognitive ability | Transgenic mice | Blood vessels | Data processing | Smooth muscle | beta -Amyloid | Glucose transport | Metabolism | Amyloid precursor protein | Blood-brain barrier | Cerebral blood flow | Lactic acid | Mutation | Original Paper
Journal Article
1991, ISBN 9780123649324, Volume 32., ix, 285
Book
Clinical & Experimental Ophthalmology, ISSN 1442-6404, 03/2009, Volume 37, Issue 2, pp. 223 - 231
Purpose:  The aim of this study is to investigate markers of inflammation and oxidative stress in an early model of diabetic retinopathy, correlate retinal and... 
pericyte | diabetic retinopathy | inflammation | microglia | oxidative stress | Diabetic retinopathy | Oxidative stress | Pericyte | Inflammation | Microglia | CELLS | ANGIOGENESIS | ANTIOXIDANTS | DEATH | DAMAGE | PATHWAY | DISEASE | OPHTHALMOLOGY | RETINOPATHY | Retina - drug effects | Diabetic Retinopathy - drug therapy | Inflammation - pathology | Microglia - metabolism | Diabetes Mellitus, Experimental - drug therapy | Retina - metabolism | Oxidative Stress | Rats, Wistar | Tumor Necrosis Factor-alpha - blood | Superoxide Dismutase - blood | Male | Diabetic Retinopathy - pathology | Immunoenzyme Techniques | Inflammation - metabolism | Pericytes - pathology | Microglia - pathology | Free Radical Scavengers - therapeutic use | Acetylcysteine - therapeutic use | Diabetes Mellitus, Experimental - metabolism | Fluorescent Antibody Technique, Indirect | Enzyme-Linked Immunosorbent Assay | Macrophages - pathology | Pericytes - metabolism | Rats | Biomarkers - blood | Diabetic Retinopathy - metabolism | Macrophages - metabolism | Animals | Endothelium, Vascular - metabolism | Diabetes Mellitus, Experimental - pathology | Endothelium, Vascular - pathology | Isoprostanes - blood | Pericytes, metabolism | Endothelium, Vascular, metabolism | Microglia, metabolism | Superoxide Dismutase, blood | Diabetic Retinopathy, pathology | Isoprostanes, blood | Tumor Necrosis Factor-alpha, blood | Acetylcysteine, therapeutic use | Endothelium, Vascular, pathology | Inflammation, metabolism | Inflammation, pathology | Diabetic Retinopathy, metabolism | Diabetes Mellitus, Experimental, drug therapy | Macrophages, pathology | Retina, drug effects | Retina, metabolism | Diabetes Mellitus, Experimental, pathology | Pericytes, pathology | Diabetic Retinopathy, drug therapy | Diabetes Mellitus, Experimental, metabolism | Biological Markers, blood | Macrophages, metabolism | Microglia, pathology | Free Radical Scavengers, therapeutic use | Streptozocin | Heme | Acetylcysteine | Superoxide dismutase | Superoxide | Ophthalmology | Universities and colleges | Macrophages | Vascular endothelial growth factor | Endothelium | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 09/2015, Volume 525, Issue 7568, pp. 247 - 250
Journal Article
Circulation, ISSN 0009-7322, 02/2015, Volume 131, Issue 6, pp. 550 - 559
BACKGROUND—Characterization of myocardial structural changes in heart failure with preserved ejection fraction (HFpEF) has been hindered by the limited... 
heart failure, diastolic | endothelium | pathology | hypertrophy | fibrosis | autopsy | microvessels | PROGNOSTIC IMPACT | CARDIAC & CARDIOVASCULAR SYSTEMS | IDIOPATHIC DILATED CARDIOMYOPATHY | CARDIAC STRUCTURE | RESTING TENSION | LEFT-VENTRICLE | ACTIVE RELAXATION | DIASTOLIC DYSFUNCTION | FLOW RESERVE | HYPERTROPHIC CARDIOMYOPATHY | PERIPHERAL VASCULAR DISEASE | ARTERY-DISEASE | Immunohistochemistry | Heart - anatomy & histology | Humans | Heart Failure - physiopathology | Microvessels - pathology | Cardiomegaly - epidemiology | Cardiomegaly - pathology | Male | Reference Values | Cause of Death | Autopsy | Coronary Artery Disease - pathology | Aged, 80 and over | Electrocardiography | Female | Endomyocardial Fibrosis - pathology | Heart Failure - diagnosis | Heart Failure - epidemiology | Coronary Vessels - pathology | Echocardiography | Comorbidity | Endomyocardial Fibrosis - epidemiology | Organ Size | Myocardium - pathology | Heart Failure - pathology | Stroke Volume | Myocytes, Cardiac - pathology | Algorithms | Diabetes Mellitus - epidemiology | Aged | Coronary Artery Disease - epidemiology | Endomyocardial Fibrosis - physiopathology | Postmortem Changes | Heart failure | Analysis | Research | Cardiac output | Risk factors | Endothelium | Hypertrophy | Index Medicus | Abridged Index Medicus | coronary microvessel | diastolic heart failure
Journal Article
Brain Research, ISSN 0006-8993, 2007, Volume 1157, Issue 1, pp. 126 - 137
Abstract The purpose of this study was to determine the ultrastructure of the blood–brain barrier (BBB) and blood–spinal cord barrier (BSCB) in G93A SOD1 mice... 
Neurology | Spinal cord | Mitochondria | Ultrastructure | Blood–spinal cord barrier | Brainstem | Motor neuron | Blood–brain barrier | G93A SOD1 mice | Endothelium | Basement membrane | Blood-brain barrier | Blood-spinal cord barrier | blood-brain barrier | MOTOR-NEURONS | endothelium | IMMUNOGLOBULIN SUPERFAMILY | blood-spinal cord barrier | basement membrane | WHITE-MATTER | mitochondria | IGG | AMYOTROPHIC-LATERAL-SCLEROSIS | ultrastructure | CEREBROSPINAL-FLUID | NEUROSCIENCES | MULTIPLE-SCLEROSIS | motor neuron | ENDOTHELIAL-CELLS | MOUSE MODEL | AQUAPORIN-4 | brainstem | spinal cord | Basement Membrane - ultrastructure | Superoxide Dismutase - genetics | Amyotrophic Lateral Sclerosis - physiopathology | Capillaries - pathology | Humans | Astrocytes - pathology | Blood-Brain Barrier - physiopathology | Basement Membrane - pathology | Mitochondria - ultrastructure | Spinal Cord - ultrastructure | Blood-Brain Barrier - ultrastructure | Motor Neurons - pathology | Motor Neurons - ultrastructure | Brain - blood supply | Endothelial Cells - ultrastructure | Spinal Cord - pathology | Brain - ultrastructure | Disease Models, Animal | Extracellular Space - physiology | Microscopy, Electron, Transmission | Capillaries - physiopathology | Capillaries - ultrastructure | Brain Edema - etiology | Brain Edema - pathology | Mice, Inbred C57BL | Mice, Transgenic | Mitochondria - pathology | Spinal Cord - blood supply | Mutation - genetics | Astrocytes - ultrastructure | Tight Junctions - ultrastructure | Blood-Brain Barrier - pathology | Amyotrophic Lateral Sclerosis - pathology | Animals | Brain Edema - physiopathology | Brain - pathology | Mice | Superoxide Dismutase-1 | Endothelial Cells - pathology | Tight Junctions - pathology | Index Medicus
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2008, Volume 172, Issue 5, pp. 1411 - 1418
Journal Article
Nature, ISSN 0028-0836, 12/2010, Volume 468, Issue 7325, pp. 824 - 828
Journal Article
Circulation, ISSN 0009-7322, 05/2009, Volume 119, Issue 19, pp. 2561 - 2567
Background-In classic Fabry patients, accelerated coronary atherosclerosis and left ventricular hypertrophy manifest in the fourth decade; however, signs of... 
Microcirculation | Pathology | Hypercholesterolemia | Ischemia | Hypertrophy | CARDIAC & CARDIOVASCULAR SYSTEMS | pathology | CHOLESTEROL | hypertrophy | CARDIOMYOPATHY | INVOLVEMENT | ATHEROSCLEROSIS | LOW-DENSITY LIPOPROTEIN | microcirculation | hypercholesterolemia | ALPHA-GALACTOSIDASE-A | ischemia | GLYCOSPHINGOLIPIDS | SERUM-LIPOPROTEINS | PERIPHERAL VASCULAR DISEASE | AGALSIDASE-ALPHA | ACCUMULATION | HEMATOLOGY | Recombinant Proteins - therapeutic use | alpha-Galactosidase - therapeutic use | Capillaries - pathology | Trihexosylceramides - analysis | Inclusion Bodies - chemistry | Humans | Lysosomes - enzymology | Male | Endocardium - pathology | Endothelium, Vascular - chemistry | Young Adult | Clinical Trials, Phase III as Topic | Adult | Female | Trihexosylceramides - metabolism | Inclusion Bodies - ultrastructure | Double-Blind Method | Capillaries - chemistry | Coronary Artery Disease - prevention & control | Fabry Disease - complications | Coronary Disease - etiology | Randomized Controlled Trials as Topic | Myocytes, Cardiac - pathology | Coronary Disease - pathology | Coronary Disease - prevention & control | Endocardium - ultrastructure | Biopsy | Adolescent | Coronary Disease - drug therapy | Myocytes, Cardiac - metabolism | Endothelium, Vascular - pathology | Prevention | Care and treatment | Heart enlargement | Atherosclerosis | Physiological aspects | Development and progression | Dosage and administration | Research | Recombinant molecules | Fabry's disease | Alpha galactosidases | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2012, Volume 7, Issue 6, pp. e38998 - e38998
Background: Kawasaki disease is recognized as the most common cause of acquired heart disease in children in the developed world. Clinical, epidemiologic, and... 
INFANTILE PERIARTERITIS-NODOSA | ADVENTITIAL FIBROBLASTS | YOUNG-ADULTS | INTRAVENOUS GAMMA-GLOBULIN | CYTOPLASMIC INCLUSION-BODIES | CORONARY-ARTERY LESIONS | ANEURYSMS | BIOLOGY | INTRAVASCULAR ULTRASOUND | PATHOLOGY | LONG | Mucocutaneous Lymph Node Syndrome - pathology | Myofibroblasts - pathology | Aneurysm - etiology | Cell Proliferation | Myocarditis - etiology | Humans | Myocytes, Smooth Muscle - pathology | Child, Preschool | Infant | Male | Aneurysm, Ruptured - etiology | Myofibroblasts - metabolism | Lymphocytes - pathology | Mucocutaneous Lymph Node Syndrome - complications | Myofibroblasts - ultrastructure | Thrombosis - etiology | Myocarditis - metabolism | Female | Myocytes, Smooth Muscle - ultrastructure | Myocarditis - pathology | Mucocutaneous Lymph Node Syndrome - etiology | Child | Myocytes, Smooth Muscle - metabolism | Neutrophils - pathology | Heart | Virus diseases | Medical research | Medicine, Experimental | Aneurysms | Transplantation | Heart diseases | Diseases | Syngeneic grafts | Lung | Aneurysm | Stenosis | Smooth muscle | Epidemiology | Arteries | Arteritis | Kawasaki disease | Vasculitis | Vascular diseases | Aorta | Children | Lesions | Thromboembolism | Autopsies | Leukocytes (eosinophilic) | Heart transplantation | Muscles | Transplants | Leukocytes (neutrophilic) | Electron microscopy | Thrombosis | Saccule | Coronary artery disease | Fever | Morbidity | Endothelium | Studies | Myocarditis | Pathology | Disease transmission | Transmission electron microscopy | Mucocutaneous lymph node syndrome | Veins & arteries | Index Medicus | Coronary heart disease
Journal Article