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Journal Article
Circulation, ISSN 0009-7322, 08/2006, Volume 114, Issue 6, pp. 597 - 605
Advanced glycation end products ( AGEs) are proteins or lipids that become glycated after exposure to sugars. AGEs are prevalent in the diabetic vasculature... 
Advanced glycosylation end products | Diabetes mellitus | Vasculature | OXIDE SYNTHASE EXPRESSION | vasculature | CARDIAC & CARDIOVASCULAR SYSTEMS | ADVANCED GLYCOSYLATION ENDPRODUCTS | MAILLARD REACTION | LOW-DENSITY-LIPOPROTEIN | advanced glycosylation end products | diabetes mellitus | FACTOR-KAPPA-B | IN-VITRO | CULTURED ENDOTHELIAL-CELLS | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | CROSS-LINK BREAKER | EXTRACELLULAR-MATRIX | HEMATOLOGY | Basement Membrane - ultrastructure | Reactive Oxygen Species - metabolism | Receptors, Immunologic - physiology | Humans | Diabetic Angiopathies - etiology | Glycation End Products, Advanced - antagonists & inhibitors | Nitric Oxide - analysis | Endothelium, Vascular - chemistry | Basement Membrane - chemistry | Diabetic Angiopathies - physiopathology | Diabetic Angiopathies - pathology | Coronary Artery Disease - physiopathology | Extracellular Matrix - physiology | Up-Regulation - physiology | Coronary Artery Disease - pathology | Monocytes - pathology | Monocytes - physiology | Receptor for Advanced Glycation End Products | NF-kappa B - analysis | Macrophages - physiology | Glycation End Products, Advanced - physiology | Coronary Artery Disease - etiology | Glycation End Products, Advanced - analysis | Macrophages - pathology | Endothelium, Vascular - physiopathology | Nitric Oxide - physiology | Receptors, Immunologic - analysis | Basement Membrane - physiopathology | Extracellular Matrix - ultrastructure | Animals | NF-kappa B - genetics | NF-kappa B - physiology | Endothelium, Vascular - pathology | Signal Transduction - physiology | Protein Binding - physiology
Journal Article
Journal Article
Pflügers Archiv - European Journal of Physiology, ISSN 0031-6768, 6/2007, Volume 454, Issue 3, pp. 345 - 359
Journal Article
Cytokine and Growth Factor Reviews, ISSN 1359-6101, 2015, Volume 26, Issue 6, pp. 673 - 685
Abstract Atherosclerosis, a chronic inflammatory disorder of the arteries, is responsible for most deaths in westernized societies with numbers increasing at a... 
Advanced Basic Science | Therapeutic avenues | Inflammation | Cytokines | Chemokines | Atherosclerosis | Atherosclerosis - immunology | Cytokines - metabolism | Atherosclerosis - physiopathology | Humans | Endothelium, Vascular - physiopathology | Extracellular Matrix - metabolism | Inflammation - immunology | Lipoproteins - blood | Molecular Targeted Therapy | Extracellular Matrix - chemistry | Macrophages - enzymology | Animals | Atherosclerosis - therapy | Muscle, Smooth, Vascular - chemistry | Myocardial Infarction - physiopathology | Chemokines - metabolism | Mice | Matrix Metalloproteinases - metabolism | Macrophages - immunology | Chemokines - immunology | Cytokines - immunology | Endothelium, Vascular - immunology | Inflammation - physiopathology | Development and progression | Blood lipids | Health aspects | GM-CSF, granulocyte macrophage colony-stimulating factor | ECM, extracellular matrix | RCT, reverse cholesterol transport | ICAM-1, intercellular adhesion molecule-1 | LDLr, low-density lipoprotein receptor | PECAM-1, platelet endothelial cell adhesion molecule-1 | ABC, ATP-binding cassette | IL-1RA, IL-1 receptor antagonist | ADRP, adipocyte differentiation related protein | SREBP, sterol response element binding protein | wt, wild type | G-CSF, granulocyte colony-stimulating factor | PAI, plasminogen activator inhibitor | CSF, colony-stimulating factor | PRR, pattern recognition receptor | TIMP, tissue inhibitor of metalloproteinases | NPC, Niemann-Pick disease, type C | TNF, tumor necrosis factor | TL1A, TNF-like protein 1A | NLR, NOD-like receptor | SMCs, smooth muscle cells | CR, chemokine receptor | Tregs, regulatory T cells | Survey | GDF-15, growth differentiation factor-15 | NK, natural killer | TNFSF12, TNF superfamily member 12 | IL, interleukin | NLRP3, NLR family, pyrin domain containing 3 | mmLDL, minimally modified LDL | LIGHT, homologous to lymphotoxin, exhibits inducible expression, and competes with HSV glycoprotein D for herpes virus entry mediator, a receptor expressed by T lymphocytes | TF, tissue factor | Th, T-helper | MMP, matrix metalloproteinase | OxLDL, oxidized LDL | CVD, cardiovascular disease | TLR, Toll-like receptor | DCs, dendritic cells | IL-18BP, IL-18 binding protein | miRNA, micro RNA | CANTOS, Canakinumab Anti-inflammatory Thrombosis Outcomes Study | TRAIL, TNF-related apoptosis-inducing ligand | ACAT-1, acyl-CoA acyl transferase-1 | ECs, endothelial cells | TWEAK, TNF-related weak inducer of apoptosis | TGF, transforming growth factor | LFA1, lymphocyte function-associated antigen 1 | ROS, reactive oxygen species | CIRT, Cardiovascular Inflammation Reduction Trial | LDL, low-density lipoprotein | IFN, interferon | MIF, macrophage migration inhibitory factor | SR, scavenger receptor | eNOS, endothelial nitric oxide synthase | STAT1, signal transducer and activator of transcription-1 | Fn14, fibroblast growth factor-inducible 14 | VCAM-1, vascular cell adhesion molecule-1 | LXR, liver X receptors | MHC, major histocompatibility complex | CCR2, CC-chemokine receptor-2 | NOD, nucleotide-binding oligomerization domain | VLA4, very late antigen 4 | BMT, bone marrow transplantation | SR-PSOX, SR that binds phosphatidyl serine and oxidized lipoprotein | ERK, extracellular signal-regulated kinase | SOCS, suppressor of cytokine signaling | Apo, apolipoprotein | M-CSF, macrophage-colony stimulating factor | ER, endoplasmic reticulum
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2004, Volume 24, Issue 12, pp. 2320 - 2325
OBJECTIVE—Atherosclerosis is a chronic inflammatory response of the arterial wall to injury. High-mobility group box 1 (HMGB1) is a DNA-binding protein, which... 
High-mobility group box 1 | Inflammation | Cytokines | Macrophages | Atherosclerosis | atherosclerosis | RHEUMATOID-ARTHRITIS | PROTEIN-KINASE-C | PHOSPHATIDYLINOSITOL 3-KINASE | IFN-GAMMA | VASCULAR-LESIONS | NECROSIS-FACTOR-ALPHA | macrophages | SMOOTH-MUSCLE | high-mobility group box 1 | inflammation | SIGNALING PATHWAY | PERIPHERAL VASCULAR DISEASE | cytokines | TNF-ALPHA | END-PRODUCTS RAGE | HEMATOLOGY | Endothelium, Vascular - cytology | Monocytes - cytology | Humans | Myocytes, Smooth Muscle - pathology | Monocytes - metabolism | Protein Transport - physiology | HMGB1 Protein - immunology | Aorta, Thoracic - chemistry | Cytokines - physiology | Endothelium, Vascular - chemistry | HMGB1 Protein - genetics | HMGB1 Protein - secretion | RNA, Messenger - biosynthesis | HMGB1 Protein - metabolism | Muscle, Smooth, Vascular - chemistry | Aorta, Abdominal - pathology | Aorta, Abdominal - metabolism | Inflammation Mediators - physiology | Myocytes, Smooth Muscle - metabolism | Aorta, Thoracic - pathology | Muscle, Smooth, Vascular - physiology | Tunica Intima - chemistry | Macrophage Activation - physiology | Macrophages - physiology | Tunica Intima - physiology | Cells, Cultured | Myocytes, Smooth Muscle - physiology | Aorta, Thoracic - metabolism | Gene Expression Regulation - physiology | Myocytes, Smooth Muscle - chemistry | Immunohistochemistry - methods | Monocytes - chemistry | Muscle, Smooth, Vascular - pathology | Tunica Intima - pathology | Aorta, Abdominal - chemistry | Endothelium, Vascular - pathology | Arteriosclerosis - metabolism | Arteriosclerosis - pathology
Journal Article
Journal Article