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Nature Cell Biology, ISSN 1465-7392, 08/2015, Volume 17, Issue 8, pp. 994 - 1003
The use of human pluripotent stem cells for in vitro disease modelling and clinical applications requires protocols that convert these cells into relevant... 
DIRECTED DIFFERENTIATION | IN-VIVO | MOUSE | DEFINITIVE ENDODERM | GROWTH-FACTOR | HUMAN BLASTOCYSTS | STROMAL CELLS | WNT | CULTURE | LINES | CELL BIOLOGY | Coculture Techniques | Humans | Endothelial Cells - transplantation | Glycogen Synthase Kinase 3 beta | Cell Lineage - drug effects | Dose-Response Relationship, Drug | Human Umbilical Vein Endothelial Cells - physiology | Transfection | Time Factors | Gene Expression Regulation, Developmental | Transcription, Genetic | Myocytes, Smooth Muscle - drug effects | Proto-Oncogene Proteins c-sis - pharmacology | Myocytes, Smooth Muscle - transplantation | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Muscle, Smooth, Vascular - physiology | Muscle, Smooth, Vascular - drug effects | Biomarkers - metabolism | Cell Line | Induced Pluripotent Stem Cells - enzymology | Induced Pluripotent Stem Cells - drug effects | Induced Pluripotent Stem Cells - physiology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Myocytes, Smooth Muscle - enzymology | Induced Pluripotent Stem Cells - transplantation | Myocytes, Smooth Muscle - physiology | Gene Expression Profiling - methods | Muscle, Smooth, Vascular - transplantation | Glycogen Synthase Kinase 3 - metabolism | Mice, SCID | Muscle, Smooth, Vascular - cytology | Metabolomics - methods | Bone Morphogenetic Protein 4 - pharmacology | Phenotype | Animals | Wnt Signaling Pathway - drug effects | Cell Differentiation - drug effects | Mice, Inbred NOD | Protein Kinase Inhibitors - pharmacology | Endothelial Cells - enzymology | Muscle, Smooth, Vascular - enzymology | Neovascularization, Physiologic | Endothelial Cells - drug effects | Usage | Cell research | Growth | Stem cells | Muscle cells | Research | Cell differentiation | Endothelium | Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 11/2017, Volume 113, Issue 13, pp. 1664 - 1676
Aims Elevation of arginase activity has been linked to vascular dysfunction in diabetes and hypertension by a mechanism involving decreased nitric oxide (NO)... 
Obesity | Arginase | Diabetes | Vascular dysfunction | Fibrosis | CARDIAC & CARDIOVASCULAR SYSTEMS | NITRIC-OXIDE SYNTHASE | DIET-INDUCED OBESITY | AORTIC STIFFNESS | HYPERTENSIVE PATIENTS | INHIBITION | INFLAMMATION | CARDIOVASCULAR-DISEASE | SMOOTH-MUSCLE-CELLS | ARGININE BIOAVAILABILITY RATIOS | ADIPOSE-TISSUE | Blood Pressure | Vascular Diseases - prevention & control | Ornithine - blood | Obesity - drug therapy | Oxidative Stress | Diabetes Mellitus, Experimental - enzymology | Diabetes Mellitus, Type 2 - genetics | Arginase - metabolism | Diabetes Mellitus, Experimental - genetics | Endothelium, Vascular - drug effects | Male | Endothelium, Vascular - enzymology | Insulin - blood | Obesity - genetics | Vasodilation | Diabetes Mellitus, Experimental - prevention & control | Diet, High-Fat | Metabolic Syndrome - physiopathology | Arginase - antagonists & inhibitors | Dietary Sucrose | Diabetes Mellitus, Experimental - physiopathology | Metabolic Syndrome - enzymology | Genetic Predisposition to Disease | Signal Transduction | Mice, Inbred C57BL | Diabetes Mellitus, Type 2 - enzymology | Diabetes Mellitus, Type 2 - prevention & control | Endothelium, Vascular - physiopathology | Enzyme Inhibitors - pharmacology | Vascular Diseases - genetics | Obesity - physiopathology | Arginase - genetics | Vascular Diseases - physiopathology | Vascular Diseases - enzymology | Mice, Knockout | Phenotype | Animals | Diabetes Mellitus, Type 2 - physiopathology | Metabolic Syndrome - genetics | Arginine - blood | Vascular Stiffness - drug effects | Endothelium, Vascular - pathology | Metabolic Syndrome - prevention & control | Obesity - enzymology | Blood Glucose - metabolism | Nitric Oxide - metabolism | Index Medicus | Original
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 06/2009, Volume 29, Issue 6, pp. 889 - 894
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 06/2014, Volume 63, Issue 24, pp. 2734 - 2741
Objectives This study sought to investigate the effect of endothelial dysfunction on the development of cardiac hypertrophy and fibrosis. Background... 
Cardiovascular | Internal Medicine | endothelial-mesenchymal transition | diastolic dysfunction | endothelium | NADPH (nicotinamide adenine dinucleotide phosphate) oxidase | angiotensin II | CELLS | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | HEART-FAILURE | ATHEROSCLEROSIS | ANGIOTENSIN-II | PATHOPHYSIOLOGY | HYPERTROPHY | MICE | CONTRIBUTES | Heart Failure, Diastolic - genetics | Mesenchymal Stromal Cells - enzymology | Humans | Cardiomegaly - pathology | Male | Endothelium, Vascular - enzymology | Membrane Glycoproteins - physiology | Ventricular Dysfunction, Left - genetics | Ventricular Dysfunction, Left - pathology | NADPH Oxidases - genetics | Ventricular Dysfunction, Left - enzymology | Inflammation Mediators - physiology | Fibrosis - genetics | Fibrosis - enzymology | Cells, Cultured | Heart Failure, Diastolic - enzymology | Mice, Transgenic | Cardiomegaly - enzymology | Heart Failure, Diastolic - pathology | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Animals | Endothelium, Vascular - pathology | Mice | Fibrosis - pathology | Mesenchymal Stromal Cells - pathology | Cardiomegaly - genetics | NADPH Oxidases - physiology | Oxidases | Heart | Fibrosis | Stem cells | Endothelium | Hypertension | Heart failure | Genotype & phenotype | Stroke | Heart attacks | Pathogenesis | Rodents | Cardiomyocytes | Variance analysis | Index Medicus | Abridged Index Medicus
Journal Article
Molecular Cell, ISSN 1097-2765, 11/2011, Volume 44, Issue 4, pp. 545 - 558
Journal Article
Circulation Research, ISSN 0009-7330, 03/2008, Volume 102, Issue 6, pp. 669 - 676
Sphingosine 1-phosphate (S1P), an abundant lipid mediator in plasma, regulates vascular and immune cells by activating S1P receptors. In this report, we... 
Plasma S1P gradient | Shear stress | S1P lyase (Sgpl) | Sphingosine 1-phosphate (S1P) | Sphingosine kinase (Sphk) | CELLS | sphingosine kinase (Sphk) | CARDIAC & CARDIOVASCULAR SYSTEMS | EXTRACELLULAR EXPORT | LIPID PHOSPHATE PHOSPHATASES | MECHANISMS | SPHINGOLIPIDS | plasma S1P gradient | KINASE-1 | SPHINGOSINE-1-PHOSPHATE | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | sphingosine 1-phosphate (S1P) | MICE | HEMATOLOGY | BLOOD | Lysophospholipids - metabolism | Whole-Body Irradiation | Liver - enzymology | Anemia - blood | Bone Marrow Cells - enzymology | Humans | Half-Life | Stress, Mechanical | Endothelium, Vascular - enzymology | Leukopenia - blood | RNA Interference | Time Factors | Bone Marrow Transplantation | Adenoviridae - genetics | Membrane Proteins - metabolism | Sphingosine - metabolism | Aldehyde-Lyases - genetics | Disease Models, Animal | Cell Line | Transduction, Genetic | Endothelial Cells - metabolism | Liver - metabolism | Mice, Inbred C57BL | Phosphotransferases (Alcohol Group Acceptor) - deficiency | Thrombocytopenia - immunology | Cells, Cultured | Anemia - chemically induced | Phosphotransferases (Alcohol Group Acceptor) - genetics | Lysophospholipids - blood | Antibodies, Monoclonal | Bone Marrow Cells - radiation effects | Phenylhydrazines | Thrombocytopenia - blood | Mice, Knockout | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Sphingosine - analogs & derivatives | Animals | Endothelium, Vascular - metabolism | Aldehyde-Lyases - metabolism | Cell Line, Tumor | Sphingosine - blood | Mice | Genetic Vectors | Phosphoric Monoester Hydrolases - metabolism | Endothelial Cells - enzymology | Bone Marrow Cells - metabolism | Platelet Glycoprotein GPIb-IX Complex - immunology | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2013, Volume 33, Issue 4, pp. 777 - 784
OBJECTIVE—To analyze the role of toll-like receptor 4 in modulating metabolism and endothelial function. APPROACH AND RESULTS—Type 2 diabetic mice with mutated... 
systolic arterial blood pressure | endothelium-derived contracting factor | nitric oxide | proinflammatory cytokines | obesity | OXIDATIVE STRESS | ACTIVATION | MACROPHAGES | VASCULAR SMOOTH-MUSCLE | SYNTHASE | INSULIN-RESISTANCE | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | NADH/NADPH OXIDASE | HYPERTENSION | HEMATOLOGY | INNATE IMMUNITY | Mesenteric Arteries - physiopathology | Oxidative Stress | Receptors, Leptin - genetics | NADPH Oxidases - metabolism | Endothelium, Vascular - drug effects | RNA, Messenger - metabolism | Toll-Like Receptor 4 - deficiency | Carotid Arteries - enzymology | NADPH Oxidases - genetics | Disease Models, Animal | NADH, NADPH Oxidoreductases - genetics | Down-Regulation | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Toll-Like Receptor 4 - genetics | Obesity - physiopathology | Toll-Like Receptor 4 - metabolism | Vasoconstriction - drug effects | Mice, Knockout | Receptors, Leptin - deficiency | 6-Ketoprostaglandin F1 alpha - metabolism | Lipopolysaccharides - pharmacology | Mice | NADH, NADPH Oxidoreductases - antagonists & inhibitors | Mutation | Vasodilation - drug effects | Mesenteric Arteries - enzymology | Muscle, Smooth, Vascular - enzymology | Phosphorylation | Diabetes Mellitus, Type 2 - genetics | Endothelium, Vascular - enzymology | Muscle, Smooth, Vascular - physiopathology | Obesity - genetics | Dose-Response Relationship, Drug | Superoxides - metabolism | Membrane Proteins - metabolism | Aorta - physiopathology | Nitric Oxide Synthase Type III - metabolism | NADH, NADPH Oxidoreductases - metabolism | Aorta - enzymology | Vasodilator Agents - pharmacology | Mice, Inbred C57BL | Diabetes Mellitus, Type 2 - enzymology | Endothelium, Vascular - physiopathology | NADPH Oxidase 4 | Mice, Inbred C3H | NADPH Oxidase 1 | Animals | Diabetes Mellitus, Type 2 - physiopathology | Carotid Arteries - physiopathology | Obesity - enzymology | Cyclooxygenase 1 - metabolism | Nitric Oxide - metabolism | Index Medicus
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 10/2004, Volume 95, Issue 8, pp. 814 - 821
To investigate the role of adenosine formed extracellularly in vascular homeostasis, mice with a targeted deletion of the cd73/ecto-5′-nucleotidase were... 
Adenosine | Vascular inflammation | Ecto-5′-nucleotidase | Thrombosis | Transgenic mice | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTECTION | ecto-5 '-nucleotidase | NITRIC-OXIDE SYNTHASE | ALKALINE-PHOSPHATASE | HUMAN ENDOTHELIAL-CELLS | adenosine | HEART | ADENOSINE RECEPTORS | ADHESION | thrombosis | INHIBITION | transgenic mice | vascular inflammation | PERIPHERAL VASCULAR DISEASE | ATP RELEASE | HEMATOLOGY | EXPRESSION | 5'-Nucleotidase - antagonists & inhibitors | 5'-Nucleotidase - genetics | Extracellular Fluid - enzymology | Platelet Activation - physiology | Male | 5'-Nucleotidase - deficiency | Leukocytes - physiology | Endothelium, Vascular - enzymology | Chemotaxis, Leukocyte - physiology | Coronary Circulation - genetics | Adenosine Triphosphate - pharmacology | Adenosine - biosynthesis | Reperfusion | Carotid Arteries - enzymology | Myocardial Ischemia - physiopathology | Female | Muscle, Skeletal - blood supply | Cyclic AMP - blood | Gene Targeting | Coronary Vessels - enzymology | Adenosine Monophosphate - metabolism | Mice, Transgenic | Receptors, Purinergic P1 - physiology | Myocardial Ischemia - enzymology | Ischemia - physiopathology | Adenosine Triphosphate - analogs & derivatives | Mice, Knockout | Animals | Cell Adhesion - physiology | Adenosine - physiology | 5'-Nucleotidase - physiology | Carotid Arteries - pathology | Mice | Blood Coagulation - physiology | Inflammation - enzymology | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 03/2016, Volume 118, Issue 5, pp. 856 - 866
Journal Article
Science, ISSN 0036-8075, 6/2002, Volume 296, Issue 5577, pp. 2391 - 2394
Journal Article