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Acta Neuropathologica, ISSN 0001-6322, 9/2011, Volume 122, Issue 3, pp. 293 - 311
...), leading to disturbed cerebral blood flow and cognitive dysfunction, posing the question how cerebrovascular pathology contributes to the pathology of AD... 
Pathology | Neurosciences | Congophilic amyloid angiopathy | Medicine & Public Health | Alzheimer’s disease | Astrocytes | Cerebral glucose metabolism | Alzheimer's disease | MICROVASCULAR PATHOLOGY | ALZHEIMERS-DISEASE | NEUROVASCULAR MECHANISMS | PATHOLOGY | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | CLINICAL NEUROLOGY | GLUCOSE | MOUSE MODEL | LACTATE | A-BETA | SMOOTH-MUSCLE-CELLS | ANGIOPATHY | Microdialysis - methods | Humans | Astrocytes - pathology | Dystroglycans - metabolism | Glucose Transporter Type 1 - metabolism | Muscle, Smooth - ultrastructure | Glial Fibrillary Acidic Protein - metabolism | Microscopy, Electron, Scanning - methods | Amyloid beta-Peptides - genetics | Amyloid beta-Peptides - metabolism | Cell Culture Techniques | Disease Models, Animal | Endothelium - pathology | Mice, Transgenic | Cerebral Arteries - ultrastructure | Basement Membrane - metabolism | Disease Progression | Symporters - metabolism | Astrocytes - ultrastructure | Blood-Brain Barrier - pathology | Cerebral Arteries - metabolism | Brain - pathology | Glucose - metabolism | Plaque, Amyloid - metabolism | Mice | Astrocytes - metabolism | Blood-Brain Barrier - physiopathology | Basement Membrane - pathology | Cerebral Amyloid Angiopathy - complications | Monocarboxylic Acid Transporters - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Hemorrhage - etiology | Amyloid beta-Protein Precursor - metabolism | Lactase - metabolism | Cerebral Amyloid Angiopathy - genetics | Astrocytes - drug effects | Plaque, Amyloid - pathology | Gene Expression Regulation - genetics | Hemorrhage - metabolism | Muscle, Smooth - metabolism | Cerebrovascular Disorders - etiology | Cerebral Amyloid Angiopathy - pathology | Animals | Endothelium - metabolism | Glucose Transporter Type 1 - genetics | Symporters - genetics | Cerebral Arteries - pathology | Laminin - metabolism | Monocarboxylic Acid Transporters - genetics | Cerebrovascular Disorders - pathology | Hemorrhage - pathology | Muscle, Smooth - pathology | Glucose transporter | Leakage | Brain | Neurodegenerative diseases | Cognitive ability | Transgenic mice | Blood vessels | Data processing | Smooth muscle | beta -Amyloid | Glucose transport | Metabolism | Amyloid precursor protein | Blood-brain barrier | Cerebral blood flow | Lactic acid | Mutation | Original Paper
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2006, Volume 116, Issue 10, pp. 2610 - 2621
Inhibitors of VEGF signaling can block angiogenesis and reduce tumor vascularity, but little is known about the reversibility of these changes after treatment... 
MEDICINE, RESEARCH & EXPERIMENTAL | NERVE REGENERATION | BASAL LAMINA | ANGIOGENESIS INHIBITORS | BLOOD-VESSELS | EXTRACELLULAR-MATRIX | MUSCLE ACTIN EXPRESSION | ENDOTHELIAL SPROUTS | BASEMENT-MEMBRANES | GROWTH IN-VIVO | ESTABLISHED TUMORS | Basement Membrane - drug effects | Pericytes - drug effects | Blood Vessels - metabolism | Blood Vessels - pathology | Actins - metabolism | Endothelium, Vascular - drug effects | Vascular Endothelial Growth Factor A - metabolism | Basement Membrane - pathology | Carcinoma, Lewis Lung - blood supply | Neovascularization, Pathologic - pathology | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Insulinoma - blood supply | Pericytes - pathology | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Organic Chemicals - pharmacology | Angiogenesis Inhibitors - therapeutic use | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Imidazoles - therapeutic use | Collagen Type IV - immunology | Receptor, Platelet-Derived Growth Factor beta - metabolism | Collagen Type IV - metabolism | Antibodies, Monoclonal - pharmacology | Mice, Inbred C57BL | Pericytes - metabolism | Insulinoma - drug therapy | Angiogenesis Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Mice, Transgenic | Neoplasms - blood supply | Treatment Outcome | Imidazoles - pharmacology | Basement Membrane - metabolism | Carcinoma, Lewis Lung - drug therapy | Neoplasms - drug therapy | Indazoles - pharmacology | Animals | Endothelium, Vascular - metabolism | Neovascularization, Pathologic - drug therapy | Blood Vessels - drug effects | Carcinoma, Lewis Lung - pathology | Matrix Metalloproteinase Inhibitors | Endothelium, Vascular - pathology | Insulinoma - pathology | Mice | Neovascularization, Pathologic - metabolism | Neoplasms - pathology | Indazoles - therapeutic use | Care and treatment | Research | Vascular endothelial growth factor | Cancer
Journal Article
Diabetes, ISSN 0012-1797, 09/2010, Volume 59, Issue 9, pp. 2297 - 2305
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 02/2014, Volume 171, Issue 3, pp. 595 - 617
Perivascular adipose tissue (PVAT) is an active endocrine and paracrine organ that modulates vascular function, with implications for the pathophysiology of cardiovascular disease (CVD... 
AMP‐activated protein kinase | atherosclerosis | adiponectin | perivascular fat | vascular reactivity | metformin | adipocytes | diabetes | leptin | hypertension | AMP-activated protein kinase | STIMULATES GLUCOSE-UTILIZATION | NITRIC-OXIDE SYNTHASE | ENDOTHELIUM-DEPENDENT RELAXATION | ENDOPLASMIC-RETICULUM STRESS | BROWN ADIPOSE-TISSUE | LOWERS BLOOD-PRESSURE | TUMOR-NECROSIS-FACTOR | AORTIC SMOOTH-MUSCLE | PHARMACOLOGY & PHARMACY | INDUCED CARDIAC-HYPERTROPHY | NF-KAPPA-B | Enzyme Activators - pharmacology | AMP-Activated Protein Kinases - metabolism | Vascular Diseases - pathology | Adipose Tissue, White - immunology | Muscle, Smooth, Vascular - metabolism | Enzyme Activators - therapeutic use | Humans | Adipose Tissue, White - metabolism | Muscle, Smooth, Vascular - immunology | Endothelium, Vascular - drug effects | Male | Autocrine Communication - drug effects | Adiposity | Female | Vascular Diseases - drug therapy | Adipose Tissue, Brown - immunology | Endothelium, Vascular - immunology | Muscle, Smooth, Vascular - drug effects | Adipose Tissue, White - pathology | Vascular Diseases - immunology | Enzyme Activation - drug effects | Adipose Tissue, Brown - pathology | Cardiovascular Agents - pharmacology | Cardiovascular Agents - therapeutic use | Muscle, Smooth, Vascular - pathology | Animals | Signal Transduction - drug effects | Endothelium, Vascular - metabolism | Models, Biological | Paracrine Communication - drug effects | Endothelium, Vascular - pathology | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Vascular Diseases - metabolism | Adipose Tissue, White - drug effects | Blood circulation disorders | Protein kinases | Leptin | Atherosclerosis | Kinases | Metabolic disorders | Muscular system | Reactive oxygen species | Adipose tissue | Paracrine signalling | Smooth muscle | Adipocytes | Contraction | Protein turnover | Vascular diseases | Diabetes mellitus (non-insulin dependent) | Obesity | AMP | Cytokines | Therapeutic applications | Blood vessels | Vascular system | Muscle contraction | Protein kinase | Arteriosclerosis | Stromal cells | Metformin | Cardiovascular diseases | Thiazolidinediones | Cell migration | Review
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 10/2013, Volume 128, Issue 18, pp. 2026 - 2038
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2015, Volume 83, pp. 112 - 121
Abstract Ageing is associated with functional, structural and mechanical changes in arteries that closely resemble the vascular alterations in hypertension... 
Cardiovascular | Vascular remodeling | Endothelial dysfunction | Oxidative stress | Mitochondria | CARDIAC & CARDIOVASCULAR SYSTEMS | ENDOPLASMIC-RETICULUM STRESS | AGE-RELATED DECLINE | HYDROGEN-SULFIDE | BLOOD-PRESSURE | CELL BIOLOGY | RENIN-ANGIOTENSIN SYSTEM | FREE-RADICAL THEORY | NITRIC-OXIDE | SMOOTH-MUSCLE-CELLS | NF-KAPPA-B | Shc Signaling Adaptor Proteins - metabolism | Oxidative Stress | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Transforming Growth Factor beta1 - metabolism | Forkhead Transcription Factors - metabolism | Myocardium - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Cell Cycle Proteins - genetics | Hypertension - genetics | Signal Transduction | Cell Cycle Proteins - metabolism | Gene Expression Regulation | Myocardium - pathology | Transforming Growth Factor beta1 - genetics | Transcription Factors - genetics | Forkhead Transcription Factors - genetics | Hypertension - pathology | Aging - pathology | Hypertension - metabolism | Transcription Factors - metabolism | Animals | Shc Signaling Adaptor Proteins - genetics | Src Homology 2 Domain-Containing, Transforming Protein 1 | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Mitogen-Activated Protein Kinases - genetics | Aged | Aging - metabolism | Mitogen-Activated Protein Kinases - metabolism | Hypertension | Review
Journal Article