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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2009, Volume 106, Issue 25, pp. 10272 - 10277
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2012, Volume 122, Issue 6, pp. 1973 - 1990
EC activation and dysfunction have been linked to a variety of vascular inflammatory disease states. The function of microRNAs (miRNAs) in vascular EC... 
CELL-ADHESION MOLECULE-1 | TRANSCRIPTION FACTORS | MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | DEPENDENT INDUCTION | LEUKOCYTE RECRUITMENT | HUMAN-ENDOTHELIAL-CELLS | ACUTE MYELOID-LEUKEMIA | PROGNOSTIC-SIGNIFICANCE | EXPRESSION PROFILES | SIGNAL-TRANSDUCTION PATHWAY | alpha Karyopherins - metabolism | Endotoxemia - therapy | Leukocytes - pathology | Vasculitis - metabolism | Acute Lung Injury - therapy | Endotoxemia - genetics | Humans | NF-kappa B - immunology | Critical Illness | Acute Lung Injury - genetics | MicroRNAs - metabolism | NF-kappa B - metabolism | Endotoxemia - pathology | Vasculitis - immunology | Leukocytes - immunology | Vasculitis - genetics | alpha Karyopherins - immunology | E-Selectin - genetics | Endotoxemia - immunology | Vascular Cell Adhesion Molecule-1 - genetics | Acute Lung Injury - metabolism | Vascular Cell Adhesion Molecule-1 - immunology | Vasculitis - therapy | Lipopolysaccharides - toxicity | Endothelial Cells - metabolism | E-Selectin - metabolism | E-Selectin - immunology | MicroRNAs - immunology | Acute Lung Injury - pathology | Endothelial Cells - immunology | Animals | NF-kappa B - genetics | alpha Karyopherins - genetics | Acute Lung Injury - immunology | Mice | MicroRNAs - genetics | Endothelial Cells - pathology | Endotoxemia - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Leukocytes - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 12/2017, Volume 21, Issue 12, pp. 3178 - 3189
The purpose of the present study was to investigate the effect of salidroside (Sal) on myocardial injury in lipopolysaccharide ( LPS )‐induced endotoxemic in... 
H9C2 | myocardial injury | ROS | mTOR | salidroside | 3K/Akt | LPS | PI3K/Akt/mTOR | MEDICINE, RESEARCH & EXPERIMENTAL | OXIDATIVE STRESS | PHOSPHATIDYLINOSITOL 3-KINASE | RATS | ACUTE LUNG INJURY | ISCHEMIA REPERFUSION INJURY | NLRP3 INFLAMMASOME | CELL BIOLOGY | MITOCHONDRIAL ROS | SIGNALING PATHWAY | TANSHINONE IIA SULFONATE | KAPPA-B PATHWAY | Superoxide Dismutase - genetics | Reactive Oxygen Species - metabolism | TOR Serine-Threonine Kinases - metabolism | Tumor Necrosis Factor-alpha - genetics | Endotoxemia - chemically induced | Endotoxemia - pathology | Interleukin-1beta - genetics | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Lipopolysaccharides | Myocardial Reperfusion Injury - pathology | Dexamethasone - pharmacology | TOR Serine-Threonine Kinases - genetics | Interleukin-1beta - metabolism | Myocardium - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Interleukin-6 - metabolism | Myocardial Reperfusion Injury - genetics | Phenols - pharmacology | Glutathione Peroxidase - metabolism | Interleukin-6 - genetics | Signal Transduction | Catalase - genetics | Endotoxemia - drug therapy | Rats | Cardiotonic Agents - pharmacology | Rats, Sprague-Dawley | Glutathione Peroxidase - genetics | Myocytes, Cardiac - pathology | Reactive Oxygen Species - antagonists & inhibitors | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - metabolism | Hemodynamics - drug effects | Nitric Oxide Synthase Type II - metabolism | Tumor Necrosis Factor-alpha - metabolism | Endotoxemia - genetics | Glutathione - metabolism | Myocardial Reperfusion Injury - chemically induced | Phosphatidylinositol 3-Kinases - metabolism | Proto-Oncogene Proteins c-akt - genetics | TOR Serine-Threonine Kinases - antagonists & inhibitors | Myocardial Reperfusion Injury - drug therapy | Superoxide Dismutase - metabolism | Cell Line | Glucosides - pharmacology | Gene Expression Regulation | Myocardium - pathology | Catalase - metabolism | Phosphatidylinositol 3-Kinases - genetics | Animals | Nitric Oxide Synthase Type II - genetics | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Mitogens | Cysteine | Heart | Flow cytometry | Creatine kinase | Interleukin | AKT protein | Superoxide dismutase | Creatine | Interleukin 6 | Antioxidants | Proteins | Salidroside | Signal transduction | Catalase | Glutathione transferase | Rodents | Interleukin 1 | Tumor necrosis factor-TNF | Peroxidase | Inhibition | Glutathione | Glutathione peroxidase | Enzymes | NF-κB protein | Dexamethasone | Cytokines | Medical treatment | Lactate dehydrogenase | Pharmacology | Embryos | Chemical compounds | L-Lactate dehydrogenase | 1-Phosphatidylinositol 3-kinase | Cytometry | Signaling | N-Acetyl-L-cysteine | In vivo methods and tests | Lactic acid | Cyclooxygenase-2 | Cardiovascular diseases | Tumors | Index Medicus | PI3K | Akt | Original
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 12/2017, Volume 21, Issue 12, pp. 3705 - 3717
Oestrogens have been reported to attenuate acute inflammation in sepsis. In this study, the effects of long‐term oestrogen replacement with 17β‐oestradiol (E 2... 
oestrogen | septic shock | autophagy | HSP70 | inflammation | endotoxaemia | HO‐1 | ovariectomy | multiple organ dysfunction syndrome | oxidative stress | HO-1 | MEDICINE, RESEARCH & EXPERIMENTAL | LUNG INJURY | SEPSIS | ANTIOXIDANT SUPPLEMENTATION | CELL BIOLOGY | PLASMINOGEN-ACTIVATOR | ESTROGEN PROTECTS | GENE-EXPRESSION | ETHYL PYRUVATE | INFLAMMATORY RESPONSE | NF-KAPPA-B | Tumor Necrosis Factor-alpha - metabolism | Heme Oxygenase-1 - metabolism | Cerebrovascular Disorders - drug therapy | Endotoxemia - genetics | Tumor Necrosis Factor-alpha - genetics | Cerebrovascular Disorders - metabolism | Endotoxemia - pathology | Transcription Factor RelA - genetics | Autophagy - drug effects | Lipopolysaccharides | Heme Oxygenase-1 - genetics | Plasminogen Activator Inhibitor 1 - metabolism | Rhabdomyolysis - drug therapy | Rhabdomyolysis - pathology | Female | Renal Insufficiency - drug therapy | Autophagy - genetics | Plasminogen Activator Inhibitor 1 - genetics | Interleukin-6 - metabolism | Estradiol - pharmacology | Renal Insufficiency - genetics | Autophagy-Related Protein 12 - genetics | Drug Administration Schedule | Interleukin-6 - genetics | Ovariectomy | Anti-Inflammatory Agents - pharmacology | Endotoxemia - drug therapy | Gene Expression Regulation | Rats | HSP70 Heat-Shock Proteins - genetics | Renal Insufficiency - metabolism | Autophagy-Related Protein 12 - metabolism | Rats, Sprague-Dawley | HSP70 Heat-Shock Proteins - metabolism | Rhabdomyolysis - metabolism | Animals | Nitric Oxide Synthase Type II - genetics | Transcription Factor RelA - metabolism | Renal Insufficiency - pathology | Rhabdomyolysis - genetics | Cerebrovascular Disorders - genetics | Endotoxemia - metabolism | Cerebrovascular Disorders - pathology | Nitric Oxide Synthase Type II - metabolism | Index Medicus | Original
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2014, Volume 211, Issue 7, pp. 1333 - 1347
Journal Article
Diabetes, ISSN 0012-1797, 01/2010, Volume 59, Issue 1, pp. 172 - 181
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2013, Volume 110, Issue 9, pp. 3507 - 3512
Journal Article
Journal of Immunology, ISSN 0022-1767, 01/2014, Volume 192, Issue 1, pp. 385 - 393
Journal Article
Journal Article