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by Locke, Adam E and Kahali, Bratati and Berndt, Sonja I and Justice, Anne E and Pers, Tune H and Day, Felix R and Powell, Corey and Vedantam, Sailaja and Buchkovich, Martin L and Yang, Jian and Croteau-Chonka, Damien C and Esko, Tonu and Fall, Tove and Ferreira, Teresa and Gustafsson, Stefan and Kutalik, Zoltán and Luan, Jian’an and Mägi, Reedik and Randall, Joshua C and Winkler, Thomas W and Wood, Andrew R and Workalemahu, Tsegaselassie and Faul, Jessica D and Smith, Jennifer A and Hua Zhao, Jing and Zhao, Wei and Chen, Jin and Fehrmann, Rudolf and Hedman, Åsa K and Karjalainen, Juha and Schmidt, Ellen M and Absher, Devin and Amin, Najaf and Anderson, Denise and Beekman, Marian and Bolton, Jennifer L and Bragg-Gresham, Jennifer L and Buyske, Steven and Demirkan, Ayse and Deng, Guohong and Ehret, Georg B and Feenstra, Bjarke and Feitosa, Mary F and Fischer, Krista and Goel, Anuj and Gong, Jian and Jackson, Anne U and Kanoni, Stavroula and Kleber, Marcus E and Kristiansson, Kati and Lim, Unhee and Lotay, Vaneet and Mangino, Massimo and Mateo Leach, Irene and Medina-Gomez, Carolina and Medland, Sarah E and Nalls, Michael A and Palmer, Cameron D and Pasko, Dorota and Pechlivanis, Sonali and Peters, Marjolein J and Prokopenko, Inga and Shungin, Dmitry and Stančáková, Alena and Strawbridge, Rona J and Ju Sung, Yun and Tanaka, Toshiko and Teumer, Alexander and Trompet, Stella and van der Laan, Sander W and van Setten, Jessica and Van Vliet-Ostaptchouk, Jana V and Wang, Zhaoming and Yengo, Loïc and Zhang, Weihua and Isaacs, Aaron and Albrecht, Eva and Ärnlöv, Johan and Arscott, Gillian M and Attwood, Antony P and Bandinelli, Stefania and Barrett, Amy and Bas, Isabelita N and Bellis, Claire and Bennett, Amanda J and Berne, Christian and Blagieva, Roza and Blüher, Matthias and Böhringer, Stefan and Bonnycastle, Lori L and Böttcher, Yvonne and Boyd, Heather A and Bruinenberg, Marcel and Caspersen, Ida H and Ida Chen, Yii-Der and Clarke, Robert and Warwick Daw, E and de Craen, Anton J. M and Delgado, Graciela and Dimitriou, Maria and ... and The PAGE Consortium and The International Endogene Consortium and The MAGIC Investigators and The ReproGen Consortium and The CKDGen Consortium and The ADIPOGen Consortium and The ICBP and The CARDIOGRAMplusC4D Consortium and The GLGC and The GENIE Consortium and The AGEN-BMI Working Group and The MIGen Consortium and The MuTHER Consortium and The LifeLines Cohort Study and ADIPOGen Consortium and PAGE Consortium and LifeLines Cohort Study and MIGen Consortium and CARDIOGRAMplusC4D Consortium and MuTHER Consortium and ICBP and CKDGen Consortium and Int Endogene Consortium and GENIE Consortium and MAGIC Investigators and ReproGen Consortium and AGEN-BMI Working Grp and GLGC and International Endogene Consortium and AGEN-BMI Working Group
Nature (London), ISSN 1476-4687, 2015, Volume 518, Issue 7538, pp. 197 - 206
Obesity is heritable and predisposes to many diseases. To understand the genetic basis of obesity better, here we conduct a genome-wide association study and Metabochip meta-analysis of body mass index (BMI... 
INDIVIDUALS | PROVIDES INSIGHTS | METAANALYSIS | VARIANTS | MULTIDISCIPLINARY SCIENCES | HIPPOCAMPAL | GLYCEMIC TRAITS | ARCHITECTURE | TOPIRAMATE | LOCI | GENOME-WIDE ASSOCIATION | Body Mass Index | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Age Factors | Humans | Male | Continental Population Groups - genetics | Adiposity - genetics | Obesity - genetics | Europe - ethnology | Obesity - metabolism | Insulin - metabolism | Synapses - metabolism | Polymorphism, Single Nucleotide - genetics | Female | Glutamic Acid - metabolism | Energy Metabolism - genetics | Adipogenesis - genetics | Insulin Secretion | Quantitative Trait Loci - genetics | Body mass index | Obesity | Genetic research | Genetic aspects | Research | Diabetes | Gene expression | Health aspects | Studies | Genomics | Genetics | Biology | Genomes | Metabolic disorders | Meta-analysis | Medical and Health Sciences | Insulin: metabolism | Medicin och hälsovetenskap | Obesity: genetics | Europe: ethnology | Glutamic Acid: metabolism | Genetic Predisposition to Disease: genetics | Single Nucleotide: genetics | Klinisk medicin | Synapses: metabolism | Adiposity: genetics | Obesity: metabolism | Clinical Medicine | Energy Metabolism: genetics | Endokrinologi och diabetes | Quantitative Trait Loci: genetics | Insulin: secretion | Adipogenesis: genetics | Polymorphism | Continental Population Groups: genetics | Endocrinology and Diabetes
Journal Article
Nature (London), ISSN 1476-4687, 2012, Volume 485, Issue 7396, pp. 123 - 127
Journal Article
Journal of Inherited Metabolic Disease, ISSN 0141-8955, 4/2008, Volume 31, Issue 2, pp. 230 - 239
Creatine deficiency syndromes, either due to AGAT, GAMT or SLC6A8 deficiencies, lead to a complete absence, or a very strong decrease, of creatine within the... 
Biochemistry, general | Human Genetics | Pediatrics | Internal Medicine | Medicine & Public Health | Metabolic Diseases | GUANIDINOACETATE METHYLTRANSFERASE DEFICIENCY | LINKED MENTAL-RETARDATION | ARGININE RESTRICTION | ENERGY HOMEOSTASIS | ENDOCRINOLOGY & METABOLISM | GENETICS & HEREDITY | IN-SITU HYBRIDIZATION | MAGNETIC-RESONANCE | CLINICAL CHARACTERISTICS | RAT-BRAIN | TRANSPORTER GENE SLC6A8 | INBORN ERROR | Glycine - analogs & derivatives | Glycine - metabolism | Prognosis | Guanidinoacetate N-Methyltransferase - genetics | Humans | Brain - enzymology | Amidinotransferases - genetics | Developmental Disabilities - genetics | Intellectual Disability - genetics | Membrane Transport Proteins - deficiency | Amino Acid Metabolism, Inborn Errors - genetics | Developmental Disabilities - enzymology | Membrane Transport Proteins - genetics | Movement Disorders - enzymology | Intellectual Disability - enzymology | Creatine - deficiency | Amidinotransferases - deficiency | Genetic Predisposition to Disease | Language Development Disorders - genetics | Speech Disorders - genetics | Language Development Disorders - enzymology | Phenotype | Animals | Guanidinoacetate N-Methyltransferase - deficiency | Movement Disorders - congenital | Speech Disorders - enzymology | Movement Disorders - genetics | Amino Acid Metabolism, Inborn Errors - enzymology | Creatine | Central nervous system
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 2016, Volume 354, Issue 6314, pp. 857 - 861
Crop leaves in full sunlight dissipate damaging excess absorbed light energy as heat. When sunlit leaves are shaded by clouds or other leaves, this protective... 
ARABIDOPSIS-THALIANA | ENERGY | PHOTOINHIBITION | FLUORESCENCE | CARBON GAIN | MULTIDISCIPLINARY SCIENCES | EXCESS LIGHT | PHOTOSYSTEM-II | ZEAXANTHIN | PLANTS | XANTHOPHYLL CYCLE | Bioengineering | Darkness | Light-Harvesting Protein Complexes - genetics | Plants, Genetically Modified - radiation effects | Plants, Genetically Modified - growth & development | Crops, Agricultural - growth & development | Light-Harvesting Protein Complexes - metabolism | Magnoliopsida - genetics | RNA, Messenger - metabolism | Magnoliopsida - metabolism | Magnoliopsida - radiation effects | Arabidopsis Proteins - metabolism | Crops, Agricultural - radiation effects | Photosystem II Protein Complex - genetics | Arabidopsis Proteins - genetics | Carbon Dioxide - metabolism | Oxidoreductases - metabolism | Plants, Genetically Modified - genetics | Oxidoreductases - genetics | Tobacco - metabolism | RNA, Messenger - genetics | Tobacco - growth & development | Tobacco - radiation effects | Photosystem II Protein Complex - metabolism | Plants, Genetically Modified - metabolism | Plant Leaves - metabolism | Plant Leaves - growth & development | Tobacco - genetics | Sunlight | Crops, Agricultural - genetics | Photosynthesis | Crops, Agricultural - metabolism | Magnoliopsida - growth & development | Phytochemistry | Crop yields | Photosynthesis research | Environmental aspects | Physiological aspects | Tobacco (Plant) | Methods | Flowers & plants | Sun | Crops | Light | Productivity | Leaves | Clouds | Dissipation | Plants (organisms)
Journal Article
Plant biotechnology journal, ISSN 1467-7644, 2011, Volume 9, Issue 8, pp. 874 - 883
Summary Increasing the energy density of biomass by engineering the accumulation of triacylglycerols (TAGs) in vegetative tissues is synergistic with efforts... 
ADP‐glucose pyrophosphorylase | carbon partitioning | triacylglycerol | WRINKLED1 | biofuels | starch | primary metabolism | Triacylglycerol | Starch | ADP-glucose pyrophosphorylase | Carbon partitioning | Primary metabolism | Biofuels | PROTEIN | SUGAR | PLANT SCIENCES | FATTY-ACID BIOSYNTHESIS | METABOLISM | THALIANA | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | DEVELOPING SEEDS | GENE-EXPRESSION | ACCUMULATION | Starch - biosynthesis | Triglycerides - biosynthesis | Plants, Genetically Modified - growth & development | Arabidopsis - growth & development | Genes, Plant | Acetyl-CoA Carboxylase - genetics | Arabidopsis Proteins - metabolism | Glucosyltransferases - metabolism | RNA Interference | Isoenzymes - metabolism | Brassica - genetics | Glucose-1-Phosphate Adenylyltransferase - metabolism | Gene Expression Regulation, Plant | Glucosyltransferases - genetics | Genetic Engineering - methods | Carbon - metabolism | Arabidopsis Proteins - genetics | Mutagenesis, Site-Directed | Seeds - metabolism | Plants, Genetically Modified - genetics | Isoenzymes - genetics | Acyl Carrier Protein - metabolism | Carbohydrate Metabolism | Electroporation | Transcription Factors - genetics | Arabidopsis - metabolism | Arabidopsis - genetics | Transcription Factors - metabolism | Microscopy, Confocal | Phenotype | DNA, Bacterial - genetics | Plants, Genetically Modified - metabolism | Acyl Carrier Protein - genetics | Plant Oils - metabolism | Agrobacterium - genetics | Acetyl-CoA Carboxylase - metabolism | Glucose-1-Phosphate Adenylyltransferase - genetics | Biomass energy | Carbohydrate metabolism | Biotin | Triglycerides | Fatty acids | Monosaccharides | Arabidopsis thaliana | Proteins | Synthesis | Physiological aspects | Genetic engineering | Universities and colleges | Sugars
Journal Article
American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, ISSN 0363-6119, 04/2008, Volume 294, Issue 4, pp. 1185 - 1196
Two recent, large whole-genome association studies (GWAS) in European populations have associated a ∼47-kb region that contains part of the FTO gene with high... 
Obesity | Adipose tissue | Hypothalamus | CUTL1 | CCAAT DISPLACEMENT PROTEIN | DNA-BINDING | PHYSIOLOGY | FTO GENE | CUT EXPRESSION | adipose tissue | DROSOPHILA | TRANSCRIPTION FACTOR | WING MARGIN | obesity | hypothalamus | FAT MASS | GENOME-WIDE ASSOCIATION | Bardet-Biedl Syndrome - metabolism | Homeodomain Proteins - metabolism | Humans | Alpha-Ketoglutarate-Dependent Dioxygenase FTO | Male | RNA, Messenger - metabolism | Obesity - genetics | Embryo, Mammalian - metabolism | Mixed Function Oxygenases | Adipose Tissue - metabolism | Transfection | Gene Expression Regulation, Developmental | Oxo-Acid-Lyases - genetics | Bardet-Biedl Syndrome - genetics | Fasting - metabolism | Oxo-Acid-Lyases - metabolism | Energy Metabolism - genetics | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Disease Models, Animal | Eating | Mice, Inbred C57BL | Stromal Cells - metabolism | Cells, Cultured | Gene Expression Regulation | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Leptin - genetics | Adiposity - genetics | Homeodomain Proteins - genetics | Obesity - metabolism | Proteins - genetics | Animals | Hypothalamus - metabolism | Proteins - metabolism | Adipocytes - metabolism | Adaptor Proteins, Signal Transducing - genetics | Hypothermia, Induced | Mice, Obese | Mice | Polymorphism, Single Nucleotide | Adaptor Proteins, Signal Transducing - metabolism
Journal Article
Cell metabolism, ISSN 1550-4131, 2011, Volume 14, Issue 3, pp. 324 - 338
.... Inhibition of BDNF signaling by genetic knockout or dominant-negative trkB reversed this phenotype... 
OBESITY | COLD-EXPOSURE | ADAPTIVE THERMOGENESIS | NEUROTROPHIC FACTOR | ENDOCRINOLOGY & METABOLISM | DIET-INDUCED THERMOGENESIS | ADULT HUMANS | MICE LACKING | REGULATES ENERGY-BALANCE | ADIPOSE-TISSUE | VENTROMEDIAL HYPOTHALAMUS | CELL BIOLOGY | Brain-Derived Neurotrophic Factor - antagonists & inhibitors | Brain-Derived Neurotrophic Factor - genetics | Adenoviridae | Adipose Tissue, White - metabolism | Ion Channels - genetics | Adipocytes - cytology | Male | Gene Expression Profiling | Mitochondrial Proteins - genetics | Receptor, trkB - genetics | MicroRNAs - pharmacology | Obesity - genetics | Brain-Derived Neurotrophic Factor - deficiency | DNA-Binding Proteins - metabolism | Thermogenesis - physiology | Adipose Tissue, White - cytology | Mitochondria - genetics | Mitochondrial Proteins - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Mitochondria - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Gene Knockout Techniques | Obesity - metabolism | Transcription Factors - metabolism | Motor Activity - genetics | Adipose Tissue, Brown - cytology | Phenotype | Animals | Hypothalamus - metabolism | Maze Learning | Hypothalamus - cytology | Ion Channels - metabolism | Adipocytes - metabolism | Adipose Tissue, Brown - metabolism | Mice | Uncoupling Protein 1 | Receptor, trkB - metabolism | Obesity | Neurosciences | Public health | Medical genetics | Enrichment | Brain-derived neurotrophic factor | Thermogenesis | Cognitive ability | Data processing | TrkB receptors | Adipocytes | Hypothalamus | Environmental effects
Journal Article
PloS one, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e54339
Background: Mitochondrial dysfunction is associated with the development and progression of age-related macular degeneration (AMD). Recent studies using... 
DRUSEN FORMATION | POPULATION | OXIDATIVE STRESS | SAUDI PATIENTS | MTDNA | MULTIDISCIPLINARY SCIENCES | FACTOR-H POLYMORPHISM | RISK | MYOSIN VIIA | DYSFUNCTION | HAPLOGROUPS | Haplotypes | Retinal Pigment Epithelium - metabolism | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Humans | Adenosine Triphosphate - biosynthesis | DNA, Mitochondrial - genetics | Mitochondria - genetics | Myosins - metabolism | Epithelial Cells - cytology | Complement C3 - genetics | Extracellular Matrix Proteins - metabolism | Hybrid Cells - metabolism | Gene Expression | DNA, Mitochondrial - metabolism | Complement C3 - metabolism | Reactive Nitrogen Species - metabolism | Extracellular Matrix Proteins - genetics | Lactic Acid - metabolism | Cells, Cultured | Mitochondria - metabolism | Signal Transduction - genetics | Myosins - genetics | Macular Degeneration - metabolism | Complement Factor H - metabolism | Hybrid Cells - pathology | Macular Degeneration - genetics | Models, Biological | Complement Factor H - genetics | Retinal Pigment Epithelium - cytology | Macular Degeneration - pathology | Macular degeneration | Cellular proteins | Care and treatment | Genetic variation | Development and progression | Genetic aspects | Mitochondrial DNA | Health aspects | Glaucoma | Diabetic retinopathy | Cybrids | Disease | Physicians | Epithelial cells | Genes | Retina | Population studies | Arthritis | Proteins | Signal transduction | Mitochondria | Pathways | Energy | Aging | Age | Deoxyribonucleic acid--DNA | Oxygen | Cell division | Metabolism | Gene expression | Age related diseases | Signaling | Retinal degeneration | Lactic acid | Deoxyribonucleic acid | DNA
Journal Article