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1. Full Text D2R striatopallidal neurons inhibit both locomotor and drug reward processes
by Durieux, PF and Bearzatto, B and Guiducci, S and Buch, T and Waisman, A and Zoli, M and Schiffmann, SN and d'Exaerde, ADK
NATURE NEUROSCIENCE, ISSN 1097-6256, 04/2009, Volume 12, Issue 4, pp. 393 - 395
The specific functions of dopamine D-2 receptor-positive (D2R) striatopallidal neurons remain poorly understood. Using a genetic mouse model, we found that... 
SYSTEM | GENE-EXPRESSION | ABLATION | NEUROSCIENCES | BASAL GANGLIA | ADDICTION | Diphtheria Toxin - pharmacology | Tyrosine 3-Monooxygenase - metabolism | Locomotion - genetics | Enkephalins - metabolism | Receptors, Dopamine D2 - deficiency | Conditioning, Operant - physiology | Receptors, Dopamine D2 - metabolism | Corpus Striatum - cytology | Enkephalins - genetics | Time Factors | Receptors, Adenosine A2 - genetics | Protein Binding - drug effects | Neurons - physiology | Neurons - drug effects | Locomotion - drug effects | Dopamine Uptake Inhibitors - pharmacology | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Amphetamine - pharmacology | Intercellular Signaling Peptides and Proteins - genetics | Glutamate Decarboxylase - genetics | Mice, Transgenic | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Autoradiography | Heparin-binding EGF-like Growth Factor | Reinforcement Schedule | Tyrosine 3-Monooxygenase - genetics | Animals | Globus Pallidus - cytology | Glutamate Decarboxylase - metabolism | Mice | Reward | Index Medicus
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2. Full Text Human 'brite/beige' adipocytes develop from capillary networks, and their implantation improves metabolic homeostasis in mice
by Min, So Yun and Kady, Jamie and Nam, Minwoo and Rojas-Rodriguez, Raziel and Berkenwald, Aaron and Kim, Jong Hun and Noh, Hye-Lim and Kim, Jason K and Cooper, Marcus P and Fitzgibbons, Timothy and Brehm, Michael A and Corvera, Silvia
Nature Medicine, ISSN 1078-8956, 03/2016, Volume 22, Issue 3, pp. 312 - 318
Uncoupling protein 1 (UCP1) is highly expressed in brown adipose tissue, where it generates heat by uncoupling electron transport from ATP production. UCP1 is... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | WHITE | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ADULT HUMANS | BROWN ADIPOSE-TISSUE | IDENTIFICATION | CELL BIOLOGY | OBESITY | GENE | INFLAMMATION | EXPRESSION | Enkephalins - metabolism | Humans | Middle Aged | Ion Channels - genetics | Male | RNA, Messenger - metabolism | Enkephalins - genetics | Mitochondrial Proteins - metabolism | Diet, High-Fat | Iodide Peroxidase - metabolism | Adipocytes, Brown - transplantation | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Oxygen Consumption | Apoptosis Regulatory Proteins - metabolism | Protein Precursors - metabolism | Mice | Iodide Peroxidase - genetics | Blood Glucose - metabolism | Integrin beta1 - genetics | Adipocytes, White - metabolism | Glucose Intolerance - metabolism | Proprotein Convertase 1 - genetics | Adipocytes, Brown - metabolism | Homeostasis | Mitochondrial Proteins - genetics | DNA-Binding Proteins - metabolism | Polymerase Chain Reaction | Apoptosis Regulatory Proteins - genetics | Adult | Female | Capillaries | Glucose Tolerance Test | Protein Precursors - genetics | Proprotein Convertase 1 - metabolism | Cell Transplantation | Adipocytes, White - transplantation | DNA-Binding Proteins - genetics | Obesity - metabolism | Integrin beta1 - metabolism | Interleukin-33 - genetics | Animals | Ion Channels - metabolism | Receptor, Platelet-Derived Growth Factor alpha - genetics | Adipocytes - metabolism | Fluorescent Antibody Technique | Interleukin-33 - metabolism | Adipocytes - transplantation | Aged | Glucose Clamp Technique | Uncoupling Protein 1 | Neovascularization, Physiologic | Adipose tissues | Physiological aspects | Genetic aspects | Research | cytokine | human adipocyte | glucose | progenitors | adipokine | capillary | adrenergic | thermogenic adipose tissue | implant
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3. Full Text Expansion of Intronic GGCCTG Hexanucleotide Repeat in NOP56 Causes SCA36, a Type of Spinocerebellar Ataxia Accompanied by Motor Neuron Involvement
by Kobayashi, Hatasu and Abe, Koji and Matsuura, Tohru and Ikeda, Yoshio and Hitomi, Toshiaki and Akechi, Yuji and Habu, Toshiyuki and Liu, Wanyang and Okuda, Hiroko and Koizumi, Akio
The American Journal of Human Genetics, ISSN 0002-9297, 07/2011, Volume 89, Issue 1, pp. 121 - 130
Autosomal-dominant spinocerebellar ataxias (SCAs) are a heterogeneous group of neurodegenerative disorders. In this study, we performed genetic analysis of a... 
NEURODEGENERATION | DOMINANT CEREBELLAR ATAXIAS | CLINICAL-FEATURES | PCR | GENETICS & HEREDITY | Haplotypes | Enkephalins - metabolism | Humans | Middle Aged | Spinocerebellar Ataxias - classification | Male | MicroRNAs - metabolism | Mutation, Missense | Lod Score | Motor Neurons - pathology | Enkephalins - genetics | Adult | Female | Nuclear Proteins - genetics | Spinocerebellar Ataxias - genetics | Introns - genetics | Protein Precursors - genetics | Amyotrophic Lateral Sclerosis - genetics | In Situ Hybridization, Fluorescence | Nuclear Proteins - metabolism | Spinocerebellar Ataxias - pathology | Protein Precursors - metabolism | Pedigree | Age of Onset | Aged | MicroRNAs - genetics | Alzheimer Disease - genetics | Cohort Studies | Genetics | Mutation | Medical screening | Neurological disorders | Fluorescence in situ hybridization | Motor neurons | Neurodegenerative diseases | RNA | Transcription | Amyotrophic lateral sclerosis | RNA-binding protein | Genetic analysis | Family studies | miRNA | Open reading frames | Gene mapping | Alzheimer's disease | Linkage analysis | Report
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4. Opioid peptides
: biology, chemistry, and genetics
by Udenfriend, Sidney and Meienhofer, Johannes
1984, ISBN 9780123042064, Volume 6., xxxv, 410
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5. Full Text Revisiting disease genes based on whole-exome sequencing in consanguineous populations
by Shamia, Ahmed and Shaheen, Ranad and Sabbagh, Nouran and Almoisheer, Agaadir and Halees, Anason and Alkuraya, Fowzan S
Human Genetics, ISSN 0340-6717, 9/2015, Volume 134, Issue 9, pp. 1029 - 1034
Assigning a causal role for genes in disease states is one of the most significant medical applications of human genetics research. The requirement for at... 
Human Genetics | Gene Function | Molecular Medicine | Biomedicine | Metabolic Diseases | GENETICS & HEREDITY | MUTATIONS | Lipoproteins - genetics | Humans | Infant | Male | Exome | Enkephalins - genetics | ATP-Binding Cassette Transporters - genetics | Serine Endopeptidases - genetics | Cell Cycle Proteins - genetics | Adult | Child | Intracellular Signaling Peptides and Proteins - genetics | Claudins - genetics | Protein Precursors - genetics | Connexins - genetics | Repressor Proteins - genetics | Amino Acid Transport Systems, Neutral - genetics | Homozygote | Phenotype | Symporters - genetics | Adolescent | Alleles | TRPM Cation Channels - genetics | Consanguinity | Genome, Human | Genome-Wide Association Study - methods | ATP Binding Cassette Transporter, Sub-Family G, Member 5 | Genes | Genomics
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6. Full Text Search for genetic markers and functional variants involved in the development of opiate and cocaine addiction and treatment
by Yuferov, Vadim and Levran, Orna and Proudnikov, Dmitri and Nielsen, David A and Kreek, Mary Jeanne
Annals of the New York Academy of Sciences, ISSN 0077-8923, 02/2010, Volume 1187, Issue 1, pp. 184 - 207
Addiction to opiates and illicit use of psychostimulants is a chronic, relapsing brain disease that, if left untreated, can cause major medical, social, and... 
genetics of drug addiction | opiate and cocaine addiction | epigenetics | pharmacogenetics | allele-specific gene expression | molecular haplotyping | Epigenetics | Opiate and cocaine addiction | Pharmacogenetics | Allele-specific gene expression | Genetics of drug addiction | Molecular haplotyping | MESSENGER-RNA EXPRESSION | MULTIDISCIPLINARY SCIENCES | GENOMIC DNA HYPERMETHYLATION | SEROTONIN TRANSPORTER GENE | FAMILY-BASED ASSOCIATION | CATECHOL-O-METHYLTRANSFERASE | POSITRON-EMISSION-TOMOGRAPHY | TRYPTOPHAN-HYDROXYLASE ISOFORM | MU-OPIOID-RECEPTOR | METHADONE-MAINTENANCE TREATMENT | SINGLE-NUCLEOTIDE POLYMORPHISMS | Haplotypes | Genome-Wide Association Study | Protein Precursors - genetics | Epigenesis, Genetic | Humans | Cocaine-Related Disorders - therapy | Methadone - therapeutic use | Methadone - metabolism | Cocaine-Related Disorders - genetics | Genetic Markers | Catechol O-Methyltransferase - genetics | Opioid-Related Disorders - therapy | Tryptophan Hydroxylase - genetics | Genetic Variation | Receptor, Melanocortin, Type 2 - genetics | Enkephalins - genetics | ATP-Binding Cassette, Sub-Family B, Member 1 - genetics | Opioid-Related Disorders - genetics | Receptor, Serotonin, 5-HT1B - genetics | ATP Binding Cassette Transporter, Sub-Family B | Receptors, Opioid, mu - genetics | Receptors, Opioid, kappa - genetics | Drug abuse | Care and treatment | Anopheles | Genetic research | Genetics | Substance abuse | Gene expression | Methylation | Substance abuse treatment | Genes | Heroin | Polymorphism
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7. Full Text Aberrant methylation of NPY, PENK, and WIF1 as a promising marker for blood-based diagnosis of colorectal cancer
by Roperch, Jean-Pierre and Incitti, Roberto and Forbin, Solène and Bard, Floriane and Mansour, Hicham and Mesli, Farida and Baumgaertner, Isabelle and Brunetti, Francesco and Sobhani, Iradj
BMC Cancer, ISSN 1471-2407, 12/2013, Volume 13, Issue 1, pp. 566 - 566
Background: DNA methylation is a well-known epigenetic mechanism involved in epigenetic gene regulation. Several genes were reported hypermethylated in CRC,... 
Epigenetic markers | QM-MSP | Circulating DNA methylation | Colorectal cancer | BLADDER-CANCER | CELLS | COST-EFFECTIVENESS | INHIBITORY FACTOR-I | DNA METHYLATION | PROMOTER METHYLATION | HEPATOCELLULAR-CARCINOMA | ONCOLOGY | FECAL OCCULT BLOOD | WNT PATHWAY | GENE-EXPRESSION | Colorectal Neoplasms - genetics | Humans | Middle Aged | Male | Neuropeptide Y - genetics | Case-Control Studies | Colorectal Neoplasms - diagnosis | DNA - blood | Enkephalins - genetics | DNA Methylation | Molecular Diagnostic Techniques | Aged, 80 and over | Female | Adenocarcinoma - genetics | Promoter Regions, Genetic | Protein Precursors - genetics | Colorectal Neoplasms - blood | Adenocarcinoma - blood | Repressor Proteins - genetics | Multiplex Polymerase Chain Reaction | Sequence Analysis, DNA | DNA - genetics | Adenocarcinoma - diagnosis | Adaptor Proteins, Signal Transducing - genetics | ROC Curve | Aged | Biomarkers, Tumor - genetics | Neoplasm Staging | Genetic markers | Physiological aspects | Genetic aspects | Diagnosis | Research | Methylation | Medical research | Epigenetic inheritance | Medical examination | Medicine, Experimental | Blood
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8. Full Text Methylation of ELOVL2 gene as a new epigenetic marker of age
by Garagnani, Paolo and Bacalini, Maria G and Pirazzini, Chiara and Gori, Davide and Giuliani, Cristina and Mari, Daniela and Di Blasio, Anna M and Gentilini, Davide and Vitale, Giovanni and Collino, Sebastiano and Rezzi, Serge and Castellani, Gastone and Capri, Miriam and Salvioli, Stefano and Franceschi, Claudio
Aging Cell, ISSN 1474-9718, 12/2012, Volume 11, Issue 6, pp. 1132 - 1134
The discovery of biomarkers able to predict biological age of individuals is a crucial goal in aging research. Recently, researchers' attention has turn toward... 
DNA | methylation | PENK | aging | FHL | biomarker | ELOVL | FHL 2 | Aging | DNA methylation | Biomarker | ELOVL 2 | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | Transcription Factors - blood | Oligonucleotide Array Sequence Analysis | Protein Precursors - blood | Epigenesis, Genetic | Humans | Middle Aged | Male | Acetyltransferases - genetics | Enkephalins - blood | Enkephalins - genetics | DNA Methylation | Muscle Proteins - blood | Aging - genetics | Fetal Blood - chemistry | Aged, 80 and over | Adult | Female | Child | Protein Precursors - genetics | Genetic Markers | Transcription Factors - genetics | Aging - blood | Muscle Proteins - genetics | LIM-Homeodomain Proteins - genetics | LIM-Homeodomain Proteins - blood | Adolescent | CpG Islands | Aged | Genome, Human | Acetyltransferases - blood | Genetic research | Methylation | Genes | Cord blood | biomarkers | epigenetics | CpG islands
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9. Full Text Mutations in rare ataxia genes are uncommon causes of sporadic cerebellar ataxia
by Fogel, Brent L and Lee, Ji Yong and Lane, Jessica and Wahnich, Amanda and Chan, Sandy and Huang, Alden and Osborn, Greg E and Klein, Eric and Mamah, Catherine and Perlman, Susan and Geschwind, Daniel H and Coppola, Giovanni
Movement Disorders, ISSN 0885-3185, 03/2012, Volume 27, Issue 3, pp. 442 - 446
Background: Sporadic-onset ataxia is common in a tertiary care setting but a significant percentage remains unidentified despite extensive evaluation. Rare... 
dominant genetic conditions | recessive genetic conditions | cerebellar ataxia | copy number variation | spinocerebellar ataxia | Recessive genetic conditions | Dominant genetic conditions | Copy number variation | Cerebellar ataxia | Spinocerebellar ataxia | DEPRESSION | PLACEBO | MOTOR FUNCTION | CLINICAL NEUROLOGY | AMOXAPINE | IMIPRAMINE | ARSACS | SENATAXIN | DISEASE RATING-SCALE | HUNTINGTONS-DISEASE | PARKINSONS-DISEASE | Genetic Predisposition to Disease - genetics | Genetic Testing | Protein Precursors - genetics | Lamin Type B - genetics | Humans | Middle Aged | Transglutaminases - genetics | Male | Mutation - genetics | Nerve Tissue Proteins - genetics | Enkephalins - genetics | Heat-Shock Proteins - genetics | Magnetic Resonance Imaging | Phenotype | Cerebellar Ataxia - genetics | Databases, Bibliographic - statistics & numerical data | Adult | Female | RNA Helicases - genetics | Aged | Nuclear Proteins - genetics | Spinocerebellar Ataxia | Dominant Genetic Conditions | Recessive Genetic Conditions | Cerebellar Ataxia | Copy Number Variation
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10. Full Text κ-opioid receptor/dynorphin system: genetic and pharmacotherapeutic implications for addiction
by Butelman, Eduardo R and Yuferov, Vadim and Kreek, Mary Jeanne
Trends in Neurosciences, ISSN 0166-2236, 2012, Volume 35, Issue 10, pp. 587 - 596
Addictions to cocaine or heroin/prescription opioids [short-acting μ-opioid receptor (MOPr) agonists] involve relapsing cycles, with experimentation/escalating... 
Neurology | prescription opioids | dynorphin | heroin | κ-opioid receptor | cocaine | addiction | Cocaine | Addiction | Prescription opioids | Heroin | Dynorphin | NOR-BINALTORPHIMINE | HEROIN-ADDICTION | PRODYNORPHIN MESSENGER-RNA | OPIATE WITHDRAWAL | NEUROSCIENCES | kappa-opioid receptor | STRIATAL DOPAMINE LEVELS | POSITRON-EMISSION-TOMOGRAPHY | RAT CAUDATE-PUTAMEN | RECEPTOR GENE | INDUCED PLACE PREFERENCE | ALCOHOL DEPENDENCE | Behavior, Addictive - drug therapy | Recurrence | Genetic Predisposition to Disease - genetics | Receptors, Opioid, kappa - antagonists & inhibitors | Protein Precursors - genetics | Behavior, Addictive - genetics | Adaptation, Biological - genetics | Humans | Receptors, Opioid, kappa - agonists | Drug Discovery - methods | Dynorphins - physiology | Adaptation, Biological - physiology | Polymorphism, Genetic | Receptors, Opioid, kappa - physiology | Enkephalins - genetics | Animals | Narcotic Antagonists - therapeutic use | Narcotic Antagonists - pharmacology | Behavior, Addictive - physiopathology | Street Drugs - pharmacology | Disease Models, Animal | Drug addiction | Opioid receptors | Withdrawal | Opioid receptors (type mu) | Depression | epigenetics | Reviews | Plasticity | Reinforcement | Anxiety | Genetic factors
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11. Full Text Association between VNTR polymorphism in promoter region of prodynorphin ( PDYN ) gene and heroin dependence
by Saify, Khyber and Saadat, Iraj and Saadat, Mostafa
Psychiatry Research, ISSN 0165-1781, 2014, Volume 219, Issue 3, pp. 690 - 692
Abstract Within the core promoter region of prodynorphin ( PDYN ), a 68-bp sequence was found to occur as a polymorphism element, either singular or as... 
Psychiatry | PDYN | Heroin dependence | VNTR | PSYCHIATRY | Genetic Predisposition to Disease | Protein Precursors - genetics | Humans | Genotype | Male | Heroin Dependence - psychology | Promoter Regions, Genetic - genetics | Case-Control Studies | Polymorphism, Genetic | Enkephalins - genetics | Enkephalin, Leucine | Alleles | Minisatellite Repeats - genetics | Heroin Dependence - genetics | Genetic research | Genetic aspects | Promoters (Genetics) | Genetic polymorphisms | Heroin habit
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12. Full Text Enriched Protein Screening of Human Bone Marrow Mesenchymal Stromal Cell Secretions Reveals MFAP5 and PENK as Novel IL-10 Modulators
by Milwid, Jack M and Elman, Jessica S and Li, Matthew and Shen, Keyue and Manrai, Arjun and Gabow, Aaron and Yarmush, Martin L and Yarmush, Joshua and Jiao, Yunxin and Fletcher, Anne and Lee, Jungwoo and Cima, Michael J and Parekkadan, Biju
Molecular Therapy, ISSN 1525-0016, 05/2014, Volume 22, Issue 5, pp. 999 - 1007
The secreted proteins from a cell constitute a natural biologic library that can offer significant insight into human health and disease. Discovering new... 
Contractile Proteins - genetics | Enkephalins - metabolism | Humans | Gene Expression Profiling | Shock, Septic - therapy | Mesenchymal Stromal Cells - immunology | Enkephalins - genetics | Proteins - secretion | Bone Marrow Cells - immunology | Interleukin-10 - metabolism | Protein Biosynthesis - genetics | Contractile Proteins - metabolism | Extracellular Matrix Proteins - metabolism | Shock, Septic - genetics | Glycoproteins - genetics | Protein Precursors - genetics | Extracellular Matrix Proteins - genetics | Mesenchymal Stromal Cells - metabolism | Protein Precursors - metabolism | Proteins - genetics | Animals | Interleukin-10 - genetics | Proteomics | Mice | Bone Marrow Cells - metabolism | Original
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13. Full Text Candidate genes for temporal lobe epilepsy: a replication study
by Salzmann, Annick and Perroud, Nader and Crespel, Arielle and Lambercy, Carmen and Malafosse, Alain
Neurological Sciences, ISSN 1590-1874, 12/2008, Volume 29, Issue 6, pp. 397 - 403
The objective of this study is to replicate previously published results regarding the involvement of several susceptibility genes in temporal lobe epilepsy... 
Neurology | Prodynorphin | Association | Medicine & Public Health | Apolipoprotein E | Interleukin 1 | Temporal lobe epilepsy | Genetics | Neurosurgery | Psychiatry | Neuroradiology | IL-1-BETA GENE | LOCALIZATION | ASSOCIATION ANALYSIS | INTERLEUKIN-1 RECEPTOR ANTAGONIST | SUSCEPTIBILITY | PROMOTER POLYMORPHISM | NEUROSCIENCES | CLINICAL NEUROLOGY | FEBRILE CONVULSIONS | HIPPOCAMPAL SCLEROSIS | EXPRESSION | ONSET | Genetic Predisposition to Disease - genetics | Genetic Testing | Protein Precursors - genetics | Gene Frequency | Humans | Epilepsy, Temporal Lobe - physiopathology | Genotype | Interleukin 1 Receptor Antagonist Protein - genetics | Epilepsy, Temporal Lobe - immunology | Interleukin-1beta - genetics | Case-Control Studies | Enkephalins - genetics | Interleukins - genetics | DNA Mutational Analysis | Apolipoproteins E - genetics | Epilepsy, Temporal Lobe - genetics | Genetic Markers - genetics | Adolescent | Adult | Polymorphism, Genetic - genetics | Interleukin-1alpha - genetics | Child | Cohort Studies | Genetic research | Interleukins | Apolipoproteins | Medical genetics
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14. Full Text Targeted drug delivery to the peripheral nervous system using gene therapy
by Wolfe, Darren and Mata, Marina and Fink, David J
Neuroscience Letters, ISSN 0304-3940, 10/2012, Volume 527, Issue 2, pp. 85 - 89
► HSV-based vectors are particularly suitable for delivering genes to the DRG. ► Preclinical studies in pain and for treatment of polyneuropathy are reviewed.... 
Gene therapy | Polyneuropathy | Pain | VIRUS | DORSAL-ROOT GANGLIA | SPINAL-CORD | DRIVEN ENKEPHALIN OVERPRODUCTION | DIABETIC-NEUROPATHY | NEUROSCIENCES | NEUROPATHIC PAIN | OPIOID RECEPTOR | NEURONS | GROWTH-FACTOR | EXPRESSION | Genetic Therapy | Pain, Intractable - therapy | Enkephalins - metabolism | Humans | Nerve Growth Factors - metabolism | Polyneuropathies - metabolism | Pain, Intractable - genetics | Diabetic Neuropathies - genetics | Enkephalins - genetics | Neoplasms - complications | Neurotransmitter Agents - genetics | Herpesviridae - genetics | Neoplasms - physiopathology | Pain, Intractable - metabolism | Chronic Pain - therapy | Pain, Intractable - etiology | Protein Precursors - genetics | Neurotransmitter Agents - metabolism | Chronic Pain - genetics | Clinical Trials as Topic | Polyneuropathies - therapy | Protein Precursors - metabolism | Chronic Pain - metabolism | Animals | Diabetic Neuropathies - therapy | Nerve Growth Factors - genetics | Peripheral Nervous System - metabolism | Polyneuropathies - genetics | Diabetic Neuropathies - metabolism | Genetic Vectors | Drugs | Care and treatment | Drug delivery systems | Genes | Genetic research | Vehicles | Indexing in process
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