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Nature (London), ISSN 1476-4687, 2019, Volume 571, Issue 7766, pp. 565 - 569
... infection wit... 
RECOGNIZE | MODELS | MULTIDISCIPLINARY SCIENCES | OCCURS | ASSOCIATION | BRAIN | CLONE | LYMPHOCYTES | Protein Kinases - genetics | CD8-Positive T-Lymphocytes - pathology | Dopaminergic Neurons - pathology | Neostriatum - physiopathology | Male | Mitochondria - immunology | Parkinson Disease - drug therapy | Citrobacter rodentium - pathogenicity | Intestines - immunology | Enterobacteriaceae Infections - immunology | Ubiquitin-Protein Ligases - immunology | Female | Protein Kinases - deficiency | Disease Models, Animal | Neostriatum - pathology | Citrobacter rodentium - immunology | Dopaminergic Neurons - immunology | Intestines - pathology | Neostriatum - microbiology | Levodopa - therapeutic use | Enterobacteriaceae Infections - microbiology | Enterobacteriaceae Infections - pathology | Mitochondria - pathology | Parkinson Disease - genetics | Antigen Presentation - immunology | Parkinson Disease - physiopathology | Enterobacteriaceae Infections - physiopathology | Animals | Intestines - microbiology | Parkinson Disease - microbiology | Protein Kinases - immunology | Autoantigens - immunology | Axons - pathology | Ubiquitin-Protein Ligases - deficiency | Mice | CD8-Positive T-Lymphocytes - immunology | Ubiquitin-Protein Ligases - genetics | Neostriatum - immunology | Ubiquitin | Brain | Animal models | Parkinson's disease | Peptides | CD8 antigen | Substantia nigra | Immune clearance | Parkinsons disease | Cytotoxicity | Lymphocytes T | Infections | Kinases | Immunity | Proteins | Mitochondria | Lymphocytes | Intestine | Neostriatum | Bacteria | Levodopa | Gram-negative bacteria | Ubiquitin-protein ligase | Age | Movement disorders | Immune system | Dopamine receptors | Spleen | Dihydroxyphenylalanine | Antigen presentation | Antigens | Repressing | Signs and symptoms | Dopamine | Bacterial infections | Cytokines | Neurodegenerative diseases | Neurons | Cloning | Inflammation | PTEN-induced putative kinase | Parkin protein | Mutation | Dopa
Journal Article
Nature communications, ISSN 2041-1723, 2017, Volume 8, Issue 1, p. 24
.... Here we show that in a Drosophila Alzheimer's disease model, enterobacteria infection exacerbated progression of Alzheimer's disease by promoting immune hemocyte recruitment to the brain, thereby... 
STROKE | SYSTEM | IMMUNE-RESPONSE | ENERGY HOMEOSTASIS | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | NEURODEGENERATION | MECHANISMS | MODEL | BRAIN | COMMUNICATION | Antimicrobial Cationic Peptides - immunology | Alzheimer Disease - complications | Brain - microbiology | Drosophila melanogaster - immunology | Hemocytes - immunology | Humans | Tumor Necrosis Factor-alpha - genetics | MAP Kinase Kinase 4 - immunology | Drosophila Proteins - immunology | Dysbiosis - microbiology | Enterobacteriaceae Infections - complications | Alzheimer Disease - pathology | Cell Movement - immunology | Enterobacteriaceae Infections - immunology | Hemocytes - pathology | Dysbiosis - immunology | Gastrointestinal Tract - immunology | Enterobacteriaceae - immunology | Leukocyte Reduction Procedures | Amyloid beta-Peptides - genetics | Peptide Fragments - immunology | Tumor Necrosis Factor-alpha - immunology | Hemocytes - microbiology | Alzheimer Disease - immunology | Antimicrobial Cationic Peptides - genetics | Peptide Fragments - genetics | Enterobacteriaceae - growth & development | Disease Models, Animal | Alzheimer Disease - microbiology | Microbiota - immunology | Signal Transduction | Enterobacteriaceae Infections - microbiology | Gene Expression Regulation | Gastrointestinal Tract - microbiology | Enterobacteriaceae Infections - pathology | Dysbiosis - complications | Disease Progression | Drosophila melanogaster - microbiology | Animals | Brain - pathology | MAP Kinase Kinase 4 - genetics | Amyloid beta-Peptides - immunology | Drosophila Proteins - genetics | Dysbiosis - pathology | Brain - immunology
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 2012, Volume 37, Issue 6, pp. 1061 - 1075
Interleukin-22 (IL-22) is central to host protection against bacterial infections at barrier sites... 
INDUCER-LIKE CELLS | MUCOSAL IMMUNITY | INTERLEUKIN 22 | T(H)17 DIFFERENTIATION | INNATE LYMPHOID-CELLS | INTESTINAL PATHOLOGY | IMMUNOLOGY | CHRONIC VIRAL-INFECTION | CITROBACTER-RODENTIUM | POTENTIAL ROLE | T-CELLS | Enterobacteriaceae Infections - prevention & control | T-Box Domain Proteins - immunology | Interleukins - metabolism | Enterobacteriaceae Infections - immunology | Interleukin-23 - metabolism | Interleukins - physiology | Th17 Cells - metabolism | T-Lymphocytes, Helper-Inducer - immunology | Inflammation Mediators - metabolism | Interleukin-6 - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Inflammation Mediators - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Citrobacter rodentium - immunology | T-Lymphocytes, Helper-Inducer - metabolism | Enterobacteriaceae Infections - mortality | Gene Expression Regulation | Receptors, Aryl Hydrocarbon - genetics | T-Box Domain Proteins - genetics | Interleukin-23 - immunology | Mice, Knockout | Animals | Mucous Membrane - immunology | Interleukin-6 - immunology | Th17 Cells - immunology | Mice | Receptors, Aryl Hydrocarbon - immunology | Mucous Membrane - microbiology | Bacteria | Bacterial infections | Interleukins | T cells | Transforming growth factors | Studies | Inflammatory bowel disease | Transcription factors | Rodents | Cloning | Gene expression | Immune system
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2013, Volume 210, Issue 6, pp. 1117 - 1124
Journal Article
European journal of immunology, ISSN 0014-2980, 2015, Volume 45, Issue 9, pp. 2553 - 2567
Orally administrated iron is suspected to increase susceptibility to enteric infections among children in infection endemic regions... 
Caenorhabditis elegans | lipocalin‐2 | gut microbiome | iron supplementation | intestinal pathogens | Iron supplementation | Intestinal pathogens | Lipocalin-2 | Gut microbiome | CONTROLLED-TRIAL | ULCERATIVE-COLITIS | INDUCED COLITIS | GELATINASE-ASSOCIATED LIPOCALIN | GUT MICROBIOTA | IMMUNOLOGY | RAT MODEL | BOWEL-DISEASE | SUPPLEMENTATION | CITROBACTER-RODENTIUM INFECTION | MICE | lipocalin-2 | Lipocalins - biosynthesis | Caenorhabditis elegans - microbiology | Intestinal Mucosa - metabolism | Iron, Dietary - administration & dosage | Leukocyte L1 Antigen Complex - immunology | Body Weight - immunology | Intestines - immunology | Enterobacteriaceae Infections - immunology | Salmonella Infections, Animal - immunology | Feces - microbiology | Salmonella Infections, Animal - mortality | Intestinal Mucosa - immunology | Oncogene Proteins - biosynthesis | Female | Diet - methods | Oncogene Proteins - immunology | Leukocyte L1 Antigen Complex - biosynthesis | Caenorhabditis elegans - metabolism | Citrobacter rodentium - immunology | Intestines - pathology | Salmonella typhimurium - immunology | Enterobacteriaceae Infections - metabolism | Salmonella Infections, Animal - metabolism | Enterobacteriaceae Infections - microbiology | Mice, Inbred C57BL | Enterobacteriaceae Infections - pathology | Intestinal Mucosa - microbiology | Immunity, Innate | Caenorhabditis elegans - immunology | Animals | Caenorhabditis elegans - drug effects | Intestines - microbiology | Lipocalins - immunology | Salmonella Infections, Animal - microbiology | Survival Analysis | Mice | Iron, Dietary - adverse effects | Acute-Phase Proteins - biosynthesis | Acute-Phase Proteins - immunology | Intestinal Mucosa - pathology | Pathogenic microorganisms | Bacterial infections | Microbiota (Symbiotic organisms) | Analysis | Gastrointestinal diseases | Escherichia coli | Health aspects | Nematoda | Salmonella | Pathology | Iron | Anemia | Bacteriology
Journal Article
Nature (London), ISSN 1476-4687, 2012, Volume 488, Issue 7410, pp. 222 - 225
.... During intestinal Citrobacter rodentium infection(4-6), a mouse model for enteropathogenic Escherichia coli infection, Hvem... 
INTESTINAL INFLAMMATION | PROTECTS MICE | HERPESVIRUS ENTRY MEDIATOR | NK CELLS | MULTIDISCIPLINARY SCIENCES | ULCERATIVE-COLITIS | RECEPTOR | CITROBACTER-RODENTIUM | CELL-DIFFERENTIATION | NF-KAPPA-B | INNATE IMMUNITY | Antigens, CD - immunology | Immunity, Mucosal - immunology | Lung - microbiology | Epithelial Cells - metabolism | Enteropathogenic Escherichia coli | Citrobacter rodentium - pathogenicity | Intestines - immunology | Antigens, CD - metabolism | Streptococcus pneumoniae - immunology | Enterobacteriaceae Infections - immunology | Pneumococcal Infections - immunology | Lymphocytes - immunology | Escherichia coli Infections | Pneumococcal Infections - microbiology | Receptors, Immunologic - immunology | Protein-Serine-Threonine Kinases - metabolism | Receptors, Tumor Necrosis Factor, Member 14 - immunology | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Lymphocytes - metabolism | Receptors, Tumor Necrosis Factor, Member 14 - genetics | Cell Line | Citrobacter rodentium - immunology | GPI-Linked Proteins - immunology | Signal Transduction | Enterobacteriaceae Infections - microbiology | Mice, Inbred C57BL | Survival Rate | Bacterial Load | GPI-Linked Proteins - metabolism | Mucous Membrane - metabolism | Receptors, Tumor Necrosis Factor, Member 14 - metabolism | Animals | Intestines - microbiology | Mucous Membrane - immunology | Epithelial Cells - immunology | Receptors, Tumor Necrosis Factor, Member 14 - deficiency | Ligands | Mice | Lung - immunology | Mucous Membrane - microbiology | Receptors, Immunologic - metabolism | Physiological aspects | Bacteria | Cellular signal transduction | Research | Health aspects | Mucous membrane | Immune system | Phosphorylation | Bacterial infections | Cytokines | Rodents | Kinases | Gene expression | Chemokines | Streptococcus infections
Journal Article
Annals of the American Thoracic Society, ISSN 2325-6621, 02/2015, Volume 12, Issue 2, pp. 221 - 229
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2012, Volume 287, Issue 20, pp. 16955 - 16964
Citrobacter rodentium is an enteric bacterial pathogen of the mouse intestinal tract that triggers inflammatory responses resembling those of humans infected... 
ACTIVATION | EFFACING PATHOGEN | MICE DEFICIENT | CASPASE-1 | INTERLEUKIN-1-BETA | BIOCHEMISTRY & MOLECULAR BIOLOGY | DISEASE | INDUCED COLITIS | SECRETION | VIRULENCE | MYD88 | Interleukin-18 - immunology | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Caspase 1 - metabolism | Caspase 1 - immunology | Interleukin-1beta - genetics | Citrobacter rodentium - metabolism | Enterobacteriaceae Infections - immunology | Calcium-Binding Proteins - immunology | Enterobacteriaceae Infections - genetics | Interleukin-1beta - metabolism | Apoptosis Regulatory Proteins - genetics | Bacterial Secretion Systems - immunology | Carrier Proteins - immunology | Macrophages - immunology | Macrophages - microbiology | Calcium-Binding Proteins - metabolism | Apoptosis Regulatory Proteins - immunology | Bacterial Secretion Systems - genetics | Citrobacter rodentium - immunology | Enterobacteriaceae Infections - metabolism | Citrobacter rodentium - genetics | Interleukin-1beta - immunology | Enterobacteriaceae Infections - pathology | Inflammasomes - genetics | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Carrier Proteins - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Caspase 1 - genetics | Inflammasomes - immunology | Mice | Interleukin-18 - genetics | Interleukin-18 - metabolism | Calcium-Binding Proteins - genetics | Host-Pathogen Interactions | Immunology | Innate Immunity | Inflammation | Inflammasome | Colitis | Citrobacter rodentium
Journal Article
Nature communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, p. 6525
.... Here we show that colonic CX3CR1(+) mononuclear phagocytes are critical inducers of the innate response to Citrobacter rodentium infection... 
HOMEOSTASIS | DENDRITIC CELLS | MACROPHAGES | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | CYTOKINE | INNATE LYMPHOID-CELLS | LY6C(HI) MONOCYTES | IL-23 | T-CELLS | CUTTING EDGE | Dendritic Cells - immunology | Immunity, Mucosal | Colon - immunology | Monocytes - immunology | Citrobacter rodentium - pathogenicity | Antigens, CD - genetics | Interleukin-23 - genetics | Interleukins - genetics | Intestinal Mucosa - immunology | Integrin alpha Chains - immunology | Monocytes - pathology | Receptors, Chemokine - genetics | Macrophages - microbiology | Monocytes - microbiology | Macrophages - pathology | Signal Transduction | Colon - pathology | Enterobacteriaceae Infections - microbiology | Mice, Transgenic | Intestinal Mucosa - microbiology | Interleukin-23 - immunology | Interleukin-12 - immunology | Survival Analysis | Dendritic Cells - microbiology | Th17 Cells - immunology | Mice | CX3C Chemokine Receptor 1 | Intestinal Mucosa - pathology | Antigens, CD - immunology | Interleukin-12 - genetics | Dendritic Cells - pathology | Homeostasis | Enterobacteriaceae Infections - immunology | CD11b Antigen - genetics | Interleukins - immunology | Interferon-gamma - genetics | Macrophages - immunology | Th17 Cells - pathology | Citrobacter rodentium - immunology | CD11b Antigen - immunology | Enterobacteriaceae Infections - mortality | Gene Expression Regulation | Enterobacteriaceae Infections - pathology | Receptors, Chemokine - immunology | Immunity, Innate | Animals | Interferon-gamma - immunology | Colon - microbiology | Integrin alpha Chains - genetics | Th17 Cells - microbiology
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 2008, Volume 29, Issue 6, pp. 958 - 970
Journal Article
Infection and immunity, ISSN 1098-5522, 2013, Volume 81, Issue 12, pp. 4649 - 4658
Journal Article
PLoS pathogens, ISSN 1553-7374, 2010, Volume 6, Issue 5, p. e1000902
...) Escherichia coli infections, the mechanisms by which the host defends against these microbes are unclear... 
DISRUPTION IN-VIVO | YERSINIA-ENTEROCOLITICA | EFFACING PATHOGEN | PURIFIED RABBIT | MICROBIOLOGY | ENTAMOEBA-HISTOLYTICA | ENTERICA SEROTYPE TYPHIMURIUM | VIROLOGY | ENTEROPATHOGENIC ESCHERICHIA-COLI | HUMAN INTESTINAL MUCIN | HOST-DEFENSE | CITROBACTER-RODENTIUM INFECTION | PARASITOLOGY | Mucin-2 - genetics | Mucin-2 - metabolism | Intestinal Mucosa - metabolism | Epithelial Cells - metabolism | Bacterial Adhesion - immunology | Enterobacteriaceae Infections - metabolism | Mice, Inbred C57BL | Bacterial Translocation - immunology | Intestinal Mucosa - microbiology | Enterobacteriaceae Infections - immunology | Animals | Colitis - microbiology | Mucin-2 - immunology | Intestinal Mucosa - immunology | Mice, Mutant Strains | Epithelial Cells - immunology | Epithelial Cells - microbiology | Colitis - metabolism | Mice | Citrobacter rodentium | Colitis - immunology | Escherichia coli | Mucins | Physiological aspects | Causes of | Development and progression | Genetic aspects | Research | Colitis | Translocation | Pathogens | Commensals | Secretion | Mortality | Mucosa | Oral administration | Diarrhea | Mucus | Epithelium | Infection | Large intestine | mucin | Fluorescence in situ hybridization | Colon | Feces | Intestinal microflora | Digestive tract | Colonization | Inflammatory bowel disease | Medical research | Bacterial infections | Microbiology | E coli | Rodents | Bacteria | Experiments | Bars
Journal Article