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Gastroenterology, ISSN 0016-5085, 2009, Volume 137, Issue 1, pp. 231 - 241.e10
Background & Aims Gastrin is a key regulator of gastric acid secretion. We aimed to isolate pure G cells to identify the mechanistic basis of luminal- and... 
Gastroenterology and Hepatology | MUCOSA | BITTER TASTE RECEPTORS | PRIMARY CULTURE | RAT | GASTROINTESTINAL-TRACT | ENTEROCHROMAFFIN-LIKE CELLS | PROLIFERATION | RELEASE | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | CANINE G-CELLS | Enzyme Activators - pharmacology | Phosphorylation | Chemoreceptor Cells - drug effects | Mechanotransduction, Cellular - drug effects | Receptors, G-Protein-Coupled - metabolism | Stress, Mechanical | Male | Gastrins - secretion | Dose-Response Relationship, Drug | Flow Cytometry | Cyclic AMP - metabolism | Gastrin-Secreting Cells - drug effects | Cyclic AMP-Dependent Protein Kinases - metabolism | Cell Separation | Cell Survival | Cells, Cultured | Enzyme Inhibitors - pharmacology | Neurotransmitter Agents - metabolism | Rats | Chemoreceptor Cells - enzymology | Gastrin-Secreting Cells - secretion | Chemoreceptor Cells - secretion | Rats, Sprague-Dawley | Animals | Bacterial Toxins - pharmacology | Gastrin-Secreting Cells - enzymology | Lipopolysaccharides - pharmacology | Enzyme Activation | Chemoreceptor Cells - ultrastructure | Gastrin-Secreting Cells - ultrastructure | Hydrogen-Ion Concentration | Mitogen-Activated Protein Kinases - metabolism | Polymerase chain reaction | Medical colleges | Oncology, Experimental | Gastrin | Esterases | Research | Peptide hormones | Protein kinases | Mitogens | Cancer
Journal Article
Neurogastroenterology and Motility, ISSN 1350-1925, 04/2009, Volume 21, Issue 4, pp. 439 - 450
Journal Article
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 01/2012, Volume 302, Issue 1, pp. G21 - G33
Fjeldbo CS, Bakke I, Erlandsen SE, Holmseth J, Laegreid A, Sandvik AK, Thommesen L, Bruland T. Gastrin upregulates the prosurvival factor secretory clusterin... 
Carcinoids | Neuroendocrine cells | Reverse transfection | Apoptosis | AR42J cells | neuroendocrine cells | PHYSIOLOGY | PROTEIN | reverse transfection | C-FOS | apoptosis | QUALITY-CONTROL | carcinoids | GENE-EXPRESSION DATA | ECL CELL | GROWTH-FACTOR-BETA | ACID INHIBITION | ENTEROCHROMAFFIN-LIKE CELL | CCK-2 RECEPTOR | GASTROENTEROLOGY & HEPATOLOGY | Neuroendocrine Cells - chemistry | Up-Regulation | Enterochromaffin Cells - drug effects | Adenocarcinoma - pathology | Pancreatic Neoplasms - metabolism | Apoptosis - drug effects | Humans | Parietal Cells, Gastric - metabolism | Stomach Neoplasms - metabolism | Clusterin - biosynthesis | Transcription Factor AP-1 - metabolism | Carcinoid Tumor - metabolism | Adenocarcinoma - metabolism | Female | Carcinoid Tumor - chemistry | Promoter Regions, Genetic | Clusterin - genetics | Pancreatic Neoplasms - pathology | Histidine Decarboxylase - metabolism | Rats | Clusterin - blood | Gastrins - metabolism | Rats, Sprague-Dawley | Clusterin - secretion | Animals | Parietal Cells, Gastric - pathology | Cell Line, Tumor | Stomach Neoplasms - chemically induced | Chromogranin A - analysis | Clusterin - antagonists & inhibitors | Neuroendocrine Cells - drug effects | Adenocarcinoma | Gastrin | Physiological aspects | Development and progression | Apolipoproteins | Health aspects | Mucous membrane
Journal Article
Endocrine, ISSN 1355-008X, 2/2013, Volume 43, Issue 1, pp. 22 - 32
Diabetes mellitus is a complex chronic disease associated with an absolute insulin deficiency in type 1 diabetes (T1D) and a progressive deterioration of... 
Incretins | Treatment | Medicine & Public Health | Internal Medicine | Proton pump inhibitors | Diabetes | Endocrinology | Science, general | DIPEPTIDYL PEPTIDASE-4 INHIBITOR | GASTRIC-ACID-SECRETION | TERMINAL TETRAPEPTIDE AMIDE | LIGATED ADULT-RATS | SOMATOSTATIN RECEPTOR LIGANDS | GLUCAGON-LIKE PEPTIDE-1 | GASTROESOPHAGEAL-REFLUX DISEASE | EPIDERMAL-GROWTH-FACTOR | ENDOCRINOLOGY & METABOLISM | BETA-CELL MASS | PANCREATIC-DUCT CELLS | Proton Pump Inhibitors - pharmacology | Enterochromaffin-like Cells - drug effects | Humans | Diabetes Mellitus, Type 1 - metabolism | Parietal Cells, Gastric - metabolism | Diabetes Mellitus, Type 2 - metabolism | Enterochromaffin-like Cells - metabolism | Hypoglycemia - prevention & control | Gastrin-Secreting Cells - metabolism | Insulin-Secreting Cells - metabolism | Somatostatin-Secreting Cells - drug effects | Parietal Cells, Gastric - drug effects | Insulin Secretion | Somatostatin - metabolism | Gastrin-Secreting Cells - drug effects | Gastrins - blood | Hypoglycemic Agents - therapeutic use | Gastrins - metabolism | Proton Pump Inhibitors - therapeutic use | Diabetes Mellitus, Type 1 - drug therapy | Hypoglycemic Agents - pharmacology | Diabetes Mellitus, Type 2 - blood | Gastrointestinal Agents - pharmacology | Insulin - metabolism | Animals | Insulin-Secreting Cells - drug effects | Gastric Emptying - drug effects | Diabetes Mellitus, Type 1 - blood | Gastrointestinal Agents - therapeutic use | Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Somatostatin-Secreting Cells - metabolism | Glucose metabolism | Research | Drug therapy | Health aspects
Journal Article
Journal Article
MOLECULES, ISSN 1420-3049, 04/2017, Volume 22, Issue 4
Adenosine is readily available to the glandular epithelium of the stomach. Formed continuously in intracellular and extracellular locations, it is notably... 
G protein-coupled receptor | SOMATOSTATIN RELEASE | HUMAN-LYMPHOCYTES | ACID-SECRETION | CHEMISTRY, ORGANIC | gastric neuroendocrine cell | adenosine | INTERNATIONAL UNION | HISTAMINE-SECRETION | PARIETAL-CELLS | adenosine deaminase | enterochromaffin-like (ECL) cell | SURFACE | ENTEROCHROMAFFIN-LIKE CELL | BINDING | ENDOCRINE-CELLS
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 06/2009, Volume 296, Issue 6, pp. 1211 - 1220
The orderly differentiation of cell lineages within gastric glands is regulated by a complicated interplay of local mucosal growth factors and hormones.... 
Enterochromaffin-like cell | Metaplasia | Spasmolytic polypeptide-expressing metaplasia | PHYSIOLOGY | metaplasia | ACUTE OXYNTIC ATROPHY | MOUSE STOMACH | EPITHELIAL-CELLS | PARIETAL-CELLS | ZYMOGENIC CELLS | GROWTH-FACTOR-ALPHA | DYNAMICS | spasmolytic polypeptide-expressing metaplasia | MENETRIERS-DISEASE | enterochromaffin-like cell | GASTROENTEROLOGY & HEPATOLOGY | HISTIDINE DECARBOXYLASE-DEFICIENT | TRANSGENIC MICE | Age Factors | Cell Count | Metaplasia - chemically induced | Gastric Mucosa - pathology | Azetidines - pharmacology | Enterochromaffin-like Cells - metabolism | Hypertrophy - pathology | Histidine Decarboxylase - deficiency | Peptides - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Enterochromaffin-like Cells - pathology | Mucins - metabolism | Muscle Proteins - metabolism | Histidine Decarboxylase - genetics | Chromogranin A - metabolism | Gastrins - genetics | Trefoil Factor-2 | Chief Cells, Gastric - metabolism | Enzyme Inhibitors - pharmacology | Piperazines - pharmacology | N-Acetylglucosaminyltransferases - metabolism | Histamine - deficiency | Metaplasia - pathology | Mice, Knockout | Hyperplasia - pathology | Animals | Parietal Cells, Gastric - pathology | Cell Differentiation - drug effects | Chief Cells, Gastric - pathology | Mice | Mice, Inbred BALB C | Gastric Fundus - pathology | Intrinsic Factor - metabolism | Mucosal Biology
Journal Article
Journal Article