X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (3225) 3225
Publication (579) 579
Book Review (40) 40
Book Chapter (32) 32
Newsletter (12) 12
Conference Proceeding (8) 8
Book / eBook (1) 1
Dissertation (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (2127) 2127
index medicus (1682) 1682
animals (1363) 1363
necrotizing enterocolitis (1336) 1336
female (926) 926
infant, newborn (847) 847
pediatrics (837) 837
male (785) 785
infants (538) 538
mice (474) 474
enterocolitis (426) 426
rats (420) 420
inflammation (404) 404
enterocolitis, pseudomembranous - microbiology (378) 378
gastroenterology & hepatology (378) 378
disease models, animal (371) 371
microbiology (357) 357
infant (355) 355
immunology (338) 338
health aspects (316) 316
infant, premature (306) 306
article (299) 299
surgery (290) 290
animals, newborn (285) 285
nutrition & dietetics (278) 278
research (274) 274
preterm infants (261) 261
enterocolitis, pseudomembranous (259) 259
analysis (258) 258
intestinal mucosa - metabolism (254) 254
disease (251) 251
intestine (249) 249
birth-weight infants (247) 247
enterocolitis, neonatal necrotizing (247) 247
diarrhea (243) 243
enterocolitis, necrotizing - metabolism (243) 243
microbiota (242) 242
adult (241) 241
clostridium difficile (240) 240
probiotics (234) 234
expression (229) 229
risk factors (226) 226
pathogenesis (213) 213
infection (211) 211
bacteria (209) 209
pregnancy (209) 209
colitis (208) 208
feces - microbiology (206) 206
intestinal mucosa - pathology (206) 206
physiological aspects (196) 196
rats, sprague-dawley (191) 191
enterocolitis, necrotizing - pathology (185) 185
infectious diseases (183) 183
proteins (182) 182
research article (181) 181
inflammatory-bowel-disease (178) 178
enterocolitis, necrotizing - prevention & control (176) 176
middle aged (175) 175
mice, inbred c57bl (173) 173
cytokines (169) 169
gestational age (166) 166
intestines - microbiology (165) 165
abridged index medicus (163) 163
clostridium difficile - metabolism (163) 163
inflammatory bowel disease (160) 160
nutrition (160) 160
medicine (159) 159
aged (158) 158
premature-infants (157) 157
metabolism (154) 154
diagnosis (149) 149
gene expression (149) 149
antibiotics (147) 147
intestines - metabolism (144) 144
mortality (144) 144
obstetrics & gynecology (144) 144
children (143) 143
prevention (143) 143
swine (143) 143
multidisciplinary sciences (141) 141
physiology (140) 140
child (139) 139
time factors (138) 138
biochemistry & molecular biology (137) 137
enterocolitis, pseudomembranous - drug therapy (137) 137
neonates (136) 136
apoptosis (135) 135
clostridium difficile - pathogenicity (134) 134
clostridium difficile - isolation & purification (133) 133
enteral nutrition (131) 131
digestive system diseases (130) 130
mice, knockout (129) 129
treatment outcome (128) 128
sepsis (127) 127
intestines - pathology (126) 126
cell biology (123) 123
intestinal mucosa - microbiology (123) 123
prospective studies (122) 122
care and treatment (121) 121
milk (120) 120
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (3123) 3123
Russian (35) 35
German (23) 23
Spanish (13) 13
French (12) 12
Japanese (12) 12
Chinese (9) 9
Polish (6) 6
Romanian (4) 4
Serbian (4) 4
Italian (3) 3
Czech (2) 2
Korean (2) 2
Bulgarian (1) 1
Danish (1) 1
Hungarian (1) 1
Portuguese (1) 1
Turkish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Molecular Aspects of Medicine, ISSN 0098-2997, 08/2017, Volume 56, pp. 54 - 65
Bile acids are synthesized from cholesterol in the liver and released into the intestine to aid the digestion of dietary lipids. The host enzymes that... 
Bile salt hydrolase (BSH) | Microbiota | Bile acid inducible (BAI) | Microbiome | Lactobacillus | MEDICINE, RESEARCH & EXPERIMENTAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | CLOSTRIDIUM-DIFFICILE | REUTERI NCIMB 30242 | BIFIDOBACTERIUM-LONGUM | OMEGA-MURICHOLIC ACID | RAT INTESTINAL MICROFLORA | SALT HYDROLASE ACTIVITY | CHOLYLGLYCINE HYDROLASE | SP STRAIN VPI-12708 | LACTOBACILLUS-ACIDOPHILUS | IRRITABLE-BOWEL-SYNDROME | Receptors, G-Protein-Coupled - metabolism | Humans | Enterocolitis, Pseudomembranous - microbiology | Gastrointestinal Microbiome - physiology | Intestines - metabolism | Receptors, Calcitriol - genetics | Biotransformation | NADH, NADPH Oxidoreductases - metabolism | Amidohydrolases - metabolism | Enterocolitis, Pseudomembranous - metabolism | NADH, NADPH Oxidoreductases - genetics | Amidohydrolases - genetics | Liver - metabolism | Bacterial Proteins - genetics | Gene Expression Regulation | Bile Acids and Salts - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Receptors, Calcitriol - metabolism | Enterocolitis, Pseudomembranous - pathology | Clostridium difficile - pathogenicity | Host-Pathogen Interactions | Animals | Intestines - microbiology | Homeostasis - physiology | Bacterial Proteins - metabolism | Liver - cytology | Receptors, G-Protein-Coupled - genetics | Clostridium difficile - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Enzymes | Vitamin D | Microbiota (Symbiotic organisms) | Analysis | Liver | Gastrointestinal diseases | Physiological aspects | Calcifediol | Bacteria | Alfacalcidol | Deoxycholic acid
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2012, Volume 287, Issue 44, pp. 37296 - 37308
Factors regulating the proliferation and apoptosis of intestinal stem cells (ISCs) remain incompletely understood. Because ISCs exist among microbial ligands,... 
PATHOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | INJURY | STEM/PROGENITOR CELLS | COLON | NECROTIZING ENTEROCOLITIS | BETA-CATENIN | PUMA | TLR4 | WNT RECEPTORS | Tumor Necrosis Factor-alpha - metabolism | Stem Cells - immunology | Ileum - pathology | Intestinal Mucosa - metabolism | Cell Proliferation | Receptors, G-Protein-Coupled - metabolism | Enterocolitis, Necrotizing - metabolism | Transcriptional Activation | Tumor Necrosis Factor-alpha - genetics | Receptors, Tumor Necrosis Factor, Type I - metabolism | Stem Cells - metabolism | Adaptor Proteins, Vesicular Transport - metabolism | Intestinal Mucosa - immunology | Tumor Suppressor Proteins - genetics | Apoptosis Regulatory Proteins - genetics | Enterocolitis, Necrotizing - pathology | Tumor Suppressor Proteins - metabolism | Signal Transduction | Mice, Inbred C57BL | Rats | Toll-Like Receptor 4 - genetics | Gene Knockout Techniques | Receptors, Tumor Necrosis Factor, Type I - genetics | Toll-Like Receptor 4 - metabolism | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Tumor Suppressor Proteins - physiology | Animals | Lipopolysaccharides - pharmacology | Apoptosis Regulatory Proteins - physiology | Stem Cells - physiology | Mice | Myeloid Differentiation Factor 88 - metabolism | Apoptosis | Intestinal Mucosa - pathology | Toll-like Receptors (TLR) | Premature | Molecular Bases of Disease | Neonate | Necrotizing Enterocolitis | Inflammation | Lipopolysaccharide (LPS)
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, p. e69620
Neonatal necrotizing enterocolitis (NEC) is a devastating gastrointestinal disease of preterm infants. Increased intestinal epithelium permeability is an early... 
ISCHEMIA-REPERFUSION INJURY | BCL-X-L | SIGNALING PATHWAYS | ISCHEMIA/REPERFUSION INJURY | HUMAN-MILK | CROHNS-DISEASE | MULTIDISCIPLINARY SCIENCES | NITRIC-OXIDE | NECROSIS-FACTOR-ALPHA | DEATH | RAT MODEL | Erythropoietin - pharmacology | Intestines - drug effects | Intestinal Mucosa - metabolism | Epithelial Cells - metabolism | Microtubule-Associated Proteins - genetics | TOR Serine-Threonine Kinases - metabolism | Microtubule-Associated Proteins - metabolism | Enterocolitis, Necrotizing - genetics | Enterocolitis, Necrotizing - metabolism | Epithelial Cells - drug effects | Humans | Caspase 3 - metabolism | Intestines - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Milk, Human - chemistry | Autophagy | Extracellular Signal-Regulated MAP Kinases - genetics | Intestinal Mucosa - drug effects | Proto-Oncogene Proteins c-akt - genetics | TOR Serine-Threonine Kinases - genetics | Caspase 3 - genetics | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | Erythropoietin - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Enterocolitis, Necrotizing - pathology | Infant, Newborn | Disease Models, Animal | Animals, Newborn | Beclin-1 | Gene Expression | Intestines - pathology | Signal Transduction | Enterocolitis, Necrotizing - prevention & control | Membrane Proteins - genetics | Rats | Epithelial Cells - pathology | Apoptosis Regulatory Proteins - metabolism | Erythropoietin - genetics | Animals | Apoptosis | Intestinal Mucosa - pathology | Enterocolitis, Pseudomembranous | Enterocolitis, Neonatal necrotizing | Erythropoietin | Infants (Premature) | Gastrointestinal diseases | Analysis | Permeability | TOR protein | Neonates | Pediatrics | Animal models | Inflammatory bowel diseases | Disease | Gastrointestinal tract diseases | Pathogenesis | Epithelial cells | Necrotizing enterocolitis | AKT protein | Infants | Kinases | Enterocolitis | Caspase-3 | Small intestine | Proteins | Signal transduction | Intestine | Rodents | Gastroenterology | Cell cycle | Supplementation | Growth factors | Milk | Extracellular signal-regulated kinase | Caspase | MAP kinase | Epithelium | Signaling | In vivo methods and tests | Phagocytosis | Breast milk
Journal Article
Haematologica, ISSN 0390-6078, 07/2015, Volume 100, Issue 7, pp. 927 - 934
We previously demonstrated vast expansion of hypoxic areas in the leukemic microenvironment and provided a rationale for using hypoxia-activated prodrugs.... 
STEM-CELLS | INHIBITION | PR-104A | ACUTE MYELOGENOUS LEUKEMIA | BONE-MARROW | I TRIAL | KETO REDUCTASE 1C3 | HIF-1-ALPHA | HEMATOLOGY | CANCER-THERAPY | CHEMOTHERAPY | Recurrence | Humans | Middle Aged | Male | Antineoplastic Agents, Alkylating - administration & dosage | Carbonic Anhydrases - metabolism | Thrombocytopenia - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - drug therapy | Anemia - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Bone Marrow - metabolism | Enterocolitis - genetics | Antigens, Neoplasm - metabolism | Bone Marrow - drug effects | Biomarkers - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - complications | Gene Expression | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Leukemia, Myeloid, Acute - pathology | Anemia - chemically induced | Hypoxia - complications | Thrombocytopenia - chemically induced | Thrombocytopenia - pathology | Nitrogen Mustard Compounds - adverse effects | Remission Induction | Hypoxia - pathology | Antineoplastic Agents, Alkylating - adverse effects | Enterocolitis - pathology | Prodrugs - administration & dosage | Hypoxia - drug therapy | Neutropenia - metabolism | Nitrogen Mustard Compounds - administration & dosage | Carbonic Anhydrases - genetics | Anemia - pathology | Leukemia, Myeloid, Acute - complications | Prodrugs - metabolism | Leukemia, Myeloid, Acute - drug therapy | Neutropenia - pathology | Adult | Female | Neutropenia - chemically induced | Neutropenia - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - pathology | Carbonic Anhydrase IX | Enterocolitis - chemically induced | Antigens, Neoplasm - genetics | Nitroimidazoles - pharmacology | Thrombocytopenia - metabolism | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | Anemia - metabolism | Prodrugs - adverse effects | Hypoxia - genetics | Nitrogen Mustard Compounds - metabolism | Bone Marrow - pathology | Aged | Enterocolitis - metabolism | Antineoplastic Agents, Alkylating - metabolism | Leukemia, Myeloid, Acute - genetics
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 11/2009, Volume 297, Issue 5, pp. G940 - G949
Khailova L, Dvorak K, Arganbright KM, Halpern MD, Kinouchi T, Yajima M, Dvorak B. Bifidobacterium bifidum improves intestinal integrity in a rat model of... 
Adherens junctions | Tight junctions | Intestinal barrier function | Mucins | PHYSIOLOGY | intestinal barrier function | TREFOIL FACTORS | PROBIOTICS | PRETERM INFANTS | mucins | PARACELLULAR PERMEABILITY | adherens junctions | IN-VITRO | INFLAMMATORY-BOWEL-DISEASE | tight junctions | TNF-ALPHA | BACTERIAL-INFECTION | GASTROENTEROLOGY & HEPATOLOGY | COLONIC EPITHELIAL-CELLS | Mucin-2 - genetics | Ileum - pathology | Cadherins - metabolism | Enterocolitis, Necrotizing - therapy | Enterocytes - metabolism | Gene Expression - genetics | Enterocolitis, Necrotizing - metabolism | Tumor Necrosis Factor-alpha - genetics | Stress, Physiological | Ileum - metabolism | Enterocolitis, Necrotizing - microbiology | Intestines - metabolism | Adherens Junctions - metabolism | Incidence | Occludin | Catenins - metabolism | Interleukins - genetics | Trefoil Factor-3 | Asphyxia - complications | Membrane Proteins - metabolism | Neuropeptides - genetics | Enterocolitis, Necrotizing - pathology | Mucin-3 - genetics | Disease Models, Animal | Animals, Newborn | Mucin-2 - metabolism | Tight Junctions - metabolism | Cold Temperature | Bifidobacterium | Intestines - pathology | Rats | Neuropeptides - metabolism | Rats, Sprague-Dawley | Claudin-3 | Animals | Intestines - microbiology | Probiotics - therapeutic use | Junctional complexes (Epithelium) | Enterocolitis, Pseudomembranous | Enterocolitis, Neonatal necrotizing | Physiological aspects | Cell junctions | Development and progression | Models | Properties | Mediators | Inflammation | Immunity
Journal Article
Journal Article
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 01/2013, Volume 304, Issue 2, pp. G132 - G141
Ganguli K, Meng D, Rautava S, Lu L, Walker WA, Nanthakumar N. Probiotics prevent necrotizing enterocolitis by modulating enterocyte genes that regulate innate... 
Toll-like receptors | Development of intestinal defense | Probiotic secretions | Innate immune response | Bifidobacterium infantis | probiotic secretions | DEVELOPING HUMAN INTESTINE | CONTROLLED-TRIAL | CELLS | PHYSIOLOGY | BIRTH-WEIGHT INFANTS | innate immune response | development of intestinal defense | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Toll-Like Receptor 2 - genetics | Enterocolitis, Necrotizing - genetics | Humans | Receptors, Interleukin-1 - genetics | Immunity, Innate - genetics | Enterocolitis, Necrotizing - microbiology | Intracellular Signaling Peptides and Proteins - metabolism | Bifidobacterium - growth & development | Intestine, Small - transplantation | RNA Interference | Bifidobacterium - metabolism | Inflammation Mediators - metabolism | Interleukin-8 - metabolism | Lactobacillus acidophilus - growth & development | Interleukin-6 - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Intestine, Small - immunology | Organ Culture Techniques | Enterocolitis, Necrotizing - prevention & control | Intestine, Small - microbiology | Cells, Cultured | Gene Expression Regulation | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 2 - metabolism | Mice, SCID | Enterocytes - microbiology | Toll-Like Receptor 4 - metabolism | Enterocytes - immunology | Receptors, Interleukin-1 - metabolism | Probiotics | Animals | Mice | Enterocolitis, Necrotizing - immunology | Primary Cell Culture | Culture Media, Conditioned - metabolism | Lactobacillus acidophilus - metabolism | Mediators | Inflammation | Immunity
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 04/2008, Volume 294, Issue 4, pp. G906 - G913
Journal Article
Gastroenterology, ISSN 0016-5085, 2008, Volume 134, Issue 3, pp. 768 - 780.e2
Background & Aims: Intestinal fibrosis and stricture formation are serious complications of Crohn’s disease, often requiring surgical intervention.... 
Gastroenterology and Hepatology | SEROVAR TYPHIMURIUM | IGF-I | INFLAMMATORY-BOWEL-DISEASE | CROHNS-DISEASE | COLITIS | ANIMAL-MODELS | MECHANISMS | GROWTH-FACTOR | LESSONS | GASTROENTEROLOGY & HEPATOLOGY | MOUSE MODELS | Enterocolitis - pathology | Up-Regulation | Species Specificity | Salmonella Infections - genetics | Transforming Growth Factor beta1 - metabolism | Salmonella Infections - metabolism | Cecum - microbiology | Salmonella typhimurium - metabolism | Immediate-Early Proteins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Time Factors | Cecum - metabolism | Enterocolitis - genetics | Cation Transport Proteins - genetics | Mice, Inbred DBA | Cation Transport Proteins - deficiency | Disease Models, Animal | Severity of Illness Index | Salmonella Infections - pathology | Streptomycin - administration & dosage | Collagen Type I - metabolism | Cytokines - metabolism | Salmonella Infections - complications | Salmonella typhimurium - genetics | Salmonella typhimurium - pathogenicity | Colon - pathology | Enterocolitis - complications | Intestinal Diseases - genetics | Fibroblasts - pathology | Enterocolitis - microbiology | Intestinal Diseases - metabolism | Colon - metabolism | Mice, Inbred C3H | Cecum - pathology | Animals | Fibrosis | Intestinal Diseases - microbiology | Salmonella Infections - microbiology | Virulence Factors - metabolism | Colon - microbiology | Mice | Enterocolitis - metabolism | Mutation | Chronic Disease | Insulin-Like Growth Factor I - metabolism | Connective Tissue Growth Factor | Intestinal Diseases - pathology | Salmonella | Transforming growth factors | Biological response modifiers | Tumor necrosis factor | Health aspects | Analysis
Journal Article
Life Sciences, ISSN 0024-3205, 02/2017, Volume 170, pp. 72 - 81
Journal Article