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Gut, ISSN 0017-5749, 04/2006, Volume 55, Issue 4, pp. 469 - 477
Background and aims: Epithelium derived interleukin (IL)-15 signalling via IL-15Rα is critical for the development, activation, and survival of intraepithelial... 
RHEUMATOID-ARTHRITIS | NATURAL-KILLER | HOMEOSTASIS | INFLAMMATORY-BOWEL-DISEASE | NK CELLS | IL-15 | RECEPTOR | BIOPSY SPECIMENS | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | T-CELLS | Intestinal Mucosa - metabolism | Enterocytes - metabolism | Humans | Middle Aged | Receptors, Interleukin-15 | Interferon-gamma - metabolism | Th1 Cells - immunology | Interleukin-15 - immunology | Chloroquine - pharmacology | Lymphocytes - immunology | Celiac Disease - immunology | Cell Division - immunology | Intestinal Mucosa - immunology | Tumor Necrosis Factor-alpha - immunology | Antibodies - immunology | Celiac Disease - complications | Adult | Female | Pore Forming Cytotoxic Proteins | Membrane Glycoproteins - immunology | Perforin | Cytotoxicity, Immunologic - drug effects | Intestine, Small - immunology | Cytokines - immunology | Caco-2 Cells | Lymphocytes - metabolism | Cytokines - metabolism | Cells, Cultured | Receptors, Interleukin-2 - metabolism | Receptors, Interleukin-2 - immunology | Enterocytes - immunology | Apoptosis - immunology | Epithelial Cells - immunology | Interferon-gamma - immunology | Interleukin-15 - metabolism | Aged | Cytotoxicity, Immunologic - immunology | Celiac disease | Epithelial cells | Lymphocytes | Interleukin-15 | Development and progression | Research | Gene expression | Disease | Cytokines | Homeostasis | Cytotoxicity | T cell receptors | Lymphomas | Histology | Gluten | Age | Apoptosis | intraepithelial lymphocyte | Coeliac Disease | interleukin 15 | T cell lymphoma | apoptosis | enterocyte | cytotoxicity
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2010, Volume 120, Issue 6, pp. 2131 - 2143
Enteropathy-associated T cell lymphoma is a severe complication of celiac disease (CD). One mechanism suggested to underlie its development is chronic exposure... 
INTESTINAL INFLAMMATION | RHEUMATOID-ARTHRITIS | MEDICINE, RESEARCH & EXPERIMENTAL | MOLECULAR TARGETS | NATURAL-KILLER-CELLS | INTERLEUKIN-15 | BCL-X | RECEPTOR | REFRACTORY SPRUE | CD8(+) T-CELLS | TRANSGENIC MICE | Phosphorylation | Apoptosis - drug effects | Enterocytes - metabolism | Humans | Intestines - metabolism | Leukemia - metabolism | STAT3 Transcription Factor - pharmacology | Interleukin-15 - immunology | Intestines - immunology | Inflammation - complications | Signal Transduction - immunology | Inflammation - metabolism | Lymphocytes - immunology | Celiac Disease - immunology | Leukemia - complications | Celiac Disease - complications | Adult | Janus Kinase 3 - metabolism | Intestine, Small - immunology | STAT3 Transcription Factor - metabolism | Cytokines - immunology | Janus Kinase 3 - immunology | Lymphocytes - metabolism | Cytokines - metabolism | Inflammation - immunology | Protein Binding - immunology | Enterocytes - immunology | Interleukin-15 - pharmacology | Apoptosis - immunology | Signal Transduction - drug effects | Leukemia - immunology | Interleukin-15 - metabolism | Intestine, Small - metabolism | STAT3 Transcription Factor - immunology | Celiac Disease - metabolism | Cytokines - pharmacology | Care and treatment | Lymphocytes | Development and progression | Interleukin-15 | Inflammation | Non-Hodgkin's lymphomas | T cells | Properties | Health aspects
Journal Article
Mucosal Immunology, ISSN 1933-0219, 09/2016, Volume 9, Issue 5, pp. 1137 - 1150
The gut CD4(+) Tcells, particularly the T helper type 17 (Th17) subset, are not completely restored in most HIV-1-infected individuals despite combined... 
MUCOSAL IMMUNE RECONSTITUTION | POOR IMMUNOLOGICAL RESPONDERS | HIV-INFECTION | INTEGRIN ALPHABETA | DENDRITIC CELLS | ACTIVE ANTIRETROVIRAL THERAPY | IMMUNOLOGY | HUMAN-IMMUNODEFICIENCY-VIRUS | TYPE-1 INFECTION | INTESTINAL EPITHELIAL-CELLS | THYMUS-EXPRESSED CHEMOKINE | Receptors, CCR6 - immunology | Humans | Middle Aged | Male | Intestinal Mucosa - drug effects | T-Lymphocytes, Regulatory - immunology | HIV Infections - immunology | Th17 Cells - drug effects | HIV-1 - growth & development | Intestinal Mucosa - immunology | Chemokine CCL20 - genetics | Chemotaxis - immunology | Intestine, Small - immunology | CD4 Antigens - genetics | Signal Transduction | HIV Infections - virology | Interleukin-17 - genetics | Chemotaxis - drug effects | Feedback, Physiological | T-Lymphocytes, Regulatory - drug effects | Enterocytes - drug effects | Interleukin-10 - genetics | Receptors, CCR6 - genetics | Th17 Cells - immunology | Interleukin-10 - immunology | T-Lymphocytes, Regulatory - virology | Forkhead Transcription Factors - immunology | Interleukin-17 - immunology | Enterocytes - virology | Th17 Cells - virology | Case-Control Studies | Chemokine CCL20 - immunology | HIV Infections - pathology | Adult | Anti-HIV Agents - therapeutic use | Female | Transforming Growth Factor beta - immunology | CD4 Antigens - immunology | HIV-1 - drug effects | Gene Expression Regulation | Receptors, CCR6 - deficiency | Intestinal Mucosa - virology | Intestine, Small - virology | Forkhead Transcription Factors - genetics | Enterocytes - immunology | Antiretroviral Therapy, Highly Active | Transforming Growth Factor beta - genetics | Intestine, Small - drug effects | Lymphocyte Count | HIV Infections - drug therapy
Journal Article
Mucosal Immunology, ISSN 1933-0219, 11/2012, Volume 5, Issue 6, pp. 646 - 657
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, p. e59259
Background: We previously showed that evaluation of anti-inflammatory activities of lactic acid bacteria in porcine intestinal epithelial (PIE) cells is useful... 
IMMUNE-RESPONSE | ACTIVATION | BACTERIA | HOMEOSTASIS | TOLL-LIKE-RECEPTOR-2 | ENTEROCYTES | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | GUT | NEGATIVE REGULATORS | NF-KAPPA-B | Bifidobacterium - immunology | Interleukin-8 - genetics | Toll-Like Receptor 2 - genetics | Chemokine CCL2 - immunology | NF-kappa B - immunology | Intestines - immunology | Swine | Biological Assay | Mitogen-Activated Protein Kinases - immunology | Epithelial Cells - cytology | Bifidobacterium - chemistry | DNA-Binding Proteins - immunology | Interleukin-6 - genetics | Signal Transduction | Anti-Inflammatory Agents - pharmacology | Immunomodulation | Cells, Cultured | Gene Expression Regulation | Toll-Like Receptor 4 - genetics | Chemokine CCL2 - genetics | Toll-Like Receptor 4 - immunology | Probiotics - pharmacology | DNA-Binding Proteins - genetics | Animals | NF-kappa B - genetics | Epithelial Cells - immunology | Interleukin-6 - immunology | Toll-Like Receptor 2 - immunology | Mitogen-Activated Protein Kinases - genetics | Interleukin-8 - immunology | Intestines - cytology | Ubiquitin | Enzymes | Interleukins | Escherichia coli | Bacteria | Mitogens | Protein kinases | Regulators | Lactic acid bacteria | Laboratories | Epithelial cells | Interleukin | Editing | Homeostasis | Inflammatory response | Activation | Food science | Kinases | Inflammatory diseases | Interleukin 6 | Proteins | Microorganisms | Immunology | Pathways | E coli | Intestine | Rodents | Modulation | Toll-like receptors | Tumor necrosis factor-TNF | A20 protein | Strains (organisms) | Immune system | NF-κB protein | Cytokines | Immunoregulation | MAP kinase | Inflammation | TLR4 protein | Inflammatory bowel disease | Probiotics | Monocytes | Molecular modelling | Protein kinase | TLR2 protein | Biomarkers | Regulation | Lactic acid
Journal Article
Immunity, ISSN 1074-7613, 2004, Volume 21, Issue 3, pp. 367 - 377
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