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Journal of neuroimmunology, ISSN 0165-5728, 07/2015, Volume 284, pp. 18 - 29
...) and D5R, and how this modulation affects CD4+ T-cell activation and differentiation. Our pharmacologic and genetic evidence shows that D3R-stimulation reduced cAMP levels and ERK2-phosphorylation, consequently increasing CD4... 
Neurology | Allergy and Immunology | Knockout mice | Th1 differentiation | Neuroimmunology | TCR signalling | T-cell activation | Dopamine receptors | Neurosciences | Immunology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | T-Lymphocytes - physiology | Receptors, Dopamine D3 - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Dose-Response Relationship, Drug | Receptors, Dopamine D3 - genetics | Cell Differentiation - genetics | Lymphocyte Activation - genetics | Phosphorylation - genetics | Dopamine Agents - pharmacology | T-Lymphocytes - drug effects | Phosphorylation - drug effects | Cell Differentiation - physiology | Cyclic AMP - metabolism | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Signal Transduction - genetics | Enzyme Activation - drug effects | Mice, Knockout | Gene Expression Regulation - drug effects | Animals | Interleukin-2 Receptor alpha Subunit - metabolism | Signal Transduction - drug effects | Cell Differentiation - drug effects | Lymphocyte Activation - drug effects | Ionomycin - pharmacology | Receptors, Dopamine D5 - metabolism | Mice | Receptors, Dopamine D5 - genetics | Enzyme Activation - genetics | CD4 Antigens - metabolism | Index Medicus
Journal Article
Nature medicine, ISSN 1546-170X, 06/2012, Volume 18, Issue 7, pp. 1095 - 1101
...) by activation of mammalian target of rapamycin (mTOR), thus maintaining proper bone microarchitecture and mass... 
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Bone Matrix - metabolism | Immunohistochemistry | Mesenchymal Stromal Cells - enzymology | Bone and Bones - pathology | Receptor, IGF Type 1 - metabolism | Insulin-Like Growth Factor I - pharmacology | Femur - pathology | TOR Serine-Threonine Kinases - metabolism | Cell Count | Humans | Aging - drug effects | Insulin-Like Growth Factor Binding Protein 3 - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Femur - diagnostic imaging | Insulin Receptor Substrate Proteins - metabolism | X-Ray Microtomography | Bone and Bones - drug effects | Insulin-Like Growth Factor Binding Protein 3 - metabolism | Bone Matrix - drug effects | Bone and Bones - metabolism | Insulin-Like Growth Factor I - administration & dosage | Bone Resorption - metabolism | Insulin-Like Growth Factor Binding Protein 3 - administration & dosage | Receptor, IGF Type 1 - deficiency | Proto-Oncogene Proteins c-akt - metabolism | Femur - growth & development | Mesenchymal Stromal Cells - drug effects | Bone Resorption - blood | Osteoblasts - enzymology | Osteoblasts - drug effects | Osteogenesis - drug effects | Rats | Enzyme Activation - drug effects | Mice, Knockout | Bone Resorption - diagnostic imaging | Osteoblasts - pathology | Organ Size - drug effects | Animals | Cell Differentiation - drug effects | Bone Resorption - pathology | Mice | Mesenchymal Stromal Cells - pathology | Insulin-Like Growth Factor I - metabolism | Aging - metabolism | Physiological aspects | Research | Insulin-like growth factor 1 | Protein kinases | Stem cells | Bone marrow | Insulin-like growth factors | Index Medicus
Journal Article
British journal of cancer, ISSN 1532-1827, 04/2009, Volume 100, Issue 9, pp. 1425 - 1433
Curcumin has been shown to inhibit the growth of various types of cancer cells; however, at concentrations much above the clinically achievable levels in... 
Curcumin | Chk1 | G2/M arrest | DNA damage | Pancreatic cancer | Life Sciences & Biomedicine | Oncology | Science & Technology | Cell Cycle Proteins - drug effects | Protein Kinases - metabolism | Gene Expression Regulation, Enzymologic - drug effects | Protein Kinases - genetics | Gene Silencing - drug effects | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | Collagen Type XI - metabolism | DNA-Binding Proteins - metabolism | Pancreatic Neoplasms - drug therapy | Transfection | Tumor Suppressor Proteins - genetics | Caspase 3 - drug effects | Cell Cycle Proteins - genetics | Gene Expression Regulation, Neoplastic - drug effects | Protein-Serine-Threonine Kinases - metabolism | DNA Damage - drug effects | Protein Kinases - drug effects | Tumor Suppressor Proteins - metabolism | Collagen Type XI - drug effects | Pancreatic Neoplasms - pathology | Cell Cycle Proteins - metabolism | Curcumin - pharmacology | DNA-Binding Proteins - drug effects | Pancreatic Neoplasms - enzymology | Protein-Serine-Threonine Kinases - genetics | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | Cell Division - drug effects | Protein-Serine-Threonine Kinases - drug effects | Cell Line, Tumor | Checkpoint Kinase 1 | Tumor Suppressor Proteins - drug effects | Cell Cycle - drug effects | Index Medicus | Translational Therapeutics | pancreatic cancer | M arrest | curcumin
Journal Article
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 11/2012, Volume 60, Issue 5, pp. 1301 - 1308
Journal Article
Journal of lipid research, ISSN 1539-7262, 08/2017, Volume 58, Issue 8, pp. 1536 - 1547
Journal Article
PloS one, ISSN 1932-6203, 08/2012, Volume 7, Issue 8, pp. e44107 - e44107
.... We investigated the capacity of resveratrol to protect RAW 264.7 cells from inflammatory insults and explored mechanisms underlying inhibitory effects of resveratrol on RAW 264.7 cells... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Dinoprostone - secretion | Sirtuin 1 - metabolism | Phosphatidylinositol 3-Kinase - antagonists & inhibitors | Tumor Necrosis Factor-alpha - genetics | Stilbenes - chemistry | Interleukin-1beta - genetics | Stilbenes - pharmacology | RNA, Messenger - metabolism | Cyclooxygenase 2 - genetics | Inflammation Mediators - metabolism | Interleukin-1beta - biosynthesis | Phosphorylation - drug effects | Phosphatidylinositol 3-Kinase - metabolism | Cell Line | Cell Survival - drug effects | RNA, Messenger - genetics | Enzyme Activation - drug effects | Macrophages - enzymology | Gene Expression Regulation - drug effects | Nitric Oxide - secretion | Animals | MAP Kinase Signaling System - drug effects | Androstadienes - pharmacology | Nitric Oxide Synthase Type II - genetics | Signal Transduction - drug effects | Models, Biological | Cyclic AMP Response Element-Binding Protein - metabolism | Cyclooxygenase 2 - metabolism | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Mice | Tumor Necrosis Factor-alpha - biosynthesis | Mitogen-Activated Protein Kinases - metabolism | Nitric Oxide Synthase Type II - metabolism | Resveratrol | Physiological aspects | Inflammation | Genetic aspects | Research | Drug therapy | Lipopolysaccharides | Mitogen-activated protein kinases | Phosphorylation | Interleukin | Prostaglandin E2 | AKT protein | mRNA | Kinases | AMP-activated protein kinase | Macrophages | Inflammatory diseases | Cell adhesion & migration | Anticancer properties | Proteins | Signal transduction | Cell activation | Metabolites | Rodents | Cascades | Tumor necrosis factor-TNF | Inhibition | Enzyme-linked immunosorbent assay | Cytokines | Cyclic AMP | Bioavailability | Tumor necrosis factor-α | IL-1β | Metabolism | Cyclic AMP response element-binding protein | SIRT1 protein | Anti-inflammatory agents | Nitric-oxide synthase | 1-Phosphatidylinositol 3-kinase | Polymerase chain reaction | Signaling | Inhibitors | Nitric oxide | Cyclooxygenase-2 | Immunofluorescence | Index Medicus | Activation
Journal Article
PloS one, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, pp. e84800 - e84800
.... The aim of our present study was to investigate whether the protective effect of LBP after I/R damage was mediated via activation of the Nrf2/HO-1-antioxidant pathway in the retina... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Amacrine Cells - drug effects | Retina - drug effects | Heme Oxygenase-1 - metabolism | Retina - metabolism | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Amacrine Cells - metabolism | Drugs, Chinese Herbal - pharmacology | Male | Retinal Ganglion Cells - metabolism | Retinal Ganglion Cells - pathology | Heme Oxygenase-1 - genetics | Cell Nucleus - metabolism | Disease Models, Animal | Reperfusion Injury | Rats | Enzyme Activation - drug effects | Protein Transport | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | NF-E2-Related Factor 2 - metabolism | Oxidative Stress - drug effects | Retina - pathology | Lycium - chemistry | Retinal Ganglion Cells - drug effects | Antioxidants | Polysaccharides | Nervous system diseases | Ischemia | Analysis | Heme | Health aspects | Glaucoma | Oxidative stress | Neuroprotection | Diabetic retinopathy | Neurosciences | Transcription factors | Laboratories | Retina | Alcohol | Activation | Kinases | Western blotting | Embryology | Reperfusion | Protoporphyrin | Education | Rodents | Aging | Carbon monoxide | Damage | Saccharides | Medical research | Enzymes | Cell survival | Neurodegenerative diseases | Heme oxygenase (decyclizing) | Gene expression | Zinc | Neurological diseases | Medicine | Studies | Oxygenase | Brain research | Inhibitors | Visual impairment | Immunofluorescence | Mercury | Apoptosis | Intraocular pressure | Animal cognition | Index Medicus
Journal Article
Journal Article