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Nature, ISSN 0028-0836, 01/2015, Volume 517, Issue 7536, pp. 583 - 588
Journal Article
Nature, ISSN 0028-0836, 10/2015, Volume 526, Issue 7575, pp. 700 - 704
Journal Article
Cancer Cell, ISSN 1535-6108, 08/2012, Volume 22, Issue 2, pp. 153 - 166
Genomic profiling has identified a subtype of high-risk B-progenitor acute lymphoblastic leukemia (B-ALL) with alteration of , a gene expression profile... 
GROWTH-FACTOR RECEPTOR | BCR-JAK2 FUSION GENE | ONCOLOGY | NUCLEAR-PORE | MYELOPROLIFERATIVE NEOPLASMS | ACUTE MYELOID-LEUKEMIA | OF-FUNCTION MUTATIONS | FLT3 MUTATIONS | CHILDHOOD | B-PROGENITOR | CHRONIC MYELOMONOCYTIC LEUKEMIA | CELL BIOLOGY | Recurrence | Humans | Molecular Sequence Data | RNA, Messenger - metabolism | Receptor, Platelet-Derived Growth Factor beta - genetics | Protein-Tyrosine Kinases - genetics | DNA Mutational Analysis | Base Sequence | Trans-Activators - genetics | Phosphorylation - drug effects | Philadelphia Chromosome | Receptors, Cytokine - genetics | Genetic Predisposition to Disease | RNA, Messenger - genetics | Risk Factors | Gene Expression Regulation, Leukemic - drug effects | Signal Transduction - genetics | Enzyme Activation - drug effects | Mutation - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - genetics | Animals | Signal Transduction - drug effects | Cell Transformation, Neoplastic | Oncogene Proteins, Fusion - genetics | Precursor Cell Lymphoblastic Leukemia-Lymphoma - enzymology | Mice | Protein Kinase Inhibitors - pharmacology | Gene Rearrangement - genetics | Sequence Deletion - genetics | Tyrosine | Platelet-derived growth factor | Cytokines | Genes | Oncology, Experimental | Genomes | Research | Gene expression | Population genetics | Hunger | Medical genetics | Genetic research | Nucleotide sequencing | Acute lymphocytic leukemia | Cancer | DNA sequencing | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2014, Volume 111, Issue 46, pp. 16508 - 16513
The pyruvate dehydrogenase complex (PDH) has been hypothesized to link lipid exposure to skeletal muscle insulin resistance through a glucose-fatty acid cycle... 
Dehydrogenases | Muscles | Insulin resistance | Lipids | Oxidation | Mice | Type 2 diabetes mellitus | Insulin | Fatty acids | Skeletal muscle | Liquid chromatography mass spectrometry | Nuclear magnetic resonance | Protein kinase C theta | Diacylglycerol | Insulin action | PROTEIN-KINASE-C | COMPLEX | FATTY LIVER-DISEASE | MULTIDISCIPLINARY SCIENCES | BLOOD-GLUCOSE | nuclear magnetic resonance | POTENTIAL MECHANISM | insulin action | DIABETES-MELLITUS | PHOSPHATIDYLINOSITOL 3-KINASE ACTIVITY | INHIBITION | protein kinase C theta | liquid chromatography mass spectrometry | RAT-HEART | diacylglycerol | GLUCOSE-TOLERANCE | Protein-Serine-Threonine Kinases - deficiency | Phosphorylation | Reactive Oxygen Species - metabolism | Oxidative Stress | Protein Kinase C-theta | Substrate Specificity | Muscle, Skeletal - metabolism | Carnitine - metabolism | Pyruvate Dehydrogenase Complex - genetics | RNA, Messenger - biosynthesis | Glycogen - metabolism | Dietary Fats - pharmacology | Insulin Resistance - physiology | Isoenzymes - metabolism | Protein Kinase C - metabolism | Nuclear Magnetic Resonance, Biomolecular | Carnitine - analogs & derivatives | Fatty Acids - metabolism | Hyperinsulinism - metabolism | Pyruvate Dehydrogenase Complex - metabolism | Oxidation-Reduction | Protein-Serine-Threonine Kinases - physiology | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Citric Acid Cycle | Mice, Knockout | Animals | Models, Biological | Glucose - metabolism | Protein Processing, Post-Translational | Enzyme Activation | Dietary Fats - toxicity | Pyruvates | Physiological aspects | Genetic research | Genetic aspects | Research | Gene expression | Enzymes | Musculoskeletal system | Signal transduction | Cells | Index Medicus | Biological Sciences
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2014, Volume 111, Issue 30, pp. 11181 - 11186
Acute glaucoma is a sight-threatening condition characterized by a sudden and substantial rise in intraocular pressure (IOP) and consequent retinal ganglion... 
Glaucoma | Reperfusion | Messenger RNA | Ischemia | Toll like receptors | Central nervous system | Retina | Brain damage | Intraocular pressure | Microglia | Retinal ischemia/reperfusion injury | Cell apoptosis | BRAIN-INJURY | MICROGLIA ACTIVATION | INTERLEUKIN-1-BETA | MULTIDISCIPLINARY SCIENCES | REPERFUSION INJURY | POPULATIONS | retinal ischemia/reperfusion injury | ANGLE-CLOSURE GLAUCOMA | cell apoptosis | TOLL-LIKE RECEPTORS | CENTRAL-NERVOUS-SYSTEM | MICE | CEREBRAL-ISCHEMIA | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Caspase 8 - metabolism | Caspase 1 - metabolism | Interleukin-1beta - genetics | Caspase 8 - genetics | Interleukin-1beta - metabolism | Apoptosis Regulatory Proteins - genetics | Eye Proteins - genetics | Glaucoma - metabolism | Disease Models, Animal | Acute Disease | Rats | Toll-Like Receptor 4 - genetics | Receptors, Cytoplasmic and Nuclear - genetics | Nerve Tissue Proteins - genetics | Rats, Sprague-Dawley | Toll-Like Receptor 4 - metabolism | Inflammasomes - genetics | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Eye Proteins - metabolism | Caspase 1 - genetics | Adaptor Proteins, Signal Transducing - genetics | Mice | Adaptor Proteins, Signal Transducing - metabolism | Enzyme Activation - genetics | Glaucoma - genetics | Receptors, Cytoplasmic and Nuclear - metabolism | Index Medicus | Biological Sciences | reperfusion injury | retinal ischemia
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 2014, Volume 124, Issue 5, pp. 2234 - 2245
Tissue inflammation in several autoimmune diseases, including SLE and MS, has been linked to an imbalance of IL-17-producing Th (Th17) cells and Tregs;... 
EPIGENETIC MECHANISMS | CRUCIAL ROLE | MEDICINE, RESEARCH & EXPERIMENTAL | KINASE TYPE-IV | SYSTEMIC-LUPUS-ERYTHEMATOSUS | MULTIPLE-SCLEROSIS | INTERLEUKIN-17 | SUPPRESSES IL-2 PRODUCTION | HELPER-CELLS | ROR-GAMMA | REGULATORY T-CELLS | RNA, Messenger - immunology | Humans | Interleukin-17 - immunology | Male | Lupus Erythematosus, Systemic - therapy | Calcium-Calmodulin-Dependent Protein Kinase Type 4 - genetics | Enzyme Activation - immunology | Proto-Oncogene Proteins c-akt - genetics | Cell Differentiation - genetics | TOR Serine-Threonine Kinases - genetics | Lupus Erythematosus, Systemic - immunology | Female | Th17 Cells - pathology | RNA, Messenger - genetics | Interleukin-17 - genetics | Multiple Sclerosis - genetics | Cyclic AMP Response Element Modulator - immunology | Multiple Sclerosis - therapy | Mice, Knockout | Cell Differentiation - immunology | TOR Serine-Threonine Kinases - immunology | Animals | Cyclic AMP Response Element Modulator - genetics | Proto-Oncogene Proteins c-akt - immunology | Lupus Erythematosus, Systemic - genetics | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Th17 Cells - immunology | Mice | Enzyme Activation - genetics | Lupus Erythematosus, Systemic - pathology | Calcium-Calmodulin-Dependent Protein Kinase Type 4 - immunology | Index Medicus | Abridged Index Medicus
Journal Article