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Circulation Research, ISSN 0009-7330, 01/2009, Volume 104, Issue 1, pp. 15 - 18
Ischemic postconditioning (IPoC) reduces infarct size following ischemia/reperfusion. Whether or not phosphorylation of RISK (reperfusion injury salvage... 
CARDIAC & CARDIOVASCULAR SYSTEMS | TRANSITION PORE | PROTECTION | REPERFUSION | reperfusion injury | NECROSIS-FACTOR-ALPHA | ADENOSINE RECEPTORS | infarct size | INHIBITION | MYOCARDIAL INFARCT SIZE | cardioprotection | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | MOUSE HEARTS | SIGNAL TRANSDUCER | Glycogen Synthase Kinase 3 - physiology | MAP Kinase Kinase 2 - physiology | Nitriles - pharmacology | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Glycogen Synthase Kinase 3 beta | Ribosomal Protein S6 Kinases, 70-kDa - physiology | MAP Kinase Kinase 1 - physiology | Myocardial Reperfusion Injury - enzymology | Ribosomal Protein S6 Kinases, 70-kDa - biosynthesis | Protein Processing, Post-Translational - drug effects | Swine | Mitogen-Activated Protein Kinase 3 - biosynthesis | Myocardial Infarction - pathology | Swine, Miniature | Phosphorylation - drug effects | Myocardial Infarction - enzymology | MAP Kinase Kinase 1 - antagonists & inhibitors | Butadienes - pharmacology | Glycogen Synthase Kinase 3 - biosynthesis | Mitogen-Activated Protein Kinase 1 - physiology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Ribosomal Protein S6 Kinases, 70-kDa - antagonists & inhibitors | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Coronary Occlusion - enzymology | Enzyme Induction | Proto-Oncogene Proteins c-akt - physiology | Enzyme Activation - drug effects | Mitogen-Activated Protein Kinase 3 - physiology | Mitogen-Activated Protein Kinase 1 - biosynthesis | Proto-Oncogene Proteins c-akt - biosynthesis | Animals | Androstadienes - pharmacology | Myocardial Reperfusion | MAP Kinase Kinase 2 - antagonists & inhibitors | Coronary Occlusion - pathology | Phosphatidylinositol 3-Kinases - physiology | Protein Kinase Inhibitors - pharmacology | Myocardial Reperfusion Injury - prevention & control | Proto-Oncogene Proteins c-akt - antagonists & inhibitors
Journal Article
Journal Article
Journal of neurochemistry, ISSN 0022-3042, 2013, Volume 127, Issue 3, pp. 303 - 313
Journal Article
Cold Spring Harbor Perspectives in Biology, ISSN 1943-0264, 06/2013, Volume 5, Issue 6, pp. a008672 - a008672
Journal Article
Journal of neurochemistry, ISSN 1471-4159, 2009, Volume 110, Issue 5, pp. 1547 - 1556
Journal Article
The Journal of neuroscience, ISSN 1529-2401, 2015, Volume 35, Issue 37, pp. 12917 - 12931
...; however, the molecular and neural circuit mechanisms are not understood. In this study, we report that KOR activation of p38 alpha MAPK in ventral tegmental (VTA... 
Depression | GIRK | Dopamine | Stress | Dynorphin | Kappa opioid receptor | MOUSE-BRAIN | SYSTEM | stress | PLACE AVERSION | dynorphin | kappa opioid receptor | TYROSINE PHOSPHORYLATION | COCAINE | SEROTONIN NEURONS | NEUROSCIENCES | IN-VIVO | ACCUMBENS | depression | DORSAL RAPHE NUCLEUS | dopamine | Potassium - metabolism | p38 Mitogen-Activated Protein Kinases - deficiency | MAP Kinase Signaling System - physiology | Recombinant Fusion Proteins - pharmacology | Nucleus Accumbens - metabolism | Analgesics, Non-Narcotic - pharmacology | Male | Ventral Tegmental Area - cytology | Ventral Tegmental Area - physiology | Gene Knockdown Techniques | Protein Processing, Post-Translational - drug effects | Dopamine - physiology | Dopaminergic Neurons - physiology | G Protein-Coupled Inwardly-Rectifying Potassium Channels - metabolism | Phosphorylation - drug effects | Action Potentials - drug effects | Dopamine - metabolism | Receptors, Opioid, kappa - genetics | Serotonergic Neurons - physiology | Conditioning, Classical - drug effects | p38 Mitogen-Activated Protein Kinases - physiology | Mice, Inbred C57BL | p38 Mitogen-Activated Protein Kinases - genetics | Avoidance Learning - physiology | Receptors, Opioid, kappa - deficiency | Conditioning, Classical - physiology | 3,4-Dichloro-N-methyl-N-(2-(1-pyrrolidinyl)-cyclohexyl)-benzeneacetamide, (trans)-Isomer - pharmacology | Receptors, Opioid, kappa - physiology | Rotarod Performance Test | Animals | Avoidance Learning - drug effects | Mice | Enzyme Activation | Ion Channel Gating - drug effects
Journal Article
Journal Article
Brain, behavior, and immunity, ISSN 0889-1591, 2014, Volume 41, Issue 1, pp. 90 - 100
.... We found that CUMS procedure significantly caused PFC nuclear factor kappa B (NF-κB) inflammatory pathway activation in rats... 
Psychiatry | Allergy and Immunology | Depression | IL-1β-related CNS inflammation | Chronic unpredictable mild stress | Prefrontal cortex | NLRP3 inflammasome | ANTIDEPRESSANT | CHRONIC MILD STRESS | CYTOKINE LEVELS | MAJOR DEPRESSION | IMMUNOLOGY | IL-1 beta-related CNS inflammation | FLUOXETINE | NEUROSCIENCES | TOLL-LIKE | DISORDER | ALTERED EXPRESSION | PITUITARY-ADRENAL AXIS | BRAIN | Microglia - metabolism | Rats, Wistar | Sucrose | Depressive Disorder - pathology | NLR Family, Pyrin Domain-Containing 3 Protein | Nerve Tissue Proteins - analysis | Stress, Physiological | Depressive Disorder - drug therapy | Male | NF-kappa B - metabolism | Receptors, Cytoplasmic and Nuclear - biosynthesis | Interleukin-1beta - blood | Prefrontal Cortex - pathology | Nerve Tissue Proteins - biosynthesis | Microglia - pathology | Interleukin-1beta - biosynthesis | Fluoxetine - therapeutic use | Carrier Proteins | Interleukin-1beta - physiology | Nerve Tissue Proteins - physiology | Calcium-Binding Proteins - analysis | Gene Expression Regulation | Interleukin-1beta - cerebrospinal fluid | Microfilament Proteins - analysis | Rats | Anhedonia | Receptors, Cytoplasmic and Nuclear - physiology | Inflammation | Random Allocation | Depressive Disorder - metabolism | Drinking Behavior | Antidepressive Agents - therapeutic use | Inflammasomes - physiology | Animals | Receptors, Cytoplasmic and Nuclear - analysis | Prefrontal Cortex - metabolism | Enzyme Activation | Chronic Disease
Journal Article