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The Journal of Immunology, ISSN 0022-1767, 03/2009, Volume 182, Issue 5, pp. 3202 - 3212
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2015, Volume 290, Issue 20, pp. 12558 - 12571
Resistance toward imatinib (IM) and other BCR/ABL tyrosine kinase inhibitors remains troublesome in the treatment of advanced stage chronic myeloid leukemia... 
TYROSINE KINASE INHIBITORS | SURVIVAL | APOPTOSIS | ACTIVATION | THERAPY | PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | BCR-ABL | FREQUENCY | MUTATIONS | EXPRESSION | Apoptosis - drug effects | Humans | Middle Aged | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Protein Kinase C beta - genetics | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Apoptosis - genetics | Male | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Sodium-Hydrogen Exchangers - metabolism | Heme Oxygenase-1 - genetics | MAP Kinase Signaling System - genetics | Cation Transport Proteins - metabolism | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Enzyme Induction - drug effects | Adult | Cation Transport Proteins - genetics | Female | NF-E2-Related Factor 2 - genetics | Sodium-Hydrogen Exchangers - genetics | Benzamides - pharmacology | Neoplasm Proteins - genetics | Heme Oxygenase-1 - biosynthesis | Oxidative Stress - genetics | Gene Expression Regulation, Leukemic | Pyrimidines - pharmacology | Imatinib Mesylate | Piperazines - pharmacology | Drug Resistance, Neoplasm - genetics | Enzyme Induction - genetics | MAP Kinase Signaling System - drug effects | NF-E2-Related Factor 2 - metabolism | K562 Cells | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Protein Kinase C beta - metabolism | Aged | Protein Kinase Inhibitors - pharmacology | Oxidative Stress - drug effects | Protein Kinase C beta - antagonists & inhibitors | Sodium-Hydrogen Exchanger 1 | Drug Resistance, Neoplasm - drug effects | heme oxygenase | H+ ion exchanger 1 | mitogen-activated protein kinase | p38 | protein kinase C (PKC) | chronic myelogenous leukemia (CML) | imatinib resistance | Na+ | drug resistance | p38 MAPK | Cell Biology
Journal Article
Blood, ISSN 0006-4971, 2009, Volume 113, Issue 25, pp. 6411 - 6418
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2015, Volume 125, Issue 2, pp. 665 - 680
Cellular lipid metabolism has been linked to immune responses; however, the precise mechanisms by which de novo fatty acid synthesis can regulate inflammatory... 
MEDICINE, RESEARCH & EXPERIMENTAL | OXIDATIVE STRESS | GLUCOSE-METABOLISM | ATP-CITRATE LYASE | UCP2 | SEPTIC SHOCK | CELL-GROWTH | CRITICALLY-ILL PATIENTS | INTENSIVE INSULIN THERAPY | CANCER | MITOCHONDRIAL UNCOUPLING PROTEINS | Fatty Acid Synthase, Type I - genetics | Fatty Acid Synthase, Type I - biosynthesis | Lipids - genetics | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Ion Channels - genetics | Caspase 1 - metabolism | Mitochondrial Proteins - genetics | Interleukin-1beta - genetics | Lipids - biosynthesis | Sepsis - pathology | Interleukin-1beta - metabolism | Mitochondrial Proteins - metabolism | Sepsis - metabolism | Macrophages - pathology | Down-Regulation - genetics | Inflammasomes - genetics | Sepsis - genetics | Mice, Knockout | Carrier Proteins - genetics | Enzyme Induction - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Sepsis - chemically induced | Ion Channels - metabolism | Caspase 1 - genetics | Sepsis - therapy | Mice | Uncoupling Protein 2 | Interleukin-18 - genetics | Interleukin-18 - metabolism | Cellular proteins | Immune response | Synthesis | Sepsis | Development and progression | Lipids | Genetic aspects | Properties | Enzymes | Cytokines | Pathogenesis | Mortality | Glucose | Metabolism | Gene expression | Fatty acids | Proteins | Studies | Metabolites | Consent | Rodents
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2013, Volume 110, Issue 27, pp. 11115 - 11120
Smoking is a major risk factor for osteoporosis and fracture, but the mechanism through which smoke causes bone loss remains unclear. Here, we show that the... 
Bone resorption | Enzymes | Cell lines | Stem cells | Bones | Osteoclasts | Cultured cells | Bone marrow cells | Cigarette smoking | Aryl hydrocarbon receptors | Osteoblast | Skeletal remodeling | Toxicology | Bone formation | toxicology | TARGET | RESORPTION | MULTIDISCIPLINARY SCIENCES | osteoblast | AHR | skeletal remodeling | CANCER | IN-VITRO | INHIBITION | METABOLISM | bone formation | CIGARETTE-SMOKE | HEALTH | OSTEOCLAST DIFFERENTIATION | Smoking - adverse effects | Cytochrome P-450 CYP1A1 - genetics | Receptors, Aryl Hydrocarbon - deficiency | Aryl Hydrocarbon Hydroxylases - biosynthesis | Humans | Aryl Hydrocarbon Hydroxylases - genetics | Male | Carcinogens - toxicity | Cytochrome P-450 CYP1A2 - genetics | Cytochrome P-450 CYP1A2 - deficiency | Bone Resorption - metabolism | Female | Cytochrome P-450 CYP1A1 - deficiency | Disease Models, Animal | Bone Resorption - etiology | Benzo(a)pyrene - toxicity | Smoke - adverse effects | Mice, Inbred C57BL | Receptors, Aryl Hydrocarbon - genetics | Smoking - genetics | Receptors, Aryl Hydrocarbon - physiology | Tobacco - toxicity | Mice, Knockout | Enzyme Induction - genetics | Animals | Aryl Hydrocarbon Hydroxylases - deficiency | Cytochrome P-450 CYP1A2 - biosynthesis | Bone Resorption - pathology | Cytochrome P-450 CYP1B1 | Polychlorinated Dibenzodioxins - toxicity | Mice | Cytochrome P-450 CYP1A1 - biosynthesis | Osteoporosis | Carcinogens | Cocarcinogens | Hydrocarbons | Physiological aspects | Health aspects | Smoking | Index Medicus | Biological Sciences
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 10/2017, Volume 118, Issue 10, pp. 3290 - 3298
ABSTRACT The aim of the present study is to investigate the effect of a natural compound crocin, one of the active components of saffron, on human multiple... 
APOPTOSIS | SHP‐1 | CROCIN | STAT3 | SHP-1 | MULTIPLE-MYELOMA | TARGET | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROLIFERATION | CANCER | CELL BIOLOGY | INFLAMMATION | ACTIVATION PATHWAY | EXPRESSION | CARCINOMA | SIGNAL TRANSDUCER | Apoptosis - drug effects | Humans | Apoptosis - genetics | G1 Phase - drug effects | Multiple Myeloma - metabolism | Neoplasm Proteins - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - genetics | Carotenoids - pharmacology | Enzyme Induction - genetics | Multiple Myeloma - drug therapy | Multiple Myeloma - pathology | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - metabolism | Enzyme Induction - drug effects | G1 Phase - genetics | HeLa Cells | Neoplasm Proteins - genetics | Multiple Myeloma - genetics | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Tyrosine | Phenols | Phosphatases | Gene expression | Vascular endothelial growth factor | Multiple myeloma | Cell proliferation | Bax protein | Bcl-2 protein | SHP-1 protein | Activation | Cyclin D1 | Kinases | Phosphatase | Nuclei | Prevention | Proteins | Angiogenesis | Signal transduction | Janus kinase 2 | Cell cycle | Janus kinase | Saffron | Inhibition | G1 phase | Translocation | Pervanadate | Stat3 protein | Src protein | CXCR4 protein | Inhibitors | Protein phosphatase | Protein-tyrosine-phosphatase | Apoptosis
Journal Article
Archives of Pharmacal Research, ISSN 0253-6269, 03/2005, Volume 28, Issue 3, pp. 249 - 268
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2013, Volume 288, Issue 47, pp. 33654 - 33666
Autophagy and apoptosis regulate cancer cell viability in response to cytotoxic stress; however, their functional relationship remains unclear.... 
INDUCED CELL-DEATH | ACTIVATION | PROTEIN | INDUCED APOPTOSIS | BH3 Mimetic | BIOCHEMISTRY & MOLECULAR BIOLOGY | TARGETING AUTOPHAGY | MIMETIC ABT-737 | Caspase | TUMOR-CELLS | Autophagy | p62 | Sequestosome 1 | Cancer Therapy | INHIBITION | DEGRADATION | Apoptosis | Bcl-2 Family Proteins | PANCREATIC-CANCER CELLS | Neoplasms - metabolism | Phagosomes - enzymology | Microtubule-Associated Proteins - genetics | Apoptosis - drug effects | Microtubule-Associated Proteins - metabolism | Sequestosome-1 Protein | Humans | Gene Expression Regulation, Neoplastic | Apoptosis - genetics | Caspase 8 - biosynthesis | Neoplasm Proteins - metabolism | Autophagy - drug effects | Gene Knockdown Techniques | Caspase 8 - genetics | Neoplasms - genetics | Enzyme Induction - drug effects | Antineoplastic Agents - pharmacology | Autophagy - genetics | Neoplasm Proteins - genetics | bcl-2-Associated X Protein - genetics | Aniline Compounds - pharmacology | Jurkat Cells | bcl-2-Associated X Protein - metabolism | Up-Regulation - genetics | Enzyme Activation - drug effects | Sulfonamides - pharmacology | TNF-Related Apoptosis-Inducing Ligand - metabolism | Up-Regulation - drug effects | Enzyme Induction - genetics | Adaptor Proteins, Signal Transducing - genetics | Adaptor Proteins, Signal Transducing - biosynthesis | Mutation | Enzyme Activation - genetics | Neoplasms - pathology | TNF-Related Apoptosis-Inducing Ligand - genetics | Phagosomes - genetics | Cell Biology
Journal Article
Journal Article