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Journal Article
The Journal of Nutritional Biochemistry, ISSN 0955-2863, 07/2016, Volume 33, pp. 54 - 62
Ursolic acid (UA), a well-known natural triterpenoid found in abundance in blueberries, cranberries and apple peels, has been reported to possess many... 
DNMT | Nrf2 | JB6 P | HDAC | Skin cancer | UA | OXIDATIVE STRESS | SKIN TUMORIGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR-SUPPRESSOR GENES | DIETARY PHYTOCHEMICALS | NUTRITION & DIETETICS | INHIBITION | PATHWAY | PROSTATE-CANCER | CANCER PREVENTION | EXPRESSION | JB6 CELLS | Antioxidants - metabolism | NF-E2-Related Factor 2 - agonists | Anticarcinogenic Agents - metabolism | Triterpenes - adverse effects | DNA (Cytosine-5-)-Methyltransferases - chemistry | Promoter Regions, Genetic - drug effects | Carcinogens - toxicity | DNA (Cytosine-5-)-Methyltransferases - metabolism | Skin Neoplasms - prevention & control | Carcinogens - antagonists & inhibitors | NF-E2-Related Factor 2 - genetics | Anticarcinogenic Agents - adverse effects | Epigenesis, Genetic - drug effects | Dietary Supplements - adverse effects | Triterpenes - metabolism | Cell Line | Cell Survival - drug effects | Epidermis - metabolism | Histone Deacetylases - genetics | Epidermis - drug effects | Histone Deacetylases - chemistry | Histone Deacetylases - metabolism | Skin Neoplasms - chemically induced | Skin Neoplasms - metabolism | DNA (Cytosine-5-)-Methyltransferases - genetics | Animals | NF-E2-Related Factor 2 - metabolism | Antioxidants - adverse effects | Mice | Cell Transformation, Neoplastic - drug effects | Enzyme Repression - drug effects | Oxidative Stress - drug effects | DNA Methylation - drug effects | Epidermis - cytology | Prevention | Epigenetic inheritance | Methyltransferases | Analysis | Skin | Methylation | Cancer | Index Medicus | skin cancer | JP6 P
Journal Article
The Journal of Nutritional Biochemistry, ISSN 0955-2863, 09/2017, Volume 47, pp. 113 - 119
Androgen receptor (AR) is a transcription factor involved in normal prostate physiology and prostate cancer (PCa) development. 3,3′-Diindolylmethane (DIM) is a... 
DIM | Epigenetic repression | Prostate cancer | DNA damage | ANDROGEN RECEPTOR | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | RETROTRANSPOSITION | INDUCTION | DISPOSITION | NUTRITION & DIETETICS | THERAPY | INDOLE-3-CARBINOL | 3,3'-DIINDOLYLMETHANE | DNA-Activated Protein Kinase - antagonists & inhibitors | Chromatin - metabolism | Enhancer of Zeste Homolog 2 Protein - antagonists & inhibitors | Prostatic Neoplasms - metabolism | Humans | Receptors, Androgen - metabolism | MRE11 Homologue Protein - antagonists & inhibitors | Male | Neoplasm Proteins - antagonists & inhibitors | Androgen Receptor Antagonists - pharmacology | Neoplasm Proteins - metabolism | Chromatin Immunoprecipitation | DNA-Activated Protein Kinase - genetics | DNA-Activated Protein Kinase - metabolism | Genomic Instability - drug effects | Epigenetic Repression - drug effects | Indoles - pharmacology | Receptors, Androgen - chemistry | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Chromatin - drug effects | MRE11 Homologue Protein - genetics | Prostatic Neoplasms - drug therapy | Prostatic Neoplasms - pathology | DNA Repair - drug effects | Enhancer of Zeste Homolog 2 Protein - metabolism | Poly (ADP-Ribose) Polymerase-1 - metabolism | Poly (ADP-Ribose) Polymerase-1 - antagonists & inhibitors | Response Elements - drug effects | Receptors, Androgen - genetics | Cell Line, Tumor | DNA Damage | Enzyme Repression - drug effects | Antineoplastic Agents, Phytogenic - pharmacology | MRE11 Homologue Protein - metabolism | Poly (ADP-Ribose) Polymerase-1 - genetics | Epigenetic inheritance | Chromatin | Genes | Genomics | DNA | Genetic aspects | Genomes | Gene expression | Cancer | Prostate-specific antigen | Genetic transcription | DNA repair | Anopheles | Index Medicus | epigenetic repression | prostate cancer
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 09/2017, Volume 140, pp. 28 - 40
Pancreatic cancer (PC) is one of the most fatal cancers worldwide. The incidence and death rates are still increasing for PC. Curcumin is the biologically... 
Cell growth | Curcumin | Pancreatic cancer | NEDD4 | Invasion | CELLS | PROTEIN | PTEN | PI3K/AKT PATHWAY | UBIQUITIN LIGASE NEDD4-1 | SIGNALING PATHWAY | GROWTH | ROS | TUMOR-SUPPRESSOR | DEGRADATION | PHARMACOLOGY & PHARMACY | Pancreatic Neoplasms - metabolism | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | Endosomal Sorting Complexes Required for Transport - genetics | Tumor Protein p73 - genetics | Ubiquitin-Protein Ligases - antagonists & inhibitors | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Reactive Oxygen Species - agonists | Tumor Protein p73 - metabolism | Pancreatic Neoplasms - drug therapy | RNA Interference | Endosomal Sorting Complexes Required for Transport - antagonists & inhibitors | Inhibitory Concentration 50 | Nedd4 Ubiquitin Protein Ligases | Neoplasm Proteins - genetics | Recombinant Proteins - metabolism | Cell Survival - drug effects | PTEN Phosphohydrolase - genetics | Neoplasm Invasiveness | Pancreatic Neoplasms - pathology | Curcumin - pharmacology | Endosomal Sorting Complexes Required for Transport - metabolism | Ubiquitin-Protein Ligases - metabolism | PTEN Phosphohydrolase - metabolism | Recombinant Proteins - chemistry | Up-Regulation - drug effects | Cell Movement - drug effects | Cell Line, Tumor | Tumor Protein p73 - agonists | Cell Proliferation - drug effects | Enzyme Repression - drug effects | Antineoplastic Agents, Phytogenic - pharmacology | Ubiquitin-Protein Ligases - genetics | PTEN Phosphohydrolase - chemistry | Ubiquitin | Medical colleges | Ligases | Analysis | Mortality | Cellular signal transduction | Apoptosis | Index Medicus
Journal Article
Endocrine Journal, ISSN 0918-8959, 2014, Volume 61, Issue 7, pp. 675 - 682
11β-hydroxysteroid dehydrogenase type 1 (11β-HSD1) is an NADPH-dependent reductase that converts cortisone to cortisol in adipose tissue. We previously... 
Insulin-like growth factor-I (IGF-I) | Hexose-6-phosphate dehydrogenase (H6PDH) | Growth hormone (GH) | 11β-hydroxysteroid dehydrogenase type 1 (11β-HSD1) | Adipocytes | Insulin-like growth factor-i (IGF-I) | CELLS | MESSENGER-RNA EXPRESSION | ENDOCRINOLOGY & METABOLISM | 11 beta-hydroxysteroid dehydrogenase type 1 (11 beta-HSD1) | MODULATION | Receptor, IGF Type 1 - metabolism | Receptor, IGF Type 1 - antagonists & inhibitors | Adipocytes, White - enzymology | Insulin-Like Growth Factor I - genetics | RNA, Messenger - metabolism | 11-beta-Hydroxysteroid Dehydrogenase Type 1 - genetics | Adipocytes, White - drug effects | Protein Processing, Post-Translational - drug effects | Phosphorylation - drug effects | Carbohydrate Dehydrogenases - genetics | Growth Hormone - metabolism | Insulin Resistance | Imidazoles - pharmacology | 3T3-L1 Cells | 11-beta-Hydroxysteroid Dehydrogenase Type 1 - antagonists & inhibitors | Receptor, IGF Type 1 - genetics | Carbohydrate Dehydrogenases - antagonists & inhibitors | Phthalazines - pharmacology | 11-beta-Hydroxysteroid Dehydrogenase Type 1 - metabolism | Insulin - metabolism | Animals | Signal Transduction - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Enzyme Repression - drug effects | Pyrazines - pharmacology | Adipocytes, White - metabolism | Insulin-Like Growth Factor I - antagonists & inhibitors | Insulin-Like Growth Factor I - metabolism | Carbohydrate Dehydrogenases - metabolism | Index Medicus
Journal Article
Biochimie, ISSN 0300-9084, 12/2015, Volume 119, pp. 192 - 203
Atherosclerosis is a major cause of coronary artery disease, which is characterized by cellular lipid accumulation. Lipoprotein lipase (LPL) is a key enzyme in... 
Oxidative stress | LPL | Betulinic acid | Atherosclerosis | PROTEIN-KINASE-C | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | INDUCTION | SUPPRESSION | BLOOD MONONUCLEAR-CELLS | NF-KAPPA-B | DERIVATIVES | MODULATION | Lipoprotein Lipase - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Triterpenes - pharmacology | Protein Transport - drug effects | RNA, Messenger - metabolism | RNA Interference | Protein Kinase C - metabolism | Lipid Droplets - metabolism | Lipoprotein Lipase - metabolism | Macrophages - immunology | Hydrogen Peroxide - toxicity | Proto-Oncogene Proteins c-fos - antagonists & inhibitors | Lipid Droplets - drug effects | Lipoprotein Lipase - genetics | Oxidants - toxicity | Proto-Oncogene Proteins c-fos - metabolism | Protein Kinase C - antagonists & inhibitors | Antioxidants - pharmacology | Hypolipidemic Agents - pharmacology | Macrophages - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Animals | MAP Kinase Signaling System - drug effects | Lipid Metabolism - drug effects | Macrophages - drug effects | Proto-Oncogene Proteins c-fos - genetics | RAW 264.7 Cells | Mice | Enzyme Repression - drug effects | Oxidative Stress - drug effects | Macrophage Activation - drug effects | Antioxidants | Enzymes | Lipoprotein lipase | Macrophages | Coronary heart disease | Yuan (China) | Physiological aspects | Index Medicus
Journal Article
Journal Article
Journal Article
Archives of Toxicology, ISSN 0340-5761, 12/2016, Volume 90, Issue 12, pp. 3061 - 3071
It was reported that 2,4-dichlorophenoxyacetic acid (2,4-D), a commonly used herbicide and a possible endocrine disruptor, can disturb spermatogenesis, but the... 
Biomedicine, general | Testosterone | Biomedicine | Environmental Health | Occupational Medicine/Industrial Medicine | Testicular toxicity | 2,4-dichlorophenoxyacetic acid | Pharmacology/Toxicology | PPARα | Cholesterol | Leydig cell | STEROIDOGENESIS | 2,4-D | PPAR alpha | ACTIVATED RECEPTOR-ALPHA | GENE | ADULT-RAT | LIVER | TOXICOLOGY | PEROXISOME PROLIFERATORS | EXPRESSION | 2,4,5-TRICHLOROPHENOXYACETIC ACID | Seminiferous Epithelium - drug effects | Hydroxymethylglutaryl-CoA Synthase - genetics | Leydig Cells - pathology | Male | Hydroxymethylglutaryl-CoA Synthase - metabolism | Leydig Cells - drug effects | Mice, 129 Strain | Cholesterol - chemistry | Dose-Response Relationship, Drug | Herbicides - administration & dosage | Testosterone - metabolism | 2,4-Dichlorophenoxyacetic Acid - administration & dosage | Hydroxymethylglutaryl CoA Reductases - metabolism | Leydig Cells - metabolism | Hydroxymethylglutaryl CoA Reductases - chemistry | Lipid Droplets - metabolism | Lipid Droplets - drug effects | Spermatogenesis - drug effects | Seminiferous Epithelium - metabolism | Lipid Droplets - pathology | Peroxisome Proliferators - toxicity | PPAR alpha - genetics | Herbicides - toxicity | Random Allocation | Infertility, Male - pathology | Seminiferous Epithelium - pathology | Endocrine Disruptors - administration & dosage | Endocrine Disruptors - toxicity | Mice, Knockout | Infertility, Male - physiopathology | Peroxisome Proliferators - administration & dosage | Animals | Infertility, Male - metabolism | Seminiferous Epithelium - physiopathology | Enzyme Repression - drug effects | PPAR alpha - metabolism | Hydroxymethylglutaryl-CoA Synthase - antagonists & inhibitors | Infertility, Male - chemically induced | 2,4-Dichlorophenoxyacetic Acid - toxicity | Hydroxymethylglutaryl CoA Reductases - genetics | Index Medicus
Journal Article
The Journal of Nutritional Biochemistry, ISSN 0955-2863, 07/2016, Volume 33, pp. 45 - 53
Our previous study demonstrated that quercetin-metabolite-enriched plasma (QP) but not quercetin itself upregulates peroxisome proliferator-activated receptor... 
Quercetin-3′-sulfate | nm23-H1 | Lung cancer cells | Peroxisome proliferator-activated receptor gamma | Metastasis | Quercetin-3-glucuronide | Nm23-H1 | Quercetin-3'-sulfate | PLASMA METABOLITES | DIETARY FLAVONOIDS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLUCURONIDES | METALLOPROTEINASE-2 | METASTASIS SUPPRESSOR | IN-VITRO | INVASION | NUTRITION & DIETETICS | Quercetin-3 '-sulfate | COLORECTAL-CANCER | TUMOR-METASTASIS | ACTIVATED-RECEPTOR-GAMMA | Lung Neoplasms - chemically induced | Humans | Lung Neoplasms - metabolism | Neoplasm Invasiveness - prevention & control | Lung Neoplasms - pathology | Male | Neoplasm Proteins - antagonists & inhibitors | Anticarcinogenic Agents - metabolism | Neoplasm Proteins - metabolism | PPAR gamma - metabolism | Glucuronides - blood | G2 Phase - drug effects | Glucuronides - metabolism | Chromans - pharmacology | RNA Interference | Anticarcinogenic Agents - administration & dosage | Neoplasm Invasiveness - pathology | Quercetin - administration & dosage | Glucuronides - administration & dosage | Matrix Metalloproteinase 2 - chemistry | Neoplasm Proteins - genetics | Thiazolidinediones - pharmacology | Anticarcinogenic Agents - pharmacology | PPAR gamma - genetics | A549 Cells | Quercetin - analogs & derivatives | Matrix Metalloproteinase 2 - metabolism | Up-Regulation - drug effects | Cell Movement - drug effects | Matrix Metalloproteinase 2 - genetics | Animals | PPAR gamma - antagonists & inhibitors | Gerbillinae | Lung Neoplasms - prevention & control | Neoplasm Proteins - agonists | Quercetin - blood | Anilides - pharmacology | PPAR gamma - agonists | Ligands | Enzyme Repression - drug effects | Dietary Supplements | Quercetin - metabolism | Index Medicus
Journal Article
Journal Article