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The Journal of Immunology, ISSN 0022-1767, 11/2009, Volume 183, Issue 10, pp. 6469 - 6477
Alternatively activated macrophages (AAM) play a crucial role in type 2 immunity. Mice deficient in ST2, a receptor for the latest member of the IL-1 family,... 
IL-1-LIKE CYTOKINE IL-33 | IMMUNE-RESPONSE | ST2 RECEPTOR | EFFECTOR FUNCTION | MAST-CELLS | IN-VIVO | INDUCED ARTHRITIS | ALVEOLAR MACROPHAGES | INTERLEUKIN-1 RECEPTOR | IMMUNOLOGY | T-CELLS | Interleukin-1 Receptor-Like 1 Protein | Asthma - metabolism | Epithelial Cells - metabolism | Ovalbumin - immunology | Humans | Middle Aged | Chemokine CCL24 - metabolism | Interleukin-13 - immunology | Male | Interleukins - metabolism | Receptors, Interleukin - metabolism | Signal Transduction - immunology | Inflammation - metabolism | Interleukins - immunology | Asthma - immunology | Chemokine CCL17 - immunology | Adult | Female | Interleukin-4 - genetics | Macrophages, Alveolar - immunology | Recombinant Proteins - metabolism | Cell Polarity - immunology | Lung - pathology | Receptors, Interleukin - immunology | Interleukin-33 | Macrophage Activation - immunology | Receptors, Interleukin-1 - immunology | Interleukin-4 - metabolism | Receptors, Interleukin-4 - metabolism | Inflammation - immunology | Receptors, Interleukin-4 - immunology | Interleukin-13 - metabolism | Chemokine CCL24 - immunology | Receptors, Interleukin - genetics | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Interleukin-4 - immunology | Animals | Eosinophilia - immunology | Recombinant Proteins - immunology | Epithelial Cells - immunology | Receptors, Interleukin-4 - genetics | Mice | Mice, Inbred BALB C | Macrophages, Alveolar - metabolism | Chemokine CCL17 - metabolism | Lung - immunology | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 06/2002, Volume 195, Issue 11, pp. 1433 - 1444
Interleukin (IL)-5 and IL-13 are thought to play key roles in the pathogenesis of asthma. Although both cytokines use eotaxin to regulate eosinophilia, IL-13... 
Allergy | Inflammation | Cytokines | Lung | Eosinophils | MEDICINE, RESEARCH & EXPERIMENTAL | TISSUE EOSINOPHILIA | BRONCHIAL BIOPSIES | MESSENGER-RNA EXPRESSION | BONE-MARROW | allergy | eosinophils | IMMUNOLOGY | BRONCHOALVEOLAR LAVAGE | lung | LUNG DAMAGE | MURINE MODEL | inflammation | ALLERGEN INHALATION CHALLENGE | cytokines | GUINEA-PIG MODEL | ATOPIC ASTHMA | Eosinophilia - metabolism | Asthma - metabolism | Eosinophils - metabolism | Eosinophils - transplantation | Bronchial Hyperreactivity - immunology | Chemokines, CC - blood | Humans | Th2 Cells - immunology | Adoptive Transfer | Interleukin-5 - genetics | RNA, Messenger - metabolism | CD4-Positive T-Lymphocytes - immunology | Eosinophilia - pathology | Interleukin-13 - biosynthesis | Gene Deletion | Asthma - immunology | Bronchial Hyperreactivity - metabolism | Disease Models, Animal | Signal Transduction | Interleukin-5 - metabolism | RNA, Messenger - genetics | CD4-Positive T-Lymphocytes - metabolism | Chemokine CCL11 | Interleukin-13 - metabolism | Bronchial Hyperreactivity - complications | Th2 Cells - metabolism | Interleukin-5 - blood | Sputum - metabolism | Bronchial Hyperreactivity - pathology | Animals | Eosinophilia - immunology | Asthma - complications | Chemokines, CC - genetics | Eosinophilia - complications | Mice | Mice, Inbred BALB C | Interleukin-18 - metabolism | Chemokines, CC - metabolism | Index Medicus
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2011, Volume 129, Issue 1, pp. 104 - 111.e9
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2014, Volume 135, Issue 2, pp. 425 - 440
Background We reported that DNA-dependent protein kinase (DNA-PK) is critical for the expression of nuclear factor κB–dependent genes in TNF-α–treated... 
Allergy and Immunology | T-cell receptor | DNA-dependent protein kinase | adhesion molecules | severe combined immunodeficiency disease | eosinophilia | asthma | GATA-3 | TH2 cytokines | 2 cytokines | POLY(ADP-RIBOSE) POLYMERASE | V(D)J RECOMBINATION | T(H)2 cytokines | ALLERGIC AIRWAY INFLAMMATION | IMMUNOLOGY | ALLERGY | GROWTH | NF-KAPPA-B | EXPRESSION | DNA-Activated Protein Kinase - antagonists & inhibitors | Spleen - immunology | Bronchial Hyperreactivity - immunology | Ovalbumin - immunology | Humans | Male | Th2 Cells - immunology | Eosinophils - immunology | CD4-Positive T-Lymphocytes - immunology | Allergens - immunology | Respiratory Mucosa - immunology | Asthma - immunology | Plasma Cells - metabolism | GATA3 Transcription Factor - metabolism | Disease Models, Animal | Gene Expression | Receptors, Antigen, T-Cell - metabolism | Lymphocyte Activation | Cell Adhesion | Genetic Heterogeneity | Mice, Knockout | Bronchial Hyperreactivity - pathology | Phenotype | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Asthma - pathology | GATA3 Transcription Factor - genetics | T-Lymphocyte Subsets - immunology | Tumor Necrosis Factor-alpha - metabolism | Asthma - metabolism | Eosinophils - metabolism | Epithelial Cells - metabolism | Adoptive Transfer | Respiratory Mucosa - pathology | Immunoglobulin E - immunology | DNA-Activated Protein Kinase - genetics | DNA-Activated Protein Kinase - metabolism | Ovalbumin - adverse effects | Bronchial Hyperreactivity - metabolism | Vascular Cell Adhesion Molecule-1 - genetics | Cytokines - metabolism | Organ Size | Th2 Cells - metabolism | Animals | Spleen - anatomy & histology | T-Lymphocyte Subsets - metabolism | Severe Combined Immunodeficiency | Respiratory Mucosa - metabolism | Plasma Cells - immunology | Pyroglyphidae - immunology | Kinases | Laboratories | Rodents | Asthma | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
International Immunopharmacology, ISSN 1567-5769, 2011, Volume 11, Issue 8, pp. 1038 - 1044
The role of tumor necrosis factor-α (TNF-α) in contributing to allergen induced airway remodeling in asthma is unknown. In this study we have utilized a mouse... 
Smooth muscle | Eosinophil | Fibronectin | CYTOKINES | MAST-CELLS | ANTIBODY | IMMUNOLOGY | REFRACTORY ASTHMA | FACTOR-ALPHA | ADHESION | TUMOR-NECROSIS-FACTOR | INHIBITION | INFLAMMATION | DOUBLE-BLIND | PHARMACOLOGY & PHARMACY | Tumor Necrosis Factor-alpha - metabolism | Asthma - metabolism | Eosinophils - metabolism | Transforming Growth Factor beta1 - antagonists & inhibitors | Pulmonary Eosinophilia - chemically induced | Receptors, Tumor Necrosis Factor, Type I - immunology | Tumor Necrosis Factor Decoy Receptors - immunology | Transforming Growth Factor beta1 - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Receptors, Tumor Necrosis Factor, Type II - immunology | Muscle, Smooth - immunology | Immunoglobulin E - blood | Muscle, Smooth - drug effects | Receptors, Tumor Necrosis Factor, Type I - deficiency | Receptors, Tumor Necrosis Factor, Type II - metabolism | Bronchi - metabolism | Mucus - metabolism | Pulmonary Fibrosis - metabolism | Bronchi - pathology | Extracellular Matrix Proteins - metabolism | Pulmonary Eosinophilia - metabolism | Tumor Necrosis Factor Decoy Receptors - deficiency | Interleukin-5 - metabolism | Mice, Inbred C57BL | Pulmonary Fibrosis - immunology | Pulmonary Fibrosis - pathology | Muscle, Smooth - metabolism | Ovalbumin - pharmacology | Pulmonary Eosinophilia - pathology | Fibronectins - metabolism | Tumor Necrosis Factor Decoy Receptors - metabolism | Mice, Knockout | Receptors, Tumor Necrosis Factor, Type II - deficiency | Collagen - metabolism | Animals | Allergens - pharmacology | Mice | Airway Remodeling - drug effects | Pulmonary Fibrosis - drug therapy | Asthma - pathology | Muscle, Smooth - pathology | Fibronectins | Allergens | Transforming growth factors | Tumor necrosis factor | Analysis | Asthma | Index Medicus | fibronectin | smooth muscle | eosinophil
Journal Article
Journal Article
PLoS Pathogens, ISSN 1553-7366, 09/2013, Volume 9, Issue 9, pp. e1003615 - e1003615
Respiratory virus infections, such as influenza, typically induce a robust type I (pro-inflammatory cytokine) immune response, however, the production of type... 
LUNG | EOSINOPHILS | MICROBIOLOGY | T(H)2-TYPE RESPONSES | INTERLEUKIN-5 | CD8(+) T-CELLS | ADAPTIVE IMMUNITY | IL-1-LIKE CYTOKINE IL-33 | VIROLOGY | IN-VIVO | TYPE-2 IMMUNITY | RESPIRATORY SYNCYTIAL VIRUS | PARASITOLOGY | Influenza A Virus, H1N1 Subtype - immunology | Up-Regulation | Eosinophils - metabolism | Macrophages, Alveolar - pathology | Killer Cells, Natural - pathology | Eosinophils - immunology | Interleukins - metabolism | Protein Subunits - metabolism | Receptors, Interleukin - metabolism | Lung - virology | Killer Cells, Natural - immunology | Eosinophils - pathology | Lung - metabolism | Respiratory Tract Infections - virology | Macrophages, Alveolar - immunology | Respiratory Tract Infections - pathology | Macrophages, Alveolar - virology | Eosinophilia - etiology | Lung - pathology | Respiratory Tract Infections - metabolism | Specific Pathogen-Free Organisms | Interleukin-33 | Interleukin-5 - metabolism | Killer Cells, Natural - virology | Orthomyxoviridae Infections - metabolism | Eosinophils - virology | Mice, Inbred C57BL | Orthomyxoviridae Infections - pathology | Cells, Cultured | Immunity, Innate | Mice, Knockout | Animals | Respiratory Tract Infections - immunology | Mice, Inbred BALB C | Killer Cells, Natural - metabolism | Macrophages, Alveolar - metabolism | Orthomyxoviridae Infections - virology | Lung - immunology | Orthomyxoviridae Infections - immunology | Killer cells | Interleukins | Influenza | Physiological aspects | Host-parasite relationships | Research | Health aspects | Index Medicus | T cell receptors | Immunology | Lungs | Rodents | Immune system
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 09/2011, Volume 184, Issue 6, pp. 662 - 671
Journal Article
Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6026, pp. 243 - 247
Journal Article