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Development (Cambridge), ISSN 0950-1991, 11/2013, Volume 140, Issue 22, pp. 4510 - 4521
Muller glia function as retinal stem cells in adult zebrafish. In response to loss of retinal neurons, Muller glia partially dedifferentiate, re-express... 
Müller glia | N-cadherin | Retinal regeneration | Alcama | Muller glia | STEM-CELLS | DEDIFFERENTIATION | PROLIFERATION | DEVELOPMENTAL BIOLOGY | GOLDFISH RETINA | ADULT ZEBRAFISH | VERTEBRATE RETINA | ROD PHOTORECEPTORS | SIGNALING PATHWAY | NEUROGENESIS | LINEAGE | Cadherins - metabolism | Multipotent Stem Cells - metabolism | Photoreceptor Cells, Vertebrate - drug effects | Ependymoglial Cells - metabolism | Neural Stem Cells - cytology | Neuroepithelial Cells - cytology | Retinal Ganglion Cells - metabolism | Retinal Neurons - cytology | Retinal Ganglion Cells - cytology | Neurogenesis - drug effects | Retinal Neurons - drug effects | Asymmetric Cell Division - drug effects | Ouabain - pharmacology | Biomarkers - metabolism | Cell Dedifferentiation - drug effects | Zebrafish Proteins - metabolism | Neural Stem Cells - drug effects | Photoreceptor Cells, Vertebrate - cytology | Cell Adhesion - drug effects | Ependymoglial Cells - drug effects | Regeneration - drug effects | Animals | Retinal Neurons - metabolism | Models, Biological | Multipotent Stem Cells - cytology | Zebrafish - metabolism | Ependymoglial Cells - cytology | Heterozygote | Neuroepithelial Cells - metabolism | Photoreceptor Cells, Vertebrate - metabolism | Cell Cycle - drug effects | Neural Stem Cells - metabolism | Retinal Ganglion Cells - drug effects | Index Medicus | Stem Cells and Regeneration
Journal Article
Nature Communications, ISSN 2041-1723, 2013, Volume 4, Issue 1, pp. 2049 - 2049
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 06/2017, Volume 141, Issue 5, pp. 750 - 765
The expression of the mRNAs of Nrf2‐related genes was increased in 661W cells after exposure to RS9. Nrf2 was detected in the nucleus of the 661W cells exposed... 
AMD | light‐induced retinal degeneration | Nrf2 | retina | photoreceptor | light-induced retinal degeneration | HO-1 | RETINAL DEGENERATION | OXIDATIVE STRESS | HUMAN-DISEASE | PROTECTION | BRAIN-INJURY | BIOCHEMISTRY & MOLECULAR BIOLOGY | MACULAR DEGENERATION | NEUROSCIENCES | BLUE-LIGHT | NEUROPROTECTION | IN-VITRO | ISCHEMIA-REPERFUSION | RNA, Small Interfering - genetics | Cell Death - radiation effects | Cell Nucleolus - radiation effects | Heme Oxygenase-1 - metabolism | NF-E2 Transcription Factor - genetics | Triterpenes - pharmacology | Cytosol - drug effects | Ependymoglial Cells - radiation effects | Male | Retinal Degeneration - etiology | NF-E2 Transcription Factor - metabolism | Retinal Degeneration - prevention & control | Heme Oxygenase-1 - genetics | Triterpenes - chemistry | Retina - cytology | Time Factors | Cell Nucleolus - drug effects | Membrane Proteins - metabolism | Cell Death - drug effects | Protein Biosynthesis - radiation effects | Membrane Proteins - genetics | Light - adverse effects | RNA, Small Interfering - pharmacology | Ependymoglial Cells - drug effects | Photoreceptor Cells - radiation effects | Gene Expression Regulation - drug effects | Animals | Photoreceptor Cells - drug effects | Ependymoglial Cells - cytology | Gene Expression Regulation - radiation effects | Protein Biosynthesis - drug effects | Mice | Cytosol - radiation effects | In Vitro Techniques | Cell Line, Transformed | Neurochemistry | Chlorophyll | Oxidative stress | Enzymes | Heme oxygenase (decyclizing) | Retina | Oxygen consumption | Exposure | Nuclei | Environmental risk | Risk factors | Macular degeneration | Oxygenase | Cell death | Heme | Photoreceptors | Eye diseases | Degeneration | In vivo methods and tests | Nuclei (cytology) | Damage | In vitro methods and tests | Age | Apoptosis | Index Medicus
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2016, Volume 22, Issue 7, pp. 800 - 806
Type 2 diabetes (T2D) is among the most common and costly disorders worldwide(1). The goal of current medical management for T2D is to transiently ameliorate... 
MEDICINE, RESEARCH & EXPERIMENTAL | HOMEOSTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MODEL | SUPPRESSION | ASTROCYTES | BALANCE | CELL BIOLOGY | INSULIN | TANYCYTES | METABOLISM | INTRAVENOUS GLUCOSE-TOLERANCE | IN-VIVO | Body Composition | Ependymoglial Cells - metabolism | Injections, Intraventricular | Male | Muscle, Skeletal - metabolism | Diabetes Mellitus, Type 2 - metabolism | Neoplasm Proteins - metabolism | Deoxyglucose | Neoplasm Proteins - drug effects | Brain - metabolism | Adipose Tissue - metabolism | Receptor, Insulin - genetics | Diet, High-Fat | Muscle, Skeletal - drug effects | Myocardium - metabolism | Hypothalamus - drug effects | Diabetes Mellitus, Experimental - metabolism | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Glucose Tolerance Test | Heat-Shock Proteins - drug effects | Heat-Shock Proteins - metabolism | Liver - metabolism | Proto-Oncogene Proteins c-fos - metabolism | Rats | Ependymoglial Cells - drug effects | Receptor, Insulin - antagonists & inhibitors | Remission Induction | Blotting, Western | Proto-Oncogene Proteins c-fos - drug effects | Mice, Knockout | Blood Glucose - drug effects | Brain - drug effects | Hyperglycemia - metabolism | Rats, Zucker | Animals | Hypothalamus - metabolism | Hypothalamus - cytology | Fibroblast Growth Factor 1 - pharmacology | Heart - drug effects | Mice, Obese | Mice | Blood Glucose - metabolism | Adipose Tissue - drug effects | Forkhead Box Protein O1 - genetics | Carbon Radioisotopes | Type 2 diabetes | Molecular targeted therapy | Care and treatment | Innovations | Development and progression | Genetic aspects | Fibroblast growth factors | Health aspects | Fibroblasts | Diabetes | Drug therapy | Rodents | Index Medicus | brain | diabetes | fibroblast growth factor
Journal Article
Journal Article
Nature, ISSN 0028-0836, 12/2017, Volume 552, Issue 7684, pp. 248 - 252
Diabetic retinopathy is an important cause of blindness in adults(1,2), and is characterized by progressive loss of vascular cells and slow dissolution of... 
BLOOD-RETINAL BARRIER | ACID | ANGIOGENESIS | CHOLESTEROL | STABILITY | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | DEPENDENT ENDOCYTOSIS | MEMBRANES | VE-CADHERIN | ASSOCIATION | Retina - drug effects | Retina - metabolism | Cadherins - metabolism | Pericytes - drug effects | Humans | Capillary Permeability - drug effects | Male | Diabetic Retinopathy - pathology | Antigens, CD - metabolism | Vitreous Body - metabolism | Pericytes - pathology | Retina - enzymology | Intercellular Junctions - pathology | Female | Cell Membrane - drug effects | Disease Models, Animal | Ependymoglial Cells | Cell Survival - drug effects | Endothelial Cells - metabolism | Mice, Inbred C57BL | Solubility | Pancreatic Elastase - metabolism | Fatty Acids, Unsaturated - metabolism | Diabetic Retinopathy - metabolism | Disease Progression | Cell Movement - drug effects | Diabetic Retinopathy - prevention & control | Animals | Carrier Proteins - metabolism | Diabetic Retinopathy - enzymology | Intercellular Junctions - drug effects | Presenilin-1 - metabolism | Mice | Endothelial Cells - pathology | Epoxide Hydrolases - antagonists & inhibitors | Retina - pathology | Docosahexaenoic Acids - metabolism | Endothelial Cells - drug effects | Prevention | Physiological aspects | Enzymes | Diabetic retinopathy | Hydrolases | Epoxy compounds | Retinopathy | Pathogenesis | Cell junctions | Retina | Glial cells | Cell interactions | Kinases | Docosahexaenoic acid | Epoxide hydrolase | Vascularization | Proteins | Signal transduction | Hyperglycemia | N-Cadherin | Rodents | Inhibition | Growth factors | Edema | Abnormalities | Diabetes mellitus | Permeability | Cadherin | Cholesterol | Endothelial cells | Acids | Presenilin 1 | Nitric oxide | Blindness | Vitreous humour | Adults | Infiltration | Diabetes | Staphylococcal enterotoxin H | Index Medicus
Journal Article
Development (Cambridge), ISSN 0950-1991, 06/2016, Volume 143, Issue 11, pp. 1859 - 1873
We investigate the roles of mTor signaling in the formation of Muller glia-derived progenitor cells (MGPCs) in the chick retina. During embryonic development,... 
Regeneration | Retina | Progenitor | Müller glia | Mtor | Muller glia | STEM-CELLS | DEVELOPMENTAL BIOLOGY | SUFFICIENT | PHOTORECEPTOR REGENERATION | CANCER PATHOGENESIS | INSULIN | EXCITOTOXICITY | ADULT ZEBRAFISH RETINA | mTor | NEURONS | NEURAL REGENERATION | RECEPTORS | Insulin-Like Growth Factor I - pharmacology | Retina - metabolism | TOR Serine-Threonine Kinases - metabolism | Ependymoglial Cells - metabolism | Fibroblast Growth Factor 2 - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Stem Cells - cytology | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Stem Cells - metabolism | N-Methylaspartate - pharmacology | Neuroglia - drug effects | PTEN Phosphohydrolase - antagonists & inhibitors | Neuroglia - cytology | Cell Nucleus - metabolism | Insulin-Like Growth Factor II - pharmacology | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Insulin - pharmacology | PAX6 Transcription Factor - metabolism | PTEN Phosphohydrolase - metabolism | Ependymoglial Cells - drug effects | Sirolimus - pharmacology | Cell Movement - drug effects | Animals | MAP Kinase Signaling System - drug effects | Signal Transduction - drug effects | Models, Biological | Chickens | Ependymoglial Cells - cytology | Stem Cells - drug effects | Neuroglia - metabolism | Cell Proliferation - drug effects | Cell Nucleus - drug effects | Retina - pathology | Index Medicus | 102 | Stem Cells and Regeneration
Journal Article