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Journal of Neurochemistry, ISSN 0022-3042, 06/2017, Volume 141, Issue 5, pp. 750 - 765
The expression of the mRNAs of Nrf2‐related genes was increased in 661W cells after exposure to RS9. Nrf2 was detected in the nucleus of the 661W cells exposed... 
AMD | light‐induced retinal degeneration | Nrf2 | retina | photoreceptor | light-induced retinal degeneration | HO-1 | RETINAL DEGENERATION | OXIDATIVE STRESS | HUMAN-DISEASE | PROTECTION | BRAIN-INJURY | BIOCHEMISTRY & MOLECULAR BIOLOGY | MACULAR DEGENERATION | NEUROSCIENCES | BLUE-LIGHT | NEUROPROTECTION | IN-VITRO | ISCHEMIA-REPERFUSION | RNA, Small Interfering - genetics | Cell Death - radiation effects | Cell Nucleolus - radiation effects | Heme Oxygenase-1 - metabolism | NF-E2 Transcription Factor - genetics | Triterpenes - pharmacology | Cytosol - drug effects | Ependymoglial Cells - radiation effects | Male | Retinal Degeneration - etiology | NF-E2 Transcription Factor - metabolism | Retinal Degeneration - prevention & control | Heme Oxygenase-1 - genetics | Triterpenes - chemistry | Retina - cytology | Time Factors | Cell Nucleolus - drug effects | Membrane Proteins - metabolism | Cell Death - drug effects | Protein Biosynthesis - radiation effects | Membrane Proteins - genetics | Light - adverse effects | RNA, Small Interfering - pharmacology | Ependymoglial Cells - drug effects | Photoreceptor Cells - radiation effects | Gene Expression Regulation - drug effects | Animals | Photoreceptor Cells - drug effects | Ependymoglial Cells - cytology | Gene Expression Regulation - radiation effects | Protein Biosynthesis - drug effects | Mice | Cytosol - radiation effects | In Vitro Techniques | Cell Line, Transformed | Neurochemistry | Chlorophyll | Oxidative stress | Enzymes | Heme oxygenase (decyclizing) | Retina | Oxygen consumption | Exposure | Nuclei | Environmental risk | Risk factors | Macular degeneration | Oxygenase | Cell death | Heme | Photoreceptors | Eye diseases | Degeneration | In vivo methods and tests | Nuclei (cytology) | Damage | In vitro methods and tests | Age | Apoptosis | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2014, Volume 111, Issue 43, pp. 15579 - 15584
Journal Article
Journal Article
Journal Article
Cell Death and Disease, ISSN 2041-4889, 09/2013, Volume 4, Issue 9, pp. e785 - e785
In many blinding diseases of the retina, loss of function and thus severe visual impairment results from apoptotic cell death of damaged photoreceptors. In an... 
Leukemia inhibitory factor (LIF) | Neuroprotection | Muller glia cell | p38 MAPK | TNF | Photoreceptor | leukemia inhibitory factor (LIF) | ACTIVATED PROTEIN-KINASE | photoreceptor | LEUKEMIA INHIBITORY FACTOR | CELL BIOLOGY | RETINAL GANGLION-CELLS | TUMOR-NECROSIS-FACTOR | neuroprotection | GENE-EXPRESSION | HYDROGEN-PEROXIDE | TNF-ALPHA | NF-KAPPA-B | LIGHT-INDUCED DEGENERATION | UP-REGULATION | Cell Death - radiation effects | Intravitreal Injections | Photoreceptor Cells, Vertebrate - drug effects | Up-Regulation - radiation effects | Ependymoglial Cells - radiation effects | Leukemia Inhibitory Factor - genetics | Down-Regulation - radiation effects | Leukemia Inhibitory Factor - metabolism | Photoreceptor Cells, Vertebrate - radiation effects | Light | Cytoprotection - drug effects | p38 Mitogen-Activated Protein Kinases - metabolism | Cell Death - drug effects | Ependymoglial Cells - pathology | Disease Models, Animal | MAP Kinase Signaling System - radiation effects | Ependymoglial Cells - enzymology | Mice, Inbred C57BL | Rats | Down-Regulation - drug effects | Enzyme Activation - drug effects | Ependymoglial Cells - drug effects | Enzyme Activation - radiation effects | Photoreceptor Cells, Vertebrate - enzymology | Up-Regulation - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Animals | MAP Kinase Signaling System - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Tumor Necrosis Factor-alpha - administration & dosage | Mice | Retinal Degeneration - pathology | Cytoprotection - radiation effects | Photoreceptor Cells, Vertebrate - pathology | Retinal Degeneration - enzymology | Index Medicus | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, pp. e80483 - e80483
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, pp. e0166348 - e0166348
Complement dysregulation plays a key role in the pathogenesis of age-related macular degeneration (AMD), but the specific mechanisms are incompletely... 
COMPLEMENT REGULATORY PROTEINS | FACTOR-H DEFICIENCY | CHELATOR DEFERIPRONE PROTECTS | OXIDATIVE STRESS | PIGMENT EPITHELIAL-CELLS | MULTIDISCIPLINARY SCIENCES | DECAY-ACCELERATING FACTOR | MACULAR DEGENERATION | FUNCTIONAL-CHARACTERIZATION | PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA | CHOROIDAL NEOVASCULARIZATION | Retinaldehyde - analysis | Retina - metabolism | Up-Regulation - radiation effects | Ependymoglial Cells - metabolism | Male | Nerve Growth Factors - metabolism | RNA, Messenger - metabolism | Rhodopsin - metabolism | Retinal Degeneration - metabolism | Phagocytosis - radiation effects | Heat-Shock Proteins - genetics | Neuroglia - cytology | Photoreceptor Cells, Vertebrate - radiation effects | Light | Cytokines - genetics | Disease Models, Animal | Electroretinography | Cytokines - metabolism | Eye Enucleation | Heat-Shock Proteins - metabolism | Mice, Inbred C57BL | CD59 Antigens - genetics | Mice, Knockout | Endoplasmic Reticulum Stress - radiation effects | Neuroglia - radiation effects | Retina - diagnostic imaging | Animals | Rhodopsin - genetics | Nerve Growth Factors - genetics | Neuroglia - metabolism | Mice | Mice, Inbred BALB C | CD59 Antigens - metabolism | Photoreceptor Cells, Vertebrate - metabolism | Retinal Degeneration - pathology | Retinal Degeneration - veterinary | Microscopy, Fluorescence | Animal experimentation | Rhodopsin | RNA | Research | Gene expression | Macular degeneration | Prevention | Analysis | Physiological aspects | Photoreceptors | Diagnosis | Comparative analysis | Health aspects | Oxidative stress | Model testing | Complement inhibitors | Laboratories | Pathogenesis | Retina | Complement | Proteins | Cell activation | Rodents | Degeneration | Damage | Growth factors | Age | Euthanasia | Damage assessment | Genetic disorders | Glial fibrillary acidic protein | Neurotrophic factors | Electroretinograms | Medicine | Retinal degeneration | Endoplasmic reticulum | Preconditioning | Structure-function relationships | Index Medicus
Journal Article
Journal Article