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index medicus (225) 225
humans (177) 177
ephrin-a1 - metabolism (129) 129
animals (114) 114
receptor, epha2 - metabolism (83) 83
ligands (78) 78
mice (74) 74
female (70) 70
cell biology (67) 67
ephrin-a1 (67) 67
ephrin-a1 - genetics (61) 61
male (59) 59
cell line, tumor (57) 57
expression (56) 56
phosphorylation (55) 55
oncology (53) 53
biochemistry & molecular biology (51) 51
receptor tyrosine kinase (51) 51
activation (50) 50
cancer (48) 48
signal transduction (45) 45
epha2 (43) 43
proteins (42) 42
receptor, epha2 - genetics (41) 41
tyrosine kinase (38) 38
immunohistochemistry (35) 35
angiogenesis (32) 32
gene expression (32) 32
cells, cultured (31) 31
cells (29) 29
ephrin (29) 29
kinases (29) 29
research (29) 29
ephrin-a1 - pharmacology (28) 28
family (28) 28
blotting, western (27) 27
ligand (27) 27
physiological aspects (27) 27
cell line (26) 26
receptors (26) 26
eph receptors (25) 25
gene (25) 25
metastasis (25) 25
middle aged (25) 25
protein (25) 25
article (24) 24
ephrins (24) 24
growth-factor (24) 24
overexpression (24) 24
proteins - metabolism (24) 24
rats (24) 24
eph (23) 23
receptor protein-tyrosine kinases - metabolism (23) 23
receptors, eph family - metabolism (23) 23
cell adhesion (22) 22
ephrin-a1 - chemistry (22) 22
molecular sequence data (22) 22
prognosis (22) 22
reverse transcriptase polymerase chain reaction (22) 22
tyrosine (22) 22
analysis (21) 21
protein binding (21) 21
adhesion (20) 20
cell movement (20) 20
receptor (20) 20
rna, messenger - metabolism (20) 20
signal transduction - physiology (20) 20
up-regulation (20) 20
adult (19) 19
amino acid sequence (19) 19
in-vivo (19) 19
membrane proteins - metabolism (19) 19
aged (18) 18
multidisciplinary sciences (18) 18
cell proliferation (17) 17
ephrin-a1 - biosynthesis (17) 17
protein-tyrosine kinase (17) 17
rna, messenger - genetics (17) 17
tumor cells, cultured (17) 17
breast-cancer (16) 16
ephrin-a1 - physiology (16) 16
gene expression regulation, neoplastic (16) 16
genetics & heredity (16) 16
research article (16) 16
abridged index medicus (15) 15
ephrina1 (15) 15
proteins - genetics (15) 15
sense organs (15) 15
biological factors (14) 14
cell adhesion & migration (14) 14
developmental biology (14) 14
epha2 protein (14) 14
epha2 receptor (14) 14
ephrin-a2 - metabolism (14) 14
genetic aspects (14) 14
mice, inbred c57bl (14) 14
receptor, epha2 (14) 14
transfection (14) 14
tumorigenesis (14) 14
base sequence (13) 13
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International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 2007, Volume 69, Issue 1, pp. 167 - 175
Purpose To investigate the expression pattern of hypoxia-induced proteins identified as being involved in malignant progression of head-and-neck squamous cell... 
Radiology | Hematology, Oncology and Palliative Medicine | Head-and-neck cancer | Hypoxia | Prognosis | Marker panel | CANCER PATIENTS | BREAST-CANCER | ENDOGENOUS MARKERS | TUMOR HYPOXIA | EPH RECEPTORS | ANGIOGENESIS | ONCOLOGY | OXYGENATION | GROWTH | GENE-EXPRESSION | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | RADIATION-THERAPY | Cell Hypoxia - physiology | Ephrin-A1 - metabolism | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Galectin 1 - metabolism | Male | Osteopontin - metabolism | Neoplasm Proteins - metabolism | Head and Neck Neoplasms - metabolism | Oxygen - metabolism | Carbonic Anhydrases - metabolism | Carcinoma, Squamous Cell - mortality | I-kappa B Kinase - metabolism | Antigens, Neoplasm - metabolism | Biomarkers, Tumor - metabolism | Female | Membrane Proteins - metabolism | Carbonic Anhydrase IX | Proto-Oncogene Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Disease Progression | Head and Neck Neoplasms - pathology | Tetrahydrofolate Dehydrogenase - metabolism | Analysis of Variance | Partial Pressure | Protein-Lysine 6-Oxidase - metabolism | Cell Line, Tumor | Head and Neck Neoplasms - mortality | Proteins | Development and progression | Squamous cell carcinoma | Index Medicus | HEMOGLOBIN | HEAD | CARBONIC ANHYDRASE | OXYGEN | GROWTH FACTORS | MULTIVARIATE ANALYSIS | THERAPY | OXIDASES | ANOXIA | CARCINOMAS | RADIOLOGY AND NUCLEAR MEDICINE | PARTIAL PRESSURE | NECK | CONNECTIVE TISSUE
Journal Article
Nature Neuroscience, ISSN 1097-6256, 02/2010, Volume 13, Issue 2, pp. 163 - 172
Journal Article
Science, ISSN 0036-8075, 3/2010, Volume 327, Issue 5971, pp. 1380 - 1385
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 05/2017, Volume 486, Issue 3, pp. 693 - 699
The Eph family of receptor tyrosine kinases serves as key modulators of various cellular functions, including inflammation, hypertrophy and fibrosis. Recent... 
Adeno-associated virus | Nephropathy | Diabetes | EphA1 | Fibrosis | CELLS | RHO KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | INJURY | INHIBITION | BIOPHYSICS | GENE | SECRETION | Dependovirus - genetics | Ephrin-A1 - metabolism | Receptor, EphA1 - metabolism | Streptozocin | Diabetes Mellitus, Experimental - genetics | Male | Genetic Vectors - therapeutic use | Diabetes Mellitus, Experimental - therapy | Diabetic Nephropathies - therapy | Hyperglycemia - genetics | Hyperglycemia - chemically induced | MAP Kinase Kinase 4 - metabolism | Islets of Langerhans - metabolism | Diet, High-Fat | Mitogen-Activated Protein Kinase 1 - genetics | Myosin-Light-Chain Phosphatase - metabolism | Hyperglycemia - pathology | Kidney Glomerulus - metabolism | Diabetes Mellitus, Experimental - chemically induced | Receptor, EphA1 - agonists | Diabetic Nephropathies - pathology | Dependovirus - metabolism | Ephrin-A1 - agonists | Genetic Vectors - chemistry | Islets of Langerhans - pathology | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Mice, Inbred C57BL | Myosin-Light-Chain Phosphatase - genetics | Diabetic Nephropathies - chemically induced | Gene Expression Regulation | Insulin Resistance | Genetic Vectors - metabolism | Diabetic Nephropathies - genetics | Kidney Glomerulus - pathology | Ephrin-A1 - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Receptor, EphA1 - genetics | Diabetes Mellitus, Experimental - pathology | MAP Kinase Kinase 4 - genetics | Mice | Hyperglycemia - therapy | Mitogen-Activated Protein Kinase 1 - metabolism | Tyrosine | Glucose metabolism | Hyperglycemia | Metabolites | Analysis | Diabetic nephropathies | Physiological aspects | Insulin resistance | Insulin | Phosphotransferases | Cardiovascular agents | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2011, Volume 6, Issue 9, pp. e24426 - e24426
Journal Article
Cancer Biology & Therapy, ISSN 1538-4047, 02/2006, Volume 5, Issue 2, pp. 228 - 233
Journal Article
The Journal of Physiology, ISSN 0022-3751, 04/2011, Volume 589, Issue 7, pp. 1725 - 1740
Non‐technical summary  Following a myocardial infarction, cardiac muscle becomes irreversibly damaged and over time this may lead to heart failure. Strategies... 
POLY(ADP-RIBOSE) POLYMERASE | HEPATOCELLULAR-CARCINOMA | PHYSIOLOGY | ENDOTHELIAL-CELL PROLIFERATION | EPH RECEPTORS | HEART-FAILURE | GROWTH-FACTOR | CARDIAC TROPONIN-I | NF-KAPPA-B | STIMULATES MIGRATION | NEUROSCIENCES | RECEPTOR TYROSINE KINASE | Neovascularization, Physiologic - drug effects | Male | Mice, 129 Strain | Immunoglobulin Fc Fragments - administration & dosage | RNA, Messenger - metabolism | DNA-Binding Proteins - metabolism | Tissue Distribution | Myocardial Infarction - pathology | Myocardium - metabolism | Receptors, Eph Family - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Recombinant Fusion Proteins - administration & dosage | Disease Models, Animal | Ephrin-A1 - administration & dosage | Gene Expression | Receptors, Eph Family - genetics | RNA, Messenger - genetics | Myocardium - pathology | Myocardial Infarction - metabolism | Ephrin-A1 - genetics | Transcription Factors - metabolism | Poly(ADP-ribose) Polymerases - metabolism | Animals | Myocardial Infarction - drug therapy | Ephrin-A1 - pharmacokinetics | Transcription Factor RelA - metabolism | Recombinant Fusion Proteins - pharmacokinetics | Recombinant Fusion Proteins - genetics | Mice | Poly (ADP-Ribose) Polymerase-1 | Immunoglobulin Fc Fragments - genetics | Troponin I - metabolism | Heart failure | Tyrosine | Physiological aspects | Gene expression | Heart attack | Index Medicus | Cardiovascular
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