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Aging Cell, ISSN 1474-9718, 08/2015, Volume 14, Issue 4, pp. 644 - 658
The healthspan of mice is enhanced by killing senescent cells using a transgenic suicide gene. Achieving the same using small molecules would have a tremendous... 
dasatinib | plasminogen‐activated inhibitor | dependence receptors | quercetin | ephrins | 3K delta | p21 | Dasatinib | Dependence receptors | Ephrins | Plasminogen-activated inhibitor | Quercetin | PI3K delta | P21 | ENDOTHELIAL DYSFUNCTION | PLASMINOGEN-ACTIVATOR INHIBITOR-1 | EXPRESSION PROFILES | plasminogen-activated inhibitor | CELLULAR SENESCENCE | CANCER-THERAPY | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | LUNG-CANCER | SET ENRICHMENT ANALYSIS | GENE-EXPRESSION | IONIZING-RADIATION | TUMOR-GROWTH | Carotid Arteries - drug effects | Endonucleases - genetics | Plasminogen Activator Inhibitor 2 - genetics | Transcriptome | Gene Expression Profiling | Adipocytes - drug effects | Aging - genetics | Osteoporosis - metabolism | Ephrins - metabolism | Plasminogen Activator Inhibitor 2 - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Osteoporosis - genetics | Intervertebral Disc - pathology | Fibroblasts - metabolism | Endothelial Cells - metabolism | Intervertebral Disc - chemistry | Fibroblasts - pathology | Mesenchymal Stem Cells - pathology | Mice, Knockout | Dasatinib - pharmacology | Osteoporosis - pathology | Ephrins - genetics | Fibroblasts - drug effects | Mice | Endothelial Cells - pathology | bcl-X Protein - metabolism | Aging - metabolism | Aging - drug effects | bcl-X Protein - genetics | Cellular Senescence - drug effects | Phosphatidylinositol 3-Kinases - metabolism | Endonucleases - metabolism | DNA-Binding Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Mesenchymal Stem Cells - drug effects | Mesenchymal Stem Cells - metabolism | Heart - physiopathology | Cellular Senescence - genetics | Osteoporosis - prevention & control | DNA-Binding Proteins - genetics | Adipocytes - pathology | Aging - pathology | Phosphatidylinositol 3-Kinases - genetics | Animals | Quercetin - pharmacology | Adipocytes - metabolism | Heart - drug effects | Carotid Arteries - pathology | Intervertebral Disc - drug effects | Drug Combinations | Endothelial Cells - drug effects | Index Medicus | Original
Journal Article
BBA - Molecular Cell Research, ISSN 0167-4889, 12/2016, Volume 1863, Issue 12, pp. 3096 - 3105
Differentiation of embryonic stem (ES) cells may be regulated by mechanical strain. Herein, signaling molecules underlying mechanical stimulation of... 
Reactive oxygen species | Vasculogenesis | Intracellular calcium | Embryonic stem cells | Mechanical strain | Nitric oxide | PROGENITOR CELLS | CARDIOMYOGENESIS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | FLOW | CELL BIOLOGY | SEMAPHORIN | SHEAR-STRESS | ENDOTHELIAL-CELLS | GROWTH | BODIES | DIFFERENTIATION | Benzoxazoles - pharmacology | RNA, Small Interfering - genetics | Reactive Oxygen Species - metabolism | Calcium - metabolism | Embryo, Mammalian | Neuropilin-1 - metabolism | Mouse Embryonic Stem Cells - cytology | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Antigens, CD - genetics | Mouse Embryonic Stem Cells - drug effects | Reactive Oxygen Species - agonists | Receptors, Immunologic - antagonists & inhibitors | Antigens, CD - metabolism | Embryoid Bodies - metabolism | Embryoid Bodies - cytology | Cell Differentiation | Semaphorins - genetics | Fibroblasts - metabolism | Calcium - agonists | Signal Transduction | Semaphorins - metabolism | Ephrin-B2 - metabolism | Embryoid Bodies - drug effects | Ephrin-B2 - genetics | Biomechanical Phenomena | Myocytes, Cardiac - drug effects | Fibroblast Growth Factor 2 - genetics | Fibroblasts - drug effects | Myocytes, Cardiac - metabolism | Fibroblasts - cytology | Mice | Receptors, Immunologic - genetics | RNA, Small Interfering - metabolism | Receptors, Cell Surface - genetics | Neuropilin-1 - genetics | Neovascularization, Physiologic - drug effects | Mouse Embryonic Stem Cells - metabolism | Neuropilin-1 - antagonists & inhibitors | Receptor, EphB4 - genetics | Ephrin-B2 - antagonists & inhibitors | Egtazic Acid - analogs & derivatives | Fibroblast Growth Factor 2 - metabolism | NG-Nitroarginine Methyl Ester - pharmacology | Nerve Tissue Proteins - antagonists & inhibitors | Proto-Oncogene Proteins c-sis - genetics | Myocytes, Cardiac - cytology | Proto-Oncogene Proteins c-sis - metabolism | Neovascularization, Physiologic - genetics | Gene Expression Regulation | Receptors, Cell Surface - metabolism | Nerve Tissue Proteins - genetics | Receptor, EphB4 - metabolism | Nitric Oxide - agonists | Nerve Tissue Proteins - metabolism | Triazoles - pharmacology | Animals | Egtazic Acid - pharmacology | Nitric Oxide - metabolism | Receptors, Immunologic - metabolism
Journal Article
Nature, ISSN 0028-0836, 04/2011, Volume 472, Issue 7343, pp. 356 - 360
Journal Article
PROTEOMICS, ISSN 1615-9853, 05/2013, Volume 13, Issue 10-11, pp. 1672 - 1686
Exosomes are small extracellular 40–100 nm diameter membrane vesicles of late endosomal origin that can mediate intercellular transfer of RNA s and proteins to... 
Cell biology | Protein complex | Secretome | Metastasis | Exosomes | Cancer | STEM-CELLS | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | BIOCHEMICAL RESEARCH METHODS | COLON-CANCER | PREMETASTATIC NICHE | LIPID RAFTS | GROWTH-FACTOR | PROTEINS | CARCINOMA | TUMOR MICROENVIRONMENT | PROGRESSION | Ephrin-B2 - secretion | Calgranulin B - metabolism | Cell Proliferation | Calgranulin A - metabolism | Membrane Microdomains - metabolism | Protein-Serine-Threonine Kinases - secretion | Receptor, Epidermal Growth Factor - secretion | Humans | Exosomes - pathology | Tenascin - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Neoplasm Metastasis | Receptor, Epidermal Growth Factor - metabolism | Serrate-Jagged Proteins | Exosomes - physiology | src-Family Kinases - metabolism | Colorectal Neoplasms | Membrane Proteins - metabolism | Intercellular Signaling Peptides and Proteins - secretion | src-Family Kinases - secretion | Endothelial Cells - physiology | Protein-Serine-Threonine Kinases - metabolism | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Membrane Proteins - secretion | Signal Transduction | Ephrin-B2 - metabolism | Animals | Proteome - secretion | Calcium-Binding Proteins - secretion | Cell Line, Tumor | Tenascin - secretion | Mice | Proteome - metabolism | Proteins | Development and progression | Electron microscopy | Cells | Colorectal cancer | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 05/2010, Volume 465, Issue 7297, pp. 483 - 486
In development, tissue regeneration or certain diseases, angiogenic growth leads to the expansion of blood vessels and the lymphatic vasculature. This involves... 
CARDIOVASCULAR DEVELOPMENT | ARTERIAL | SMOOTH-MUSCLE | TRANSGENIC MOUSE | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | AXON GUIDANCE | MOLECULAR REGULATION | RECEPTORS | EXPRESSION | ADULT | Humans | rac GTP-Binding Proteins - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Embryo, Mammalian - metabolism | Receptor, EphB4 - genetics | Endocytosis | Lymphatic Vessels | Female | Proto-Oncogene Proteins c-akt - metabolism | Ephrin-B2 - deficiency | Receptor, EphB4 - deficiency | Signal Transduction | Endothelial Cells - metabolism | Neovascularization, Physiologic - genetics | Embryo, Mammalian - blood supply | Cells, Cultured | Mice, Transgenic | Neuropeptides - metabolism | Zebrafish | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Ephrin-B2 - metabolism | Receptor, EphB4 - metabolism | Ephrin-B2 - genetics | Pregnancy | Embryo Loss | Animals | Endothelial Cells - cytology | Vascular Endothelial Growth Factor C - metabolism | Mice | rac1 GTP-Binding Protein | Lymphangiogenesis - genetics | Ephrins | Control | Physiological aspects | Genetic aspects | Research | Neovascularization | Vascular endothelial growth factor | Angiogenesis | Kinases | Rodents | Cell adhesion & migration | Index Medicus | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 09/2017, Volume 110, pp. 368 - 380
Disruption of mitochondrial reactive oxygen species (mtROS) homeostasis is a key factor inducing UCB-MSC senescence. Accordingly, preventing mtROS accumulation... 
Mesenchymal stem cell | Ephrin | MnSOD | Aging | Ephrin receptor | Mitochondrial ROS | Sirtuin | OXIDATIVE STRESS | DECREASED SIRT3 | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROLIFERATION | SIRTUINS | CELLULAR SENESCENCE | CANCER | MESENCHYMAL STEM-CELLS | METABOLISM | MANGANESE SUPEROXIDE-DISMUTASE | ENDOCRINOLOGY & METABOLISM | CALORIE RESTRICTION | RNA, Small Interfering - genetics | Phosphorylation | Superoxide Dismutase - genetics | Reactive Oxygen Species - metabolism | Sirtuin 3 - metabolism | Skin - metabolism | Humans | Male | Sirtuin 3 - antagonists & inhibitors | Skin - injuries | Mesenchymal Stromal Cells - cytology | Cellular Senescence | src-Family Kinases - metabolism | NF-E2-Related Factor 2 - genetics | Superoxide Dismutase - metabolism | Sirtuin 3 - genetics | Signal Transduction | Cells, Cultured | Gene Expression Regulation | Mesenchymal Stromal Cells - metabolism | Mitochondria - metabolism | Fetal Blood - cytology | Receptor, EphB2 - genetics | Receptor, EphB2 - metabolism | Fetal Blood - metabolism | Ephrin-B2 - metabolism | Mice, Inbred ICR | Ephrin-B2 - genetics | Animals | NF-E2-Related Factor 2 - metabolism | Mice | Wound Healing - physiology | src-Family Kinases - genetics | Mesenchymal Stem Cell Transplantation | RNA, Small Interfering - metabolism | Homeostasis | Care and treatment | Research institutes | Lysine | Wounds and injuries | Superoxide | Stem cells | Index Medicus
Journal Article
International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 2007, Volume 69, Issue 1, pp. 167 - 175
Purpose To investigate the expression pattern of hypoxia-induced proteins identified as being involved in malignant progression of head-and-neck squamous cell... 
Radiology | Hematology, Oncology and Palliative Medicine | Head-and-neck cancer | Hypoxia | Prognosis | Marker panel | CANCER PATIENTS | BREAST-CANCER | ENDOGENOUS MARKERS | TUMOR HYPOXIA | EPH RECEPTORS | ANGIOGENESIS | ONCOLOGY | OXYGENATION | GROWTH | GENE-EXPRESSION | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | RADIATION-THERAPY | Cell Hypoxia - physiology | Ephrin-A1 - metabolism | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Galectin 1 - metabolism | Male | Osteopontin - metabolism | Neoplasm Proteins - metabolism | Head and Neck Neoplasms - metabolism | Oxygen - metabolism | Carbonic Anhydrases - metabolism | Carcinoma, Squamous Cell - mortality | I-kappa B Kinase - metabolism | Antigens, Neoplasm - metabolism | Biomarkers, Tumor - metabolism | Female | Membrane Proteins - metabolism | Carbonic Anhydrase IX | Proto-Oncogene Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Disease Progression | Head and Neck Neoplasms - pathology | Tetrahydrofolate Dehydrogenase - metabolism | Analysis of Variance | Partial Pressure | Protein-Lysine 6-Oxidase - metabolism | Cell Line, Tumor | Head and Neck Neoplasms - mortality | Proteins | Development and progression | Squamous cell carcinoma | Index Medicus | HEMOGLOBIN | HEAD | CARBONIC ANHYDRASE | OXYGEN | GROWTH FACTORS | MULTIVARIATE ANALYSIS | THERAPY | OXIDASES | ANOXIA | CARCINOMAS | RADIOLOGY AND NUCLEAR MEDICINE | PARTIAL PRESSURE | NECK | CONNECTIVE TISSUE
Journal Article
Science Signaling, ISSN 1945-0877, 12/2017, Volume 10, Issue 508, p. eaan4667
Journal Article
Journal of Proteome Research, ISSN 1535-3893, 11/2015, Volume 14, Issue 11, pp. 4674 - 4686
Recent epidemiological data indicate that radiation doses as low as those used in computer tomography may result in long-term neurocognitive side effects. The... 
memory | proteomics | miR-132 | mitochondria | dendritic spine | Rac1 | synapse | brain | cerebellum | ionizing radiation | ADULT NEUROGENESIS | ALZHEIMERS-DISEASE | BIOCHEMICAL RESEARCH METHODS | AXONAL-TRANSPORT | CEREBRAL-CORTEX | DENDRITIC SPINES | BRAIN MITOCHONDRIA | MITOCHONDRIAL DYNAMICS | LONG-TERM POTENTIATION | IONIZING-RADIATION | UP-REGULATION | Memory - drug effects | Whole-Body Irradiation | Proteome - genetics | Actin Depolymerizing Factors - genetics | Gamma Rays - adverse effects | MicroRNAs - metabolism | Synaptosomes - metabolism | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Actin Depolymerizing Factors - metabolism | Oxidative Phosphorylation - radiation effects | rho-Specific Guanine Nucleotide Dissociation Inhibitors - genetics | rho-Specific Guanine Nucleotide Dissociation Inhibitors - metabolism | Axons - ultrastructure | Ephrins - metabolism | Mitochondria - radiation effects | Female | Neuropeptides - genetics | Animals, Newborn | Mice, Inbred C57BL | Axons - metabolism | Neuropeptides - metabolism | Electron Transport Chain Complex Proteins - genetics | Mitochondria - metabolism | Long-Term Potentiation - radiation effects | Cerebral Cortex - radiation effects | Cyclic AMP Response Element-Binding Protein - genetics | Hippocampus - metabolism | Synaptosomes - radiation effects | Animals | Synaptic Transmission - radiation effects | Electron Transport Chain Complex Proteins - metabolism | Ephrins - genetics | Cyclic AMP Response Element-Binding Protein - metabolism | Hippocampus - radiation effects | Axons - radiation effects | Mice | MicroRNAs - genetics | Hippocampus - physiopathology | Proteome - metabolism | rac1 GTP-Binding Protein - metabolism | rac1 GTP-Binding Protein - genetics | Index Medicus
Journal Article