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Oncogene, ISSN 0950-9232, 09/2010, Volume 29, Issue 36, pp. 4989 - 5005
Hepatocellular carcinoma (HCC) is a highly prevalent, treatment-resistant malignancy with a multifaceted molecular pathogenesis. Current evidence indicates... 
Signaling | Hepatocellular carcinoma | Multikinase inhibitor | Vascular endothelial growth factor | Sorafenib | Epidermal growth factor receptor | FACTOR-RECEPTOR | signaling | sorafenib | MESSENGER-RNA EXPRESSION | epidermal growth factor receptor | BIOCHEMISTRY & MOLECULAR BIOLOGY | C-MET PROTOONCOGENE | CELL BIOLOGY | CELL LUNG-CANCER | PHASE-II TRIAL | ONCOLOGY | vascular endothelial growth factor | GENETICS & HEREDITY | TYROSINE KINASE INHIBITOR | PROGNOSTIC-SIGNIFICANCE | ACTIVATED PROTEIN-KINASES | multikinase inhibitor | TUMOR-GROWTH | hepatocellular carcinoma | ENDOTHELIAL GROWTH-FACTOR | Receptor, Epidermal Growth Factor - genetics | Receptors, Vascular Endothelial Growth Factor - physiology | MAP Kinase Signaling System - physiology | Humans | Liver Neoplasms - therapy | Liver Neoplasms - etiology | Somatomedins - physiology | Hepatocyte Growth Factor - physiology | Carcinoma, Hepatocellular - genetics | Hepatocyte Growth Factor - genetics | Liver Neoplasms - pathology | Carcinoma, Hepatocellular - etiology | Receptors, Vascular Endothelial Growth Factor - genetics | Receptor, Epidermal Growth Factor - physiology | Liver Neoplasms - genetics | Signal Transduction - genetics | Proto-Oncogene Proteins c-met - genetics | Liver Neoplasms - ethnology | Proto-Oncogene Proteins c-met - physiology | Animals | Models, Biological | Somatomedins - genetics | Carcinoma, Hepatocellular - pathology | Signal Transduction - physiology | Carcinoma, Hepatocellular - therapy | Care and treatment | Gene mutations | Cellular signal transduction | Genetic aspects | Research | Hepatoma | Health aspects | Signal transduction | Genetics | Liver diseases | Mutation | Cancer
Journal Article
Journal Article
Biochemical Journal, ISSN 0264-6021, 03/2009, Volume 418, Issue 3, pp. 491 - 506
The multicellular nature of metazoans means that all cellular processes need to be tuned by adhesive interactions between cells and their local... 
Growth factor receptor | Integrin | Adhesion complex | Integrin signalling | Adhesome | Syndecan | Cytokine receptor | FOCAL ADHESION FORMATION | cytokine receptor | EPIDERMAL-GROWTH | BIOCHEMISTRY & MOLECULAR BIOLOGY | adhesion complex | adhesome | growth factor receptor | HUMAN BREAST-CANCER | CELL-CYCLE PROGRESSION | GROWTH-FACTOR RECEPTOR | FACTOR EGF RECEPTOR | ALPHA-6-BETA-4 INTEGRIN | integrin signalling | syndecan | EXTRACELLULAR-MATRIX | integrin | NF-KAPPA-B | PROTEIN-TYROSINE-PHOSPHATASE | Neovascularization, Physiologic - drug effects | Integrin beta3 - physiology | Integrin alpha6beta4 - physiology | Receptor Protein-Tyrosine Kinases - physiology | Receptors, Platelet-Derived Growth Factor - physiology | Receptors, Cytokine - physiology | Cell Movement - physiology | Integrin beta1 - physiology | Prolactin - physiology | Extracellular Matrix - physiology | Receptors, Vitronectin - physiology | Receptor, Epidermal Growth Factor - physiology | Cell Survival - physiology | Gene Expression Regulation, Developmental - physiology | Integrins - physiology | Receptors, Interleukin - physiology | Neoplasms, Glandular and Epithelial - physiopathology | Neoplasm Invasiveness - physiopathology | Neovascularization, Physiologic - physiology | Receptors, Growth Factor - physiology | Animals | Cell Adhesion - physiology | Syndecans - physiology | Cell Cycle - physiology | Integrin alphaVbeta3 - physiology | Signal Transduction - physiology | Interferons - physiology
Journal Article
Nature Medicine, ISSN 1078-8956, 05/2011, Volume 17, Issue 5, pp. 589 - 595
Hepatitis C virus (HCV) is a major cause of liver disease, but therapeutic options are limited and there are no prevention strategies. Viral entry is the first... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | B TYPE-I | NEUTRALIZING ANTIBODIES | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | FACTOR RECEPTOR | CELL BIOLOGY | EPIDERMAL-GROWTH-FACTOR | CELL TRANSMISSION | HUMAN LIVER | INFECTION | ERLOTINIB | Erlotinib Hydrochloride | RNA, Small Interfering - genetics | Receptor, Epidermal Growth Factor - genetics | Hepatitis C - therapy | Humans | Virus Internalization - drug effects | RNA Interference | Membrane Proteins - physiology | Base Sequence | Hepacivirus - physiology | Hepatitis C - prevention & control | Receptor, Epidermal Growth Factor - physiology | Hepacivirus - drug effects | Cell Line | Antiviral Agents - pharmacology | Receptor, EphA2 - physiology | Tetraspanin 28 | Claudin-1 | Animals | Hepatitis C - virology | Receptor, EphA2 - genetics | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Antigens, CD - physiology | Hepatitis C - physiopathology | Ligands | Mice | Protein Kinase Inhibitors - pharmacology | Receptor, EphA2 - antagonists & inhibitors | Quinazolines - pharmacology | Host-Pathogen Interactions - physiology | Ephrin A | Antiviral agents | Care and treatment | Patient outcomes | Physiological aspects | Research | Hepatitis C | Hepatitis | Viruses | Drug therapy | Antiviral drugs | Receptor, EphA2 | Hépatology and Gastroenterology | Antigens, CD81 | Antiviral Agents | Quinazolines | Virus Internalization | Membrane Proteins | Host-Pathogen Interactions | Life Sciences | Human health and pathology | Protein Kinase Inhibitors | RNA, Small Interfering | Hepacivirus | Antigens, CD | Receptor, Epidermal Growth Factor | Liver | HCV escape variants | Cell-cell transmission | genetics | pharmacology | Antiviral | therapy | physiology | drug effects | prevention & control | antagonists & inhibitors | physiopathology | Phosphotyrosine kinase | virology
Journal Article
Nature Genetics, ISSN 1061-4036, 04/2007, Volume 39, Issue 4, pp. 503 - 512
Journal Article
Journal Article
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