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PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 9, pp. e23833 - e23833
Epithelial to mesenchymal transition (EMT) is a critical event in cancer progression and is closely linked to the breast epithelial cancer stem cell phenotype.... 
SURVIVAL | DENSITY | BASAL-LIKE | MORPHOGENESIS | TUMOR PROGRESSION | MICROENVIRONMENT | MULTIDISCIPLINARY SCIENCES | CARCINOMAS | MESENCHYMAL TRANSITION | CANCER | EXPRESSION | Cell Line | Neoplastic Stem Cells - cytology | Epithelial Cells - metabolism | Humans | Transforming Growth Factor beta1 - metabolism | Cells, Cultured | Cell Dedifferentiation | Epidermal Growth Factor - metabolism | Immunochemistry | Blotting, Western | Hepatocyte Growth Factor - metabolism | Fibroblast Growth Factors - metabolism | Flow Cytometry | Neoplastic Stem Cells - metabolism | Epithelial-Mesenchymal Transition | Cell Line, Tumor | Female | Apoptosis - physiology | Epithelial Cells - cytology | In Vitro Techniques | Analysis | Stem cells | Endothelium | Keratins | Cell culture | Health sciences | Leukocyte migration | Mesenchyme | Laboratories | Epithelial cells | Metastasis | E-cadherin | Morphogenesis | Liver cancer | Genotype & phenotype | Angiogenesis | N-Cadherin | Epidermal growth factor | Rodents | Fibroblasts | Vascular endothelial growth factor | Inducers | Medical research | Hematology | Invasiveness | Breast cancer | Endothelial cells | Signaling | Properties (attributes) | Hospitals | Breast | Hypoxia | Three dimensional models | Cell migration | Tumors | Apoptosis | Cancer | Index Medicus | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Cancer and Oncology | Medicin och hälsovetenskap | Cancer och onkologi
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2010, Volume 285, Issue 6, pp. 4213 - 4223
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, pp. e33788 - e33788
Background: To improve cancer therapy, it is critical to target metastasizing cells. Circulating tumor cells (CTCs) are rare cells found in the blood of... 
EPITHELIAL-MESENCHYMAL TRANSITION | GROWTH-FACTOR-BETA | TGF-BETA | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | CLINICAL-IMPLICATIONS | HER2 STATUS | STEM-CELL | PROGNOSTIC-SIGNIFICANCE | NF-KAPPA-B | RT-QPCR | Microarray Analysis - methods | Microfluidic Analytical Techniques | Humans | Gene Expression Regulation, Neoplastic | Neoplastic Cells, Circulating - metabolism | Gene Expression Profiling | Breast Neoplasms - metabolism | Lymphoma - blood | Single-Cell Analysis - instrumentation | Neoplasm Metastasis | Breast Neoplasms - genetics | Single-Cell Analysis - methods | Breast Neoplasms - blood | Cell Line, Tumor | Female | Care and treatment | Genes | Breast cancer | Genetic transcription | Metastasis | Transforming growth factors | Analysis | Stem cells | Genetic aspects | Drug discovery | Health aspects | Microfluidics | Cancer | Bioengineering | Therapy | Biotechnology | Motility | Profiling | Transcription | Mesenchyme | S100A4 protein | Feasibility analysis | Genomes | Biochemistry | Kinases | Blood | Subgroups | Metastases | Heterogeneity | Epidermal growth factor | Rodents | Surgery | Electrical engineering | Medical research | Antigens | Tumor cells | Feasibility studies | Tumor cell lines | Gene expression | Patients | Contamination | CXCR4 protein | Medicine | White blood cells | Blood circulation | Biopsy | Pancreatic cancer | Cell lines | Breast | Solid tumors | Molecular biology | Prostate | Tumors | Index Medicus
Journal Article
Journal of Dental Research, ISSN 0022-0345, 6/2015, Volume 94, Issue 6, pp. 843 - 852
Journal Article
Nature, ISSN 0028-0836, 10/2015, Volume 526, Issue 7575, pp. 715 - 718
Journal Article
Cancer Letters, ISSN 0304-3835, 2010, Volume 296, Issue 2, pp. 216 - 224
Abstract We found that among four master epithelial-to-mesenchymal transition (EMT)-inducing genes ( ZEB1 , SIP1 , Snail , and Slug ) ZEB1 expression was most... 
Hematology, Oncology and Palliative Medicine | MicroRNA | RNA interference | Anchorage-independent growth | EMT | Lung cancer | Epidermal growth factor receptor | METASTASIS | E-CADHERIN EXPRESSION | PANCREATIC-CANCER | MIR-200 FAMILY | LINES | BREAST-CANCER | INVASION | ONCOLOGY | CARCINOMA | TRANSCRIPTIONAL REPRESSOR | SNAIL | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | RNA, Small Interfering - genetics | Receptor, Epidermal Growth Factor - genetics | Mesothelioma - pathology | Carcinoma, Non-Small-Cell Lung - genetics | Humans | Apoptosis - genetics | Epithelial Cells - pathology | Lung Neoplasms - pathology | Mesothelioma - genetics | Transcription Factors - genetics | Homeodomain Proteins - genetics | Gene Knockdown Techniques | Vimentin - genetics | Cell Division | Cell Line, Tumor | MicroRNAs - genetics | Mutation | Cadherins - genetics | Mesoderm - pathology | Zinc Finger E-box-Binding Homeobox 1 | RNA | Growth | Oncology, Experimental | Respiratory agents | Research | Lung cancer, Non-small cell | Epidermal growth factor | Stem cells | Genetic research | Genetic aspects | Universities and colleges | Intermediate filament proteins | Cancer | Proteins | Genotype & phenotype | Transcription factors | Genes | Medical prognosis | MicroRNAs | Values | Breast cancer | Apoptosis | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2016, Volume 11, Issue 9, pp. e0163149 - e0163149
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2017, Volume 12, Issue 2, pp. e0172574 - e0172574
During wound healing, skin function is restored by the action of several cell types that undergo differentiation, migration, proliferation and/or apoptosis.... 
BREAST-CANCER | GROWTH-FACTOR RECEPTOR | HEALING ACTIVITY | SIGNALING PATHWAYS | TGF-BETA | MULTIDISCIPLINARY SCIENCES | GELATINASE-B | HUMAN KERATINOCYTE MIGRATION | INHIBITS PROLIFERATION | C-JUN | WOUND REPAIR | Enzyme Activators - pharmacology | Cell Line | Epithelial Cells - metabolism | ErbB Receptors - metabolism | Epithelial Cells - drug effects | Humans | Oleanolic Acid - pharmacology | Enzyme Activation - drug effects | Gene Expression Regulation - drug effects | Cell Movement - drug effects | MAP Kinase Kinase 4 - metabolism | MAP Kinase Signaling System - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Epithelial Cells - cytology | ErbB Receptors - agonists | Mitogen-Activated Protein Kinases - metabolism | Wound healing | Epithelial cells | Analysis | Cell migration | Mitogen-activated protein kinases | Cell culture | Transcription factors | Phosphorylation | Physiological effects | Mesenchyme | Lung | Activation | Kinases | Cell adhesion & migration | Oleanolic acid | Proteins | Lipophilic | Signal transduction | Cell growth | Epidermal growth factor | Fibroblasts | Polyphenols | Extracellular matrix | Physiology | Localization | Medicinal plants | Epidermal growth factor receptors | Extracellular signal-regulated kinase | c-Jun protein | Breast cancer | Pharmacology | Gene expression | Plant extracts | Signaling | Flavonoids | Molecular modelling | Acids | Cell lines | Healing | Skin | Apoptosis | Cancer | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 10/2018, Volume 562, Issue 7727, pp. 429 - 433
Despite the efficacy of Hedgehog pathway inhibitors in the treatment of basal cell carcinoma (BCC)(1) , residual disease persists in some patients and may... 
STEM-CELLS | VISMODEGIB | HAIR FOLLICLE | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | KERATINOCYTES | OPEN-LABEL | SKIN | DIFFERENTIATION | CARCINOMA | BASAL-CELL | Anilides - therapeutic use | Skin Neoplasms - drug therapy | Humans | Epidermal Cells - metabolism | Hedgehog Proteins - metabolism | Smoothened Receptor - metabolism | Stem Cells - metabolism | Hair Follicle - pathology | Epidermal Cells - drug effects | Pyridines - therapeutic use | Skin Neoplasms - pathology | Pyridines - administration & dosage | Carcinoma, Basal Cell - pathology | Hedgehog Proteins - antagonists & inhibitors | Carcinoma, Basal Cell - metabolism | Skin Neoplasms - metabolism | Hair Follicle - drug effects | Animals | Wnt Signaling Pathway - drug effects | Anilides - administration & dosage | Signal Transduction - drug effects | Cell Differentiation - drug effects | Hair Follicle - metabolism | Anilides - pharmacology | Carcinoma, Basal Cell - drug therapy | Epidermal Cells - pathology | Stem Cells - drug effects | Stem Cells - pathology | Cell Proliferation - drug effects | Mice | Pyridines - pharmacology | Basal cell carcinoma | Enzyme inhibitors | Research | Genetic transcription | Oncology, Experimental | Cancer | Chromatin | Transcription factors | Substance abuse treatment | Wnt protein | Medical treatment | Genomics | Stem cell transplantation | Epidermis | Activation | Genomes | Gene expression | Enhancers | Inhibitors | Transcription activation | Stem cells | Tumors | Index Medicus
Journal Article