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Nature cell biology, ISSN 1476-4679, 02/2010, Volume 12, Issue 3, pp. 299 - 305
For most types of cancers, the cell at the origin of tumour initiation is still unknown. Here, we used mouse genetics to identify cells at the origin of basal... 
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Epithelial Cells - metabolism | Cadherins - metabolism | Receptors, G-Protein-Coupled - metabolism | Skin - metabolism | Cell Count | Ear, External - pathology | Integrin beta4 - metabolism | Tail - pathology | Hair Follicle - pathology | Hedgehog Proteins - genetics | Neoplastic Stem Cells - metabolism | Kruppel-Like Transcription Factors - metabolism | Smoothened Receptor | Neoplastic Stem Cells - pathology | Cell Differentiation | Patched Receptors | Skin - pathology | Keratin-10 - metabolism | Epidermis - metabolism | Epidermis - pathology | RNA, Untranslated | Bacterial Proteins - genetics | Receptors, Cell Surface - metabolism | Epithelial Cells - pathology | Genes, Reporter - genetics | Mice, Transgenic | Carcinoma, Basal Cell - pathology | Mice, Inbred Strains | Clone Cells - metabolism | Keratin-14 - genetics | Carcinoma, Basal Cell - metabolism | Keratin-15 - metabolism | Keratin-19 - genetics | Proteins - genetics | Cell Lineage | Animals | Clone Cells - pathology | Proteins - metabolism | Models, Biological | Hair Follicle - metabolism | Bacterial Proteins - metabolism | Luminescent Proteins - genetics | Mice | Receptors, G-Protein-Coupled - genetics | Integrases - genetics | Keratin-15 - genetics | Luminescent Proteins - metabolism | Basal cell carcinoma | Stem cells | Genetic aspects | Cellular signal transduction | Research | Health aspects | Risk factors | Index Medicus
Journal Article
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 06/2014, Volume 133, Issue 6, pp. 1626 - 1634
.... Methods CsA's effects on AD skin pathology were evaluated by using gene expression and immunohistochemistry... 
Allergy and Immunology | cyclosporine | immune | S100 proteins | Atopic dermatitis | epidermal abnormalities | T cell | Allergy | Immunology | Life Sciences & Biomedicine | Science & Technology | T-Lymphocyte Subsets - immunology | Inflammation - pathology | Dermatitis, Atopic - genetics | Cyclosporine - pharmacology | Dendritic Cells - immunology | Humans | Hyperplasia | Immunosuppressive Agents - therapeutic use | Middle Aged | Male | Gene Expression Profiling | Young Adult | Inflammation - drug therapy | T-Lymphocyte Subsets - drug effects | Dermatitis, Atopic - immunology | Adult | Dendritic Cells - drug effects | Female | Immunosuppressive Agents - pharmacology | Dendritic Cells - metabolism | Epidermis - pathology | Epidermis - drug effects | Treatment Outcome | Dermatitis, Atopic - pathology | Inflammation - immunology | Gene Expression Regulation - drug effects | Cyclosporine - therapeutic use | Phenotype | Dermatitis, Atopic - drug therapy | Epidermis - immunology | Signal Transduction - drug effects | T-Lymphocyte Subsets - metabolism | Adolescent | Biomarkers | Inflammation - genetics | Aged | Cluster Analysis | Medical colleges | Pharmacogenetics | Care and treatment | Analysis | T cells | Immunosuppressive agents | Hypothesis testing | Cytokines | Pathogenesis | Proteins | Studies | Pathology | Signal transduction | Genotype & phenotype | Biopsy | Lymphocytes | Quality control | Skin | Index Medicus | Abridged Index Medicus
Journal Article
Experimental dermatology, ISSN 0906-6705, 07/2011, Volume 20, Issue 7, pp. 589 - 594
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2013, Volume 182, Issue 3, pp. 806 - 818
Journal Article
Journal of investigative dermatology, ISSN 0022-202X, 02/2008, Volume 128, Issue 2, pp. 352 - 360
Nitric oxide (NO) plays an important role in the cutaneous response to UV radiation and in cutaneous inflammation. The presence of inducible NO synthase... 
Life Sciences & Biomedicine | Dermatology | Science & Technology | Erythema - pathology | Inflammation Mediators - administration & dosage | Langerhans Cells - pathology | Dermatitis - immunology | Humans | Male | Nitric Oxide - adverse effects | Skin Ulcer - immunology | Inflammation Mediators - adverse effects | Cell Movement - immunology | Erythema - chemically induced | T-Lymphocytes - drug effects | Blister - chemically induced | Female | T-Lymphocytes - pathology | Langerhans Cells - drug effects | Nitric Oxide Donors - adverse effects | Zeolites - adverse effects | Erythema - immunology | Neutrophils - pathology | Nitric Oxide - immunology | Epidermis - pathology | Edema - immunology | Macrophages - pathology | Epidermis - drug effects | Neutrophils - drug effects | Dermatitis - prevention & control | Nitric Oxide - administration & dosage | Skin Ulcer - chemically induced | Zeolites - administration & dosage | Cell Movement - drug effects | Dermatitis - pathology | Nitric Oxide Donors - administration & dosage | Skin Ulcer - pathology | Acids | Epidermis - immunology | Blister - immunology | Macrophages - drug effects | Blister - pathology | Edema - chemically induced | Administration, Topical | Edema - pathology | CD8 antigen | U.V. radiation | Inflammation | Leukocytes (neutrophilic) | Lymphocytes T | Nitrite | Zeolite | Macrophages | Nitric-oxide synthase | CD4 antigen | Interleukin 4 | Microdialysis | Nitric oxide | gamma -Interferon | Skin | Internet | Topical application | Erythema | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 10/2012, Volume 109, Issue 45, pp. 18384 - 18389
A chronic inflammatory microenvironment favors tumor progression through molecular mechanisms that are still incompletely defined. In inflammation-induced skin... 
Myeloid cells | Cytokines | Skin cancers | Keratinocytes | Epidermis | Mice | Skin | Inflammation | Tumors | Cancer | Innate immunity | Interleukin-1 | Epithelial skin cancer | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Inflammation - pathology | Tetradecanoylphorbol Acetate | Cell Proliferation | Inflammasomes - metabolism | Humans | Tumor Microenvironment | Caspase 1 - metabolism | Cytoskeletal Proteins - deficiency | Skin Neoplasms - prevention & control | Receptors, Interleukin-1 - deficiency | Cytoskeletal Proteins - metabolism | Neoplasms, Squamous Cell - pathology | Skin - pathology | Skin Neoplasms - pathology | Epithelium - pathology | Epithelium - metabolism | Down-Regulation | Tumor Suppressor Protein p53 - metabolism | Organ Specificity | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Skin Neoplasms - metabolism | Keratinocytes - pathology | Animals | Apoptosis Regulatory Proteins | Keratinocytes - metabolism | CARD Signaling Adaptor Proteins | Caspase 1 - deficiency | Myeloid Cells - metabolism | 9,10-Dimethyl-1,2-benzanthracene | Myeloid Cells - pathology | Adaptor Proteins, Signal Transducing - metabolism | Cell Transformation, Neoplastic - pathology | Cytokines - biosynthesis | Epithelial tumors | Development and progression | Research | Health aspects | Carcinogenesis | Skin cancer | Index Medicus | Biological Sciences | interleukin-1 | innate immunity | epithelial skin cancer
Journal Article
Nature (London), ISSN 1476-4687, 01/2017, Volume 541, Issue 7638, pp. 494 - 499
We are just beginning to understand how translational control affects tumour initiation and malignancy. Here we use an epidermis-specific, in vivo ribosome... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Genetic aspects | Metastasis | Genetic translation | Health aspects | Tumors | Proteins | Phosphorylation | Peptides | Protein synthesis | Genomes | Tumorigenesis | Cancer | Index Medicus
Journal Article