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Nature Cell Biology, ISSN 1465-7392, 03/2010, Volume 12, Issue 3, pp. 299 - 305
For most types of cancers, the cell at the origin of tumour initiation is still unknown. Here, we used mouse genetics to identify cells at the origin of basal... 
POPULATION | STEM-CELLS | SONIC HEDGEHOG | EPIDERMIS | HAIR FOLLICLE BULGE | KERATINOCYTES | CANCER | EXPRESSION | HUMAN HOMOLOG | MOUSE SKIN | CELL BIOLOGY | Epithelial Cells - metabolism | Cadherins - metabolism | Receptors, G-Protein-Coupled - metabolism | Skin - metabolism | Cell Count | Ear, External - pathology | Integrin beta4 - metabolism | Tail - pathology | Hair Follicle - pathology | Hedgehog Proteins - genetics | Neoplastic Stem Cells - metabolism | Kruppel-Like Transcription Factors - metabolism | Smoothened Receptor | Neoplastic Stem Cells - pathology | Cell Differentiation | Patched Receptors | Skin - pathology | Keratin-10 - metabolism | Epidermis - metabolism | Epidermis - pathology | RNA, Untranslated | Bacterial Proteins - genetics | Receptors, Cell Surface - metabolism | Epithelial Cells - pathology | Genes, Reporter - genetics | Mice, Transgenic | Carcinoma, Basal Cell - pathology | Mice, Inbred Strains | Clone Cells - metabolism | Keratin-14 - genetics | Carcinoma, Basal Cell - metabolism | Keratin-15 - metabolism | Keratin-19 - genetics | Proteins - genetics | Cell Lineage | Animals | Clone Cells - pathology | Proteins - metabolism | Models, Biological | Hair Follicle - metabolism | Bacterial Proteins - metabolism | Luminescent Proteins - genetics | Mice | Receptors, G-Protein-Coupled - genetics | Integrases - genetics | Keratin-15 - genetics | Luminescent Proteins - metabolism | Basal cell carcinoma | Stem cells | Genetic aspects | Cellular signal transduction | Research | Health aspects | Risk factors | Index Medicus
Journal Article
Journal of the European Academy of Dermatology and Venereology, ISSN 0926-9959, 05/2015, Volume 29, Issue 5, pp. 991 - 997
BackgroundProgression from actinic keratosis (AK) to invasive squamous cell carcinoma (iSCC) of the skin is thought to occur after the development of full... 
PROPOSAL | SOLAR KERATOSIS | DISEASE | DERMATOLOGY | Skin Neoplasms - pathology | Epidermis - pathology | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Male | Skin Ulcer - pathology | Keratinocytes - pathology | Aged, 80 and over | Female | Aged | Cell Transformation, Neoplastic - pathology | Keratosis, Actinic - pathology | Index Medicus
Journal Article
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2014, Volume 133, Issue 6, pp. 1626 - 1634
Background Atopic dermatitis (AD) is the most common inflammatory disease. Evolving disease models link changes in epidermal growth and differentiation to TH... 
Allergy and Immunology | cyclosporine | immune | S100 proteins | Atopic dermatitis | epidermal abnormalities | T cell | BARRIER | KERATINOCYTES | A MICROEMULSION | IMMUNOLOGY | INHIBITION | ALLERGY | TH2 CYTOKINES | DISEASE | THERAPEUTIC RESPONSE | SKIN | EXPRESSION | T-CELLS | T-Lymphocyte Subsets - immunology | Inflammation - pathology | Dermatitis, Atopic - genetics | Cyclosporine - pharmacology | Dendritic Cells - immunology | Humans | Hyperplasia | Immunosuppressive Agents - therapeutic use | Middle Aged | Male | Gene Expression Profiling | Young Adult | Inflammation - drug therapy | T-Lymphocyte Subsets - drug effects | Dermatitis, Atopic - immunology | Adult | Dendritic Cells - drug effects | Female | Immunosuppressive Agents - pharmacology | Dendritic Cells - metabolism | Epidermis - pathology | Epidermis - drug effects | Treatment Outcome | Dermatitis, Atopic - pathology | Inflammation - immunology | Gene Expression Regulation - drug effects | Cyclosporine - therapeutic use | Phenotype | Dermatitis, Atopic - drug therapy | Epidermis - immunology | Signal Transduction - drug effects | T-Lymphocyte Subsets - metabolism | Adolescent | Biomarkers | Inflammation - genetics | Aged | Cluster Analysis | Medical colleges | Pharmacogenetics | Care and treatment | Analysis | T cells | Immunosuppressive agents | Hypothesis testing | Cytokines | Pathogenesis | Proteins | Studies | Pathology | Signal transduction | Genotype & phenotype | Biopsy | Lymphocytes | Quality control | Skin | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2013, Volume 8, Issue 9, pp. e75696 - e75696
Journal Article
Cancer Research, ISSN 0008-5472, 07/2013, Volume 73, Issue 14, pp. 4212 - 4221
VEGF-C and VEGF-D were identified as lymphangiogenic growth factors and later shown to promote tumor metastasis, but their effects on carcinogenesis are poorly... 
VESSELS | ANGIOGENESIS | SPECIFICITY | ONCOLOGY | TUMOR LYMPHANGIOGENESIS | MOUSE | SQUAMOUS-CELL CARCINOMA | FACTOR RECEPTOR-3 | CANCER | LYMPHATIC METASTASIS | ENDOTHELIAL GROWTH-FACTOR | Inflammation - pathology | Leukocytes - pathology | Skin Neoplasms - drug therapy | Skin - metabolism | Carcinogenesis - chemically induced | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Carcinoma, Squamous Cell - chemically induced | Carcinogenesis - metabolism | Neovascularization, Pathologic - pathology | Carcinogens | Inflammation - metabolism | Inflammation - drug therapy | Female | Skin - pathology | Skin Neoplasms - pathology | Epidermis - metabolism | Epidermis - pathology | Vascular Endothelial Growth Factor D - antagonists & inhibitors | Cytokines - metabolism | Macrophages - pathology | Epidermis - drug effects | Mice, Transgenic | Skin Neoplasms - chemically induced | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Skin Neoplasms - metabolism | Carcinogenesis - drug effects | Keratinocytes - pathology | Macrophages - metabolism | Vascular Endothelial Growth Factor D - metabolism | Animals | Carcinoma, Squamous Cell - drug therapy | Vascular Endothelial Growth Factor C - antagonists & inhibitors | Keratinocytes - drug effects | Keratinocytes - metabolism | Neovascularization, Pathologic - drug therapy | Vascular Endothelial Growth Factor C - metabolism | Leukocytes - drug effects | Macrophages - drug effects | Cell Proliferation - drug effects | Mice | Neovascularization, Pathologic - metabolism | Leukocytes - metabolism | Skin - drug effects | Index Medicus
Journal Article
Experimental Dermatology, ISSN 0906-6705, 07/2011, Volume 20, Issue 7, pp. 589 - 594
Journal Article