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Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2014, Volume 111, Issue 34, pp. 12426 - 12431
Pluripotency can be induced in somatic cells by overexpressing transcription factors, including POU class 5 homeobox 1 (OCT3/4), sex determining region Y-box 2... 
Phenotypes | Induced pluripotent stem cells | Somatic cells | Stem cells | Retroviridae | Embryonic stem cells | Pluripotent stem cells | Cellular differentiation | Cells | Mesenchymal stem cells | Epigenetics | Retrotransposon | Evolution | NONCODING RNA-ROR | ROADBLOCK | DNA METHYLATION | MULTIDISCIPLINARY SCIENCES | TRANSPOSABLE ELEMENTS | evolution | INDUCTION | epigenetics | IPS CELLS | PLURIPOTENT STEM-CELLS | PATHWAY | HUMAN FIBROBLASTS | retrotransposon | EXPRESSION | Octamer Transcription Factor-3 - physiology | Endogenous Retroviruses - physiology | Embryonic Stem Cells - cytology | Epigenesis, Genetic | Humans | Endogenous Retroviruses - genetics | Pluripotent Stem Cells - virology | Gene Knockdown Techniques | Octamer Transcription Factor-3 - genetics | Cell Differentiation - genetics | RNA, Viral - antagonists & inhibitors | RNA, Viral - genetics | SOXB1 Transcription Factors - genetics | Embryonic Stem Cells - virology | SOXB1 Transcription Factors - physiology | Induced Pluripotent Stem Cells - cytology | Cell Differentiation - physiology | Cellular Reprogramming - genetics | Gene Expression | Pluripotent Stem Cells - cytology | Induced Pluripotent Stem Cells - physiology | Induced Pluripotent Stem Cells - virology | Pluripotent Stem Cells - physiology | Kruppel-Like Transcription Factors - physiology | RNA, Long Noncoding - genetics | Embryonic Stem Cells - physiology | RNA, Long Noncoding - antagonists & inhibitors | Kruppel-Like Transcription Factors - genetics | Cellular Reprogramming - physiology | Epigenetic inheritance | Transcription factors | Retroviruses | Physiological aspects | Genetic research | Genetic aspects | Research | Cell differentiation | Gene expression | Virus research | Biological Sciences
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2013, Volume 123, Issue 12, pp. 5231 - 5246
Epigenetic dysregulation has emerged as a major contributor to tumorigenesis. Histone methylation is a well-established mechanism of epigenetic regulation that... 
MEDICINE, RESEARCH & EXPERIMENTAL | HISTONE LYSINE METHYLATION | REGULATES CELL-PROLIFERATION | EMBRYONIC FIBROBLASTS | PATHWAY | TYROSINE-PHOSPHATASE VHR | ADENOCARCINOMA | DIMETHYLATION | CANCER | DEMETHYLASES | GENOME | Prognosis | Humans | Neoplasm Proteins - physiology | Protein Processing, Post-Translational - genetics | Jumonji Domain-Containing Histone Demethylases - physiology | Lung Neoplasms - pathology | Male | Neoplasm Proteins - antagonists & inhibitors | Dual Specificity Phosphatase 3 - genetics | F-Box Proteins - antagonists & inhibitors | MAP Kinase Signaling System | Epigenesis, Genetic - physiology | RNA, Messenger - biosynthesis | Heterografts | RNA Interference | Cell Division | Jumonji Domain-Containing Histone Demethylases - antagonists & inhibitors | Female | Neoplasm Proteins - genetics | Gene Expression Regulation, Neoplastic - physiology | Gene Expression Regulation, Neoplastic - genetics | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Promoter Regions, Genetic | Dual Specificity Phosphatase 3 - physiology | Carcinoma, Non-Small-Cell Lung - genetics | Neoplasm Invasiveness | Neoplasm Proteins - biosynthesis | RNA, Messenger - genetics | F-Box Proteins - biosynthesis | Jumonji Domain-Containing Histone Demethylases - genetics | Protein Processing, Post-Translational - physiology | Dual Specificity Phosphatase 3 - biosynthesis | Animals | F-Box Proteins - physiology | RNA, Neoplasm - biosynthesis | Jumonji Domain-Containing Histone Demethylases - biosynthesis | Epigenesis, Genetic - genetics | Mice, Nude | Cell Line, Tumor | RNA, Neoplasm - genetics | Mice | Histones - metabolism | Methylation | F-Box Proteins - genetics | Genetic aspects | Research | Lung cancer, Non-small cell | Genetic regulation | Tumors | Cancer | Proteins | Survival analysis | Gene amplification | Lung cancer | Epigenetics | Kinases | Gene expression
Journal Article
Nature reviews. Gastroenterology & hepatology, ISSN 1759-5045, 05/2016, Volume 13, Issue 5, pp. 261 - 280
Journal Article
Molecular cell, ISSN 1097-2765, 2018, Volume 71, Issue 1, pp. 25 - 41.e6
Journal Article
Nature (London), ISSN 1476-4687, 2014, Volume 509, Issue 7501, pp. 447 - 452
Journal Article
Nature reviews. Cancer, ISSN 1474-1768, 2007, Volume 7, Issue 11, pp. 823 - 833
Translocations that involve the mixed lineage leukaemia (MLL) gene identify a unique group of acute leukaemias, and often predict a poor prognosis. The MLL... 
CHROMOSOMAL TRANSLOCATIONS | ONCOLOGY | GENE-EXPRESSION | METHYLTRANSFERASE ACTIVITY | ALL-1 GENE | ACUTE MYELOID-LEUKEMIA | FUSION PARTNERS | HOX EXPRESSION | BREAKPOINT CLUSTER REGION | PARTIAL TANDEM DUPLICATION | HEMATOPOIETIC STEM | Protein Methyltransferases | Neoplastic Stem Cells - cytology | Prognosis | Humans | Myeloid-Lymphoid Leukemia Protein - deficiency | Infant | Myeloid-Lymphoid Leukemia Protein - chemistry | Epigenesis, Genetic - physiology | Oncogene Proteins, Fusion - physiology | Oncogene Proteins, Fusion - chemistry | Myeloid-Lymphoid Leukemia Protein - physiology | Cell Transformation, Neoplastic - genetics | Gene Expression Regulation, Developmental | Adult | Child | Leukemia - genetics | Chromatin - ultrastructure | Histone-Lysine N-Methyltransferase - genetics | Gene Expression Regulation - genetics | Gene Expression Regulation - physiology | Hematopoietic Stem Cells - metabolism | Cell Division - physiology | Animals | Histone Methyltransferases | Epigenesis, Genetic - genetics | Myeloid-Lymphoid Leukemia Protein - genetics | Oncogene Proteins, Fusion - genetics | Hematopoietic Stem Cells - cytology | Mice | Protein Processing, Post-Translational | Histones - metabolism | Methylation | Histone-Lysine N-Methyltransferase - physiology | Leukemia | Stem cells | Histones | Physiological aspects | Genetic aspects | Research | Gene expression
Journal Article
PLoS genetics, ISSN 1553-7390, 06/2008, Volume 4, Issue 6, pp. e1000116 - e1000116
Journal Article