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PLoS ONE, ISSN 1932-6203, 09/2011, Volume 6, Issue 9, p. e25178
Imbalance of the excitatory neurotransmitter glutamate and of the inhibitory neurotransmitter GABA is one of several causes of seizures. ATP has also been... 
HIPPOCAMPAL SLICES | ADENOSINE KINASE | P2X RECEPTOR | HEMICHANNELS | BIOLOGY | ASTROCYTE PROGENITORS | CENTRAL-NERVOUS-SYSTEM | RAT HIPPOCAMPUS | CHANNELS | CELL-CELL COMMUNICATION | ATP RELEASE | Potassium - metabolism | Seizures - prevention & control | Connexins - physiology | Epilepsy - metabolism | Neurons - cytology | Seizures - metabolism | Seizures - chemically induced | Brain - metabolism | Adenosine Triphosphate - metabolism | Behavior, Animal - drug effects | Neurons - metabolism | Astrocytes - cytology | Epilepsy - prevention & control | Brain - cytology | Nerve Tissue Proteins - physiology | Kainic Acid - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Hippocampus - cytology | Blotting, Western | Hippocampus - metabolism | Animals | Epilepsy - chemically induced | Fluorescent Antibody Technique | Mice | Status Epilepticus - metabolism | Astrocytes - metabolism | Brain | Neurons | Epilepsy | GABA | Genetic engineering | Seizures (Medicine) | Glutamate | Neurosciences | Hyperactivity | Nervous system | Membrane channels | Channels | γ-Aminobutyric acid | Membrane conductance | Metabolites | Rodents | Cooperation | Physiology | Seizures | Seizing | Adenosine | Astrocytes | Transgenic mice | Pharmacology | Metabolism | Embryos | Resistance | Medicine | Signaling | Conductance | ATP | Kainic acid | Hippocampus
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 9, p. e44789
Increasing evidence supports the involvement of microRNAs (miRNA) in the regulation of inflammation in human neurological disorders. In the present study we... 
INTERLEUKIN-1 | CORTICAL DYSPLASIA | ACTIVATION | MALFORMATIONS | MULTIDISCIPLINARY SCIENCES | TOLL-LIKE RECEPTOR | DISEASE | EPILEPSY | NF-KAPPA-B | EXPRESSION | RAT MODEL | Brain Diseases - metabolism | Interleukin-1 Receptor-Associated Kinases - metabolism | Interleukin-1beta - pharmacology | Humans | Astrocytes - pathology | Brain Diseases - genetics | Epilepsy - metabolism | Male | MicroRNAs - metabolism | NF-kappa B - metabolism | Brain Diseases - immunology | Ganglioglioma - pathology | Inflammation Mediators - metabolism | Epilepsy - genetics | Female | Toll-Like Receptors - metabolism | Malformations of Cortical Development - immunology | Malformations of Cortical Development - pathology | Interleukin-6 - metabolism | Astrocytes - drug effects | Malformations of Cortical Development, Group I | Immunity, Innate - drug effects | Malformations of Cortical Development - metabolism | Malformations of Cortical Development - genetics | Receptors, Interleukin-1 - metabolism | Gene Expression Regulation - drug effects | Pregnancy | Ganglioglioma - genetics | Brain Diseases - pathology | Signal Transduction - drug effects | Cyclooxygenase 2 - metabolism | TNF Receptor-Associated Factor 6 - metabolism | Cell Line, Tumor | MicroRNAs - genetics | Ganglioglioma - metabolism | Epilepsy - pathology | Astrocytes - metabolism | Medical research | Nervous system diseases | Immune response | MicroRNA | Cytokines | Epilepsy | Medicine, Experimental | Inflammation | Research | Enzyme-linked immunosorbent assay | Neurosciences | Downstream effects | Disorders | Inflammatory response | Nervous system | Kinases | Interleukin 6 | Transfection | Modulation | miRNA | Lesions | Immune system | IRAK protein | Encephalitis | Immunomodulation | Astrocytes | Tumor necrosis factor-α | Gene expression | Ribonucleic acid--RNA | Neurological diseases | Pathology | Signaling | Brain research | MicroRNAs | Cyclooxygenase-2 | RNA | Ribonucleic acid
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2018, Volume 115, Issue 28, pp. E6640 - E6649
Endosomes have emerged as a central hub and pathogenic driver of Alzheimer’s disease (AD). The earliest brain cytopathology in neurodegeneration, occurring... 
Histone deacetylase | Trichostatin A | Amyloid beta | ApoE4 | exchanger | Na+/H+ exchanger | CHRISTIANSON SYNDROME | HUMAN BRAIN | trichostatin A | MULTIDISCIPLINARY SCIENCES | SPORADIC ALZHEIMERS-DISEASE | histone deacetylase | SYNAPTIC PLASTICITY | amyloid beta | APOLIPOPROTEIN-E | LYSOSOMAL DYSFUNCTION | DOWN-SYNDROME | A-BETA | GENE-EXPRESSION | PRECURSOR PROTEIN | Microcephaly - genetics | Epigenesis, Genetic | Humans | Astrocytes - pathology | Epilepsy - metabolism | Endosomes - metabolism | Alzheimer Disease - pathology | Intellectual Disability - metabolism | Amyloid beta-Peptides - genetics | Amyloid beta-Peptides - metabolism | Ataxia - drug therapy | Genetic Diseases, X-Linked - genetics | Ataxia - genetics | Ataxia - metabolism | Microcephaly - drug therapy | Ataxia - pathology | Ocular Motility Disorders - pathology | Tumor Suppressor Proteins - metabolism | Apolipoprotein E4 - metabolism | Endosomes - genetics | Histone Deacetylases - genetics | Ocular Motility Disorders - metabolism | Intellectual Disability - pathology | Alzheimer Disease - drug therapy | Receptors, LDL - metabolism | Mice, Knockout | Genetic Diseases, X-Linked - pathology | Epilepsy - drug therapy | Histone Deacetylase Inhibitors - pharmacology | Mice | Astrocytes - metabolism | Hydrogen-Ion Concentration | Apolipoprotein E4 - genetics | Ocular Motility Disorders - genetics | Intellectual Disability - genetics | Sodium-Hydrogen Exchangers - metabolism | Genetic Diseases, X-Linked - drug therapy | Microcephaly - pathology | Tumor Suppressor Proteins - genetics | Epilepsy - genetics | Sodium-Hydrogen Exchangers - genetics | Receptors, LDL - genetics | Microcephaly - metabolism | Histone Deacetylases - metabolism | Endosomes - pathology | Genetic Diseases, X-Linked - metabolism | Intellectual Disability - drug therapy | Ocular Motility Disorders - drug therapy | Animals | Alzheimer Disease - metabolism | Alzheimer Disease - genetics | Epilepsy - pathology | Physiological aspects | Epigenetic inheritance | Glycoproteins | Genetic aspects | Observations | Astrocytes | Biological Sciences | PNAS Plus | H+ exchanger | Na+
Journal Article
Biochemical journal, ISSN 1470-8728, 2009, Volume 418, Issue 3, pp. 475 - 489
Journal Article
Brain, ISSN 0006-8950, 09/2017, Volume 140, Issue 9, pp. 2460 - 2474
Journal Article
Science (American Association for the Advancement of Science), ISSN 0036-8075, 9/2006, Volume 313, Issue 5794, pp. 1792 - 1795
Journal Article
International journal of molecular sciences, ISSN 1422-0067, 2019, Volume 20, Issue 14, p. 3596
WW domain-containing oxidoreductase (Wwox) is a putative tumor suppressor. Several germline mutations of Wwox have been associated with infant neurological... 
Wwox | SPERMATOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | oligodendrocyte | neuron | DOMAIN-CONTAINING OXIDOREDUCTASE | CANCER | CHEMISTRY, MULTIDISCIPLINARY | cerebral cortex | MYELIN | SEIZURES | GENE | MUTATION | EXON DELETION | BASIC-PROTEIN | EXPRESSION | Glial Fibrillary Acidic Protein - genetics | Cell Count | Astrocytes - pathology | Cerebral Cortex - pathology | Epilepsy - metabolism | Male | Glial Fibrillary Acidic Protein - metabolism | WW Domain-Containing Oxidoreductase - genetics | Myelin Basic Protein - genetics | Neurogenesis - genetics | Dwarfism - metabolism | Tumor Suppressor Proteins - deficiency | Germ-Line Mutation | Neurons - metabolism | Prosencephalon - metabolism | Disease Models, Animal | Adenomatous Polyposis Coli Protein - genetics | WW Domain-Containing Oxidoreductase - deficiency | Myelin Basic Protein - metabolism | Rats, Transgenic | Signal Transduction | Psychomotor Disorders - pathology | Rats | Antiporters - metabolism | Hereditary Central Nervous System Demyelinating Diseases - metabolism | Oligodendroglia - pathology | Hereditary Central Nervous System Demyelinating Diseases - pathology | 2',3'-Cyclic Nucleotide 3'-Phosphodiesterase - metabolism | Adenomatous Polyposis Coli Protein - metabolism | Cerebral Cortex - growth & development | Astrocytes - metabolism | Mitochondrial Diseases - pathology | Neurons - pathology | Oligodendroglia - metabolism | Amino Acid Transport Systems, Acidic - metabolism | Mitochondrial Diseases - metabolism | Amino Acid Transport Systems, Acidic - deficiency | Cerebral Cortex - metabolism | Psychomotor Disorders - metabolism | Psychomotor Disorders - genetics | 2',3'-Cyclic Nucleotide 3'-Phosphodiesterase - genetics | Gene Expression Regulation, Developmental | Tumor Suppressor Proteins - genetics | Antiporters - genetics | Epilepsy - genetics | Dwarfism - pathology | Hereditary Central Nervous System Demyelinating Diseases - genetics | Prosencephalon - growth & development | Prosencephalon - pathology | Antiporters - deficiency | Mitochondrial Diseases - genetics | Dwarfism - genetics | Animals | Amino Acid Transport Systems, Acidic - genetics | Epilepsy - pathology
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 9, p. e74458
Status epilepticus (SE) induces vasogenic edema in the piriform cortex with disruptions of the blood-brain barrier (BBB). However, the mechanisms of vasogenic... 
NADPH-OXIDASE | PERMEABILITY | MULTIDISCIPLINARY SCIENCES | SEIZURE | RAT PIRIFORM CORTEX | NITRIC-OXIDE | RECEPTOR | KAINIC ACID | ENDOTHELIN | BLOOD-BRAIN-BARRIER | FACTOR-ALPHA | Tumor Necrosis Factor-alpha - metabolism | Neurons - pathology | Reactive Oxygen Species - metabolism | Tumor Necrosis Factor-alpha - genetics | Astrocytes - pathology | Cerebral Cortex - pathology | NADPH Oxidases - metabolism | Male | Status Epilepticus - genetics | Cerebral Cortex - metabolism | Brain Edema - genetics | NADPH Oxidases - genetics | Status Epilepticus - complications | Neurons - metabolism | Nitric Oxide Synthase Type III - metabolism | Disease Models, Animal | Brain Edema - metabolism | Brain Edema - etiology | Signal Transduction | Endothelial Cells - metabolism | Endothelin-1 - genetics | Endothelin-1 - metabolism | Brain Edema - pathology | Gene Expression Regulation | Status Epilepticus - pathology | Rats | Receptor, Endothelin A - genetics | Nitric Oxide Synthase Type III - genetics | Rats, Sprague-Dawley | Blood-Brain Barrier - metabolism | Stereotaxic Techniques | Blood-Brain Barrier - pathology | Animals | Microdialysis | Receptor, Endothelin A - metabolism | Endothelial Cells - pathology | Status Epilepticus - metabolism | Astrocytes - metabolism | Reactive oxygen species | Neurosciences | Phosphorylation | Laboratories | Epilepsy | Neurobiology | Oxidase | Membrane permeability | Activation | Cortex (piriform) | NAD(P)H oxidase | Blood-brain barrier | Rodents | Surgery | Tumor necrosis factor-TNF | Edema | Stroke | Oxygen | Astrocytes | Mortality | Endothelin 1 | Permeability | Tumor necrosis factor-α | Nitric-oxide synthase | Endothelial cells | Neurological diseases | Medicine | Brain research | Nitric oxide | TNF inhibitors | Receptor mechanisms
Journal Article