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Journal of Neuroscience Methods, ISSN 0165-0270, 07/2008, Volume 172, Issue 2, pp. 143 - 157
Understanding the pathophysiogenesis of temporal lobe epilepsy (TLE) largely rests on the use of models of (SE), as in the case of the pilocarpine model. The... 
Temporal lobe epilepsy | Animal models | Pilocarpine | Entorhinal cortex | Hippocampus | animal models | temporal lobe epilepsy | CONVULSIVE STATUS EPILEPTICUS | BIOCHEMICAL RESEARCH METHODS | entorhinal cortex | NEURONAL LOSS | GUINEA-PIG BRAIN | NEUROSCIENCES | IN-VITRO | hippocampus pilocarpine | MEDIAL ENTORHINAL CORTEX | INDUCED STATUS EPILEPTICUS | IMMATURE RATS | SPONTANEOUS RECURRENT SEIZURES | ANTIEPILEPTIC DRUG LEVETIRACETAM | LITHIUM-PILOCARPINE | Species Specificity | Epilepsy, Temporal Lobe - chemically induced | Humans | Nerve Degeneration - physiopathology | Status Epilepticus - chemically induced | Status Epilepticus - pathology | Epilepsy, Temporal Lobe - physiopathology | Hippocampus - pathology | Hippocampus - drug effects | Nerve Degeneration - chemically induced | Nerve Degeneration - pathology | Dose-Response Relationship, Drug | Animals | Time Factors | Pilocarpine - pharmacology | Status Epilepticus - physiopathology | Convulsants - pharmacology | Epilepsy, Temporal Lobe - pathology | Hippocampus - physiopathology | Disease Models, Animal | Anticonvulsants | Index Medicus | AEDs, antiepileptic drugs | P, postnatal | EEG, electroencephalogram | TLE, temporal lobe epilepsy | SE, status epilepticus | Invited Review | MRI, magnetic resonance imaging | SRSs, spontaneous recurrent seizures | i.p., intraperitoneal | s.c., subcutaneous | CA, Cornu Ammonis
Journal Article
1987, Clinics in developmental medicine., ISBN 0632017589, Volume no.103, xii, 129
Book
Epilepsy Research, ISSN 0920-1211, 2009, Volume 88, Issue 1, pp. 23 - 45
Summary Mitochondrial oxidative stress and dysfunction are contributing factors to various neurological disorders. Recently, there has been increasing evidence... 
Neurology | Epileptogenesis | Seizure | Oxidative damage | Hyperexcitability | BASE-EXCISION-REPAIR | ACID-INDUCED SEIZURES | CA2+-ACTIVATED K+ CHANNELS | HUMAN EPILEPTOGENIC HIPPOCAMPUS | CLINICAL NEUROLOGY | RAT HIPPOCAMPAL-NEURONS | MANGANESE SUPEROXIDE-DISMUTASE | INDUCED STATUS EPILEPTICUS | GLUTAMINE-SYNTHETASE ACTIVITY | OXYGEN-FREE-RADICALS | METHYL-D-ASPARTATE | DNA, Mitochondrial - metabolism | Humans | Lipids - physiology | Oxidative Stress - physiology | Epilepsy, Temporal Lobe - drug therapy | Epilepsy, Temporal Lobe - physiopathology | Ion Channels - drug effects | Ion Channels - physiology | Antioxidants - pharmacology | Mitochondria - drug effects | Electron Transport Complex I - metabolism | Antioxidants - therapeutic use | Animals | Neurons - ultrastructure | Models, Biological | Neurons - physiology | Oxidation-Reduction - drug effects | Glutamic Acid - metabolism | Mitochondria - physiology | Neurons - drug effects | Epilepsy, Temporal Lobe - pathology | Oxidative stress | Coenzymes | Hydrogen peroxide | Superoxide dismutase | Mitochondrial DNA | Superoxide | Permeability | Glutamate | Fatty acids | Antioxidants | Methyl aspartate | Ligases | Radicals (Chemistry) | Nitric oxide | DNA polymerases | Physiological aspects | Temporal lobe epilepsy | Peroxidase | Seizures (Medicine) | Adenosine triphosphate | Glutathione | Glutamine | Porphyrins
Journal Article