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PLoS ONE, ISSN 1932-6203, 07/2017, Volume 12, Issue 7, pp. e0180834 - e0180834
Patients with Ulcerative Colitis (UC) have an increased risk to develop colitis-associated colorectal cancer (CAC). Here, we found that protein expression of... 
MULTIDRUG-RESISTANT CELLS | COLORECTAL-CANCER RISK | COLON-CANCER | INTESTINAL INFLAMMATION | INFLAMMATORY-BOWEL-DISEASE | NEOPLASTIC PROGRESSION | MULTIDISCIPLINARY SCIENCES | ULCERATIVE-COLITIS | HUMAN B-CELLS | P-GLYCOPROTEIN EXPRESSION | PLATELET-ACTIVATING-FACTOR | Epiregulin - immunology | ATP Binding Cassette Transporter, Sub-Family B - deficiency | Colorectal Neoplasms - genetics | Humans | Colitis, Ulcerative - genetics | Gene Expression Regulation, Neoplastic | Male | Monocyte Chemoattractant Proteins - immunology | Colitis, Ulcerative - immunology | DNA-Binding Proteins - deficiency | Leukocyte Common Antigens - immunology | Cyclooxygenase 2 - genetics | Intestinal Mucosa - immunology | ATP Binding Cassette Transporter, Sub-Family B - immunology | B-Lymphocytes - pathology | Cyclooxygenase 2 - immunology | Disease Models, Animal | Colitis, Ulcerative - complications | DNA-Binding Proteins - immunology | Signal Transduction | Carcinogenesis - immunology | Genes, Immunoglobulin Light Chain - genetics | Carcinogenesis - genetics | Interleukin-11 - genetics | Colitis, Ulcerative - pathology | TNF-Related Apoptosis-Inducing Ligand - immunology | DNA-Binding Proteins - genetics | Epiregulin - genetics | Chemotaxis | Mice, Knockout | Carcinogenesis - pathology | Monocyte Chemoattractant Proteins - genetics | Animals | B-Lymphocytes - immunology | Colorectal Neoplasms - immunology | Leukocyte Common Antigens - genetics | Colorectal Neoplasms - complications | Interleukin-11 - immunology | Mice | Colorectal Neoplasms - pathology | ATP Binding Cassette Transporter, Sub-Family B - genetics | TNF-Related Apoptosis-Inducing Ligand - genetics | Intestinal Mucosa - pathology | Patient outcomes | Colorectal cancer | Development and progression | Research | B cells | Gene expression | Health aspects | Risk factors | Ulcerative colitis | Protein binding | Cell culture | Mucosa | Proteins | Tumorigenesis | Light levels | Damage | Interleukin 11 | Glycoproteins | Patients | Inflammatory bowel disease | Injury prevention | Hospitals | B220 antigen | DNA microarrays | Cell injury | Ligands | Animal models | Inflammatory bowel diseases | Colorectal carcinoma | Genes | Inflammatory response | Carcinogenesis | Medical schools | Carcinogens | Colon cancer | Lymphocytes | Intestine | Rodents | Hepatology | Gastroenterology | Colon | Dysplasia | Immune response | CD19 antigen | Multidrug resistance | Health risks | Inflammation | Spheroids | Lymphocytes B | Cell death | Cyclooxygenase-2 | ATP | Chemokines | Tumors | Cancer | Apoptosis | Index Medicus
Journal Article
Laboratory Investigation, ISSN 0023-6837, 09/2010, Volume 90, Issue 9, pp. 1295 - 1305
Epiregulin (EPI) and amphiregulin (AR) are epidermal growth factor receptor (EGFR) ligands implicated in mucosal repair and tumorigenesis. We have shown that... 
MEDICINE, RESEARCH & EXPERIMENTAL | FACTOR FAMILY | COLORECTAL TUMORS | intestine | EPIREGULIN | CANCER CELLS | epidermal growth factor receptor | mucosal repair | PATHOLOGY | intestinal bacteria | INFLAMMATORY-BOWEL-DISEASE | EPITHELIAL-CELLS | INCREASED SUSCEPTIBILITY | SULFATE-INDUCED COLITIS | IMMUNE-RESPONSES | NF-KAPPA-B | Toll-like receptor | Receptor, Epidermal Growth Factor - genetics | Epithelial Cells - metabolism | Colitis - genetics | Humans | Lipopolysaccharides - metabolism | Epiregulin | Amphiregulin | Lipopolysaccharides - immunology | Receptor, Epidermal Growth Factor - metabolism | EGF Family of Proteins | Dextran Sulfate - metabolism | Epidermal Growth Factor - immunology | Colitis - immunology | Antibodies, Neutralizing | Dextran Sulfate - pharmacology | Cell Line | Receptor, Epidermal Growth Factor - immunology | Enzyme-Linked Immunosorbent Assay | Epidermal Growth Factor - genetics | Intercellular Signaling Peptides and Proteins | Toll-Like Receptor 4 - genetics | Epidermal Growth Factor - metabolism | Glycoproteins | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Mucous Membrane - metabolism | Up-Regulation - drug effects | Animals | Dextran Sulfate - immunology | Epithelial Cells - immunology | Colitis - metabolism | Cell Proliferation - drug effects | Mice
Journal Article
Journal of Immunology, ISSN 0022-1767, 02/2015, Volume 194, Issue 3, pp. 1039 - 1046
Journal Article
STEM CELLS, ISSN 1066-5099, 12/2012, Volume 30, Issue 12, pp. 2631 - 2644
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Experimental Cell Research, ISSN 0014-4827, 2005, Volume 306, Issue 1, pp. 216 - 229
Regeneration of the urothelium is rapid and effective in order to maintain a barrier to urine following tissue injury. Whereas normal human urothelial (NHU)... 
Regeneration | Wound healing | Migration | Bladder | Proliferation | Amphiregulin | Epidermal growth factor receptor | Urothelium | wound healing | ACTIVATION | proliferation | regeneration | URINARY-BLADDER | EGF | ERBB RECEPTOR FAMILY | MEMBRANE-PROTEIN | KERATINOCYTES | urothelium | CELL BIOLOGY | amphiregulin | epidennal growth factor receptor | ONCOLOGY | bladder | migration | NEU DIFFERENTIATION FACTOR | GROWTH-FACTOR | EXPRESSION | CARCINOMA | Cell Cycle - genetics | Receptor, Epidermal Growth Factor - genetics | MAP Kinase Signaling System - physiology | Urothelium - cytology | Cell Proliferation | Gene Expression - genetics | Humans | Growth Substances - deficiency | Epiregulin | Glycoproteins - pharmacology | Cell Movement - physiology | EGF Family of Proteins | Epidermal Growth Factor - immunology | Receptor, Epidermal Growth Factor - physiology | Wound Healing - drug effects | Transforming Growth Factor alpha - pharmacology | Glycoproteins - genetics | Epidermal Growth Factor - genetics | Autocrine Communication - physiology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Intercellular Signaling Peptides and Proteins - genetics | Transforming Growth Factor alpha - genetics | Antibodies - pharmacology | Transforming Growth Factor alpha - immunology | Cell Movement - drug effects | Heparin-binding EGF-like Growth Factor | Urothelium - physiology | Glycoproteins - immunology | Regeneration - drug effects | Intercellular Signaling Peptides and Proteins - pharmacology | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Wound Healing - physiology | Epidermal Growth Factor - pharmacology | Quinazolines - pharmacology | Intercellular Signaling Peptides and Proteins - immunology | Transforming growth factors | Analysis | Epidermal growth factor | ANTIBODIES | ELECTRON SPIN RESONANCE | CATTLE | PHENOTYPE | CELL PROLIFERATION | HEALING | HEPARIN | 60 APPLIED LIFE SCIENCES | INOSITOL | GROWTH FACTORS | LIGANDS | IN VITRO | WOUNDS | URINE | BIOLOGICAL REPAIR | CELL CYCLE | BLADDER | GENE REGULATION | RECEPTORS
Journal Article
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 02/2014, Volume 348, Issue 2, pp. 236 - 245
Rheumatoid arthritis (RA) is a chronic autoimmune disease with high morbidity and mortality. Within the inflammatory milieu, resident fibroblast-like... 
OVEREXPRESSION | CIGARETTE-SMOKING | MMP EXPRESSION | VEGF | DIOXIN RECEPTOR | PHARMACOLOGY & PHARMACY | LIGAND | METALLOPROTEINASES | INHIBITORY FACTOR | T-CELLS | 2,3,7,8-TETRACHLORODIBENZO-P-DIOXIN | Receptors, Aryl Hydrocarbon - antagonists & inhibitors | Humans | Synovial Membrane - pathology | Vascular Endothelial Growth Factor A - metabolism | Epiregulin | Interleukin-1beta - genetics | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Promoter Regions, Genetic - drug effects | Vascular Endothelial Growth Factor A - genetics | Vascular Endothelial Growth Factor A - secretion | Arthritis, Rheumatoid - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Arthritis, Rheumatoid - drug therapy | Interleukin-1beta - metabolism | Gelatinases - metabolism | Interleukin-1beta - antagonists & inhibitors | Glycoproteins - genetics | Basic Helix-Loop-Helix Transcription Factors - genetics | Gelatinases - antagonists & inhibitors | Receptors, Aryl Hydrocarbon - genetics | Recombinant Proteins - chemistry | Cell Movement - drug effects | Fibroblast Growth Factor 2 - antagonists & inhibitors | Fibroblast Growth Factor 2 - genetics | Epidermal Growth Factor - secretion | Glycoproteins - metabolism | Synovial Membrane - immunology | Synovial Membrane - drug effects | Amphiregulin | Molecular Targeted Therapy | Intercellular Signaling Peptides and Proteins - metabolism | EGF Family of Proteins | Fibroblast Growth Factor 2 - metabolism | Receptors, Aryl Hydrocarbon - metabolism | Indoles - pharmacology | Antirheumatic Agents - pharmacology | Recombinant Proteins - metabolism | Cytokines - metabolism | Epidermal Growth Factor - genetics | Purines - pharmacology | Cells, Cultured | Gene Silencing | Glycoproteins - antagonists & inhibitors | Intercellular Signaling Peptides and Proteins - genetics | Epidermal Growth Factor - metabolism | Basic Helix-Loop-Helix Transcription Factors - antagonists & inhibitors | Down-Regulation - drug effects | Arthritis, Rheumatoid - pathology | Response Elements - drug effects | Synovial Membrane - secretion | Gelatinases - genetics | Epidermal Growth Factor - antagonists & inhibitors | Cell Proliferation - drug effects | Cytokines - antagonists & inhibitors | Arthritis, Rheumatoid - immunology | Cellular and Molecular
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