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Cell Stem Cell, ISSN 1934-5909, 09/2012, Volume 11, Issue 3, pp. 401 - 414
The integrity of the epidermis and mucosal epithelia is highly dependent on resident self-renewing stem cells, which makes them vulnerable to physical and... 
MAMMALIAN TARGET | CANCER PATIENTS | RAPAMYCIN | HEAD | AGING PHENOTYPES | MANGANESE SUPEROXIDE-DISMUTASE | STRESS-RESPONSE | TUMOR | NECK | SKIN | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Cell Death - radiation effects | TOR Serine-Threonine Kinases - metabolism | Keratinocytes - radiation effects | Carcinoma, Squamous Cell - pathology | Epithelial Cells - drug effects | Humans | Cellular Senescence - drug effects | Cell Compartmentation - radiation effects | Mucositis - prevention & control | Mouth Mucosa - drug effects | TOR Serine-Threonine Kinases - antagonists & inhibitors | Mouth Mucosa - radiation effects | Mucositis - pathology | Stem Cells - enzymology | Mucositis - enzymology | Cellular Senescence - radiation effects | Cytoprotection - drug effects | Clone Cells | Oxidative Stress - radiation effects | Cell Death - drug effects | Head and Neck Neoplasms - enzymology | Mouth Mucosa - pathology | Keratinocytes - enzymology | Superoxide Dismutase - metabolism | Radiation, Ionizing | Carcinoma, Squamous Cell - enzymology | Epithelial Cells - radiation effects | Cells, Cultured | Stem Cells - radiation effects | Epithelial Cells - pathology | Radiation Injuries - enzymology | Sirolimus - pharmacology | Head and Neck Neoplasms - pathology | Keratinocytes - pathology | Animals | Keratinocytes - drug effects | Radiation Injuries - prevention & control | Epithelial Cells - enzymology | Cell Compartmentation - drug effects | Stem Cells - drug effects | Stem Cells - pathology | Radiation Injuries - pathology | Cell Proliferation - drug effects | Mice | Oxidative Stress - drug effects | Cytoprotection - radiation effects | Cell Proliferation - radiation effects
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2007, Volume 104, Issue 2, pp. 618 - 623
Journal Article
Journal of Immunology, ISSN 0022-1767, 07/2015, Volume 195, Issue 2, pp. 661 - 671
CD47, a self recognition marker expressed on tissue cells, interacts with immunoreceptor SIRP alpha expressed on the surface of macrophages to initiate... 
SIGNAL | IN-VITRO | PHAGOCYTOSIS | INHIBITION | CALRETICULIN | THERAPEUTIC TARGET | REGULATORY PROTEIN-ALPHA | RED-BLOOD-CELLS | NEUTROPHIL TRANSMIGRATION | IMMUNOLOGY | EXPRESSION | Spleen - immunology | Apoptosis - drug effects | Apoptosis - radiation effects | Membrane Microdomains - radiation effects | Epithelial Cells - drug effects | Humans | Protein Transport - drug effects | Spleen - drug effects | beta-Cyclodextrins - pharmacology | CD47 Antigen - immunology | Phagocytosis - radiation effects | Ultraviolet Rays | Leukocytes, Mononuclear - immunology | Leukocytes, Mononuclear - radiation effects | Macrophages - radiation effects | Receptors, Immunologic - immunology | Epithelial Cells - cytology | Binding Sites | Macrophages - immunology | CD47 Antigen - genetics | Epithelial Cells - radiation effects | Phagocytosis - drug effects | Leukocytes, Mononuclear - drug effects | Signal Transduction | Mice, Inbred C57BL | CD47 Antigen - chemistry | Gene Expression Regulation | Spleen - cytology | Macrophages - cytology | Animals | Fibroblasts - radiation effects | Membrane Microdomains - drug effects | Protein Transport - radiation effects | Epithelial Cells - immunology | Fibroblasts - drug effects | Cell Line, Tumor | Protein Binding | Fibroblasts - immunology | Leukocytes, Mononuclear - cytology | Macrophages - drug effects | Spleen - radiation effects | Antigens, Differentiation - genetics | Fibroblasts - cytology | Mice | Primary Cell Culture | Receptors, Immunologic - genetics | Antigens, Differentiation - immunology
Journal Article
Cancer Research, ISSN 0008-5472, 12/2011, Volume 71, Issue 23, pp. 7155 - 7167
Glioblastoma multiforme (GBM) is a highly aggressive primary brain tumor that tends to be resistant to the ionizing radiotherapy used to treat it. Because... 
EPITHELIAL-MESENCHYMAL TRANSITION | GLIOMA-CELLS | INITIATING CELLS | VITRO | APOPTOSIS | GROWTH-FACTOR-BETA | STEM-CELLS | ONCOLOGY | PATHWAY | CANCER STEM | EXPRESSION | Apoptosis - drug effects | Apoptosis - radiation effects | Neoplastic Stem Cells - drug effects | Humans | Brain Neoplasms - pathology | Smad2 Protein - antagonists & inhibitors | Glioblastoma - radiotherapy | Brain Neoplasms - metabolism | Neoplastic Stem Cells - metabolism | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Neoplastic Stem Cells - pathology | Transforming Growth Factor beta - antagonists & inhibitors | Glioblastoma - metabolism | Signal Transduction - radiation effects | Brain Neoplasms - radiotherapy | Protein-Serine-Threonine Kinases - metabolism | Neoplastic Stem Cells - radiation effects | Pyrazoles - pharmacology | Microarray Analysis - methods | Mesenchymal Stromal Cells - drug effects | Neoplasm Invasiveness | Radiation-Sensitizing Agents - pharmacology | Smad2 Protein - metabolism | Brain Neoplasms - drug therapy | Mice, SCID | Radiation Tolerance - drug effects | Xenograft Model Antitumor Assays | Pyrroles - pharmacology | Animals | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Neovascularization, Pathologic - drug therapy | DNA Repair | Glioblastoma - pathology | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | Protein Kinase Inhibitors - pharmacology | DNA Damage | Glioblastoma - drug therapy | Transforming Growth Factor beta - metabolism | Cell Proliferation - radiation effects
Journal Article
Nature Medicine, ISSN 1078-8956, 02/2008, Volume 14, Issue 2, pp. 199 - 204
Rhinoviruses cause serious morbidity and mortality as the major etiological agents of asthma exacerbations and the common cold. A major obstacle to... 
MEDICINE, RESEARCH & EXPERIMENTAL | BRONCHIAL EPITHELIAL-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | ICAM-1 | EFFICIENT REPLICATION | CHILDREN | CELL BIOLOGY | POTENT ANTIVIRAL ACTIVITY | IN-VIVO | INFECTION | EXPRESSION | SEVERITY | VIRUS-INDUCED ASTHMA | Respiratory System - pathology | Bronchial Hyperreactivity - immunology | Dendritic Cells - immunology | Humans | Th2 Cells - radiation effects | Hypersensitivity - immunology | Th2 Cells - immunology | Rhinovirus - physiology | Th1 Cells - immunology | Picornaviridae Infections - virology | Dendritic Cells - radiation effects | Respiratory System - virology | Virus Inactivation - radiation effects | Chemotactic Factors - biosynthesis | Respiratory System - radiation effects | Rhinovirus - radiation effects | Ultraviolet Rays | Neutrophils - radiation effects | Th1 Cells - radiation effects | Mucus - metabolism | Respiratory System - immunology | Chemokines - immunology | Disease Models, Animal | Inflammation Mediators - immunology | Chemokines - biosynthesis | Antibody Formation - radiation effects | Intercellular Adhesion Molecule-1 - immunology | Neutrophils - immunology | Mice, Transgenic | Inflammation | Hypersensitivity - virology | Bronchial Hyperreactivity - virology | Animals | Immunity, Innate - radiation effects | Virus Replication - radiation effects | Mice | Usage | Care and treatment | Rhinoviruses | Models | Research | Health aspects | Risk factors | Asthma | Pathology | Medical research | Cellular biology | Rodents | Viruses | Colds | Allergies
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 12/2017, Volume 21, Issue 12, pp. 3264 - 3276
Radiation‐induced lung injury (RILI) is one of the most common and fatal complications of thoracic radiotherapy. It is characterized with two main features... 
radiation‐induced lung injury | free radicals | epithelial–mesenchymal transition (EMT) | polydatin (PD) | radiation-induced lung injury | MEDICINE, RESEARCH & EXPERIMENTAL | APOPTOSIS | SMAD3 | INDUCED FIBROSIS | CANCER CELLS | IRRADIATED MICE | DAMAGE | IMPAIRMENT | CELL BIOLOGY | epithelial-mesenchymal transition (EMT) | TH2-LIKE IMMUNE-RESPONSE | GROWTH | EXPRESSION | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - genetics | Epithelial Cells - metabolism | Epithelial-Mesenchymal Transition - radiation effects | Radiation Pneumonitis - pathology | Epithelial Cells - drug effects | Humans | Acute Lung Injury - genetics | Epithelial-Mesenchymal Transition - drug effects | Smad3 Protein - immunology | Stilbenes - pharmacology | Radiation Pneumonitis - prevention & control | NF-E2-Related Factor 2 - immunology | Smad3 Protein - genetics | Transforming Growth Factor beta1 - immunology | Acute Lung Injury - prevention & control | Radiation-Protective Agents - pharmacology | Female | Lung - radiation effects | NF-E2-Related Factor 2 - genetics | Lung - metabolism | Th1-Th2 Balance - drug effects | Disease Models, Animal | Cell Line | Epithelial Cells - radiation effects | Glucosides - pharmacology | Lung - pathology | Sirtuin 3 - genetics | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Epithelial Cells - pathology | Transforming Growth Factor beta1 - genetics | Acute Lung Injury - pathology | Radiation Pneumonitis - immunology | Sirtuin 3 - immunology | Animals | Th1-Th2 Balance - radiation effects | Lung - drug effects | Radiation Pneumonitis - genetics | Acute Lung Injury - immunology | Mice | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - immunology | Transforming growth factors | Lung diseases | Stem cells | Radiation | Cell culture | Radiation effects | Mesenchyme | Complications | Lymphocytes T | Smad3 protein | Thorax | Radiation therapy | Tissues | Signaling | Signal transduction | Pneumonitis | Beta irradiation | Lungs | Fibrosis | Radiation injuries | Irradiation | Inhibition | Injuries | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2012, Volume 7, Issue 8, p. e42224
Ionizing radiation-induced oxidative stress is attributed to generation of reactive oxygen species (ROS) due to radiolysis of water molecules and is short... 
NADPH-OXIDASE | INFLAMMATORY-BOWEL-DISEASE | BIOLOGICAL EFFECTIVENESS | INDUCED GENOMIC INSTABILITY | EPITHELIAL-CELLS | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | ANTIOXIDANT ENZYMES | MITOCHONDRIAL DYSFUNCTION | IONIZING-RADIATION | REPLICATIVE SENESCENCE | Epithelial Cells - radiation effects | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Gamma Rays - adverse effects | Intestines - radiation effects | Mice, Inbred C57BL | Cells, Cultured | NADPH Oxidases - metabolism | Animals | Cosmic Radiation - adverse effects | Female | Mice | Oxidative Stress - radiation effects | DNA Damage - radiation effects | Heavy Ions - adverse effects | Intestines - cytology | Gamma rays | Oxidative stress | Reactive oxygen species | Physiological aspects | Causes of | Development and progression | Research | Immunohistochemistry | Flow cytometry | Transformation | Senescence | Laboratories | Epithelial cells | DNA damage | Oxidase | Irradiated | Biology | Biochemistry | NAD(P)H oxidase | Cosmic rays | Antioxidants | Iron isotopes | Mitochondria | Ionizing radiation | Linear energy transfer (LET) | Intestine | Rodents | Animal tissues | Radiolysis | Tumorigenesis | Membrane potential | Heavy ion radiation | Deoxyribonucleic acid--DNA | Enzymes | Radiation effects | Damage assessment | Water chemistry | γ Radiation | Superoxide | Exposure | Radiation therapy | Gene expression | Radiation dosage | Studies | Cytometry | Cell death | Intracellular | Energy transfer | Radiation damage | Cancer | Deoxyribonucleic acid | DNA
Journal Article
Molecular Cancer, ISSN 1476-4598, 01/2017, Volume 16, Issue 1, p. 10
Journal Article