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1. Full Text Induction of KLF4 in basal keratinocytes blocks the proliferation-differentiation switch and initiates squamous epithelial dysplasia
by Foster, K. Wade and Liu, Zhaoli and Nail, Clinton D and Li, Xingnan and Fitzgerald, Thomas J and Bailey, Sarah K and Frost, Andra R and Louro, Iuri D and Townes, Tim M and Paterson, Andrew J and Kudlow, Jeffrey E and Lobo-Ruppert, Susan M and Ruppert, J. Michael
Oncogene, ISSN 0950-9232, 02/2005, Volume 24, Issue 9, pp. 1491 - 1500
KLF4/GKLF normally functions in differentiating epithelial cells, but also acts as a transforming oncogene in vitro. To examine the role of this zinc finger... 
Dysplasia | Squamous cell carcinoma | KLF4 | MMTV | Doxycycline | BIOCHEMISTRY & MOLECULAR BIOLOGY | C-MYC | doxycycline | NECK-CANCER | CELL BIOLOGY | dysplasia | BREAST-CANCER | ONCOLOGY | COLORECTAL-CANCER | MOUSE MODEL | GENETICS & HEREDITY | GENE-EXPRESSION | KRUPPEL-LIKE FACTOR | SKIN | MALIGNANT PROGRESSION | squamous cell carcinoma | TRANSGENIC MICE | Apoptosis - drug effects | Humans | Mice, Inbred C57BL | Epithelial Cells - pathology | Male | Transcription Factors - biosynthesis | Keratinocytes - cytology | DNA Primers | Cell Division - drug effects | Kruppel-Like Transcription Factors | Keratinocytes - pathology | Animals | Transfection | Keratinocytes - drug effects | Polymerase Chain Reaction | Mice | Cell Differentiation - physiology | DNA-Binding Proteins - biosynthesis | Doxorubicin - pharmacology | Skin - pathology | Crosses, Genetic | Index Medicus
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2. Full Text Synergy between Bacterial Infection and Genetic Predisposition in Intestinal Dysplasia
by Yiorgos Apidianakis and Chrysoula Pitsouli and Norbert Perrimon and Laurence Rahme and Frederick M. Ausubel
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2009, Volume 106, Issue 49, pp. 20883 - 20888
Accumulating evidence suggests that hyperproliferating intestinal stem cells (SCs) and progenitors drive cancer initiation, maintenance, and metastasis. In... 
Intestines | Enterocytes | Midgut | Drosophila | Stem cells | Infections | Progenitor cells | Cellular differentiation | Enteroendocrine cells | Apoptosis | Tumor | Cell polarity | Cytokines | Virulence factors | Cancer | HUMAN COLON | STEM-CELLS | HOMEOSTASIS | JNK | MULTIDISCIPLINARY SCIENCES | NOTCH | virulence factors | DROSOPHILA MIDGUT | tumor | DISEASE | cancer | cytokines | cell polarity | TOLL | Cell Proliferation | Cell Count | Hyperplasia | Homeostasis | JNK Mitogen-Activated Protein Kinases - metabolism | Pseudomonas aeruginosa - physiology | Drosophila Proteins - metabolism | Gastrointestinal Tract - pathology | Cell Division | Stem Cells - microbiology | Cell Differentiation | Enterocytes - pathology | Genetic Predisposition to Disease | Intestinal Neoplasms - microbiology | Intestines - pathology | Gastrointestinal Tract - microbiology | Pseudomonas Infections - complications | Enterocytes - microbiology | Intestinal Neoplasms - pathology | Drosophila melanogaster - microbiology | Animals | Intestines - microbiology | Pseudomonas Infections - pathology | Drosophila melanogaster - enzymology | Stem Cells - pathology | Enzyme Activation | Genes, ras | Pseudomonas Infections - enzymology | Evaluation | Dysplasia | Development and progression | Genetic aspects | Polarity (Biology) | Virulence (Microbiology) | Research | Properties | Bacteria | Genetics | Kinases | Cell proliferation | Pathogens | Epithelial cells | Chronic infection | Opportunist infection | Inflammation | Synergism | Metastases | c-Jun amino-terminal kinase | Intestine | Polarity | Tumorigenesis | Digestive tract | Oncogenes | Index Medicus | Biological Sciences
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3. Full Text Stepwise radical endoscopic resection versus radiofrequency ablation for Barrett's oesophagus with high-grade dysplasia or early cancer: a multicentre randomised trial
by van Vilsteren, Frederike G I and Pouw, Roos E and Seewald, Stefan and Alvarez Herrero, Lorenza and Sondermeijer, Carine M T and Visser, Mike and ten Kate, Fiebo J W and Yu Kim Teng, Karl C and Soehendra, Nib and Rösch, Thomas and Weusten, Bas L A M and Bergman, Jacques J G H M
Gut, ISSN 0017-5749, 06/2011, Volume 60, Issue 6, pp. 765 - 773
ObjectiveAfter focal endoscopic resection (ER) of high-grade dysplasia (HGD) or early cancer (EC) in Barrett's oesophagus (BO), eradication of all remaining BO... 
SYSTEM | GASTROINTESTINAL EPITHELIAL NEOPLASIA | FOCAL ABLATION | TERM-FOLLOW-UP | MUCOSAL RESECTION | COMPLETE REMOVAL | ERADICATION | INTRAEPITHELIAL NEOPLASIA | GASTROENTEROLOGY & HEPATOLOGY | INTRAMUCOSAL CARCINOMA | EARLY ADENOCARCINOMA | Acute Disease | Prospective Studies | Follow-Up Studies | Humans | Middle Aged | Esophagoscopy - adverse effects | Male | Treatment Outcome | Remission Induction | Barrett Esophagus - surgery | Esophageal Neoplasms - pathology | Catheter Ablation - methods | Precancerous Conditions - surgery | Barrett Esophagus - pathology | Esophageal Neoplasms - surgery | Aged, 80 and over | Female | Precancerous Conditions - pathology | Aged | Esophagoscopy - methods | Catheter Ablation - adverse effects | Esophageal Stenosis - etiology | Neoplasm Staging | Endoscopic surgery | Care and treatment | Patient outcomes | Barrett's esophagus | Radiofrequency ablation | Research | Endoscopy | Comparative analysis | Studies | Lymphatic system | Clinical medicine | Cancer | Index Medicus | Abridged Index Medicus
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4. Full Text Epithelial dysplasia in pterygium postoperative granuloma
by Wang, Guoliang and Wang, Yanzi and Dong, Fei and Dong, Nuo and Li, Cheng and Li, Jing and Jeyalatha, Vimalin and Wu, Jingwen and Shen, Mei and Yang, Qichen and Chen, Pei and Xue, Yuhua and Liu, Zuguo
Experimental Eye Research, ISSN 0014-4835, 10/2018, Volume 175, pp. 199 - 206
Pterygium postoperative granuloma (PPG) is one of the common complications of pterygium surgery. In order to provide the structural features of PPG, and to... 
Cytokeratin | Epithelial cell | Pterygium postoperative granuloma | Collagen | Epithelial mesenchymal transition (EMT) | SURGERY | CELLS | Collagen IV | COLLAGEN | TISSUE | MESENCHYMAL TRANSITION | BASEMENT-MEMBRANES | INVASION | OPHTHALMOLOGY | EXPRESSION | Keratin | Granuloma | Dysplasia | Dendritic cells | Mucins | Stem cells | Neovascularization | Index Medicus
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5. Full Text Gastric High-grade Dysplasia Can Be Associated With Submucosal Invasion: Evaluation of its Prevalence in a Series of 121 Endoscopically Resected Specimens
by Sakurai, Urara and Lauwers, Gregory Y and Vieth, Michael and Sawabe, Motoji and Arai, Tomio and Yoshida, Tatsuya and Aida, Junko and Takubo, Kaiyo
The American Journal of Surgical Pathology, ISSN 0147-5185, 11/2014, Volume 38, Issue 11, pp. 1545 - 1550
Gastric neoplasms classified as high-grade dysplasia (HGD) by Western pathologists are frequently diagnosed as tubular adenocarcinoma in Japan. Here, we... 
Intramucosal adenocarcinoma | Endoscopic resection | Gastric neoplasm | Gastric dysplasia | SURGERY | WESTERN PATHOLOGISTS | DIAGNOSIS | gastric dysplasia | intramucosal adenocarcinoma | GASTROINTESTINAL EPITHELIAL NEOPLASIA | gastric neoplasm | JAPANESE | PATHOLOGY | endoscopic resection | BIOPSIES | Predictive Value of Tests | Adenocarcinoma - pathology | Neoplasm Invasiveness | Adenocarcinoma - classification | Blood Vessels - pathology | Humans | Middle Aged | Terminology as Topic | Gastric Mucosa - pathology | Male | Stomach Neoplasms - pathology | Lymphatic Metastasis | Gastroscopy | Adenocarcinoma - secondary | Gastrectomy - methods | Neoplasm Grading | Biopsy | Aged, 80 and over | Female | Aged | Stomach Neoplasms - classification | Stomach Neoplasms - surgery | Adenocarcinoma - surgery | Gastric Mucosa - surgery | Index Medicus
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6. Full Text Inhibition of β-catenin signaling improves alveolarization and reduces pulmonary hypertension in experimental bronchopulmonary dysplasia
by Alapati, Deepthi and Rong, Min and Chen, Shaoyi and Hehre, Dorothy and Hummler, Stefanie C and Wu, Shu
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 2014, Volume 51, Issue 1, pp. 104 - 113
Bronchopulmonary dysplasia (BPD) is the most common and serious chronic lung disease of preterm infants. The development of pulmonary hypertension (PH)... 
β-catenin | Bronchopulmonary dysplasia | Neonatal lung injury | Hyperoxia | Pulmonary hypertension | beta-catenin | ACTIVATION | hyperoxia | pulmonary hypertension | BIOCHEMISTRY & MOLECULAR BIOLOGY | INDUCED LUNG INJURY | PROLIFERATION | bronchopulmonary dysplasia | VASCULAR-DISEASE | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | CADHERIN | PATHOGENESIS | neonatal lung injury | RESPIRATORY SYSTEM | GROWTH | WNT | EXPRESSION | Apoptosis - drug effects | Humans | Bronchopulmonary Dysplasia - pathology | Myocytes, Smooth Muscle - pathology | Extracellular Matrix - metabolism | Hypertension, Pulmonary - prevention & control | Immunoenzyme Techniques | Hyperoxia - metabolism | Pulmonary Alveoli - metabolism | Pulmonary Alveoli - drug effects | Pyrimidinones - pharmacology | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Animals, Newborn | Pulmonary Alveoli - pathology | Bronchopulmonary Dysplasia - prevention & control | Extracellular Matrix - drug effects | RNA, Messenger - genetics | Rats | Hypertension, Pulmonary - metabolism | Hyperoxia - prevention & control | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | beta Catenin - metabolism | Blotting, Western | beta Catenin - genetics | Hyperoxia - pathology | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | Animals | beta Catenin - antagonists & inhibitors | Bronchopulmonary Dysplasia - metabolism | Fluorescent Antibody Technique | Cell Proliferation - drug effects | Extracellular Matrix - pathology | Hypertension, Pulmonary - pathology | Index Medicus
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7. Full Text Association of TLR polymorphisms with bronchopulmonary dysplasia
by Malash, Amr Hosny and Ali, Aliaa Adel and Samy, Rania Mohamed and Shamma, Radwa Ahmed
Gene, ISSN 0378-1119, 10/2016, Volume 592, Issue 1, pp. 23 - 28
Bronchopulmonary dysplasia (BPD) remains a leading cause of morbidity and mortality during infancy. Evidence suggests that the Toll-like receptor ( ) signaling... 
Toll-like receptors | Bronchopulmonary dysplasia | Sepsis | Oxygen therapy | Neonatal mortality | Preterm neonates | FLAGELLIN | RECOGNITION | INTESTINAL EPITHELIAL-CELLS | GENETIC SUSCEPTIBILITY | GENETICS & HEREDITY | LUNG-DISEASE | PRETERM | EXPRESSION | INNATE IMMUNITY | Bronchopulmonary Dysplasia - genetics | Toll-Like Receptor 5 - genetics | Humans | Infant, Premature | Receptors, Interleukin-1 - genetics | Bronchopulmonary Dysplasia - pathology | Female | Male | Polymorphism, Single Nucleotide | Case-Control Studies | Infant, Newborn | Membrane Glycoproteins - genetics | Genetic aspects | Single nucleotide polymorphisms | Infants (Newborn) | Endothelial growth factors | Patient outcomes | Disease susceptibility | Infants | Organic acids | Surface active agents | Interleukins | Lectins | Children | Acetic acid | Health aspects | Chromosomes | Index Medicus
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8. Full Text Dysplasia Should Not Be Ignored in Lichenoid Mucositis
by Rock, L.D and Laronde, D.M and Lin, I and Rosin, M.P and Chan, B and Shariati, B and Zhang, L
Journal of Dental Research, ISSN 0022-0345, 7/2018, Volume 97, Issue 7, pp. 767 - 772
Oral lichen planus is categorized as a potentially malignant condition by the World Health Organization; however, some argue that only lichen planus with... 
oral epithelial dysplasia | epithelial neoplasms | precancerous conditions | oral lichen planus | leukoplakia | oral pathology | PLANUS | DENTISTRY, ORAL SURGERY & MEDICINE | RISK | CANCER | LESIONS | Index Medicus | Dentistry
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9. Full Text Hyperoxia modulates TGF-β/BMP signaling in a mouse model of bronchopulmonary dysplasia
by Miguel A. Alejandre-Alcázar and Grazyna Kwapiszewska and Irwin Reiss and Oana V. Amarie and Leigh M. Marsh and Julia Sevilla-Pérez and Malgorzata Wygrecka and Bastian Eul and Silke Köbrich and Mareike Hesse and Ralph T. Schermuly and Werner Seeger and Oliver Eickelberg and Rory E. Morty
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 02/2007, Volume 292, Issue 2, pp. 537 - 549
Prematurely born infants who require oxygen therapy often develop bronchopulmonary dysplasia (BPD), a debilitating disorder characterized by pronounced... 
Alveolarization | Lung development | Bone morphogenetic | Neonatal chronic lung disease | Transforming growth factor-β | Protein | PHYSIOLOGY | NEWBORN RAT LUNG | TISSUE INHIBITORS | bone morphogenetic protein | alveolarization | CHRONIC LUNG-DISEASE | transforming growth factor-beta | GROWTH-FACTOR-BETA | DIRECT BINDING | EPITHELIAL-CELLS | MATRIX METALLOPROTEINASE-1 | RESPIRATORY SYSTEM | TRANSFORMING GROWTH-FACTOR-BETA-1 | neonatal chronic lung disease | SMOOTH-MUSCLE-CELLS | EXTRACELLULAR-MATRIX | lung development | NIH 3T3 Cells | Apoptosis - drug effects | Epithelial Cells - drug effects | Humans | Protein Transport - drug effects | RNA, Messenger - metabolism | Bone Morphogenetic Proteins - metabolism | Hyperoxia - metabolism | Respiration - drug effects | Pulmonary Alveoli - drug effects | Epithelial Cells - cytology | Myocytes, Smooth Muscle - drug effects | Bone Morphogenetic Proteins - pharmacology | Myocytes, Smooth Muscle - cytology | Extracellular Matrix Proteins - metabolism | Infant, Newborn | Disease Models, Animal | Animals, Newborn | Pulmonary Alveoli - pathology | Extracellular Matrix Proteins - genetics | RNA, Messenger - genetics | Pulmonary Artery - drug effects | Pulmonary Artery - cytology | Gene Expression Regulation - drug effects | Hyperoxia - pathology | Transforming Growth Factor beta - pharmacology | Animals | Signal Transduction - drug effects | Bronchopulmonary Dysplasia - metabolism | Fibroblasts - drug effects | Survival Analysis | Cell Proliferation - drug effects | Fibroblasts - cytology | Mice | Lung Diseases - pathology | Transforming Growth Factor beta - metabolism | Index Medicus
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10. Full Text Risk of high-grade dysplasia or carcinoma in gastric biopsy-proven low-grade dysplasia: an analysis using the Vienna classification
by Cho, S.-J and Choi, I. J and Kim, C. G and Lee, J. Y and Kook, M.-C and Park, S and Ryu, K. W and Lee, J. H and Kim, Y.-W
Endoscopy, ISSN 0013-726X, 06/2011, Volume 43, Issue 6, pp. 465 - 471
Background and aims: Therapeutic guidelines have not yet been established for low-grade gastric adenomas/dysplasias (LGD), which have a low risk of progression... 
Original article | CRITERIA | TRANSFORMATION | SURGERY | ENDOSCOPIC MUCOSAL RESECTION | EPITHELIAL DYSPLASIA | MULTICENTER | NONINVASIVE NEOPLASIA | TERM-FOLLOW-UP | GASTROENTEROLOGY & HEPATOLOGY | CANCER | ADENOMAS | CLINICAL-SIGNIFICANCE | Multivariate Analysis | Predictive Value of Tests | Adenoma - surgery | Humans | Middle Aged | Gastric Mucosa - pathology | Male | Stomach Neoplasms - pathology | Precancerous Conditions - classification | Aged, 80 and over | Female | Precancerous Conditions - pathology | Retrospective Studies | Carcinoma - pathology | Gastric Mucosa - surgery | Risk Assessment | Carcinoma - surgery | Risk Factors | Logistic Models | Gastroscopy | Precancerous Conditions - surgery | Adenoma - classification | Biopsy | Adenoma - pathology | Aged | Stomach Neoplasms - surgery | Index Medicus
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11. Full Text Ectopic Expression of Oct-4 Blocks Progenitor-Cell Differentiation and Causes Dysplasia in Epithelial Tissues
by Hochedlinger, Konrad and Yamada, Yasuhiro and Beard, Caroline and Jaenisch, Rudolf
Cell, ISSN 0092-8674, 2005, Volume 121, Issue 3, pp. 465 - 477
The POU-domain transcription factor Oct-4 is normally expressed in pluripotent cells of the mammalian embryo. In addition, germ-cell tumors and a few somatic... 
BREAST-CANCER CELLS | SOMATIC-CELLS | INTESTINAL EPITHELIUM | HAIR FOLLICLE | BIOCHEMISTRY & MOLECULAR BIOLOGY | EMBRYONIC STEM-CELLS | GENE-EXPRESSION | SELF-RENEWAL | BETA-CATENIN | ES CELLS | TRANSCRIPTION FACTOR | CELL BIOLOGY | Intestines - drug effects | Octamer Transcription Factor-3 | Neoplasms - etiology | Gene Expression - drug effects | Cytoskeletal Proteins - genetics | Gene Expression - genetics | Skin - metabolism | Green Fluorescent Proteins - genetics | Intestines - metabolism | Stem Cells - metabolism | Gastrointestinal Tract - pathology | DNA-Binding Proteins - metabolism | Cell Differentiation - genetics | Doxycycline - administration & dosage | Neoplasms - genetics | Trans-Activators - genetics | Cytoskeletal Proteins - metabolism | Cell Lineage - genetics | Skin - pathology | Doxycycline - toxicity | Epithelium - pathology | Intestines - pathology | Epithelium - metabolism | beta Catenin | Gastrointestinal Tract - drug effects | Mice, Transgenic | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Gastrointestinal Tract - metabolism | Transcription Factors - metabolism | Animals | Stem Cells - pathology | Trans-Activators - metabolism | Mice | Neoplasms - pathology | Skin - drug effects | Dysplasia | Research | Cell differentiation | Epithelial cells | Index Medicus
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12. Full Text Tracheal dysplasia precedes bronchial dysplasia in mouse model of N-nitroso trischloroethylurea induced squamous cell lung cancer
by Ghosh, Moumita and Dwyer-Nield, Lori D and Kwon, Jennifer B and Barthel, Lea and Janssen, William J and Merrick, Daniel T and Keith, Robert L
PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, pp. e0122823 - e0122823
Squamous cell lung cancer (SCC) is the second leading cause of lung cancer death in the US and has a 5-year survival rate of only 16%. Histological changes in... 
IN-VITRO | REPAIR | EPITHELIAL-CELLS | AIRWAY STEM-CELLS | MULTIDISCIPLINARY SCIENCES | QUANTITATIVE ASSESSMENT | CHEMOPREVENTION | DIFFERENTIATION | EXPRESSION | LESIONS | BASAL-CELLS | Lung Neoplasms - chemically induced | Carcinoma, Squamous Cell - pathology | Carcinoma, Squamous Cell - chemically induced | Lung Neoplasms - pathology | Cell Transformation, Neoplastic - chemically induced | Biomarkers, Tumor | Cell Transformation, Neoplastic - metabolism | Respiratory Mucosa - pathology | Trachea - pathology | Mitotic Index | Animals | Neoplasm Grading | Female | Carmustine - analogs & derivatives | Mice | Respiratory Mucosa - metabolism | Bronchi - pathology | Carmustine - adverse effects | Disease Models, Animal | Care and treatment | Dysplasia | Lung cancer | Analysis | Cell proliferation | Pediatrics | Epithelial cells | Lung | Bromodeoxyuridine | Respiratory tract | Proteins | Keratin | Time dependence | Histopathology | Trachea | Phenotypes | Invasiveness | Basal cells | Epithelium | Medicine | Metaplasia | Chemotherapy | Medical prognosis | Stem cells | Topical application | Laboratory animals | Cancer | Apoptosis | Index Medicus
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13. Full Text Ki67, CD105, and α‐SMA expressions better relate the binary oral epithelial dysplasia grading system of World Health Organization
by Gadbail, Amol Ramchandra and Chaudhary, Minal S and Sarode, Sachin C and Gawande, Madhuri and Korde, Sheetal and Tekade, Satyajit Ashok and Gondivkar, Shailesh and Hande, Alka and Maladhari, Rupesh
Journal of Oral Pathology & Medicine, ISSN 0904-2512, 11/2017, Volume 46, Issue 10, pp. 921 - 927
BackgroundThe binary system of oral epithelial dysplasia (OED) has never been investigated with reference to the carcinogenesis-related biomarkers. Hence,... 
α | CD | oral submucous fibrosis | SMA | 105 | oral epithelial dysplasia | Ki67 | areca nut | α-SMA | CD105 | LEUKOPLAKIA | POTENTIAL TARGET | TUMOR ANGIOGENESIS | PATHOLOGY | LESIONS | SUBMUCOUS FIBROSIS | VASCULATURE | DENTISTRY, ORAL SURGERY & MEDICINE | SQUAMOUS-CELL CARCINOMA | IMPROVEMENT | MYOFIBROBLASTS | ENDOGLIN CD105 | Actins - biosynthesis | World Health Organization | Epithelium - metabolism | Humans | Middle Aged | Mouth - metabolism | Male | Mouth Neoplasms - metabolism | Young Adult | Adolescent | Aged, 80 and over | Adult | Endoglin - biosynthesis | Female | Ki-67 Antigen - biosynthesis | Aged | Carcinoma in Situ - metabolism | Index Medicus | Dentistry
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