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Oncogene, ISSN 1476-5594, 2013, Volume 33, Issue 18, pp. 2307 - 2316
Signals from the tumor microenvironment trigger cancer cells to adopt an invasive phenotype through epithelial-mesenchymal transition (EMT... 
Vimentin | Epithelial-mesenchymal transition | Signal transduction | Breast cancer | Calcium | STAT3 | MIGRATION | ACTIVATION | calcium | METASTASIS | signal transduction | BIOCHEMISTRY & MOLECULAR BIOLOGY | PHENOTYPE | breast cancer | vimentin | CELL BIOLOGY | ONCOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | epithelial-mesenchymal transition | TRPM7 | CHANNELS | UP-REGULATION | CONTRIBUTES | PROGRESSION | RNA, Small Interfering - genetics | Phosphorylation | Vimentin - biosynthesis | Calcium - metabolism | Epithelial-Mesenchymal Transition - physiology | Humans | Protein-Serine-Threonine Kinases | Epidermal Growth Factor - metabolism | Epithelial-Mesenchymal Transition - drug effects | Epithelial-Mesenchymal Transition - genetics | Breast Neoplasms - metabolism | Cell Hypoxia | TRPM Cation Channels - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - metabolism | Breast Neoplasms - pathology | Cell Line, Tumor | TRPM Cation Channels - genetics | Female | TRPM Cation Channels - metabolism | Epidermal Growth Factor - pharmacology | Calcium Signaling | STAT3 Transcription Factor - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Care and treatment | Calcium channels | Physiological aspects | Genetic aspects | Cellular signal transduction | Research | Risk factors | Cellular biology | Epithelial-mesenchymal transition (EMT)
Journal Article