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The Journal of immunology (1950), ISSN 0022-1767, 01/2014, Volume 192, Issue 2, pp. 623 - 629
.... Although the precise origin of the unchecked inflammatory response in obesity is unclear, it is known that overproduction of proinflammatory cytokines by innate immune cells affects metabolism... 
IMMUNE-SYSTEM | INFLAMMATION | DISEASE | RESISTANCE | FAT | AKT | IMMUNOLOGY | DIET-INDUCED OBESITY | KEY PLAYERS | ADIPOSE-TISSUE | CUTTING EDGE | Tumor Necrosis Factor-alpha - metabolism | Antigens, CD - immunology | T-Lymphocytes, Regulatory - metabolism | Epithelial Cells - metabolism | TOR Serine-Threonine Kinases - metabolism | Obesity - immunology | Insulin - immunology | Interferon-gamma - metabolism | Antigens, CD - metabolism | T-Lymphocytes, Regulatory - immunology | Epithelium - immunology | Signal Transduction - immunology | Inflammation - metabolism | Tumor Necrosis Factor-alpha - immunology | Interleukin-10 - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Receptor, Insulin - immunology | Macrophages - immunology | Hyperinsulinism - metabolism | Intra-Abdominal Fat - immunology | Transforming Growth Factor beta - immunology | Epithelium - metabolism | Mice, Inbred C57BL | Apyrase - metabolism | Cells, Cultured | Apyrase - immunology | CTLA-4 Antigen - metabolism | Inflammation - immunology | CTLA-4 Antigen - immunology | Intra-Abdominal Fat - metabolism | Obesity - metabolism | TOR Serine-Threonine Kinases - immunology | Insulin - metabolism | Macrophages - metabolism | Animals | Hyperinsulinism - immunology | Proto-Oncogene Proteins c-akt - immunology | Epithelial Cells - immunology | Interferon-gamma - immunology | Interleukin-10 - antagonists & inhibitors | Receptor, Insulin - metabolism | Mice | Interleukin-10 - immunology | Transforming Growth Factor beta - metabolism
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2010, Volume 107, Issue 28, pp. 12611 - 12616
Asbestos carcinogenesis has been linked to the release of cytokines and mutagenic reactive oxygen species (ROS) from inflammatory cells. Asbestos is cytotoxic... 
Actinomycin | Asbestos | Cell death | Secretion | Plasma cells | Inflammation | Macrophages | Carcinogenesis | Necrosis | Apoptosis | Mesothelioma | Biomarker | Tumor necrosis factor-alpha | Chemoprevention | TRANSFORMATION | POLY(ADP-RIBOSE) POLYMERASE | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | CROCIDOLITE ASBESTOS | HMGB1 | biomarker | MAMMALIAN-CELLS | tumor necrosis factor-alpha | PATHOGENESIS | chemoprevention | NECROTIC CELLS | carcinogenesis | TNF-ALPHA | INHIBITORS | mesothelioma | Tumor Necrosis Factor-alpha - metabolism | Asbestos - metabolism | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Humans | Reactive Oxygen Species - pharmacology | Adenosine Diphosphate Ribose - metabolism | Adenosine Diphosphate Ribose - pharmacology | Carcinogens - metabolism | Asbestos - pharmacology | Pleural Neoplasms - metabolism | Inflammation - metabolism | Carcinogens - pharmacology | Cell Nucleus - metabolism | HMGB Proteins - pharmacology | HMGB1 Protein - pharmacology | HMGB1 Protein - metabolism | Cell Death | Mesocricetus | Female | HMGB Proteins - metabolism | Poly Adenosine Diphosphate Ribose - pharmacology | Epithelium - drug effects | Cricetinae | Cytokines - metabolism | Epithelium - metabolism | Hydrogen Peroxide - pharmacology | Necrosis - metabolism | Hydrogen Peroxide - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Macrophages - metabolism | Poly(ADP-ribose) Polymerases - metabolism | Animals | Mesothelioma - metabolism | Poly(ADP-ribose) Polymerases - pharmacology | Cells - metabolism | Mice | Mice, Inbred BALB C | Cytokines - pharmacology | Prevention | Mesothelium | Chromosomal proteins | Research | Chemical properties | Health aspects | Reactive oxygen species | Transformation | Deposits | Hydrogen peroxide | Cytokines | Cytotoxicity | HMGB1 protein | Nuclei | Carcinogens | Tumor necrosis factor-a | ATP | Cytoplasm | Biological Sciences
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2011, Volume 141, Issue 2, pp. 621 - 632
Background & Aims Epithelial cells that cover the intestinal mucosal surface maintain immune homeostasis and tolerance in the gastrointestinal tract. However,... 
Gastroenterology and Hepatology | IgA | Inflammatory Bowel Disease | T Cell | Immune Response | CELLS | COMPLEX | ACTIVATION | POLYMERIC IMMUNOGLOBULIN RECEPTOR | INTESTINAL EPITHELIUM | TRANSPORT | INFLAMMATORY-BOWEL-DISEASE | ADAPTER | HOST-DEFENSE | INFECTION | GASTROENTEROLOGY & HEPATOLOGY | Tumor Necrosis Factor-alpha - metabolism | Intestinal Mucosa - metabolism | Adaptor Protein Complex mu Subunits - metabolism | Epithelial Cells - metabolism | Adaptor Protein Complex beta Subunits - deficiency | Humans | Cell Membrane Permeability | Crohn Disease - metabolism | Cell Membrane - physiology | Ribonucleases - metabolism | Adaptor Protein Complex 1 - metabolism | Th17 Cells - metabolism | Intestinal Mucosa - immunology | alpha-Defensins - metabolism | Adaptor Protein Complex beta Subunits - immunology | Cell Membrane - metabolism | beta-Defensins - metabolism | Colon | Receptors, Cytokine - metabolism | Colitis - immunology | Adaptor Protein Complex 1 - deficiency | Lipocalins - metabolism | Adaptor Protein Complex 1 - immunology | Immunoglobulin A - metabolism | Signal Transduction | Acute-Phase Proteins - metabolism | Down-Regulation | Oncogene Proteins - metabolism | Homeostasis - immunology | Epithelial Cells - pathology | S100 Proteins - metabolism | Muramidase - metabolism | Intestinal Mucosa - microbiology | Mice, Knockout | Interleukin-17 - metabolism | Adaptor Protein Complex beta Subunits - metabolism | Animals | Colitis - microbiology | Proteins - metabolism | Ribonuclease, Pancreatic - metabolism | Lipocalin-2 | Cathelicidins - metabolism | Epithelial Cells - immunology | Receptors, Cytokine - immunology | Mice | Intestinal Mucosa - pathology | Medical colleges | Fc receptors | Gastrointestinal diseases | Homeostasis | Biological response modifiers | Immunoglobulin A | T cells
Journal Article
Clinical & Experimental Ophthalmology, ISSN 1442-6404, 01/2006, Volume 34, Issue 1, pp. 64 - 73
Journal Article
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 05/2017, Volume 12, Issue 5, p. e0178525
Mastitis is the most prevalent disease in dairy cattle worldwide and not only causes huge economic losses in the dairy industry but also threatens public... 
LIPOPOLYSACCHARIDE-INDUCED MASTITIS | DIAGNOSIS | OXIDATIVE STRESS | ACTIVATION | ACUTE-PHASE PROTEINS | PEROXYNITRITE | CHEMOKINES | MULTIDISCIPLINARY SCIENCES | AMNIOTIC-FLUID | EXPRESSION | NITRATION | Tumor Necrosis Factor-alpha - metabolism | Heme Oxygenase-1 - metabolism | Epithelial Cells - metabolism | Mammary Glands, Animal | Membrane Glycoproteins - metabolism | Epithelial Cells - drug effects | Colony-Stimulating Factors - metabolism | Lipopolysaccharide Receptors - metabolism | C-Reactive Protein - metabolism | Cattle Diseases - metabolism | Mastitis - metabolism | Mastitis - drug therapy | Cattle | Interleukin-1beta - metabolism | Interleukin 1 Receptor Antagonist Protein - metabolism | Female | Cattle Diseases - drug therapy | Interleukin-6 - metabolism | Acute-Phase Proteins - metabolism | Anti-Inflammatory Agents - pharmacology | Antioxidants - pharmacology | Toll-Like Receptor 4 - metabolism | Animals | Carrier Proteins - metabolism | Signal Transduction - drug effects | Lipopolysaccharides - pharmacology | Melatonin - pharmacology | Mammary glands | Melatonin | Research | Health aspects | Lipopolysaccharides | Cell culture | Oxidative stress | C-reactive protein | Zoology | Glands | Cerebrospinal fluid | Cell surface | Antioxidants | Interleukin 6 | Proteins | Microorganisms | Metabolites | Amniotic fluid | Rodents | Interleukin 1 | Biosphere | Bacteria | Tumor necrosis factor-TNF | Amyloid | Gram-negative bacteria | Mammary gland | Astrocytoma | Public health | Mastitis | Advanced glycosylation end products | Antiinflammatory agents | Cytokines | Animal sciences | Inflammation | Epithelium | Gene expression | Pregnancy | Monocytes | Acute phase substances | Cell death | Ligands | Chemokines | Cancer | Apoptosis
Journal Article
Nature cell biology, ISSN 1476-4679, 2008, Volume 10, Issue 5, pp. 593 - 601
Journal Article
Journal of cell science, ISSN 0021-9533, 01/2014, Volume 127, Issue 1, pp. 158 - 171
Here, we show that epithelial-mesenchymal status influences how cells deposit extracellular matrix. Retinal pigmented epithelial (RPE) cells that expressed... 
Fibrillin-1 | Cell-cell junction | Integrin | Perlecan | Epithelial cell | Mesenchymal cell | Fibronectin | Syndecan | FOCAL ADHESION FORMATION | ACTIVATION | SYNDECAN-4 | FIBRONECTIN EXTRACELLULAR-MATRIX | LATENT TGF-BETA | CELL BIOLOGY | TRANSITION | EVOLUTION | HEPARAN-SULFATE | BINDING-SITES | Retinal Pigment Epithelium - metabolism | Epithelial Cells - metabolism | Cadherins - metabolism | Humans | Syndecan-4 - genetics | Actins - metabolism | Integrins - genetics | Epithelial-Mesenchymal Transition - genetics | Mammary Glands, Human - metabolism | Fibroblasts - ultrastructure | Heparitin Sulfate - metabolism | Integrins - metabolism | Actins - genetics | Actomyosin - genetics | Retinal Pigment Epithelium - ultrastructure | Female | Microfilament Proteins - metabolism | Zonula Occludens-1 Protein - metabolism | Cadherins - genetics | Microfilament Proteins - genetics | Fibroblasts - metabolism | Microfibrils - ultrastructure | Zonula Occludens-1 Protein - genetics | Cell Line | Podocytes - metabolism | Syndecan-4 - metabolism | Gene Expression Regulation | Intercellular Junctions - metabolism | Epithelial Cells - ultrastructure | Fibrillins | Organ Specificity | Actomyosin - metabolism | beta Catenin - metabolism | Fibronectins - metabolism | beta Catenin - genetics | Transforming Growth Factor beta - pharmacology | Podocytes - ultrastructure | Mammary Glands, Human - ultrastructure | Fibronectins - genetics | Intercellular Junctions - ultrastructure | Microfibrils - metabolism | Cell–cell junction
Journal Article
Nature (London), ISSN 1476-4687, 2014, Volume 512, Issue 7515, pp. 387 - 392
Journal Article