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1. Full Text G-quadruplexes regulate Epstein-Barr virus-encoded nuclear antigen 1 mRNA translation
by Murat, Pierre and Zhong, Jie and Lekieffre, Lea and Cowieson, Nathan P and Clancy, Jennifer L and Preiss, Thomas and Balasubramanian, Shankar and Khanna, Rajiv and Tellam, Judy
Nature Chemical Biology, ISSN 1552-4450, 2014, Volume 10, Issue 5, pp. 358 - 364
Viruses that establish latent infections have evolved unique mechanisms to avoid host immune recognition. Maintenance proteins of these viruses regulate their... 
HUMAN TELOMERIC DNA | MAINTENANCE | T-CELL RECOGNITION | CD8(+) | INHIBITION | SMALL-MOLECULE | BIOCHEMISTRY & MOLECULAR BIOLOGY | VIRAL PROTEIN | ENDOGENOUS PRESENTATION | EVASION | EXPRESSION | G-Quadruplexes | Protein Biosynthesis | Epstein-Barr Virus Nuclear Antigens - genetics | Base Sequence | RNA, Messenger - genetics | Antigens | Viruses | Nuclear energy | Ribonucleic acid--RNA
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2. Full Text Genome diversity of epstein-barr virus from multiple tumor types and normal infection
by Palser, Anne L and Grayson, Nicholas E and White, Robert E and Corton, Craig and Correia, Samantha and Ba abdullah, Mohammed M and Watson, Simon J and Cotten, Matthew and Arrand, John R and Murray, Paul G and Allday, Martin J and Rickinson, Alan B and Young, Lawrence S and Farrell, Paul J and Kellam, Paul
Journal of Virology, ISSN 0022-538X, 2015, Volume 89, Issue 10, pp. 5222 - 5237
Epstein-Barr virus (EBV) infects most of the world's population and is causally associated with several human cancers, but little is known about how EBV... 
STRAINS | LYMPHOMAGENESIS | INDIVIDUALS | AMINO-ACID | PHYLOGENIES | VIROLOGY | INTERTYPIC RECOMBINANTS | SEQUENCE | ALGORITHMS | SELECTION | CHINESE POPULATION | Amino Acid Sequence | Genome, Viral | Viral Matrix Proteins - genetics | Herpesvirus 4, Human - genetics | Epstein-Barr Virus Infections - virology | Epstein-Barr Virus Nuclear Antigens - genetics | Humans | Antigens, Viral - genetics | Epitopes, T-Lymphocyte - genetics | Phylogeny | Genetic Variation | Herpesvirus 4, Human - isolation & purification | Carrier State - virology | Recombination, Genetic | Cell Line, Tumor | Polymorphism, Single Nucleotide | Herpesvirus 4, Human - classification | DNA, Viral - genetics
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3. Full Text Epstein–Barr virus nuclear antigen 3A partially coincides with EBNA3C genome-wide and is tethered to DNA through BATF complexes
by Stefanie C. S. Schmidt and Sizun Jiang and Hufeng Zhou and Bradford Willox and Amy M. Holthaus and Peter V. Kharchenko and Eric C. Johannsen and Elliott Kieff and Bo Zhao
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2015, Volume 112, Issue 2, pp. 554 - 559
Epstein–Barr Virus (EBV) conversion of B-lymphocytes to Lymphoblastoid Cell Lines (LCLs) requires four EBV nuclear antigen (EBNA) oncoproteins: EBNA2, EBNALP,... 
EBNA3A | CDKN2A | IRF4 | RUNX3 | BATF | RECOMBINATION | SIGNAL-BINDING-PROTEIN | RBP-J-KAPPA | MULTIDISCIPLINARY SCIENCES | LYMPHOBLASTOID CELL-GROWTH | NONCODING RNA | BURKITTS-LYMPHOMA | 3C BINDS | TRANSCRIPTIONAL REGULATION | EXPRESSION | HISTONE DEACETYLASE | Cyclin D2 - genetics | Core Binding Factor Alpha 3 Subunit - metabolism | Basic-Leucine Zipper Transcription Factors - metabolism | Herpesvirus 4, Human - genetics | Humans | Interferon Regulatory Factors - metabolism | Genes, myc | Viral Proteins - metabolism | B-Lymphocytes - virology | Herpesvirus 4, Human - pathogenicity | Epstein-Barr Virus Nuclear Antigens - metabolism | B-Lymphocytes - metabolism | Cell Line | Genome, Viral | Immunoglobulin J Recombination Signal Sequence-Binding Protein - metabolism | Promoter Regions, Genetic | Epstein-Barr Virus Nuclear Antigens - genetics | Herpesvirus 4, Human - physiology | Viral Proteins - genetics | Binding Sites - genetics | DNA - metabolism | Genes, bcl-2 | Chemokine CXCL9 - genetics | Genes, p16 | DNA - genetics | Enhancer Elements, Genetic | Host-Pathogen Interactions - genetics | Chemokine CXCL10 - genetics | Genome, Human | Cell proliferation | Physiological aspects | Epstein-Barr virus | Biological Sciences
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4. Full Text Epstein-Barr virus latent genes
by Kang, Myung-Soo and Kieff, Elliott
Experimental and Molecular Medicine, ISSN 1226-3613, 01/2015, Volume 47, Issue 1, pp. e131 - e131
Latent Epstein-Barr virus (EBV) infection has a substantial role in causing many human disorders. The persistence of these viral genomes in all malignant... 
MEDICINE, RESEARCH & EXPERIMENTAL | NUCLEAR-PROTEIN-2 ACIDIC DOMAIN | GASTRIC-CARCINOMA CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | NUCLEAR ANTIGEN 1 | SMALL RNA EBER-1 | ORAL HAIRY LEUKOPLAKIA | NASOPHARYNGEAL EPITHELIAL-CELLS | ORIGIN-BINDING PROTEIN | MEMBRANE-PROTEIN 2A | NF-KAPPA-B | LYMPHOCYTE GROWTH TRANSFORMATION | Viral Matrix Proteins - genetics | Neoplasms - etiology | Epstein-Barr Virus Infections - virology | Epstein-Barr Virus Nuclear Antigens - genetics | Virus Latency | Herpesvirus 4, Human - physiology | Humans | RNA, Viral - genetics | Genes, Viral | Protein Binding | Epstein-Barr Virus Infections - complications | Viral Matrix Proteins - metabolism | MicroRNAs - genetics | Epstein-Barr Virus Nuclear Antigens - metabolism | Review
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5. Full Text Epstein–Barr Virus nuclear antigen 1 (EBNA1) confers resistance to apoptosis in EBV-positive B-lymphoma cells through up-regulation of survivin
by Lu, Jie and Murakami, Masanao and Verma, Subhash C and Cai, Qiliang and Haldar, Sabyasachi and Kaul, Rajeev and Wasik, Mariusz A and Middeldorp, Jaap and Robertson, Erle S
Virology, ISSN 0042-6822, 2011, Volume 410, Issue 1, pp. 64 - 75
Abstract Resistance to apoptosis is an important component of the overall mechanism which drives the tumorigenic process. EBV is a ubiquitous human... 
Infectious Disease | EBNA1 | Survivin | Sp1 | Oncogenic process | Apoptosis | LATENT INFECTION | PROTEIN SURVIVIN | CANCER | METAPHASE CHROMOSOMES | VIROLOGY | GASTRIC-CARCINOMA | GENE-EXPRESSION | PROMOTER | TRANSCRIPTION FACTOR | BINDING | TERMINAL REPEATS | Protein Structure, Tertiary | Promoter Regions, Genetic | Herpesvirus 4, Human - genetics | Epstein-Barr Virus Nuclear Antigens - genetics | Inhibitor of Apoptosis Proteins | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Humans | Transcriptional Activation | Herpesvirus 4, Human - immunology | Sp1 Transcription Factor - metabolism | Up-Regulation - drug effects | RNA Interference | Sp1 Transcription Factor - genetics | Up-Regulation - physiology | B-Lymphocytes - virology | Cell Line, Tumor | Apoptosis - physiology | Epstein-Barr Virus Nuclear Antigens - metabolism | B-Lymphocytes - metabolism | Antigens | Epstein-Barr virus | Protein binding | MICROORGANISMS | APOPTOSIS | PARASITES | ORGANIC COMPOUNDS | ANTIGENS | ONCOGENIC VIRUSES | PROTEINS | 60 APPLIED LIFE SCIENCES | VIRUSES | apoptosis | oncogenic process
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6. Full Text Epstein-Barr nuclear antigen 1 contributes to nasopharyngeal carcinoma through disruption of PML nuclear bodies
by Sivachandran, Nirojini and Sarkari, Feroz and Frappier, Lori
PLoS Pathogens, ISSN 1553-7366, 10/2008, Volume 4, Issue 10, pp. e1000170 - e1000170
Latent Epstein-Barr virus (EBV) infection is strongly associated with several cancers, including nasopharyngeal carcinoma (NPC), a tumor that is endemic in... 
UBIQUITIN-SPECIFIC PROTEASE | CELL-CYCLE REGULATION | P53-MDM2 PATHWAY | DNA | SIMPLEX-VIRUS-INFECTION | GENE-EXPRESSION | BINDING DOMAIN | TUMOR-SUPPRESSOR | MICROBIOLOGY | PARASITOLOGY | P53 | USP7/HAUSP | Tumor Virus Infections - genetics | Nasopharyngeal Neoplasms - genetics | Nasopharyngeal Neoplasms - metabolism | Herpesvirus 4, Human - genetics | Humans | Nasopharyngeal Neoplasms - virology | Tumor Suppressor Protein p53 - genetics | Tumor Suppressor Proteins - genetics | Plasmids - genetics | Epstein-Barr Virus Infections - genetics | Ubiquitin Thiolesterase - metabolism | Nuclear Proteins - genetics | Epstein-Barr Virus Nuclear Antigens - metabolism | Cell Line | Tumor Suppressor Proteins - metabolism | Tumor Virus Infections - metabolism | Epstein-Barr Virus Nuclear Antigens - genetics | Intranuclear Inclusion Bodies - genetics | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | Transcription Factors - genetics | Ubiquitin Thiolesterase - genetics | Plasmids - metabolism | Transcription Factors - metabolism | Herpesvirus 4, Human - metabolism | Epstein-Barr Virus Infections - metabolism | Intranuclear Inclusion Bodies - metabolism | Promyelocytic Leukemia Protein | Ubiquitin-Specific Peptidase 7 | Proteins | DNA damage | Infections | Experiments | DNA repair | Cancer | Tumors | Apoptosis
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7. Full Text Transcription factors operate across disease loci, with EBNA2 implicated in autoimmunity
by Harley, John B and Chen, Xiaoting and Pujato, Mario and Miller, Daniel and Maddox, Avery and Forney, Carmy and Magnusen, Albert F and Lynch, Arthur and Chetal, Kashish and Yukawa, Masashi and Barski, Artem and Salomonis, Nathan and Kaufman, Kenneth M and Kottyan, Leah C and Weirauch, Matthew T
Nature Genetics, ISSN 1061-4036, 05/2018, Volume 50, Issue 5, pp. 699 - 707
Explaining the genetics of many diseases is challenging because most associations localize to incompletely characterized regulatory regions. Using new... 
REED-STERNBERG CELLS | SYSTEMIC-LUPUS-ERYTHEMATOSUS | RISK LOCI | SIGNAL-BINDING-PROTEIN | GENETICS & HEREDITY | BARR-VIRUS INFECTION | MOLECULAR MIMICRY | NF-KAPPA-B | HUMAN GENE-EXPRESSION | TRAIT LOCI | GENOME-WIDE ASSOCIATION | Epstein-Barr Virus Nuclear Antigens - immunology | Cell Line | Herpesvirus 4, Human - genetics | Epstein-Barr Virus Nuclear Antigens - genetics | Humans | Regulatory Sequences, Nucleic Acid | Viral Proteins - immunology | Autoimmunity - genetics | Viral Proteins - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Maximum Tolerated Dose | Phenotype | Neoplasms - genetics | Lupus Erythematosus, Systemic - genetics | Cell Line, Tumor | Protein Binding | Autoimmunity | Multiple sclerosis | Genes | Arthritis | B cells | DNA binding proteins | Gene expression | Rheumatoid factor | Systemic lupus erythematosus | Analysis | Genetic aspects | Epstein-Barr virus | Prostate cancer | Transcription factors | Inflammatory bowel diseases | Disease | Downstream effects | Viruses | Genomes | Diabetes mellitus (insulin dependent) | Regulatory sequences | Datasets | Proteins | Consortia | Intestine | Genetics | Deoxyribonucleic acid--DNA | Binding | Lupus | Phenotypes | Nominations | Diabetes mellitus | Health risks | Breast cancer | Loci | Celiac disease | Chronic conditions | Androgens | Rheumatoid arthritis | Computer applications | GATA-3 protein | Lymphomas | Autoimmune diseases | Prostate | Cancer
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8. Full Text Efficient replication of Epstein-Barr virus in stratified epithelium in vitro
by Rachel M. Temple and Junjia Zhu and Lynn Budgeon and Neil David Christensen and Craig Meyers and Clare E. Sample
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2014, Volume 111, Issue 46, pp. 16544 - 16549
Epstein-Barr virus is a ubiquitous human herpesvirus associated with epithelial and lymphoid tumors. EBV is transmitted between human hosts in saliva and must... 
Epithelial cells | B lymphocytes | Rafts | Cell lines | Viruses | Cultured cells | Infections | Epstein Barr virus infections | Epithelium | Human herpesvirus 4 | GENOME AMPLIFICATION | PROTEIN | VIVO | MULTIDISCIPLINARY SCIENCES | epithelial | organotypic culture | productive replication | B-CELLS | REACTIVATION | GENE-EXPRESSION | EBV | HAIRY LEUKOPLAKIA | Epstein-Barr virus | INFECTION | CELL-DIFFERENTIATION | Viral Matrix Proteins - genetics | Keratinocytes - virology | Protein Precursors - analysis | Virus Latency | Humans | Cytopathogenic Effect, Viral | DNA, Viral - analysis | Palatine Tonsil - cytology | Keratins - analysis | RNA, Viral - genetics | Plasmids - genetics | Trans-Activators - genetics | Cell Differentiation | Trans-Activators - biosynthesis | Cell Culture Techniques | Keratinocytes - ultrastructure | Viral Proteins - biosynthesis | Viral Matrix Proteins - biosynthesis | Virus Replication - drug effects | Antiviral Agents - pharmacology | Epstein-Barr Virus Nuclear Antigens - genetics | Herpesvirus 4, Human - physiology | RNA, Viral - biosynthesis | Acyclovir - pharmacology | Viral Proteins - genetics | Epstein-Barr Virus Nuclear Antigens - biosynthesis | Gene Expression Regulation, Viral | Keratinocytes - metabolism | Gingiva - cytology | Virus Cultivation | DNA, Viral - genetics | Genetic research | Genetic aspects | Virus research | Health aspects | Biological Sciences | Epstein–Barr virus
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9. Full Text Epstein-Barr virus exploits intrinsic B-lymphocyte transcription programs to achieve immortal cell growth
by Bo Zhao and James Zou and Hongfang Wang and Eric Johannsen and Chih-wen Peng and John Quackenbush and Jessica C. Mar and Cynthia Casson Morton and Matthew L. Freedman and Stephen C. Blacklow and Jon C. Aster and Bradley E. Bernstein and Elliott Kieff
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2011, Volume 108, Issue 36, pp. 14902 - 14907
Epstein-Barr virus nuclear antigen 2 (EBNA2) regulation of transcription through the cell transcription factor RBPJ is essential for resting B-lymphocyte (RBL)... 
T lymphocytes | Up regulation | Chromatin | B lymphocytes | DNA | Nucleosomes | Gene expression regulation | Epstein Barr virus infections | Human herpesvirus 4 | Binding sites | Development | Lymphoma | Leukemia | Notch | leukemia | development | SIGNAL-BINDING-PROTEIN | CIS-ELEMENT | MULTIDISCIPLINARY SCIENCES | lymphoma | C-MYC | NUCLEAR ANTIGEN-2 | FATE | GENOME | LONG-RANGE INTERACTION | GENE | PROMOTER | J-KAPPA | Cell Proliferation | Herpesvirus 4, Human - genetics | Proto-Oncogene Proteins c-ets - genetics | Genome, Viral - genetics | Humans | Transcription Factor RelA - genetics | Viral Proteins - metabolism | Trans-Activators - genetics | B-Lymphocytes - virology | Epstein-Barr Virus Infections - genetics | Core Binding Factor alpha Subunits - metabolism | Transcription, Genetic | Proto-Oncogene Proteins c-ets - metabolism | Epstein-Barr Virus Nuclear Antigens - metabolism | Immunoglobulin J Recombination Signal Sequence-Binding Protein - genetics | B-Lymphocytes - metabolism | Proto-Oncogene Proteins - metabolism | Immunoglobulin J Recombination Signal Sequence-Binding Protein - metabolism | Epstein-Barr Virus Nuclear Antigens - genetics | Response Elements | Nucleosomes - metabolism | Viral Proteins - genetics | Nucleosomes - genetics | Proto-Oncogene Proteins - genetics | Proto-Oncogene Proteins c-myc - metabolism | Core Binding Factor alpha Subunits - genetics | Transcription Factor RelA - metabolism | Cell Line, Tumor | Trans-Activators - metabolism | Proto-Oncogene Proteins c-myc - genetics | Herpesvirus 4, Human - metabolism | Epstein-Barr Virus Infections - metabolism | Physiological aspects | Transcription factors | Epstein-Barr virus | Genetic aspects | Lymphocytes | Health aspects | Biological Sciences
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10. Full Text Epstein-Barr virus nuclear antigen 3C facilitates cell proliferation by regulating cyclin D2
by Pei, Yonggang and Singh, Rajnish Kumar and Shukla, Sanket Kumar and Lang, Fengchao and Zhang, Shengwei and Robertson, Erle S
Journal of Virology, ISSN 0022-538X, 09/2018, Volume 92, Issue 18
Cell cycle regulation is one of the hallmarks of virus-mediated oncogenesis. Epstein-Barr virus (EBV)-induced lymphomas express a repertoire of essential viral... 
Cell proliferation | Epstein-Barr virus | EBNA3C | Cyclin D2 | PRB | cell proliferation | COMPLEXES | LATENT MEMBRANE PROTEIN-1 | VIROLOGY | RETINOBLASTOMA PROTEIN | ROLES | GROWTH | cyclin D2 | DEGRADATION | TRANSCRIPTION FACTOR | EXPRESSION | Cyclin D2 - genetics | Cell Proliferation - genetics | Cell Nucleus - metabolism | Epstein-Barr Virus Nuclear Antigens - genetics | Herpesvirus 4, Human - physiology | Humans | Cell Cycle Proteins - metabolism | Gene Expression Regulation | B-Lymphocytes - virology | Cell Transformation, Viral | Index Medicus
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