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Nature Communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, p. 6377
Tyrosine kinase inhibitors are effective treatments for non-small-cell lung cancers (NSCLCs) with epidermal growth factor receptor (EGFR) mutations. However,... 
GROWTH-FACTOR RECEPTOR | DRUG-SENSITIVITY | GEFITINIB | AMPLIFICATION | MODELS | GENE | MULTIDISCIPLINARY SCIENCES | MUTATION | ACQUIRED-RESISTANCE | KINASE INHIBITORS | CHEMOTHERAPY | Lung Neoplasms - drug therapy | Recurrence | Adenocarcinoma - pathology | Adenocarcinoma of Lung | Humans | Lung Neoplasms - metabolism | ErbB Receptors - genetics | Gene Expression Regulation, Neoplastic | bcl-X Protein - genetics | Lung Neoplasms - pathology | Small Cell Lung Carcinoma - drug therapy | Small Cell Lung Carcinoma - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Adenocarcinoma - metabolism | Adenocarcinoma - genetics | Antineoplastic Agents - pharmacology | Lung - metabolism | Gefitinib | ErbB Receptors - deficiency | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Lung - pathology | Aniline Compounds - pharmacology | ErbB Receptors - antagonists & inhibitors | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Retinoblastoma Protein - deficiency | Small Cell Lung Carcinoma - genetics | Sulfonamides - pharmacology | Adenocarcinoma - drug therapy | Disease Progression | Drug Resistance, Neoplasm - genetics | Small Cell Lung Carcinoma - pathology | Afatinib | Retinoblastoma Protein - genetics | Erlotinib Hydrochloride - pharmacology | Lung - drug effects | Cell Line, Tumor | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Quinazolines - pharmacology | bcl-X Protein - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Drug Resistance, Neoplasm - drug effects
Journal Article
European Journal of Medicinal Chemistry, ISSN 0223-5234, 06/2019, Volume 172, pp. 36 - 47
Journal Article
Journal of Cancer Research and Clinical Oncology, ISSN 0171-5216, 8/2018, Volume 144, Issue 8, pp. 1413 - 1422
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 10/2011, Volume 31, Issue 42, pp. 15173 - 15187
Astrocytes are both detrimental and beneficial for repair and recovery after spinal cord injury (SCI). These dynamic cells are primary contributors to the... 
NEURAL STEM-CELLS | FUNCTIONAL RECOVERY | NEURITE OUTGROWTH | MONODELPHIS-DOMESTICA | AXON REGENERATION | REACTIVE ASTROCYTES | CENTRAL-NERVOUS-SYSTEM | RAT-BRAIN | FACTOR RECEPTOR | RADIAL GLIA | NEUROSCIENCES | Enzyme-Linked Immunosorbent Assay - methods | Locomotion - genetics | Recovery of Function - drug effects | Humans | Axons - physiology | Cell Transdifferentiation - genetics | Ganglia, Spinal - cytology | Green Fluorescent Proteins - genetics | Glial Fibrillary Acidic Protein - metabolism | Cell Movement - genetics | Spinal Cord Injuries - pathology | Transfection - methods | Cell Transdifferentiation - drug effects | Lamins - metabolism | Female | Bromodeoxyuridine - metabolism | Spinal Cord - cytology | Spinal Cord Injuries - therapy | ErbB Receptors - deficiency | Disease Models, Animal | Locomotion - drug effects | Transforming Growth Factor alpha - pharmacology | Astrocytes - drug effects | Green Fluorescent Proteins - metabolism | Recovery of Function - genetics | ErbB Receptors - metabolism | Mice, Inbred C57BL | Cells, Cultured | Neural Stem Cells - drug effects | Axons - drug effects | Up-Regulation - genetics | Transforming Growth Factor alpha - genetics | Mice, Knockout | Up-Regulation - drug effects | Cell Movement - drug effects | Phenotype | Animals | Analysis of Variance | Cell Proliferation - drug effects | Mice | Spinal Cord Injuries - physiopathology | Neurofilament Proteins - metabolism
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 02/2009, Volume 29, Issue 8, pp. 2404 - 2413
Neuregulin-1s (NRG-1s) are a family of growth and differentiation factors with multiple roles in the development and function in different organs including the... 
Cerebellum | Development | Granule cell | Neuregulin | Synapse formation | NMDA receptor | PURKINJE-CELLS | development | DENDRITIC GROWTH | SYNAPSE ELIMINATION | neuregulin | RECEPTOR SUBUNIT EXPRESSION | NEUROSCIENCES | synapse formation | cerebellum | granule cell | MESSENGER-RNAS | POSTNATAL-DEVELOPMENT | RAT-BRAIN | DIFFERENTIATION | GRANULE CELLS | CLIMBING-FIBER | Electric Stimulation | Receptors, N-Methyl-D-Aspartate - metabolism | Gene Expression Regulation, Developmental - genetics | Male | Receptors, N-Methyl-D-Aspartate - genetics | Excitatory Postsynaptic Potentials - physiology | Neurons - physiology | Female | Tetrodotoxin - pharmacology | ErbB Receptors - deficiency | Organ Culture Techniques | Gene Expression Regulation, Developmental - physiology | Animals, Newborn | Receptors, GABA - genetics | Cerebellum - metabolism | Valine - analogs & derivatives | Enzyme Inhibitors - pharmacology | Neuregulins - physiology | Sodium Channel Blockers - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Mice, Knockout | Patch-Clamp Techniques | Animals | Cerebellum - cytology | Receptors, GABA - metabolism | Cerebellum - growth & development | Signal Transduction - physiology | Mice | Valine - pharmacology | Receptor, ErbB-4 | Receptor, ErbB-2 - deficiency | Index Medicus | Tetrodotoxin | Enzyme Inhibitors | Neurons and Cognition | Life Sciences | Neuregulins | Receptors, GABA | Excitatory Postsynaptic Potentials | Gene Expression Regulation, Developmental | Signal Transduction | Neurons | Receptors, N-Methyl-D-Aspartate | Receptor, erbB-2 | Valine | Sodium Channel Blockers | Excitatory Amino Acid Antagonists | Receptor, Epidermal Growth Factor
Journal Article
Journal Article
International Journal of Cancer, ISSN 0020-7136, 11/2018, Volume 143, Issue 10, pp. 2458 - 2469
Journal Article
International Journal of Oncology, ISSN 1019-6439, 2008, Volume 33, Issue 6, pp. 1165 - 1176
Three prominent hallmarks of triple-negative/ basal-like breast carcinomas, a subtype of breast cancer gene phenotype associated with poor relapse-free and... 
BRCA1 | Basal-like | Cetuximab | Cisplatin | Epidermal growth factor receptor | Mitogen-activated protein kinases | basal-like | epidermal growth factor receptor | TYROSINE KINASE | MONOCLONAL-ANTIBODY | EXPRESSION PROFILES | COMBINATION | BASAL PHENOTYPE | TUMORS | REPAIR | ONCOLOGY | mitogen-activated protein kinases | cisplatin | RECEPTOR-ENHANCED CHEMOSENSITIVITY | ERBB RECEPTORS | Up-Regulation | Phosphorylation | Apoptosis - drug effects | Humans | Drug Resistance, Neoplasm | Receptors, Estrogen - deficiency | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Breast Neoplasms - metabolism | Cisplatin - administration & dosage | Dose-Response Relationship, Drug | Receptors, Progesterone - deficiency | Antibodies, Monoclonal, Humanized | Mitogen-Activated Protein Kinase Kinases - metabolism | Breast Neoplasms - enzymology | Receptor, Epidermal Growth Factor - metabolism | BRCA1 Protein - metabolism | Female | PTEN Phosphohydrolase - deficiency | Tumor Suppressor Protein p53 - deficiency | Cross-Linking Reagents - administration & dosage | Drug Synergism | Protein Kinase Inhibitors - administration & dosage | Antibodies, Monoclonal - administration & dosage | Breast Neoplasms - pathology | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Cell Proliferation - drug effects | Quinazolines - pharmacology | Receptor, ErbB-2 - deficiency
Journal Article
International Journal of Cancer, ISSN 0020-7136, 02/2019, Volume 144, Issue 4, pp. 777 - 787
Journal Article